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4 6 sections each from 25 hearts with typical (fibrofatty) ARVC, 7 hearts with fat replacement of the r
5 characterize plaque broadly as calcified or fibrofatty but was limited in its ability to more precis
6 cintigraphy, and finally, attenuation of the fibrofatty changes of the skin, the final consequences o
7 ed myocyte apoptosis in skeletal muscle; and fibrofatty connective tissue proliferation around joints
8 population, the mean percentage of fibrous, fibrofatty, dense calcified, and necrotic core plaques i
10 m RyR2(R176Q/+) mice revealed no evidence of fibrofatty infiltration or structural abnormalities char
13 acking is associated with the severity of LA fibrofatty myocardial remodeling at histologic analysis.
14 ion was found between PLAS and the degree of fibrofatty myocardial replacement at histologic analysis
16 ), mesenteric signs such as hyperemia (n=9), fibrofatty proliferation (n=8) and lymphadenopathy (n=28
18 myopathy, characterized by right ventricular fibrofatty replacement and arrhythmias, causes sudden de
22 athy (ARVD/C) is a disorder characterized by fibrofatty replacement of cardiac myocytes that typicall
23 eritable myocardial disorder associated with fibrofatty replacement of myocardium and ventricular arr
24 lial arrhythmogenic disease characterized by fibrofatty replacement of myocytes with scattered foci o
25 ogical features include loss of myocytes and fibrofatty replacement of right ventricular myocardium;
27 genetic myocardial disease characterized by fibrofatty replacement of the myocardium and a predispos
28 rdiomyopathy characterized pathologically by fibrofatty replacement primarily of the RV and clinicall
30 ht ventricle, subepicardial left ventricular fibrofatty replacements (64%), myocyte atrophy (96%), an
33 nvolution were telangiectasias (145, 84.3%), fibrofatty tissue (81, 47.1%), and anetodermic skin (56,
34 ncluding fibre size variability, presence of fibrofatty tissue of varying severity, without specific
35 r disorder characterized by myocyte loss and fibrofatty tissue replacement of the right ventricle.
36 lcium progression, regression of fibrous and fibrofatty tissue, and excessive expansive remodeling, s
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