ライフサイエンスコーパス: first instar

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1 ss susceptible to Cry1Ab toxin compared with first instars.

2 n all populations, and in salivary glands of first instars.

3 l morphology and connectivity change between first instar and third instar larval stages using serial

4 y nontoxic to monarch first instars, whereas first instars are sensitive to Cry1Ab and Cry1Ac protein

5 Potted host plants were infested with first instar black swallowtails and placed at intervals

6 -1KO-associated paralysis and death; starved first instar DmiR-1KO larvae are essentially normal.

7 ession of pnr and Iro-C is initiated in late first instar dorsal eye margin cells.

8 between brain and ventral nerve cord of the first-instar Drosophila larva.

9 Ac reduced insect mortality by 25.5-55.6% to first instar H. virescens and M. sexta larvae, suggestin

10 meless (tim) expression are initiated at the first instar (L1) larval stage or during metamorphosis,

11 tion, morphogenetic movements that shape the first instar larva.

12 We hypothesize that the first-instar larva (L1) of F. occidentalis mounts a resp

13 addition to adult dispersal, wind-dispersed first instar larvae also contribute to lifetime dispersa

14 ds to a dramatic delay in the growth rate of first instar larvae and ultimately death.

15 The adult males and the first instar larvae in the Mengenillidia and Stylopidia

16 The same situation exists in first instar larvae of holometabolous insects, in which

17 ion and the viviparous female to release the first instar larvae when the next generation of the host

18 Maternal HP1 is still detectable in first instar larvae, but disappears by third instar, sug

19 fragmentation, and lethality at the stage of first instar larvae.

20 hibited liquid clearance from the trachea of first instar larvae.

21 hal phenotype and die as stage 17 embryos or first instar larvae.

22 y required for embryos to hatch into feeding first instar larvae.

23 s, but they die within 2 days of hatching as first instar larvae.

24 embryos from null mothers fail to hatch into first instar larvae.

25 imilarly we are able to rescue the embryonic/first instar larval lethality of an alphaPS1 null mutati

26 now identified Notch expressing HSCs in the first instar larval lymph gland.

27 ion is required in mesoderm beginning at the first instar larval stage of development.

28 We show that mge mutations lead to first-instar larval lethality and that Mge protein is si

29 null PSI mutation is recessive lethal at the first-instar larval stage, and lethality is fully rescue

30 First-instar mantispids use two strategies to locate spi

31 ehavior, by recording from photoreceptors of first instar nymphs and adult animals using the patch-cl

32 Molting after the normal first instar period was restored to various degrees by f

33 omatin localized gene leads to arrest at the first instar stage which can be rescued by feeding the l

34 ing Aedes aegypti, do not develop beyond the first instar when fed a nutritionally complete diet in t

35 proteins are relatively nontoxic to monarch first instars, whereas first instars are sensitive to Cr

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