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1 n in a chronic helminth infection (lymphatic filariasis).
2 North Americans with no history of lymphatic filariasis.
3 ecology and pathogenesis of human lymphatic filariasis.
4 (H2(d)), the only fully permissive model of filariasis.
5 seases such as river blindness and lymphatic filariasis.
6 n of programs aimed at eliminating lymphatic filariasis.
7 symbionts into the blood after treatment for filariasis.
8 malayi, a causative agent of human lymphatic filariasis.
9 or nonantigenemic individuals with clinical filariasis.
10 ofile are well-established features of human filariasis.
11 gamma- and IL-4-producing cells in lymphatic filariasis.
12 infection and morbidity rates in bancroftian filariasis.
13 e of the causative agents of human lymphatic filariasis.
14 sociated with cytokine patterns in lymphatic filariasis.
15 rugia malayi, a causative agent of lymphatic filariasis.
16 ntial to accelerate elimination of lymphatic filariasis.
17 diseases like river blindness and lymphatic filariasis.
18 i, one of the nematodes that cause lymphatic filariasis.
19 nia africana, which are vectors of lymphatic filariasis.
20 as well as of nematodes that cause lymphatic filariasis.
21 mosquitoes that transmit West Nile fever and filariasis.
22 erlying T cell immune tolerance in lymphatic filariasis.
23 fic T-cell response seen in patent lymphatic filariasis.
24 he vector-borne parasitic disease, lymphatic filariasis.
25 account for the T cell hyporesponsiveness in filariasis.
26 erlying the dysfunctional immune response in filariasis.
27 concept of protective immunity in lymphatic filariasis, 19 adult residents of a Wuchereria bancrofti
28 oncepts of disease progression (asymptomatic filariasis = 25%; clinical filariasis with active infect
29 with a low baseline prevalence of lymphatic filariasis (5%), the triple-drug regimen reduced the num
30 ases including river blindness and lymphatic filariasis affect hundreds of millions of people annuall
32 associated with disease in chronic lymphatic filariasis and could potentially have an important role
33 ore effective than DEC + ALB for Bancroftian filariasis and has the potential to accelerate eliminati
37 d by filarial nematodes, including lymphatic filariasis and onchocerciasis (river blindness) has tran
40 ger-scale ivermectin treatment for lymphatic filariasis and onchocerciasis in areas where L. loa infe
42 in the global effort to eliminate lymphatic filariasis and possibly for the control of other mosquit
43 enya, where BCG is administered at birth and filariasis and schistosomiasis are endemic, were examine
44 administration for elimination of lymphatic filariasis and soil-transmitted helminth infections in R
45 ticle reviews available diagnostic tests for filariasis and their potential use as tools for differen
49 h as guinea worm, schistosomiasis, lymphatic filariasis, and onchocerciasis, suggests that many of th
51 lymphoedema, and elephantiasis in lymphatic filariasis, and skin disease and blindness in onchocerci
52 an area highly endemic for schistosomiasis, filariasis, and tuberculosis in Kenya would either fail
53 within-host population dynamics of lymphatic filariasis, and use a simulated goodness-of-fit (GOF) me
54 d Th1 responses observed in patent lymphatic filariasis are associated with decreased expression of T
56 mpaigns against onchocerciasis and lymphatic filariasis being conducted in areas where Onchocerca vol
57 ential for school-based control of lymphatic filariasis by investigating the efficacy and tolerabilit
58 th ivermectin plus albendazole for lymphatic filariasis cannot be applied in central Africa, because
62 ver, few such programmes exist for lymphatic filariasis, despite evidence that single-dose treatment
63 s, whether with subclinical or with clinical filariasis, distinct and limited T cell populations are
64 idence that protective immunity to lymphatic filariasis does occur and that it is probably T cell-med
67 immunocompromised mouse models suggest that filariasis elicits a complex host immune response involv
68 Improved diagnostic tests are needed for filariasis elimination programs (to identify areas of en
70 in A is an antibiotic to develop further for filariasis elimination without concern for cross-resista
71 es, including dengue, malaria, and lymphatic filariasis, exact a devastating toll on global health an
74 ting Global Programme to Eliminate Lymphatic Filariasis (GPELF) is largely based on a strategy of mas
75 The Global Program to Eliminate Lymphatic Filariasis (GPELF) was launched in 2000 with the princip
76 l Programme for the Elimination of Lymphatic Filariasis (GPELF), as interaction dynamics may change w
78 e elimination of onchocerciasis or lymphatic filariasis has been delayed in Central Africa because of
79 l to accelerate the elimination of lymphatic filariasis if high population coverage of mass drug admi
80 l to accelerate the elimination of lymphatic filariasis if high population coverage of mass drug admi
82 ration of albendazole to eliminate lymphatic filariasis in areas where loiasis is co-endemic and iver
85 ns is the cause of the second most prevalent filariasis in Gabon, but so far reports on the presence
86 cellular parasites responsible for lymphatic filariasis in humans--and the APC with which they come i
88 is a severe asthmatic syndrome of lymphatic filariasis, in which an allergic response is induced to
91 to be qualitatively different from those of filariasis-infected subjects; whereas filarial antigens
96 ent global initiative to eliminate lymphatic filariasis is a major renewed commitment to reduce or el
105 ic T-cell unresponsiveness seen in lymphatic filariasis is mediated, in part, by diminished antigen-p
106 d from individuals from North America, where filariasis is not endemic, were also positive for anti-W
111 One of the causative agents of lympahtic filariasis is the nematode parasite Brugia malayi that r
116 possible strategy for eliminating lymphatic filariasis (LF) in post-conflict countries such as the D
119 tropical diseases (NTD), including lymphatic filariasis (LF), scaled up dramatically after the signin
120 ommunities tasked with eliminating lymphatic filariasis (LF), the underlying cause of elephantiasis a
124 ce with Wuchereria bancrofti due to maternal filariasis may influence susceptibility to infection.
127 the data available for five NTDs (lymphatic filariasis, onchocerciasis, intestinal helminthiasis, sc
128 amples from patients with loiasis, lymphatic filariasis, onchocerciasis, mansonellosis, or other helm
129 oadministration of drugs to target lymphatic filariasis, onchocerciasis, schistosomiasis, soil-transm
130 from filarial nematodes that cause lymphatic filariasis or onchocerciasis, resulting in blocked worm
131 - to 10-year-old children of mothers without filariasis or schistosomiasis produced 10-fold more IFN-
132 duals, antigenemic individuals with clinical filariasis, or nonantigenemic individuals with clinical
136 est this hypothesis in bancroftian lymphatic filariasis, pedigree data were collected twice during an
139 rugia malayi, a causative agent of lymphatic filariasis, resulting in the identification of more than
142 s, could be administered as short courses to filariasis target populations with potential to reduce a
143 the nonantigenemic individuals with clinical filariasis than in the asymptomatic microfilaremic indiv
144 and Th2 immune responses occurs in lymphatic filariasis that is governed at the transcriptional level
145 h the putatively immune state in bancroftian filariasis (that is, both microfilaria and antigen free)
146 site products has been investigated in human filariasis to understand immune hyporesponsiveness.
148 nitor populations for possible resurgence of filariasis transmission following suspension of MDA.
149 study of diseases as diverse as lymphedema, filariasis, transplant rejection, obesity, and tumor met
151 Culex pipiens mosquito group (including the filariasis vector C. quinquefasciatus) a very unusual de
152 ite of disease activity in human bancroftian filariasis, we have compared the repertoire of TCR Vbeta
153 ls and antigenemic individuals with clinical filariasis were grouped together to constitute all activ
156 , tuberculosis, leishmaniasis, and lymphatic filariasis, which impose tremendous public health burden
157 ion (asymptomatic filariasis = 25%; clinical filariasis with active infection = 60%; clinical filaria
158 ymptomatic microfilaremic or having clinical filariasis with active infection or without current acti
160 e and well-tolerated treatment for lymphatic filariasis with significant activity against adult worms
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