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1 asal epithelial, luminal epithelial, stromal fibromuscular, and endothelial - and for prostate precur
2 , more lipid accumulates and is covered by a fibromuscular cap to form a fibrous plaque.
3                         In addition, stromal fibromuscular cells associated with primary prostate can
4  The rectus extraocular muscles pass through fibromuscular connective tissue pulleys that stabilize m
5 rteriography in diagnosing renal artery (RA) fibromuscular dysplasia (FMD) and correlating with the h
6 ously described a strong association between fibromuscular dysplasia (FMD) and spontaneous coronary a
7                                              Fibromuscular dysplasia (FMD) involving the coronary art
8                                              Fibromuscular dysplasia (FMD) is a heterogeneous group o
9                                              Fibromuscular dysplasia (FMD) is a nonatheromatous, noni
10                                              Fibromuscular dysplasia (FMD) is a noninflammatory arter
11                                              Fibromuscular dysplasia (FMD) is a rare, nonatherosclero
12  on histology, the diagnosis of renal artery fibromuscular dysplasia (FMD) is now based mostly on ang
13 ce of intracranial aneurysm in patients with fibromuscular dysplasia (FMD) is uncertain.
14                                              Fibromuscular dysplasia (FMD), a noninflammatory disease
15 oderate to severe arterial changes caused by fibromuscular dysplasia (FMD), mild and distal moderate
16 ed with extracoronary vasculopathy including fibromuscular dysplasia (P<0.05 for both).
17                  The most common causes were fibromuscular dysplasia and atherosclerosis.
18 , P-SCAD was less likely with a diagnosis of fibromuscular dysplasia and extracoronary vascular abnor
19 anial aneurysms, cervical artery dissection, fibromuscular dysplasia and moyamoya disease.
20                  An association of CeAD with fibromuscular dysplasia and reversible cerebral vasocons
21        Majority of patients with NA-SCAD had fibromuscular dysplasia and type 2 angiographic SCAD.
22 dissections, aneurysms, atherosclerosis, and fibromuscular dysplasia can be identified on both MR ima
23 igorous program of prospective screening for fibromuscular dysplasia combined with intervention signi
24        Some patients may be asymptomatic and fibromuscular dysplasia could only be discovered by imag
25  been little new information published about fibromuscular dysplasia in the past 30 years.
26                                              Fibromuscular dysplasia is a noninflammatory, nonatheros
27                                              Fibromuscular dysplasia is a novel association and poten
28                                              Fibromuscular dysplasia is a pathologic diagnosis, but t
29                                              Fibromuscular dysplasia is an uncommon angiopathy that o
30                                              Fibromuscular dysplasia is an underdiagnosed and misunde
31                                              Fibromuscular dysplasia of the iliac artery was identifi
32 of primary RA repair correlated with complex fibromuscular dysplasia requiring branch ex vivo reconst
33                                              Fibromuscular dysplasia screening of renal, iliac, and c
34                                              Fibromuscular dysplasia was diagnosed in 72.0%.
35                                              Fibromuscular dysplasia was present in 62.7%, connective
36  of arterialized saphenous venous graft, and fibromuscular dysplasia) revealed a distinct increase in
37 al imaging registered in the US Registry for Fibromuscular Dysplasia, an observational disease-based
38 graine headache, cervical artery dissection, fibromuscular dysplasia, and hypertension.
39 d with extracoronary vasculopathy, including fibromuscular dysplasia.
40  or aneurysm may also occur in patients with fibromuscular dysplasia.
41 erial disease, is fundamentally a variant of fibromuscular dysplasia.
42 s and discrete, nonostial stenoses caused by fibromuscular dysplasia.
43 mplantation, reconstructed tissues displayed fibromuscular fatty structures typical of the alar lobul
44 pletely, the skin biopsy did show arteriolar fibromuscular intimal proliferation, which is usually se
45 infiltrated with macrophages than the stable fibromuscular lesion.
46 IVUS, lipid-laden lesions appear hypoechoic, fibromuscular lesions generate low-intensity echoes, and
47 focally into, and frequently compromise, the fibromuscular sheath.
48 ared with 8.6 for prazosin), 6.9 in anterior fibromuscular stroma (prazosin, 8.9), and 7.1 in bladder
49            Positive staining was seen in the fibromuscular stroma in normal prostates but not in the
50                                Moreover, the fibromuscular stroma surrounding prostatic glands was re
51 ing, remodeling, and hypercellularity of the fibromuscular stroma.
52 inal secretory epithelial, basal epithelial, fibromuscular stromal, nerve sheath, and endothelial cel

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