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2 n normal and human breast cancer cell lines (fibrocystic and metastatic states) as well as normal and
3 and the n-7 saturation index against benign fibrocystic breast changes and the progression of prolif
6 eoporosis, breast cancer, endometriosis, and fibrocystic breast disease and has been hypothesized to
7 stic algorithm to distinguish normal tissue, fibrocystic change (FCC), fibroadenoma, and breast cance
8 ules and ducts, at higher levels in areas of fibrocystic change and papillomas, in all benign breast
9 t LOH occurs frequently in the components of fibrocystic change as well as in normal TDLUs and sugges
10 2 cases containing the various components of fibrocystic change in the absence of malignancy were stu
11 vivo samples of human breast tissue (normal, fibrocystic change, fibroadenoma, and infiltrating carci
14 Eight patients (4.4%) had benign results: fibrocystic changes in four, atypical ductal hyperplasia
15 These lesions display characteristics of fibrocystic changes observed in breasts of women, and in
16 RhoC was not expressed in any of the normal, fibrocystic changes, atypical hyperplasia, or ductal car
17 ystic conditions (PFCs) and nonproliferative fibrocystic conditions (NPFCs) in the breast and to eval
18 s in the development of benign proliferative fibrocystic conditions (PFCs) and nonproliferative fibro
19 l papilloma as the source of discharge, with fibrocystic disease and ductal ectasia providing the nex
22 ve polycystic kidney disease is a hereditary fibrocystic disease that involves the kidneys and the bi
29 n of cancer as compared with both normal and fibrocystic tumor tissue was measured in all the samples
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