ライフサイエンスコーパス: fibroproliferation

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1 a potential mechanism for ventilator-induced fibroproliferation.

2 , and absent perfusion prior to the onset of fibroproliferation.

3 e regulation of angiogenesis-mediated airway fibroproliferation.

4 on of the airway epithelium and intraluminal fibroproliferation.

5 more effective than lower doses in reducing fibroproliferation (0% in high dose versus 29% in low do

6 near complete obliteration of the lumen with fibroproliferation (96.9% occlusion, =0.001) and absent

7 Pathological fibroproliferation after tissue injury is harmful and ma

8 serves as a physiological restraint to limit fibroproliferation after tissue injury.

9 providing a physiological mechanism to limit fibroproliferation after tissue injury.

10 lveolar angiogenesis and fibrogenesis (i.e., fibroproliferation and deposition of extracellular matri

11 and creating a local environment that favors fibroproliferation and matrix deposition.

12 crotic cores within the plaques, and reduced fibroproliferation and neovascularization in the aortic

13 important role in the development of airway fibroproliferation and obliteration.

14 nredundant role for CXCR3 in limiting tissue fibroproliferation and suggest that this effect may be m

15 ays, biochemical markers of inflammation and fibroproliferation, and infectious complications.

16 eptide III (PCP III), a biological marker of fibroproliferation, and with increased fatality rates.

17 gens, chronic inflammation, and unrestrained fibroproliferation are likely to be part of a dynamic, u

18 The role of CXCR3 in fibroproliferation has not been investigated.

19 CsA markedly reduced the development of fibroproliferation in allografts (19% in treated allogra

20 Abnormal vascular fibroproliferation in CAV occurs as a result of coronary

21 ents results in epithelial abnormalities and fibroproliferation in the airway lumen, changes not seen

22 use of CsA in our model system would reduce fibroproliferation in tracheal allografts.

23 yndrome (ARDS) is accompanied by progressive fibroproliferation, inability to improve lung injury sco

24 Exuberant fibroproliferation is a common complication after injury

25 data are consistent with the hypothesis that fibroproliferation is an early response to lung injury a

26 We hypothesized that fibroproliferation is initiated early in ARDS, character

27 ibroblast migration is an initiating step in fibroproliferation; its involvement during acute lung in

28 mmatory/immunologic process characterized by fibroproliferation, matrix deposition, and obliteration

29 eated transgenic mice demonstrated increased fibroproliferation, myofibroblast persistence, and impai

30 yndrome (ARDS) is characterized by excessive fibroproliferation, ongoing inflammation, prolonged mech

31 Diseases associated with pathological fibroproliferation represent a major cause of morbidity

32 There was little fibroproliferation seen in either of these groups.

33 ithelial loss correlated with progression to fibroproliferation, suggesting that the epithelium plays

34 ch is to promote resolution of physiological fibroproliferation that follows injury before it becomes

35 ors, activation of cell cycle signaling, and fibroproliferation, the central events in immunopathogen

36 on and airway ischemia in the development of fibroproliferation, we used a murine orthotopic tracheal

37 Lung transplantation is complicated by fibroproliferation, which is likely mediated in part by

38 ptosis but exhibit exaggerated CHI3L1-driven fibroproliferation, which together promote HPS fibrosis.

39 cipients, is characterized histologically by fibroproliferation within small airways.

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