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1 n potentials (adipogenesis, osteogenesis and fibrogenesis).
2 ns and may be promising targets for treating fibrogenesis.
3 in-Ang II-AT1 cascade in promoting pulmonary fibrogenesis.
4 n multiple pathological processes, including fibrogenesis.
5 R), a second portal phenomenon implicated in fibrogenesis.
6 liver sinusoidal endothelial cells abrogated fibrogenesis.
7 native LYCAT expression by siRNA accentuated fibrogenesis.
8 e identified that may be important during PD fibrogenesis.
9 by Gram-negative bacteria stimulate hepatic fibrogenesis.
10 trogens inhibit stellate cell activation and fibrogenesis.
11 culating markers of hepatocyte apoptosis and fibrogenesis.
12 ameliorates hepatocellular damage and liver fibrogenesis.
13 whether microbiota may interfere with liver fibrogenesis.
14 y the protective effects of estrogen against fibrogenesis.
15 ine models to establish the role of LYCAT in fibrogenesis.
16 hat play vital roles in liver physiology and fibrogenesis.
17 ial cell-derived mesenchymal proteins during fibrogenesis.
18 liferation, apoptosis, oxidative stress, and fibrogenesis.
19 ecto-enzymes CD39 and CD73, promotes dermal fibrogenesis.
20 gests they play an important role in hepatic fibrogenesis.
21 in FFC-fed mice abrogated indices of hepatic fibrogenesis.
22 ignals perpetuating hepatic inflammation and fibrogenesis.
23 l mechanism contributing to inflammation and fibrogenesis.
24 f ongoing parenchymal injury by disease, not fibrogenesis.
25 and survival of HSCs and protects from liver fibrogenesis.
26 d myofibroblast activation, thereby limiting fibrogenesis.
27 response in the liver and promoting hepatic fibrogenesis.
28 ism of TLR4-mediated fibroblast responses in fibrogenesis.
29 ation of lung leukocyte subsets in pulmonary fibrogenesis.
30 y diseases associated with TGF-beta-mediated fibrogenesis.
31 tor were used to assess the role of SphKs in fibrogenesis.
32 rise to HSCs and myofibroblasts during liver fibrogenesis.
33 y potential key regulatory proteins of liver fibrogenesis.
34 stitial spaces and are key effectors of lung fibrogenesis.
35 e cell viability, promoting inflammation and fibrogenesis.
36 d glomerular flow is a critical initiator of fibrogenesis.
37 lung development and may be involved in lung fibrogenesis.
38 g initial inflammatory responses and hepatic fibrogenesis.
39 henyleneiodonium was associated with reduced fibrogenesis.
40 synthesis of extracellular matrix and liver fibrogenesis.
41 GFR significantly decreased TGFbeta-mediated fibrogenesis.
42 intracellular renin receptor that stimulates fibrogenesis.
43 eral blood cells in hepatic inflammation and fibrogenesis.
44 10R2 and IL-22R1, thereby ameliorating liver fibrogenesis.
45 tic regulatory mechanisms impacting on liver fibrogenesis.
46 ial therapeutic target for modulating tissue fibrogenesis.
47 role of Shh signaling in renal interstitial fibrogenesis.
48 rarenal mRNA expression of genes involved in fibrogenesis.
49 ctors that are known to drive diabetic renal fibrogenesis.
50 dulates hepatic stellate cell activation and fibrogenesis.
51 its sustained activation is associated with fibrogenesis.
52 tenin signaling in abnormal wound repair and fibrogenesis.
53 ne response accompanied by tissue injury and fibrogenesis.
54 gen, which may provide a molecular basis for fibrogenesis.
55 of oxidative stress are warranted to inhibit fibrogenesis.
56 y may also have key functions independent of fibrogenesis.
57 increased MF-HSC viability, and exacerbated fibrogenesis.
58 ng that NOX2-expressing HSC are important in fibrogenesis.
59 elming orderly repair and creating sustained fibrogenesis.
60 effects of myofibroblast contraction on lung fibrogenesis.
61 ctivation is an essential event during liver fibrogenesis.
62 ls, suggesting a direct role of HIV in liver fibrogenesis.
63 ive stress appear to be warranted to inhibit fibrogenesis.
64 genetic silencing of MMP genes during tissue fibrogenesis.
65 stellate cells (HSC) are critical events in fibrogenesis.
66 echanisms; and assess if NKT cells stimulate fibrogenesis.
67 a1) production play important roles in liver fibrogenesis.
68 ight into intrahepatic immunity during liver fibrogenesis.
69 sm that promotes DNA hypermethylation during fibrogenesis.
70 ely up-stream of TGF-betas in the process of fibrogenesis.
71 in procollagen I secretion in HSCs and liver fibrogenesis.
72 xplored how a PNPLA3 variant impacts hepatic fibrogenesis.
73 ic Chagas disease, favoring inflammation and fibrogenesis.
74 e acid-induced ductular reaction (DR) drives fibrogenesis.
75 activated, they are contractile and promote fibrogenesis.
76 an important role in the reversal of hepatic fibrogenesis.
77 and emerging blood and imaging biomarkers of fibrogenesis.
78 asingly recognized as an important driver of fibrogenesis.
79 during carbon tetrachloride induced hepatic fibrogenesis.
80 dent determinant of chemically induced liver fibrogenesis.
81 the contributions of HIV and HCV to hepatic fibrogenesis.
82 UPR in facilitating collagen I secretion and fibrogenesis.
83 nt, immunoregulation, regeneration, and also fibrogenesis.
84 stress, which play important roles in liver fibrogenesis.
85 egy that blocks TGF-beta signaling and renal fibrogenesis.
86 and tissue resident cell populations invokes fibrogenesis.
87 Hedgehog (Hh) signalling regulates hepatic fibrogenesis.
88 ly targeting apelin, a critical repressor of fibrogenesis.
89 s and identify Tc2 cells as key mediators of fibrogenesis.
90 verify in vivo the relevance of AR in NAFLD fibrogenesis.
91 s a source of myofibroblast formation during fibrogenesis.
92 ohepatitis (NASH) is associated with hepatic fibrogenesis.
93 ription 3 (STAT3) had been involved in liver fibrogenesis.
94 inflammation, but may paradoxically promote fibrogenesis.
95 tricate links between activation of HPCs and fibrogenesis.
96 Ia with IPF HLF suggests its contribution to fibrogenesis.
97 sponses, as well as pattern recognition, and fibrogenesis.
100 Thus, SP, via NK-1R, promotes intestinal fibrogenesis after chronic colitis by stimulating fibrot
102 pha, hydrogen peroxide, hydroxynonenal), and fibrogenesis (alpha-smooth muscle actin, picrosirius red
103 .IMPORTANCE Endoglin plays a crucial role in fibrogenesis and angiogenesis and is an important protei
106 HF/MCD+leptin lean rats, significant hepatic fibrogenesis and cirrhosis, marked portal hypertension,
107 /SMAD genomic circuit that regulates hepatic fibrogenesis and define a role for VDR as an endocrine c
108 ogically soft silicone substrates suppresses fibrogenesis and desensitizes mesenchymal stem cells (MS
109 may modulate the immune response and inhibit fibrogenesis and discuss the current evidence for their
110 ized by an alteration of the balance between fibrogenesis and fibrinolysis, which results in accumula
113 receptor (EGFR) inhibitor erlotinib on liver fibrogenesis and hepatocellular transformation in three
114 aradigm for the regulation of CCN2-dependent fibrogenesis and identifies fibrotic pathways as targets
115 uncover a new molecular mechanism of tissue fibrogenesis and identify sEphrin-B2, its receptors EphB
117 nding of the roles of vitamin D in pulmonary fibrogenesis and in the treatment of pulmonary fibrosis.
118 the renin-angiotensin system contributes to fibrogenesis and increased hepatic resistance in patient
120 e activation of each homolog modulates renal fibrogenesis and inflammation has not been established.
121 R-21 was protective against TGF-beta-induced fibrogenesis and inflammation in glomerular and intersti
123 vasculogenesis, wound healing responses, and fibrogenesis and is upregulated in fibroblasts and myofi
126 e cells and in mice following injury reduced fibrogenesis and matrix accumulation; this effect was pa
127 ion state of eosinophils in EoE with altered fibrogenesis and motility of esophageal fibroblasts and
130 ion as a fundamental epigenetic mechanism in fibrogenesis and place Egr-1 upstream in TGF-beta-driven
132 mising therapeutic approach for reduction of fibrogenesis and prevention of HCC in high-risk cirrhosi
133 T-cell responses, which may encourage liver fibrogenesis and progression to end-stage liver disease.
135 iated induction of IL-6 contributes to renal fibrogenesis and shows potential therapeutic targets for
136 icate that TRPV4 activity mediates pulmonary fibrogenesis and suggest that manipulation of TRPV4 chan
138 n the kidney and other organs, their role in fibrogenesis and their role in regulation of the microva
141 TCS exposure, especially on enhancing liver fibrogenesis and tumorigenesis, and the relevance of TCS
143 O-1 interferes with chronic inflammation and fibrogenesis and, in consequence, might delay progressio
144 ars critical to the induction of progressive fibrogenesis and, ultimately, the ominous complications
149 ated by the methyltransferase Dnmt1 in renal fibrogenesis, and kidney fibrosis is ameliorated in Dnmt
150 ajor source of extracellular proteins during fibrogenesis, and may directly, or via secreted products
152 iral-mediated gene transfer on angiogenesis, fibrogenesis, and portal hypertension-associated hemodyn
153 ain receptor-2, contributes significantly to fibrogenesis, and promotes resolution of lung inflammati
154 levels are a critical determinant of EMT and fibrogenesis, and suggest regulation of axin levels as a
155 Changes in hepatic vasculature accompany fibrogenesis, and targeting angiogenic molecules often a
156 They act as a brake on the processes driving fibrogenesis, and they dismantle and degrade established
159 and liver endothelial cells (LECs) modulates fibrogenesis, angiogenesis, and portal hypertension.
160 and elucidating the final common pathways of fibrogenesis are critical for the development of efficac
163 matory infiltrate may contribute directly to fibrogenesis as well as influence the fate of the DR hep
165 were protected against CCl(4)-mediated liver fibrogenesis, as evidenced by reduced collagen type I al
166 stellate cells (HSC) play a critical role in fibrogenesis associated with HCC onset and progression,
167 f CAV1 contributed to the hyperactivation of fibrogenesis-associated RUNX2, a transcription factor ge
169 of liver fibrosis resolution, VEGF promoted fibrogenesis, but was also required for hepatic tissue r
170 adapted to mice, thus allowing the study of fibrogenesis by genetic approaches in transgenic mice.
171 n hepatic stellate cells (HSCs), could drive fibrogenesis by modulating the HSC pro-fibrogenic phenot
172 SPHK2 plays an important role in kidney fibrogenesis by modulating transforming growth factor-be
173 impedes ductular morphogenesis and enhances fibrogenesis by promoting accumulation of immature ductu
174 ithelial cells (AECs) directly contribute to fibrogenesis by secreting mesenchymal proteins, such as
175 source of myofibroblasts that participate in fibrogenesis by way of synthesis of proinflammatory cyto
176 se to kidney injury, resulting in attenuated fibrogenesis, capillary rarefaction, and inflammation.
178 fects of galectin-3 inhibition on myocardial fibrogenesis, cultured fibroblasts were treated with gal
180 cle actin (alpha-SMA) signaling pathway, and fibrogenesis despite similar fat accretion with diet-ind
181 ates both diet-induced hepatic steatosis and fibrogenesis, despite the observation that L-Fabp parado
182 GTPase activity selectively in HSC enhances fibrogenesis, driven at least in part through up-regulat
183 HSC activation contributes to termination of fibrogenesis during fibrosis resolution, but results in
185 role in modulating hepatic inflammation and fibrogenesis during NASH progression, suggesting the pos
188 endocannabinoids production, promote hepatic fibrogenesis, enhance the hepatic vasoconstrictive respo
189 ribe here a novel method to quantify hepatic fibrogenesis flux rates both directly in liver tissue an
190 out specific cellular contributors mediating fibrogenesis hampers the design of effective antifibroti
194 nto the cellular and molecular mechanisms of fibrogenesis herald the promise of new therapies to slow
199 mal transition (EndMT) and spontaneous liver fibrogenesis in EC-specific constitutive hemi-deficient
208 ctivation of Hif suppressed inflammation and fibrogenesis in mice subjected to unilateral ureteral ob
213 gest a novel model to account for persistent fibrogenesis in scleroderma, in which activation of fibr
216 ent lysine deacetylase, sirtuin 1 (SIRT1) in fibrogenesis in the cell culture, animal model, and huma
217 shows consistent and reproducible allograft fibrogenesis in the context of single-lung transplantati
219 ng cell culture expansion protects MSCs from fibrogenesis in the host wound environment and increases
222 halting the progression of liver injury and fibrogenesis in various liver pathogeneses driven by NLR
224 on in HSCs would decrease HSC activation and fibrogenesis in vivo by disrupting receptor tyrosine kin
225 , depletion of mononuclear phagocytes during fibrogenesis in vivo resulted in suppressed NF-kappaB ac
227 reviously unidentified regulatory pathway in fibrogenesis in which a macrophage scavenger receptor pr
230 Firstly, we estimated in mice with acute fibrogenesis induced by a single CCl4 injection the half
231 iver injuries and functions to inhibit liver fibrogenesis induced by either carbon tetrachloride into
233 iciency of Fn14 protected mouse kidneys from fibrogenesis, inflammation, and associated vascular inst
234 ve endotoxin producers, may accelerate liver fibrogenesis, introducing dysbiosis as a cofactor contri
239 s, we hypothesized that this perpetuation of fibrogenesis is caused by epigenetic modifications.
240 g studies suggest that their contribution to fibrogenesis is limited compared with that of hepatic st
243 senchymal features during carcinogenesis and fibrogenesis is regulated by several mesenchymal transcr
249 of GSK-3beta in cardiac fibroblasts leads to fibrogenesis, left ventricular dysfunction, and excessiv
250 of GSK-3beta in cardiac fibroblasts leads to fibrogenesis, left ventricular dysfunction, and excessiv
252 brosis progression, but do not contribute to fibrogenesis or stabilization of the collagen matrix.
253 dase (NADPH) oxidase induction is central to fibrogenesis, our aim was to study the phagocytic NADPH
254 onger administered, despite normalization of fibrogenesis parameters; these findings confirm reversal
255 athologic angiogenesis, attenuation of liver fibrogenesis partly mediated through inhibition of hepat
258 cells, and myeloid leukocytes contribute to fibrogenesis predominantly by producing key fibrogenic c
259 ic programs that control multiple aspects of fibrogenesis, ranging from growth factor expression and
263 o mesenchymal transition (EMT) and pulmonary fibrogenesis require epithelial integrin alpha3beta1-med
264 on of myofibroblasts, which are critical for fibrogenesis, requires both a mechanical signal and acti
268 any cellular processes being associated with fibrogenesis such as cell migration and contraction.
270 idal remodeling is believed to contribute to fibrogenesis, the impact of sinusoidal angiogenesis on t
271 ponents of the pathophysiological process of fibrogenesis; these target molecules include collagen ty
272 mplex and context-dependent, and may promote fibrogenesis through coregulation of fibrogenic gene tar
274 steatosis-induced HSC activation and hepatic fibrogenesis through mitigating inflammation and oxidati
275 enetic confounders in the recipient on graft fibrogenesis, thus explaining significantly different gr
276 ibroblasts correlates with regions of active fibrogenesis, thus representing a pathologically relevan
277 invasively detect and quantify the extent of fibrogenesis using magnetic resonance imaging (MRI).
279 our work supports that controlling pulmonary fibrogenesis via inhibition of miR-497-5p expression may
285 lial growth factor (VEGF), angiogenesis, and fibrogenesis was determined through the analysis of huma
286 patic stellate cells (HSC) were examined and fibrogenesis was evaluated in CD1d-deficient mice that l
287 ve importance of each PAI-1 function in lung fibrogenesis, we administered mutant PAI-1 proteins that
288 from proximal tubule is sufficient for renal fibrogenesis, we generated a novel mouse strain with ind
289 Because TGF-betas play critical roles in fibrogenesis, we initiated efforts to define the role of
290 ta's pleiotropic effects on inflammation and fibrogenesis, we investigated potential effect modificat
292 tential function of E-type cyclins for liver fibrogenesis, we repetitively treated constitutive CcnE1
293 use certain ciliopathies are associated with fibrogenesis, we sought to explore the fate and potentia
294 ole of the vitamin D receptor (VDR) in renal fibrogenesis, we subjected VDR-null mice to unilateral u
295 ematopoietic cell-derived type I collagen to fibrogenesis, we use a double-transgenic system to speci
297 te into myofibroblasts and promote pulmonary fibrogenesis, while inhibition of its expression could e
298 ouse model of chronic liver inflammation and fibrogenesis with progression to hepatocellular carcinom
300 GFbeta1 may reveal therapeutics to attenuate fibrogenesis yet preserve the important homeostatic func
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