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1 l tumor, chondroblastoma, osteoblastoma, and fibrous dysplasia.
2 (111)In-pentetreotide was noted in areas of fibrous dysplasia.
3 at this pattern of expression is retained in fibrous dysplasia.
4 atient with axial and peripheral polyostotic fibrous dysplasia and cafe au lait spots on clinical exa
5 was to assess the prevalence of polyostotic fibrous dysplasia and McCune-Albright syndrome among pat
9 ere retrospectively screened for polyostotic fibrous dysplasia based on their preoperative abdominal
12 OC complex: progenitor cell differentiation (fibrous dysplasia), extracellular matrix production (ost
13 sative role of Wnt/beta-catenin signaling in fibrous dysplasia (FD) of bone, a disease that exhibits
14 pective review of patients with craniofacial fibrous dysplasia (FD), the clinical and radiological fi
17 Sixty-seven optic canals were affected by fibrous dysplasia; in 49 of them (73 percent) there was
19 ave previously been shown to be the cause of fibrous dysplasia of bone (FD)/McCune-Albright syndrome
20 nt found in certain tumors and patients with fibrous dysplasia of bone and McCune-Albright syndrome g
21 Our data indicate the need to reinterpret fibrous dysplasia of bone as a disease of cells in the o
23 entation patterns and hyperendocrinopathies, fibrous dysplasia of bone is a major finding in the McCu
25 be indicated on the basis of the presence of fibrous dysplasia on diagnostic images alone, since it d
26 n McCune-Albright syndrome, characterized by fibrous dysplasia, precocious puberty, and cafe au lait
27 h the de novo deposition of lesional bone in fibrous dysplasia produce a bone matrix enriched in cert
28 lting in their defective differentiation and fibrous dysplasia, we identify Galphas as a key regulato
29 al desmoid tumor, osteofibrous dysplasia, or fibrous dysplasia were examined by cytogenetic analysis
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