ライフサイエンスコーパス: fibrocyte

1 to the enhanced basal migratory motility of fibrocytes.

2 (SAP), and healthy tissues contain very few fibrocytes.

3 asons, very few monocytes differentiate into fibrocytes.

4 ferentiate into fibroblast-like cells called fibrocytes.

5 i polarization and generation of circulating fibrocytes.

6 (+)) showed selective expression of CD206 on fibrocytes.

7 raction of cockroach allergens with CD206 in fibrocytes.

8 otein kinase (p38), ERK, and JNK in cultured fibrocytes.

9 ne marrow-derived mesenchymal stem cells and fibrocytes.

10 monocytes into fibroblast-like cells called fibrocytes.

11 h levels of functional CD40 are displayed by fibrocytes.

12 XCL2 upregulation in the rat spiral ligament fibrocytes.

13 ionine are incorporated into Tg expressed by fibrocytes.

14 s substantial activity when transfected into fibrocytes.

15 markedly increased CD34(+)/procollagen 1(+) fibrocytes.

16 migration of bone marrow-derived circulating fibrocytes.

17 ce and correlated with high numbers of liver fibrocytes.

18 oplastic collagen- and fibronectin-producing fibrocytes.

19 diseases is associated with monocyte-derived fibrocytes.

20 l proliferation once they differentiate into fibrocytes.

21 is known about signaling from fibroblasts to fibrocytes.

22 d 2.78 +/- 0.36-fold compared with Ccr5(-/-) fibrocytes (1.0 +/- 0.06; p < 0.05).

23 ress syndrome patients to differentiate into fibrocytes; 2) the influence of the acute respiratory di

24 d fibrosing diseases is the monocyte-derived fibrocyte, a collagen-secreting profibrotic cell.

25 tients with Graves' disease (GD), as well as fibrocyte abundance, were determined by flow cytometry.

26 Bone marrow-derived fibrocytes accumulate in the dermis.

27 characterized the role of CTGF in promoting fibrocyte accumulation and regulation after AngII exposu

28 omal ERalpha to FSP1+, CD34+, SMA- precursor fibrocytes adjacent to normal and precancerous CIN epith

29 Monocyte-derived cells called fibrocytes also activate fibroblasts, and we found that

30 We now report that fibrocytes also express Tg, which resolves as a 305-kDa

31 Injection of WT enhanced GFP(+) fibrocytes also increased the number of Gr-1(Int), CD11b

32 WT and Ccl2(-/-) fibrocytes also stimulated Ccl2 expression in the lung b

33 exicanin I increased the number of MDSCs and fibrocytes and accelerated the wound healing.

34 entified two bone marrow-derived cell types, fibrocytes and alternatively activated M2 macrophages, a

35 Levels of CD206 expression on human fibrocytes and CD206 mediated signaling and cytokine pro

36 ng flow cytometry, we quantified circulating fibrocytes and characterized their chemokine receptor ex

37 The functional properties of fibrocytes and dermal fibroblasts were tested by using r

38 uced the expression of CTGF and alpha-SMA in fibrocytes and fibrocyte differentiation in patients wit

39 mice resulted in a significant reduction in fibrocytes and in skin fibrosis.

40 Previously, leukocyte progenitors termed fibrocytes and myofibroblasts generated from epithelial

41 CD40 levels on cultivated fibrocytes and orbital fibroblasts (GOFB) from patients

42 EAV has specific tropism for stromal cells (fibrocytes and possibly tissue macrophages) and CD8(+) T

43 was observed between CD45(+)Col1(+)CXCR4(+) fibrocytes and the activation phenotypes CD45(+)Col1(+)p

44 r ability of monocytes to differentiate into fibrocytes and the inhibitory effect of the alveolar env

45 ading to reduction in the number of cultured fibrocytes and total nonadherent non-T cells from health

46 talline basophils deposits of minerals, rare fibrocytes and very few vessels brought in discussion a

47 found two populations: (i) CD45(+) CD34(+) (fibrocytes) and (ii) CD45(+) CD34(-) [myeloid-derived su

48 ells, CD45(+) CD34(+) collagen I(+) CXCR4(+) fibrocytes, and HSP47(+) activated fibroblasts that were

49 decision of monocytes to differentiate into fibrocytes, and indicate that modulating lumican signali

50 decision of monocytes to differentiate into fibrocytes, and may indicate that modulating Slit2 signa

51 monocytes into fibroblast-like cells called fibrocytes, and promotes phagocytosis of cell debris by

52 mesenchymal cells, attraction of circulating fibrocytes, and stimulation of the epithelial to mesench

53 ulted in decreased accumulation of MDSCs and fibrocytes, and wound healing was significantly delayed.

54 Fibrocytes are bone marrow-derived circulating progenito

55 Fibrocytes are circulating bone marrow-derived cells tha

56 Fibrocytes are circulating progenitor cells that are inc

57 Fibrocytes are circulating, hematopoietic cells that exp

58 Fibrocytes are detectable in bronchoalveolar lavage duri

59 Circulating CD40(+)CD45(+)Col1(+) fibrocytes are far more frequent in vivo in donors with

60 Fibrocytes are hematopoietic derived stem cells that may

61 Fibrocytes are hematopoietic stem cell-derived fibroblas

62 Fibrocytes are hematopoietic-derived cells with mesenchy

63 CD45(+) and collagen I-positive (Col(+)) fibrocytes are implicated in fibrogenesis in skin, lungs

64 These data may explain why fibrocytes are increased in fibrotic tissues, suggest th

65 Fibrocytes are mesenchymal progenitors involved in norma

66 Alveolar fibrocytes are monocyte-derived mesenchymal cells associ

67 mice injured with bleomycin indicating that fibrocytes are not a necessary source of type I collagen

68 ese findings demonstrate that high levels of fibrocytes are present in the peripheral blood of patien

69 These data may explain why fibrocytes are rarely observed in healthy tissues, may s

70 Blood fibrocytes are recruited during COPD exacerbations and r

71 kines to recruit these fibrocytes or whether fibrocytes are stimulated to home to the affected tissue

72 at the inner ear fibrocytes (spiral ligament fibrocytes) are able to recognize nontypeable Haemophilu

73 Fibroblast progenitor cells (fibrocytes) are important to the development of myocardi

74 compared to control subjects nor were blood fibrocytes associated with lung function or qCT, but wer

75 ignificantly increased number of circulating fibrocytes at V1 compared with control subjects.

76 The percentage of fibrocytes at V2 was negatively correlated with FEV1, fo

77 r the phenotypic and functional hallmarks of fibrocytes but mediate immune suppression.

78 , GLAST was expressed in the spiral ligament fibrocytes but was not detected in the satellite cells o

79 ferentiate into fibroblast-like cells called fibrocytes, but fibrocyte differentiation is strongly in

80 Peripheral blood fibrocytes can accelerate wound healing by stimulating c

81 Fibrocytes can be recruited to inflamed tissues to promo

82 luorescence-activated cell sorting analysis, fibrocytes (CD45(+) and collagen 1 [Col1](+)) were enume

83 Flow cytometric analyses of human fibrocytes (CD45(+) and collagen-1(+)) showed selective

84 Fibrocytes (CD45+/collagen 1+) were quantified in bronch

85 [myeloid-derived suppressor cell (MDSC)-like fibrocytes] cells in stable COPD (n = 41) and control (n

86 Here we quantify CD40 expression on fibrocytes, circulating, and bone marrow-derived progeni

87 om neighboring cells account for much of the fibrocyte collagen.

88 Fibrocytes collected from patients with COPD at V1 had i

89 fold increased ability to differentiate into fibrocytes compared to ventilated controls or non-ventil

90 CXCR4(+) fibrocyte concentration may be useful as a biomarker for

91 nd final values of peripheral blood CXCR4(+) fibrocyte concentration were strongly associated with de

92 a cross-sectional analysis, peripheral blood fibrocyte concentrations were markedly elevated in a sub

93 These data support our hypothesis that fibrocytes contribute to premetastatic conditioning by r

94 llectively, our data suggest that neoplastic fibrocytes contribute to the induction of BM fibrosis in

95 ion and activation state of peripheral blood fibrocytes correlates with asthma severity.

96 We measured circulating fibrocyte counts and chemokine levels in a cohort of sub

97 nd marked episodic elevations in circulating fibrocyte counts over a median follow-up period of 614 d

98 The number of circulating fibrocytes decreased at V2.

99 ds in diabetic mice were treated either with fibrocytes, dermal fibroblasts, or phosphate buffered sa

100 thout (n = 11) COPD was collected for tissue fibrocyte detection.

101 Furthermore, WT fibrocytes did not increase Ly-6C(+) monocytes in Ccr2(-

102 at aa Q55 and E126 in human SAP affect human fibrocyte differentiation and SAP binding to FcgammaRI.

103 that tryptase and thrombin potentiate human fibrocyte differentiation at biologically relevant conce

104 n the concentration of SAP needed to inhibit fibrocyte differentiation by 95%.

105 veolar lavage fluid inhibited by 71% (55-94) fibrocyte differentiation compared to saline control.

106 vital for CTGF expression, which results in fibrocyte differentiation in COA, and suggests that an E

107 sion of CTGF and alpha-SMA in fibrocytes and fibrocyte differentiation in patients with COA.

108 Lumican competes with the serum fibrocyte differentiation inhibitor serum amyloid P, but

109 proliferation in the myocardium and enhances fibrocyte differentiation into a myofibroblast phenotype

110 fibroblast-like cells called fibrocytes, but fibrocyte differentiation is strongly inhibited by the p

111 NaCl-increased fibrocyte differentiation may thus contribute to NaCl-in

112 However, the mechanism of fibrocyte differentiation remains unclear.

113 nitial trigger to override SAP inhibition of fibrocyte differentiation to initiate scar tissue format

114 uction of BM fibrosis in PMF, and inhibiting fibrocyte differentiation with SAP may interfere with th

115 P binds to FcgammaRI on monocytes to inhibit fibrocyte differentiation, and binds to FcgammaRIIa on n

116 combinant LGALS3BP inhibits monocyte-derived fibrocyte differentiation, and immunodepletion of LGALS3

117 an IgG-blocking Ab reduces the SAP effect on fibrocyte differentiation, and ligating FcgammaRIIa with

118 rotein called serum amyloid P (SAP) inhibits fibrocyte differentiation, and sialidases attenuate SAP

119 ocytes, and unlike other factors that affect fibrocyte differentiation, lumican has no detectable eff

120 ctivity that inhibits human monocyte-derived fibrocyte differentiation, whereas less aggressive breas

121 nditioned media removes the monocyte-derived fibrocyte differentiation-inhibiting activity.

122 ta2 integrins are needed for lumican-induced fibrocyte differentiation.

123 ecrete factors that inhibit monocyte-derived fibrocyte differentiation.

124 sts, and we found that sialidases potentiate fibrocyte differentiation.

125 protein Slit2 and that Slit2 inhibits human fibrocyte differentiation.

126 ther, our data suggest that NaCl potentiates fibrocyte differentiation.

127 lasma protein Serum Amyloid P (SAP) inhibits fibrocyte differentiation.

128 oncho-alveolar lavage fluid had no effect on fibrocyte differentiation.

129 chloride and sodium nitrate also potentiate fibrocyte differentiation.

130 rum and SAP that normally completely inhibit fibrocyte differentiation.

131 related proteoglycan decorin, promotes human fibrocyte differentiation.

132 ers of LGALS3BP, potentiate monocyte-derived fibrocyte differentiation.

133 ry distress syndrome alveolar environment on fibrocyte differentiation; and 3) mediators involved in

134 ized that if these chemokines are recruiting fibrocytes, disrupting their signaling will reduce early

135 pha transgenic mouse model demonstrated that fibrocytes do not transform into WT1(+) mesenchymal cell

136 tudy demonstrate a novel association between fibrocyte-driven WT1(+) cell accumulation and severe fib

137 A high percentage of circulating fibrocytes during exacerbation was associated with incre

138 nstrated that two circulating populations of fibrocyte exist in COPD, with distinct clinical associat

139 The degree of fibrocyte expansion observed in individual subjects dire

140 These findings indicate that human fibrocytes express multiple "thyroid-specific" proteins,

141 Fibrocytes express several chemokine receptors (CCR7 and

142 Furthermore, fibrocytes express TSLP receptor (TSLPR), and TSLP direc

143 ing collagen-promoter GFP mice, we find that fibrocytes express type I collagen.

144 ound that CD34(+) progenitor cells, known as fibrocytes, express functional TSHR, infiltrate the orbi

145 To do so, we isolated fibrocytes expressing CD45, CD11b, CD13, and Col1a1 from

146 herent non-T (NANT) cells were isolated, and fibrocytes expressing CD45, collagen I, CTGF, ETAR, or a

147 The blood concentration of fibrocytes expressing the chemokine receptor CXCR4 corre

148 Fibrocyte expression of both proinflammatory and profibr

149 Fibrocyte expression of collagens and profibrotic growth

150 ferentially expressed chemokines (i) promote fibrocyte (FC) migration towards ASM and (ii) are increa

151 nters and augment immune reactivity, whereas fibrocytes from cancer subjects suppressed anti-CD3-medi

152 Intriguingly, collagen-producing fibrocytes from hematopoietic lineages were observed att

153 Circulating fibrocytes from nonadherent non-T-cell mononuclear cell

154 ated alpha-SMA expression induced by ET-1 in fibrocytes from normal participants.

155 0A9 enhances the basal migratory motility of fibrocytes from patients in the Asthma AE group and pati

156 Expression of EGFR was increased in fibrocytes from patients with COA compared with that see

157 Fibrocytes from patients with COA have a greater capacit

158 After being cultured, CTGF was increased in fibrocytes from patients with COA, but not from those of

159 ronchial walls and overexpression of CTGF in fibrocytes from patients with COA.

160 Dexamethasone reduced CCR7 expression in fibrocytes from patients with nonsevere asthma but not f

161 ticoid receptor expression was attenuated in fibrocytes from patients with severe asthma.

162 In cocultures, fibrocytes from the mdx(5cv) diaphragm stimulated a high

163 bits the differentiation of monocyte-derived fibrocytes from wild-type mouse spleen cells, but not fr

164 13-independent novel mechanisms by promoting fibrocyte functions.

165 In the injured liver, fibrocytes gave rise to (myo)fibroblasts.

166 Unlike fibrocytes, GD orbital fibroblasts, which comprise a mix

167 Fibrocytes gradually lose their hematopoietic cell marke

168 Fibrocytes have been considered to play a role in allerg

169 pulmonary cell types and bone marrow-derived fibrocytes have been implicated as contributors to fibro

170 Fibrocytes have been proposed as an important direct con

171 onocyte-derived fibroblast-like cells called fibrocytes help to form scar tissue.

172 Fibrocytes highly expressed CXCR4 and CCR3, the chemokin

173 Blood MDSC-like fibrocytes, however, are increased and associated with p

174 erized the function of CD45(+)/collagen I(+) fibrocytes in acutely injured skeletal muscle of wild-ty

175 We showed the accumulation of fibrocytes in bronchial walls and overexpression of CTGF

176 The aims of this study were: 1) to quantify fibrocytes in bronchoalveolar lavage fluid from patients

177 The role of fibrocytes in chronic obstructive pulmonary disease (COP

178 increased number of activated/differentiated fibrocytes in circulating blood of asthmatic patients ex

179 We sought to enumerate blood and tissue fibrocytes in COPD and determine the association of bloo

180 Col(+) cells capable of differentiation into fibrocytes in culture was identified.

181 suggesting roles for TLR7/8 in induction of fibrocytes in HCV infection.

182 uated by flow cytometry, and the presence of fibrocytes in HP and normal lungs by confocal microscopy

183 There were higher numbers of circulating fibrocytes in patients in the Asthma AE group and patien

184 CR4/CXCL12 axis contributes to chemotaxis of fibrocytes in patients in the Asthma AE group, whereas t

185 FR) activation mediated the proliferation of fibrocytes in patients with COA and whether oxidative st

186 udy was to evaluate the recruitment of blood fibrocytes in patients with COPD during exacerbations an

187 We propose that fibrocytes in severe asthma are different from those in

188 study, we investigated the role of TSLP and fibrocytes in the generation of IL-13-induced skin fibro

189 primary auditory neurons, and, finally, the fibrocytes in the lateral wall.

190 The presence of fibrocytes in the lung during acute respiratory distress

191 ar SP-D regulates numbers of macrophages and fibrocytes in the lungs, profibrotic cytokine expression

192 WT but not Ccr5(-/-) fibrocytes increased the number of metastatic foci when

193 In co-cultures, fibrocytes induced on lung fibroblasts a significant inc

194 Likewise, fibrocytes induced the up-regulation of CCL2 in HP lymph

195 d AngII + AMD3100 animals showed exacerbated fibrocyte infiltration and fibrosis compared with AngII

196 ng their signaling will reduce early (3-day) fibrocyte infiltration and, consequently, fibrosis in th

197 Treatment of the xenograft mice with the fibrocyte inhibitor serum amyloid P (SAP; pentraxin-2) s

198 , but dominates over the fibroblast-secreted fibrocyte inhibitor Slit2.

199 Basal CD40 expression on fibrocytes is greater than that on GOFB.

200 The frequency of circulating CD40(+) fibrocytes is markedly increased in patients with TAO, s

201 Fibrocytes isolated from BLM-treated iSP-D mice off Dox

202 The conditioned medium from fibrocyte-like cells (FcCM) has been shown to stimulate

203 In addition, the influx of fibrocyte-like cells into brain abscesses immediately pr

204 Thus, alterations in the ability of IUGR fibrocyte-like cells to stimulate angiogenesis may contr

205 decrease the ability of both normal and IUGR fibrocyte-like cells to stimulate angiogenesis.

206 in the angiogenic zones of the placenta are fibrocyte-like cells.

207 Desmoid tumors also contained a subclass of fibrocytes linked to wound healing, angiogenesis, and fi

208 Fibrocytes may participate in the pathogenesis of HP, am

209 The application of fibrocytes may represent a potential clinical solution f

210 The mesenchymal progenitors, fibrocytes, may be involved in the remodeling of asthmat

211 It has been proposed that circulating fibrocytes mediate the disease.

212 The cell markers are consistent with fibrocytes mediating the disease.

213 In conclusion, in response to liver injury, fibrocytes migrate from BM to the liver.

214 Bone marrow-derived fibrocytes migrate to injured tissues and contribute to

215 Fibrocytes migrated towards recombinant CCL2 and ASM sup

216 Early CTGF expression occurred before fibrocyte migration (1 day) into the myocardium or ECM d

217 , respectively, by 50% and 25% inhibition of fibrocyte migration in Col-Luc(CCR2-/-)-->wt and Col-Luc

218 Fibrocyte migration in response to liver injury was inve

219 4, a chemokine receptor that is important in fibrocyte migration into the lungs.

220 Plerixafor, a CXCR4 antagonist, decreased fibrocyte migration to plasma from patients with exacerb

221 e role of the chemokines CXCL12 and CCL11 in fibrocyte migration was investigated by using a chemotax

222 Our findings suggest that i.m. fibrocytes most likely originate from infiltrating monoc

223 ion of bone marrow-derived progenitor cells (fibrocytes) occurs before deposition of extracellular ma

224 upregulated expression of S100A9 and RAGE in fibrocytes of patients in the Asthma AE group and those

225 posed of smooth muscle cells and surrounding fibrocytes of the tunica adventitia and the lamina propr

226 The effect of fibrocytes on lung fibroblasts and T lymphocytes was exa

227 lso noted the presence of collagen-producing fibrocytes on the epicardial surface that resulted at le

228 s (6 hours), well before the accumulation of fibrocytes or TGF-beta mRNA up-regulation.

229 organs liberate chemokines to recruit these fibrocytes or whether fibrocytes are stimulated to home

230 A bronchoalveolar lavage fibrocyte percentage >6% provides an additive prognostic

231 everity of illness, a bronchoalveolar lavage fibrocyte percentage >6% was independently associated wi

232 Comparison of bronchoalveolar lavage fibrocyte percentage from patients with or without acute

233 Addition of bronchoalveolar lavage fibrocyte percentage in a clinical model predicting mort

234 e prognostic value of bronchoalveolar lavage fibrocyte percentage in patients with acute lung injury

235 Therefore, we sought to determine whether fibrocytes play a role in the induction of BM fibrosis i

236 We examined circulating fibrocyte populations for correlations with clinical par

237 nce involves continuous viral replication in fibrocytes (possibly including tissue macrophages) and T

238 Fibrocyte potentiation by thrombin and tryptase is media

239 In this report, we demonstrate that fibrocytes predispose the lung to B16-F10 metastasis by

240 rs also report that TSLP is able to activate fibrocytes, probably by inducing stromal cell-derived fa

241 Fibrocytes produce high levels of cytokines, including i

242 F on isolated fibrocytes suggested a role in fibrocyte proliferation (twofold; P < 0.05) and collagen

243 inhibitors of EGFR tyrosine kinase, reduced fibrocyte proliferation and myofibroblast transformation

244 Enhanced fibrocyte proliferation and transformation found in pati

245 Increased expression of EGFR and fibrocyte proliferation and transformation were induced

246 In addition, CTGF contributes to fibrocyte proliferation in the myocardium and enhances f

247 Although fibrocytes promote collagen production by fibroblasts, l

248 The CXCL12/CXCR4 axis is involved in such fibrocyte recruitment (Firebrob study; ClinicalTrials NC

249 In conclusion, early fibrocyte recruitment cannot be inhibited through modula

250 The CXCL12/CXCR4 axis is involved in such fibrocyte recruitment.

251 of smooth muscle precursors and adventitial fibrocytes, respectively, by E13.5.

252 we show that fibroblasts stimulated with the fibrocyte-secreted inflammatory signal tumor necrosis fa

253 However, classical fibrocytes serve as antigen presenters and augment immun

254 sly, we have demonstrated that the inner ear fibrocytes (spiral ligament fibrocytes) are able to reco

255 Fibrocytes stimulate wound healing by dermal cell prolif

256 There was correlation between circulating fibrocyte subsets and asthma severity, and there was an

257 metastatic risk to conditions that mobilize fibrocytes, such as inflammation and wound repair.

258 The effect of CTGF on isolated fibrocytes suggested a role in fibrocyte proliferation (

259 ional cockroach allergen-CD206 axis in human fibrocytes, suggesting a role for CD206 in regulating al

260 asthma have elevated numbers of circulating fibrocytes that show enhanced myofibroblastic differenti

261 Like classical fibrocytes, they display cell surface alpha smooth muscl

262 reports regarding the direct contribution of fibrocytes to collagen deposition.

263 PR), and TSLP directly promotes PBMC-derived fibrocytes to produce collagen.

264 ts (RAGE; ie, its receptor), are involved in fibrocyte trafficking in patients with chronic obstructi

265 estinal permeability have important roles in fibrocyte trafficking.

266 CXCL12/CXCR4 and CCL19/CCR7 enhanced fibrocyte transmigration in the Asthma AE group and in p

267 ollagen-I and alpha-SMA) were upregulated in fibrocyte-treated wounds.

268 on of CCL19 in bronchial tissues and CCR7 in fibrocytes was higher in patients with COA.

269 n bronchial tissues and CXCR4 in circulating fibrocytes was higher in the Asthma AE group and, to a l

270 Interestingly, the number of fibrocytes was highly increased in the skin samples of A

271 pression of CCR7, CXCR4, S100A9, and RAGE in fibrocytes was measured by using flow cytometry.

272 -dependent uptake of FITC labeled Bla g 2 by fibrocytes was observed, but was significantly inhibited

273 that collagen type 1 intensity for MDSC-like fibrocytes was positively associated with lung function

274 increase in total and CD45(+)Col1(+)CXCR4(+) fibrocytes was primarily seen in patients with severe as

275 Migration of CD45(+)Col(+) fibrocytes was regulated by chemokine receptors CCR2 and

276 median percentage of bronchoalveolar lavage fibrocytes was significantly higher in patients with acu

277 The percentage of circulating fibrocytes was significantly increased in patients with

278 enotypic expression of peripheral blood (PB) fibrocytes was stained with anti-CD11b, anti-CD45, anti-

279 Fibrocytes were counted at basal condition and after cul

280 Fibrocytes were detected in 90 of 92 (98%) bronchoalveol

281 Fibrocytes were detected in acutely injured muscles and

282 g and cytokine production in Bla g 2 treated fibrocytes were determined.

283 CD45(+)/CXCR4(+)/Col-I(+) circulating fibrocytes were evaluated by flow cytometry, and the pre

284 Numerous fibrocytes were found infiltrating the HP lungs near fib

285 The rat spiral ligament fibrocytes were found to release CXCL2 in response to no

286 Total and differentiating fibrocytes were identified by their expression of CD45,

287 d (alpha-smooth muscle actin [alpha-SMA](+)) fibrocytes were increased in asthmatic patients compared

288 lating alpha-SMA(+) and alpha-SMA(+)CXCR4(+) fibrocytes were increased in asthmatic patients experien

289 Myeloid-derived suppressor cell-like fibrocytes were increased in COPD compared to controls [

290 Fibrocytes were not detected in peripheral blood of WT m

291 Blood and tissue fibrocytes were not increased compared to control subjec

292 ls of ET-1 and the expression of the ETAR in fibrocytes were significantly higher in patients with CO

293 Isolated circulating fibrocytes were used for migration assay.

294 Fibrocytes, which are bone marrow-derived mesenchymal pr

295 MDSCs have been shown to differentiate into fibrocytes, which serve as emerging effector cells that

296 injury, lymphocyte subsets, and circulating fibrocytes, will be presented.

297 COPD and determine the association of blood fibrocytes with clinical features of disease.

298 However, fibrocytes with confirmed deletion of the type I collage

299 onors cultured with IL-4 differentiated into fibrocytes with the same phenotypic profile and immunosu

300 Increased numbers of circulating fibrocytes, with greater myofibroblastic differentiation