ライフサイエンスコーパス: first-pass metabolism

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1 r deliver drugs systemically without hepatic first pass metabolism.

2 , by inhibiting intestinal (but not hepatic) first-pass metabolism.

3 f verapamil, a drug that undergoes extensive first-pass metabolism.

4 ufficient alcohol to account for the bulk of first-pass metabolism.

5 sm in keeping with its inhibition of in vivo first-pass metabolism.

6 ministered alcohol is incomplete, indicating first-pass metabolism.

7 ain barrier-permeant, but suffers from rapid first-pass metabolism.

8 ch has the metabolic capacity to account for first-pass metabolism.

9 substrates that undergo extensive intestinal first-pass metabolism.

10 okinetics are remarkable for almost complete first pass metabolism after oral administration, resulti

11 Based on this difference in first pass metabolism, an increase of 2% in bioavailabil

12 and extensive distal binding in all tissues, first-pass metabolism and activation of NNK in the airwa

13 Intranasal administration avoids first-pass metabolism and provides a reproducible, easil

14 There is debate regarding the site of first-pass metabolism and specifically whether the stoma

15 Astemizole undergoes extensive first-pass metabolism, and its dominant metabolite, desm

16 azole, 8.0% of the tacrolimus dose underwent first pass metabolism (E(H)), whereas in the presence of

17 rful new tool for preclinical predictions of first-pass metabolism for new drugs in development.

18 aempferol to cultured hepatocytes, mimicking first pass metabolism, greatly diminished the inhibitory

19 thanol from reaching the liver by activating first-pass metabolism in the stomach.

20 First-pass metabolism is a common cause of incomplete an

21 aining monooxygenase enzymes involved in the first-pass metabolism of drugs and foreign chemicals in

22 The effect of glycine on the first-pass metabolism of ethanol was also examined in vi

23 The first-pass metabolism of foreign compounds in the lung i

24 tients with TIPS, there was a marked loss in first-pass metabolism of midazolam as a result of dimini

25 intestinal P450 expression and capacity for first-pass metabolism of oral drugs.

26 en et al. show in this issue of the JCI that first-pass metabolism of the anticancer agent docetaxel

27 and poor oral bioavailability, due to rapid first-pass metabolism of the phenol moieties.

28 9 was not active orally, likely due to rapid first-pass metabolism of the phenol moiety.

29 y influencing either the absorption phase or first-pass metabolism of TVR.

30 henols undergo extensive modification during first-pass metabolism so that the forms reaching the blo

31 )), whereas in the presence of ketoconazole, first pass metabolism was 6.2% (P=0.01).

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