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1 pical region, but was less extensive than in fibrofatty ARVC and FaRV.
2                                              Fibrofatty ARVC was characterized by right ventricular m
3                                Patients with fibrofatty ARVC were younger than those with FaRV (31+/-
4 6 sections each from 25 hearts with typical (fibrofatty) ARVC, 7 hearts with fat replacement of the r
5  characterize plaque broadly as calcified or fibrofatty but was limited in its ability to more precis
6 cintigraphy, and finally, attenuation of the fibrofatty changes of the skin, the final consequences o
7 ed myocyte apoptosis in skeletal muscle; and fibrofatty connective tissue proliferation around joints
8  population, the mean percentage of fibrous, fibrofatty, dense calcified, and necrotic core plaques i
9      ARVC is characterized pathologically by fibrofatty infiltration and clinically by arrhythmias an
10 m RyR2(R176Q/+) mice revealed no evidence of fibrofatty infiltration or structural abnormalities char
11                In scar, myocytes adjacent to fibrofatty interfaces demonstrated increased connexin43
12 ydroxylase positive, were present within the fibrofatty matrix and within the myocardial tracts.
13 acking is associated with the severity of LA fibrofatty myocardial remodeling at histologic analysis.
14 ion was found between PLAS and the degree of fibrofatty myocardial replacement at histologic analysis
15 001) and regression of fibrous (P<0.001) and fibrofatty (P<0.001) tissue.
16 ), mesenteric signs such as hyperemia (n=9), fibrofatty proliferation (n=8) and lymphadenopathy (n=28
17 t correlate with the histologic degree of LA fibrofatty replacement (r = -0.35, P = .330).
18 myopathy, characterized by right ventricular fibrofatty replacement and arrhythmias, causes sudden de
19 PLAS, correlates strongly with the degree of fibrofatty replacement at histologic analysis.
20 reas the heart showed extensive fibrosis and fibrofatty replacement in both ventricles.
21 hen junctions are disrupted, cell death, and fibrofatty replacement occur.
22 athy (ARVD/C) is a disorder characterized by fibrofatty replacement of cardiac myocytes that typicall
23 eritable myocardial disorder associated with fibrofatty replacement of myocardium and ventricular arr
24 lial arrhythmogenic disease characterized by fibrofatty replacement of myocytes with scattered foci o
25 ogical features include loss of myocytes and fibrofatty replacement of right ventricular myocardium;
26                An ARVD/C is characterized by fibrofatty replacement of RV myocardium and RV dilation.
27  genetic myocardial disease characterized by fibrofatty replacement of the myocardium and a predispos
28 rdiomyopathy characterized pathologically by fibrofatty replacement primarily of the RV and clinicall
29  characteristic of ARVC is myocyte loss with fibrofatty replacement.
30 ht ventricle, subepicardial left ventricular fibrofatty replacements (64%), myocyte atrophy (96%), an
31 atal growth followed by slow regression to a fibrofatty residuum.
32 c right ventricular cardiomyopathy (ARVC) is fibrofatty scar replacement.
33 nvolution were telangiectasias (145, 84.3%), fibrofatty tissue (81, 47.1%), and anetodermic skin (56,
34 ncluding fibre size variability, presence of fibrofatty tissue of varying severity, without specific
35 r disorder characterized by myocyte loss and fibrofatty tissue replacement of the right ventricle.
36 lcium progression, regression of fibrous and fibrofatty tissue, and excessive expansive remodeling, s
37 n which the right ventricle is "replaced" by fibrofatty tissue, resulting in lethal arrhythmias.
38 rdial tracts (Marshall Bundles) insulated by fibrofatty tissue.

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