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1 n)-for paediatric patients with desmoid-type fibromatosis.
2 deficiency and maternally inherited gingival fibromatosis.
3 old boy suffering from an aggressive form of fibromatosis.
4 well tolerated in children with desmoid-type fibromatosis.
5 is the most common genetic form of gingival fibromatosis.
6 e for autosomal-dominant hereditary gingival fibromatosis.
7 of the underlying genetic basis of gingival fibromatosis.
8 autosomal dominant form of isolated gingival fibromatosis.
9 rential diagnosis of GIST, including desmoid fibromatosis (0 of 17) and Schwannoma (0 of 3), were imm
10 A syndromic association between gingival fibromatosis and a wide variety of other genetically inh
12 ns for understanding other forms of gingival fibromatosis and corrective gingival-tissue management.
16 s is the first report of hereditary gingival fibromatosis associated with aggressive periodontitis.
17 We previously identified retroperitoneal fibromatosis-associated herpesvirus (RFHV) as a simian h
18 The complete sequence of retroperitoneal fibromatosis-associated herpesvirus Macaca nemestrina (R
19 ed macaque homologs of KSHV, retroperitoneal fibromatosis-associated herpesvirus-Macaca nemestrina (R
21 ltiple subcutaneous nodular tumors, gingival fibromatosis, flexion contractures of the joints, and an
22 ries, patients (<25 years) with desmoid-type fibromatosis from 57 centres in eight countries were pro
23 tion of ERK signaling in hereditary gingival fibromatosis gingival fibroblasts that was associated wi
28 Although non-syndromic hereditary gingival fibromatosis (HGF) is genetically heterogeneous, etiolog
31 ced gingival overgrowth, hereditary gingival fibromatosis (HGF), and neurofibromatosis I (von Recklin
32 ost common genetic form, hereditary gingival fibromatosis (HGF), is usually transmitted as an autosom
36 ssociated with maternally inherited gingival fibromatosis is an allelic disorder with cardiac arrhyth
40 heterodimer partner Maf (muscle aponeurosis fibromatosis) is sufficient and necessary for robust tra
47 ) were evaluated and diagnosed with gingival fibromatosis suggesting that this is a dominant trait in
49 sue tumors: desmoid tumor (DT) or aggressive fibromatosis, tenosynovial giant cell tumor (TGCT) or di
50 is the most common genetic form of gingival fibromatosis that develops as a slowly progressive, beni
51 otrexate (Mtx) in children with desmoid-type fibromatosis that is recurrent or not amenable to treatm
52 ON: In paediatric patients with desmoid-type fibromatosis, the EpSSG conservative strategy did not co
53 SOS1 is responsible for hereditary gingival fibromatosis type 1, a benign overgrowth condition of th
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