ライフサイエンスコーパス: fimbria-fornix

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1 ptum following unilateral transection of the fimbria fornix.

2 cellularis, or unilateral transection of the fimbria fornix.

3 the septum 3 weeks before transection of the fimbria fornix.

4 ubcortical inputs to the hippocampus via the fimbria-fornix.

5 ore auditory gating following lesions of the fimbria-fornix.

6 ased dramatically in the corpus callosum and fimbria/fornix.

7 ections from the ventral hippocampus via the fimbria/fornix.

8 We have lesioned the fimbria fornix, a major pathway of septal cholinergic fi

9 gnocellularis, radiofrequency lesions of the fimbria-fornix, and aspiration lesions of the frontal co

10 atter regions including the corpus callosum, fimbria/fornix, and cerebellar deep white matter.

11 entorhinal cortices, the corpus callosum and fimbria/fornix, and cerebellar white matter.

12 h factor, can also prevent the loss of these fimbria fornix axotomised cholinergic neurons, where GM1

13 sly, we demonstrated that transection of the fimbria/fornix blocked the excitatory effect of corticot

14 11 and continuing until p28, the ipsilateral fimbria fornix degenerates.

15 Lesions of the fimbria-fornix disrupt auditory gating by preventing cho

16 er-matched controls along the left and right fimbria-fornix (FF), parahippocampal WM bundle (PWMB), a

17 thalamic nuclei (ATN) or transection of the fimbria-fornix (FF).

18 ll animals received a complete lesion of the fimbria-fornix (FF).

19 (approximately 100 msec duration) evoked by fimbria/fornix (FF) stimulation in a majority of neurons

20 Results demonstrate the importance of the fimbria-fornix fiber system in spatial short-term memory

21 region (PARA) or electrolytic lesions of the fimbria-fornix (FNX) and were tested for their (a) discr

22 pal white matter bundle, and the ipsilateral fimbria-fornix in regions located within the medial temp

23 ast, patterned electrical stimulation of the fimbria-fornix increased TGC in amnestic animals and par

24 Transection of the fimbria fornix leads to retrograde degeneration of axoto

25 control animals 2 weeks following unilateral fimbria-fornix lesion.

26 of trkA (RTA) into the lateral ventricle of fimbria-fornix lesioned animals.

27 toration of auditory gating by DMXB-A in the fimbria-fornix-lesioned rats was blocked by intracerebro

28 DMXB-A restored auditory gating in the fimbria-fornix-lesioned rats, indicating that activation

29 pal injections of 192 IgG-saporin (SAP-HPC), fimbria-fornix lesions (FF), or sham control surgeries w

30 ions on hippocampal physiology compared with fimbria-fornix lesions and septal inactivation, we obser

31 From preoperative imaging, the fimbria-fornix, parahippocampal white matter bundle and

32 s have demonstrated that DBS targeted to the fimbria-fornix, the region that appears to regulate hipp

33 migrated into corpus callosum, striatum, and fimbria fornix to differentiate into the NG2-positive no

34 of colchicine-pretreated, control, untreated fimbria-fornix-transected (5 days), as well as perforant

35 Fimbria-fornix transection resulted in a marked loss of

36 Similar to the effects of fimbria-fornix transection, Pb exposure resulted in a lo

37 is (cynomolgous) monkeys received unilateral fimbria fornix transections followed by chronic intracra

38 Fimbria-fornix transections (FFTs), conducted 1 day afte

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