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1 upstream regulators Sonic hedgehog, Car and Fibroblast Growth Factor 8.
2 d its putative receptor, patched, as well as fibroblast growth factor 8 and bone morphogeneic protein
4 her, these malformations result from loss of fibroblast growth factor 8 and sonic hedgehog expression
5 toderm, defined by the juxtaposed domains of Fibroblast growth factor 8 and Sonic hedgehog, which pre
7 that we connect to intron retention in fgf8 (fibroblast growth factor 8) and fgfr2 (fgf receptor 2) t
8 n of 4 of these genes (Tbx14/15, Dickkopf-1, Fibroblast Growth Factor 8, and Keratin-18) was further
10 dermal ridge (AER)-specific genes, including fibroblast growth factor 8 (FGF8) and FGF4 occurred norm
12 a boundary between Sonic hedgehog (Shh) and Fibroblast growth factor 8 (Fgf8) expression domains.
15 te regrowth of neural processes, the role of fibroblast growth factor 8 (FGF8) in mammalian spiral ga
16 a precise and dynamic expression pattern of fibroblast growth factor 8 (Fgf8) in the HAA anlage, whi
22 During development of the urogenital tract, fibroblast growth factor 8 (Fgf8) is expressed in mesone
24 terior to posterior (AP) patterning, whereas fibroblast growth factor 8 (Fgf8) is produced by the api
25 e show that the spatiotemporal expression of Fibroblast growth factor 8 (Fgf8) is strategically poise
26 iated during the third day of development by Fibroblast Growth Factor 8 (FGF8) produced by the newly
29 of cells expressing sonic hedgehog (Shh) and fibroblast growth factor 8 (Fgf8) that is responsible fo
30 dditionally, we observed that, in the brain, fibroblast growth factor 8 (Fgf8) was downregulated, and
32 face plasmon resonance analysis reveals that fibroblast growth factor 8 (Fgf8), a key mediator of cel
36 mic organizer and its key effector molecule, fibroblast growth factor 8 (Fgf8), have been cornerstone
37 n of the morphogens sonic hedgehog (Shh) and fibroblast growth factor 8 (Fgf8), the possibility that
38 n of Bone morphogenetic protein 4 (Bmp4) and Fibroblast growth factor 8 (Fgf8), two genes that are kn
39 were attributable in part to a reduction of fibroblast growth factor 8 (Fgf8), which was shown to be
41 those of null mutants for the gene encoding fibroblast growth factor 8 (Fgf8): an inconsistent start
45 ic hedgehog (Shh), but the expression of the fibroblast growth factor-8 gene (Fgf-8) appears as norma
46 the potential role of the signaling protein, fibroblast growth factor 8, in the early patterning of a
50 , overexpression of a mitogenic polypeptide (fibroblast growth factor 8, isoform b or FGF8b) and loss
51 that were exposed to both sonic hedgehog and fibroblast growth factor 8 with telomerase-immortalized
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