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1 neurostimulation techniques (e.g., cutaneous field stimulation).
2 release of [3H]acetylcholine in response to field stimulation.
3 ains by employing weak, alternating magnetic field stimulation.
4 to cholecystokinin octapeptide or electrical field stimulation.
5 ulated with a muscarinic agonist or electric field stimulation.
6 rebound responses similar to the response to field stimulation.
7 ory thalamic neurons to peripheral receptive field stimulation.
8 sponses to non-noxious and noxious receptive field stimulation.
9 e potential caused by extracellular electric field stimulation.
10 ted by ipsilateral rostral scratch receptive field stimulation.
11 rical brain responses associated with visual field stimulation.
12 se of [3H]NE was elicited through electrical field stimulation.
13 ncholinergic nerve stimulation by electrical field stimulation.
14 ual-evoked potentials associated with visual field stimulation.
15 enhanced contractile response to electrical field stimulation.
16 ergic neurostimulation initiated by electric field stimulation.
17 ntractile responses to CCh and to electrical field stimulation.
18 rmal spontaneous Ca(2+) release events after field stimulation.
19 es of anesthetized mice in response to local field stimulation.
20 PY overflow increased 0 s and 30 s following field stimulation.
21 nd centralized rotors by optical S1-S2 cross-field stimulation.
22 arterioles was assessed at 0 s and 30 s post-field stimulation.
23 ng bilateral compared with unilateral visual field stimulation.
24 stimulation and phase two started 35 s after field stimulation.
25 and prVEP amplitude evoked by full- and half-field stimulation.
26 e of FM4-64 only during the initial phase of field stimulation.
27 cted to increasingly faster pacing rates via field stimulation.
28 NEP) using alternating current (ac)-magnetic field stimulation.
29 , and their frequency could be elevated with field stimulation.
33 d 1000 ms) and SNA was initiated using local field stimulation (30-50 V; 1 ms at 2, 8 and 16 Hz).
34 on with elevated KCl or patterned electrical field stimulation (50 biphasic rectangular pulses of 25
35 ntitate the overflow of ADPR upon electrical field stimulation (8 Hz, 0.3 ms, 15 V, 1-2 min) of both
37 ty were evaluated using video microscopy and field stimulation after exposure to osmotic stress at 37
38 ning both a nearly normal twitch response to field stimulation and a robust positive inotropic respon
39 interactive effects of pulsatile electrical field stimulation and substrate topography on cell diffe
40 ion channels using extracellular electrical field stimulation and voltage-sensitive fluorescent prob
41 lar [Ca2+] was monitored using fura-2 during field stimulation and while membrane potential was contr
42 e cell membrane under conditions of electric field stimulation, and a counterbalancing interplay betw
43 Delta) preparations, responses to electrical field stimulation are nearly abolished despite persisten
44 contraction of bladder strips to electrical field stimulation, as well as activation with carbachol,
48 om wild-type (WT) mice, [Na](i) increased on field stimulation at 2 Hz from 11.1+/-1.8 mmol/L to a pl
50 We investigated the effects of electrical field stimulation (average frequency 2 Hz) in an irregul
51 In the normal homozygous mice, electrical field stimulation caused a biphasic response, an initial
52 auditory cortex (AC stimulation) under free field stimulation conditions using a two-tone inhibition
56 P, overflow of beta-NAD evoked by electrical field stimulation correlated with stimulation frequency
58 cholinergic responses elicited by electrical field stimulation (EFS) (8 Hz, 1 ms, 8 V for 10 s every
59 ergic (NANC) nerve stimulation by electrical field stimulation (EFS) (either short-train or continuou
60 ular transmission was achieved by electrical field stimulation (EFS) (frequency, 10 Hz; pulse voltage
61 es min(-1) using 0.1 ms pulses of electrical field stimulation (EFS) (three pulses delivered at 3-30
62 of the guinea pig vas deferens to electrical field stimulation (EFS) and norepinephrine (NE) exocytos
63 t treatment vs. neurostimulation by electric field stimulation (EFS) in bovine tracheal smooth muscle
64 xation responses were assessed by electrical field stimulation (EFS) in presence of A(2B)AR agonists,
65 S was induced by CO, NO, VIP, and electrical field stimulation (EFS) in the presence and absence of n
69 rgic stimulation by NO donors and electrical field stimulation (EFS) of intrinsic enteric inhibitory
70 ne, guanethidine and propranolol, electrical field stimulation (EFS) of the atrial preparations evoke
72 s in an organ bath in response to electrical field stimulation (EFS) under nonadrenergic, noncholiner
74 one, vasoconstrictor responses to electrical field stimulation (EFS) were not attenuated by vitamin E
75 ses, frequency-response curves to electrical field stimulation (EFS) were performed with results expr
76 resides in the combination of the electrical field stimulation (EFS) with data acquisition in spatial
77 measured in the basal state, after electric field stimulation (EFS), ganglionic stimulant dimethyl d
79 lium acts as a crucial sensor for electrical field stimulation (EFS)-enhanced osteogenic response in
81 gs of (i) caffeine-induced-, (ii) electrical field stimulation (EFS)-induced force, (iii) pCa-force,
85 n to ATP and norepinephrine (NE), electrical field stimulation (EFS, 4-16 Hz, 0.1-0.3 ms, 15 V, 60-12
86 ons were stimulated for 60 s with electrical field stimulation (EFS; vas deferens), dimethylphenylpip
90 e epilepsy were exposed to weak, DC magnetic field stimulation following computer-controlled protocol
91 eased the contractile response to electrical field stimulation for 48 hours after administration.
94 after spontaneous contraction or short-term field stimulation fusion activity increased in cardiomyo
97 tage (V(m)) of cardiac cells during electric field stimulation have a complex spatial- and time-depen
98 tylcholine release in response to electrical field stimulation in both the stomach and small intestin
99 siologically relevant patterns of electrical field stimulation in culture, including tonic stimulatio
100 spatially resolved Ca(2+) transients during field stimulation in left ventricular (LV) myocytes from
101 lateral and unilateral left and right visual field stimulation in normal and compromised systems.
104 oth muscle hyperresponsiveness to electrical field stimulation, indicating that IgE/CD23-mediated imm
108 de of the somatic Ca2+ increase triggered by field stimulation is independent of the extracellular Ca
109 urred spontaneously and could be evoked with field stimulation: large, rapid, global Ca2+ transients
113 results are consistent with predictions from field stimulation of continuous representations of myoca
114 much has been learned about BDNF function by field stimulation of hippocampal neurons, it is not know
115 ectrical activity or responses to electrical field stimulation of intrinsic nerves in these mice.
116 ansmission have used indiscriminate electric field stimulation of neural elements within the gastric
120 the ciliary nerves in the eye, or electrical field stimulation of the cornea, evoked Ca2+ transients
122 testinal peptide (VIP) and direct electrical field stimulation of the intrinsic innervation; inhibiti
123 ing, and function was assessed by electrical field stimulation of tracheas in the presence/absence of
125 inantly observed in the contralateral visual field, stimulation of hMST also affected the ipsilateral
128 ng fluorescence technologies with electrical field stimulation or the patch-clamp technique in beatin
129 ty to neuropeptide Y (P<0.05) and electrical field stimulation (P<0.05) in mesenteric arteries was al
130 USA) delivers percutaneous electrical nerve field stimulation (PENFS) in the external ear to modulat
132 ion of skeletal muscle fibres via electrical field stimulation produced a vasodilatation (19.4 +/- 1.
133 so demonstrate that near-threshold diastolic field stimulation produces activation of deep myocardial
136 imetic actuation when controlled by electric field stimulation results in movement similar to that of
137 teries subjected to low-frequency electrical field stimulation revealed Ca2+ transients in perivascul
138 OFF-centre RGC responses were evoked by full-field stimulation, significantly fewer converted that re
140 ATP, and a reduced contraction to electrical field stimulation, suggesting altered P2X1 receptor func
141 (IJPs) and relaxations evoked in response to field stimulation (supramaximal voltage, 0.1 ms, single
142 mbining fluorescence imaging with electrical field stimulation, the patch-clamp method and knock-out
143 Primary cilia are sensors of electrical field stimulation to induce osteogenesis of human adipos
145 owed similar patterns of surface labeling by field stimulation to those shown previously by high pota
146 emonstrate that transcranial static magnetic field stimulation (tSMS) over the somatosensory parietal
148 Maximum sarcomere shortening elicited by field stimulation was graded in the order: LVendo > LVep
149 oline (ACh) or noradrenaline (NA) release to field stimulation was measured 4 months after gene trans
150 ction potentials (S-IJPs) evoked by electric field stimulation was significantly higher in CSE-KO mou
151 cy-dependent relaxation evoked by electrical field stimulation was significantly lower in gallbladder
154 cell activity responses to somatic receptive fields stimulation, was compared between anesthetized ca
155 onophosphate elevations evoked by electrical field stimulation were diminished markedly in both nNOSD
156 C-acetylcholine (ACh) released by electrical field stimulation were measured simultaneously in strips
157 C]acetylcholine (ACh) released by electrical field stimulation were measured simultaneously in strips
159 art cell contractility inducible by electric field stimulation with directionally dependent electrica
161 tment spiking neurons responding to electric field stimulation with small incremental polarization.
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