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1 ed the inhibitory effect of ethanol on NMDAR field excitatory postsynaptic potentials.
2 pal slices during extracellular recording of field excitatory postsynaptic potentials.
3 ifts in net synaptic efficacy as measured by field excitatory postsynaptic potentials.
4 ic depression of the simultaneously recorded field excitatory postsynaptic potential and was greatly
5 the SC) using voltage-sensitive dye imaging, field excitatory postsynaptic potentials and whole cell
6 impairment of mGlu II-mediated depression of field excitatory postsynaptic potentials at mossy fiber-
7 were measured as the change in layer II/III field excitatory postsynaptic potentials by a multielect
8 ow-frequency voltage fluctuations (presumed "field excitatory postsynaptic potentials") during 89% of
9 creased excitatory postsynaptic currents and field excitatory postsynaptic potentials (EPSPs) with th
12 on protocol altered both the [Zn2+]t and the field excitatory postsynaptic potential (fEPSP) coordina
13 determined the cornu ammonis region 1 (CA1) field excitatory postsynaptic potential (fEPSP) response
14 pplication had different actions on both the field excitatory postsynaptic potentials (fEPSPS) and LT
15 effects of isoflurane were similar on evoked field excitatory postsynaptic potentials (fEPSPs) and pa
16 acologically isolated NMDA-receptor-mediated field excitatory postsynaptic potentials (fEPSPs) in the
18 inspired oxygen and the amplitude of evoked field excitatory postsynaptic potentials (fEPSPs) record
23 nhanced the size (amplitude and duration) of field excitatory postsynaptic potentials in hippocampal
24 es demonstrated that ethanol inhibited NMDAR field excitatory postsynaptic potential slope and amplit
25 orded from control mice and (ii) the plot of field excitatory postsynaptic potential slope versus the
26 developing nicotine-induced potentiation of field excitatory postsynaptic potential that appeared to
27 bility, and slow-wave sleep, produce a large field excitatory postsynaptic potential throughout strat
29 ubunit and ethanol's inhibition of the NMDAR field excitatory postsynaptic potential was attenuated b
30 er collateral pathway was stimulated and the field excitatory postsynaptic potentials were recorded i
31 t 60 min post-tetanization the slopes of the field excitatory postsynaptic potentials were significan
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