ライフサイエンスコーパス: filaria

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1 Extracts from the rodent filaria, Acanthocheilonema viteae, which is not infected

2 ddition of an IL-10-neutralizing antibody to filaria antigen-stimulated cultures resulted in signific

3 iotic Wolbachia in B. malayi and O. volvulus filaria are dependent on TLR2-TLR6 interactions and are

4 ofilaremic (MF; n = 16) and in patients with filaria-associated lymphatic pathology (LP; n = 23).

5 ompare against the Wolbachia-dependent model filaria, Brugia malayi.

6 To explore the role of filaria-derived parasite antigen in differentiation of h

7 implicated in a variety of roles, including filaria development and fecundity and the pathogenesis o

8 pared with uninfected controls from the same filaria-endemic region of Mali.

9 unomodulatory roles of IL-4 and IFN-gamma in filaria-induced AHR and pulmonary inflammation using mic

10 e that IL-4 is required for the induction of filaria-induced AHR, whereas IFN-gamma suppresses AHR.

11 cells (Tr1s)]) CD4(+) and CD8(+) T cells in filaria-infected (Fil(+)) and -uninfected (Fil(-)) indiv

12 examined the cytokine profile of a cohort of filaria-infected (n = 10) and uninfected (n = 10) indivi

13 tude of malaria-specific T cell responses in filaria-infected and -uninfected individuals with concom

14 , TLR4, and TLR9 on B cells and monocytes of filaria-infected and uninfected individuals.

15 lower in B cells but not in monocytes of the filaria-infected group compared with the uninfected grou

16 t that, when Ag-specific IgE is present in a filaria-infected individual, basophils function to ampli

17 ression was performed on purified T cells of filaria-infected individuals (n = 6) and uninfected cont

18 s of circulating mDCs (CD11c(+)CD123(lo)) in filaria-infected individuals compared with uninfected co

19 Our data indicate that filaria-infected individuals had a lower percentage of c

20 IgE ELISA confirmed that filaria-infected individuals had higher IgE prevalences

21 ed at significantly lower levels in cells of filaria-infected individuals in response to BmA compared

22 Monocytes from filaria-infected individuals were studded with intracell

23 expression is diminished on B and T cells of filaria-infected individuals, we examined the effect of

24 ral blood mononuclear cells from children of filaria-infected mothers (n = 33) had higher levels of c

25 ikely to be from newborns of schistosome- or filaria-infected mothers than from uninfected mothers (p

26 Using serum samples from a filaria-infected patient who also had dust mite (Dermato

27 uction and gene expression in monocytes from filaria-infected patients and uninfected healthy donors

28 function and gene expression of monocytes in filaria-infected patients are altered but that this dysf

29 Furthermore, PBMC from 6 filaria-infected patients before antifilarial treatment

30 Serum from filaria-infected patients did not cross-react when teste

31 PBMC from filaria-infected patients produced less RANTES (P=.02) b

32 producing IL-10 was significantly greater in filaria-infected patients than in uninfected control sub

33 oward increased HIV replication in PBMC from filaria-infected patients was observed.

34 Most IL-10-producing cells in filaria-infected patients were T cells, with CD4(+) and

35 n basophils in the peripheral circulation of filaria-infected patients, the absolute numbers of basop

36 In experiments with fresh blood drawn from filaria-infected patients, we found no association betwe

37 hil populations from 20 patients with active filaria infections correlated strongly with total serum

38 function using the Litomosoides sigmodontis filaria model of chronic helminth infection in BALB/c mi

39 tored pretreatment serum was used to measure filaria-specific antibody responses, eosinophil-related

40 ariae, few clinical symptoms, and diminished filaria-specific IgG.

41 ood susceptibility to W. bancrofti and skews filaria-specific immunity toward a Th2-type cytokine res

42 ly elevated eosinophil counts, and increased filaria-specific immunoglobulin G (IgG) levels; these fi

43 e cells adhere to L3 only in the presence of filaria-specific sera or IgM purified from them.

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