ライフサイエンスコーパス: fine particulate matter

1 erprints for source apportionment of ambient fine particulate matter.

2 lfate measured by ion chromatography (IC) in fine particulate matter.

3 alue of statistical life and the toxicity of fine particulate matter.

4 dfire smoke, including those associated with fine particulate matter.

5 o estimate the annual mean concentrations of fine particulate matter.

6 confounders, including co-pollutants such as fine particulate matter.

7 e 25th and 75th percentiles as cutpoints for fine particulate matter.

8 measurements of the chemical composition of fine particulate matter.

9 An average increase of 6 microg/m(3) in fine particulate matter 2 days before onset was associat

10 d Analysis Methods for Airborne Carbonaceous Fine Particulate Matter" (2003) and "Source Apportionmen

11 artile-range increase in average exposure to fine particulate matter (3.2 microg/m(3)) during pregnan

12 en maternal exposure to nitrogen dioxide and fine particulate matter (aerodynamic diameter </=2.5 mic

13 y (COWO), the role of early life exposure to fine particulate matter (aerodynamic diameter <2.5 mum;

14 Prenatal exposure to ambient PM2.5, (i.e., fine particulate matter, aerodynamic diameter </= 2.5 mu

15 Exposure to ambient fine particulate matter air pollution (PM(2.5); < 2.5 mi

16 ic studies have linked long-term exposure to fine particulate matter air pollution (PM) to broad caus

17 at further public policy efforts that reduce fine particulate matter air pollution are likely to have

18 Associations between source-specific fine particulate matter and emergency department visits

19 associations between source contributions of fine particulate matter and emergency department visits

20 (SOA) constitutes a substantial fraction of fine particulate matter and has important impacts on cli

21 ous compounds are a significant component of fine particulate matter and haze in national parks and w

22 e findings confirm the link between OHCA and fine particulate matter and introduce evidence of a simi

23 For fine particulate matter and NO2, we used land use regres

24 Long-term exposure to ambient fine particulate matter and renal function in older men:

25 us work has examined the association between fine particulate matter and risk of kidney disease; howe

26 tent association between increased levels of fine particulate matter and risk of primary cardiac arre

27 l cardiac arrest (OHCA) are inconsistent for fine particulate matter, and, although pathophysiologica

28 (diet)/biomedical (drug) and environmental (fine particulate matter) applications of sub-mg of C sam

29 easurements were made for both ultrafine and fine particulate matter as well as black carbon inside a

30 to HEPA-filtered air or concentrated ambient fine particulate matter (CAP, 30-100 microg/m(3)) from d

31 racers of primary and secondary carbonaceous fine particulate matter collected at four midwestern Uni

32 sual Environments) network has characterized fine particulate matter composition at locations through

33 The 2003 annual average fine particulate matter concentration was not associated

34 e improve the accuracy of daily ground-level fine particulate matter concentrations (PM(2.5)) derived

35 (GWR) statistical model to represent bias of fine particulate matter concentrations (PM2.5) derived f

36 and major roads were calculated, and annual fine particulate matter concentrations, derived from a s

37 conducive to the formation of peak ozone and fine particulate matter concentrations.

38 5% confidence interval (CI): 1.04, 1.39) and fine particulate matter (diameter </=2.5 mum) on lag day

39 Air pollution health studies of fine particulate matter (diameter </=2.5 mum, PM2.5) oft

40 Life cycle fine particulate matter emissions are higher for ethanol

41 Airborne fine particulate matter exhibits spatiotemporal variabil

42 Fine particulate matter exposure from both ambient air p

43 Fine particulate matter exposure was associated with CAC

44 nal age, race/ethnicity, education, prenatal fine particulate matter exposure, prenatal smoking expos

45 We used weekly fine-particulate-matter exposure data for 238 births in

46 and duration of exposure to fire-originated fine particulate matter (fire-PM2.5) and CHVI.

47 tted against estimated average daily dose of fine particulate matter from cigarette smoke along with

48 udies utilizing source apportionment (SA) of fine particulate matter have shown that particles from c

49 re is consistent evidence that the levels of fine particulate matter in the air are associated with t

50 n period (OR = 1.35, 95% CI: 1.09, 1.66) and fine particulate matter in the spring (OR = 1.28, 95% CI

51 We propose that the nicotine and fine particulate matter in tobacco smoke lead to increas

52 between cardiovascular disease mortality and fine particulate matter is relatively steep at low level

53 is strong evidence of an association between fine particulate matter less than 2.5 mum (PM2.5) in aer

54 Long-term exposure to fine particulate matter less than 2.5 mum in diameter (P

55 re is a positive association between ambient fine particulate matter </= 2.5 mum in aerodynamic diame

56 the association between prenatal exposure to fine particulate matter </= 2.5 mum in aerodynamic diame

57 Growing evidence indicates that toxicity of fine particulate matter </= 2.5 mum in diameter (PM2.5)

58 Elevated levels of fine particulate matter <2.5 microm in aerodynamic diame

59 olar epithelial cells in culture and mice to fine particulate matter <2.5 microm in diameter (PM(2.5)

60 Previous studies have examined fine particulate matter (</= 2.5 mum; PM2.5) and preterm

61 Both short- and long-term exposures to fine particulate matter (</= 2.5 mum; PM2.5) are associa

62 Outdoor fine particulate matter (</= 2.5 mum; PM2.5) has been id

63 Given that fine particulate matter (</= 2.5 mum; PM2.5) is a mixtur

64 o satellite-based nitrogen dioxide (NO2) and fine particulate matter (</= 2.5 mum; PM2.5) predictions

65 90 days before birth for temporally resolved fine particulate matter (</= 2.5 mum; PM2.5), black carb

66 primary cardiac arrest and daily measures of fine particulate matter (</=2.5 micro m) using a case-cr

67 Laboratory studies suggest that exposure to fine particulate matter (</=2.5 mum in diameter) (PM2.5)

68 We hypothesized that exposure to ambient fine particulate matter (<2.5 mum; PM(2.5)) exaggerates

69 ad more symptoms associated with exposure to fine particulate matter measuring less than 2.5 mum in d

70 ve risk for a 13.8- micro g/m(3) increase in fine particulate matter (nephelometry: 0.54 x 10(-1) km(

71 Ambient fine particulate matter, nitrogen dioxide, and preterm b

72 d a unified modeling approach for predicting fine particulate matter, nitrogen dioxide, oxides of nit

73 Exposure to fine particulate matter, O3, and NO2 was positively asso

74 nt cohort, we found positive associations of fine particulate matter, O3, and NO2 with mortality.

75 Health risks of fine particulate matter of 2.5 microm or less in aerodyn

76 he-art air pollution exposure assessments of fine particulate matter, oxides of nitrogen, and black c

77 ontextual covariates, including exposures to fine particulate matter, ozone, and nitrogen dioxide.

78 Data on concentrations of fine particulate matter (particles that are < or = 2.5 m

79 Annual averages of fine particulate matter (particles with a mass median ae

80 nalyses used daily cardiovascular mortality, fine particulate matter (particles with an aerodynamic d

81 eses that subchronic and chronic exposure to fine particulate matter (particulate matter < or = 2.5 m

82 sible joint effects of cigarette smoking and fine particulate matter (particulate matter less than or

83 The impact of chronic exposure to fine particulate matter (particulate matter with an aero

84 vestigated the short-term effects of UFP and fine particulate matter (particulate matter with an aero

85 Estimates of fine particulate matter (particulate matter with an aero

86 th effects associated with acute exposure to fine particulate matter (particulate matter with an aero

87 ile exhaust contains precursors to ozone and fine particulate matter (PM </= 2.5 microm in aerodynami

88 ozone and black carbon (BC), a component of fine particulate matter (PM </= 2.5 microm in aerodynami

89 Exposure to fine particulate matter (PM with diameter </= 2.5 mum; P

90 iabetes mellitus associated with exposure to fine particulate matter (PM(2.5)) and nitrogen oxides in

91 d health effects of greenhouse gas (GHG) and fine particulate matter (PM(2.5)) emissions from gasolin

92 egional decreases in ozone (0.2-0.7 ppb) and fine particulate matter (PM) (0.1-0.7 mug/m(3)).

93 the search for AD risk factors; exposure to fine particulate matter (PM) air pollution increases car

94 Fine particulate matter (PM) air pollution is associated

95 of magnitude higher than ambient coarse and fine particulate matter (PM) collected from downtown Tor

96 are thought to be related to exposure to the fine particulate matter (PM) component of HAP, but it is

97 anic compounds are important constituents of fine particulate matter (PM) in the troposphere.

98 Airborne fine particulate matter (PM) is responsible for the most

99 mine the putative adverse effects of ambient fine particulate matter (PM2.5 : PM with aerodynamic dia

100 Fine particulate matter (PM2.5) air pollution exposure h

101 s with changes in nitrogen dioxide (NO2) and fine particulate matter (PM2.5) air pollution.

102 Measurements of fine particulate matter (PM2.5) and absorbance were cond

103 ions relative to baseline of 45% and 47% for fine particulate matter (PM2.5) and carbon monoxide (CO)

104 and 2013, we measured personal exposures to fine particulate matter (PM2.5) and carbon monoxide (CO)

105 Data on exposures to fine particulate matter (PM2.5) and ozone (O3) were obta

106 alth by increasing ambient concentrations of fine particulate matter (PM2.5) and ozone (O3).

107 tion between short-term exposures to ambient fine particulate matter (PM2.5) and ozone, and at levels

108 Vehicle emissions contribute to fine particulate matter (PM2.5) and tropospheric ozone a

109 te phase-out scenario reduces annual average fine particulate matter (PM2.5) by an estimated 0.71 mug

110 hrough December 2011 and annual estimates of fine particulate matter (PM2.5) by census tract.

111 Exposure to ozone and fine particulate matter (PM2.5) can cause adverse health

112 Short-term exposure to ambient fine particulate matter (PM2.5) concentrations has been

113 We estimated global fine particulate matter (PM2.5) concentrations using inf

114 model routinely underpredicts peak ozone and fine particulate matter (PM2.5) concentrations.

115 In highly polluted environments, fine particulate matter (PM2.5) containing redox-active

116 inorganic sulfate concentrations in ambient fine particulate matter (PM2.5) data from 2005 to 2012 a

117 ween ambient species in high time resolution fine particulate matter (PM2.5) data to form clusters th

118 gy, could increase ambient concentrations of fine particulate matter (PM2.5) due to its ammonia emiss

119 lected and analyzed within-city variation of fine particulate matter (PM2.5) elements.

120 These fuel additives also reduce fine particulate matter (PM2.5) emissions and alter the

121 nt air pollution, accounting for over 25% of fine particulate matter (PM2.5) emissions in Canada.

122 Long-term fine particulate matter (PM2.5) exposure is linked with

123 nd temporally resolved black carbon (BC) and fine particulate matter (PM2.5) exposures, residential p

124 Exposure to fine particulate matter (PM2.5) from indoor and outdoor

125 ntion advisories were designed to occur when fine particulate matter (PM2.5) from smoke, forecasted 2

126 Ambient fine particulate matter (PM2.5) has a large and well-doc

127 Fine particulate matter (PM2.5) has been linked to cardi

128 ollution, up to 11 mug m(-3) for annual mean fine particulate matter (PM2.5) in northern Vietnam and

129 idence suggests that exposure to ambient air fine particulate matter (PM2.5) increases the risk of de

130 bution of DICE-Africa to annual mean surface fine particulate matter (PM2.5) is >5 mug m(-3) in popul

131 Ambient fine particulate matter (PM2.5) is a leading environment

132 Ambient fine particulate matter (PM2.5) is among the most preval

133 Exposure to atmospheric fine particulate matter (PM2.5) is associated with cardi

134 It is unknown if ambient fine particulate matter (PM2.5) is associated with lower

135 tigating whether chronic exposure to ambient fine particulate matter (PM2.5) is associated with morta

136 Fine particulate matter (PM2.5) is thought to be respons

137 ons, can significantly impact ozone (O3) and fine particulate matter (PM2.5) pollution across the U.S

138 ls to estimate premature mortality caused by fine particulate matter (PM2.5) pollution as a result of

139 dverse health effects of chronic exposure to fine particulate matter (PM2.5) requires accurate estima

140 summer (April-September) surface ozone (O3), fine particulate matter (PM2.5), and maximum temperature

141 develop land use regression (LUR) models for fine particulate matter (PM2.5), black carbon (BC), and

142 s than 1 ppb for ozone and 0.5 mug m(-3) for fine particulate matter (PM2.5), but widespread.

143 nitrogen oxides (NOx), carbon monoxide (CO), fine particulate matter (PM2.5), organic aerosol (OA), a

144 and assessed short-term exposure to ambient fine particulate matter (PM2.5), sulfates, black carbon

145 ic health impact modeling for ozone (O3) and fine particulate matter (PM2.5).

146 r use in a chemical mass balance (CMB) SA of fine particulate matter (PM2.5).

147 s a substantial mass fraction to atmospheric fine particulate matter (PM2.5).

148 O, black carbon (BC), NO, NO2, NOx, SO2, and fine particulate matter (PM2.5)] decreased with increasi

149 l studies have provided strong evidence that fine particulate matter (PM2.5; aerodynamic diameter </=

150 , the temporal pattern of the association of fine particulate matter (PM2.5; particulate matter </= 2

151 d normal chow or a high-fat diet to airborne fine particulate matters (PM2.5), and then investigated

152 ase (GBD) assessments estimated that outdoor fine-particulate matter (PM2.5) is a causal factor in ov

153 Long-term exposure to fine-particulate-matter (PM2.5) air pollution may accele

154 ns (particle number [PN], black carbon [BC], fine particulate matter [PM2.5], particle size).

155 The Salt Lake Valley experiences severe fine particulate matter pollution episodes in winter dur

156 Black carbon (BC) is fine particulate matter produced by the incomplete combu

157 particles (DEP), a major component of urban fine particulate matter, suppresses antimycobacterial hu

158 crog per cubic meter in the concentration of fine particulate matter was associated with an estimated

159 Exposure to fine particulate matter was positively associated with h

160 NO2 (a marker for traffic pollution) and fine particulate matter were also associated with mortal

161 ng the long-term health impacts of ozone and fine particulate matter with a diameter smaller than 2.5

162 CI, 1.101-1.147) on days of lower values of fine particulate matter with a median aerodynamic diamet

163 cal studies have linked residual oil fly ash fine particulate matter with aerodynamic diameter <2.5 m

164 ated costs; the GAINS model, which estimates fine particulate matter with aerodynamic diameter 2.5 mi

165 s investigated whether short-term effects of fine particulate matter with an aerodynamic diameter < o

166 ociated with concentrations of black carbon, fine particulate matter with an aerodynamic with diamete

167 3) is a key precursor species to atmospheric fine particulate matter with strong implications for reg

168 ong-term residential exposure to traffic and fine particulate matter with the degree of coronary athe

169 nt by other traffic-related pollutants: NO2, fine particulate matter (with aerodynamic diameter < or