ライフサイエンスコーパス: flaccid

1 increase of 30% and 40% in mean diameter in flaccid and erectile states, respectively.

2 serotypes A-G) and tetanus neurotoxin elicit flaccid and spastic paralysis, respectively.

3 In 9 days, flaccid areflexic quadriparesis and bulbar palsy develop

4 gnosed based on its clinical manifestations (flaccid blisters and erosions on skin and oral mucosa),

5 sitization of CC smooth muscle maintains the flaccid (contracted) state.

6 We hypothesize that the flaccid cortex of mhcA- cells cannot generate sufficient

7 nificant adhesive interaction to distort the flaccid cortex of mhcA- cells mhcA- cells were excluded

8 Flaccid giant liposomes showed no measurable elasticity.

9 Flaccid hearts (no left ventricular balloon) served as c

10 reement with aspiration measurements made on flaccid human erythrocytes, but the prestressed model al

11 terminal sequences lead to the production of flaccid, infertile eggs with a soluble, rather than inso

12 ne membrane as sV23 protein null mutants lay flaccid, infertile eggs.

13 ant cause of epidemic viral encephalitis and flaccid limb paralysis, yet the mechanism by which it en

14 Neurological deficits included flaccid limb weakness (n=10; asymmetric n=7), bulbar wea

15 Acute flaccid myelitis refers to acute flaccid limb weakness with spinal cord gray matter lesio

16 t toxic substances known to humankind, cause flaccid muscle paralysis by blocking acetylcholine relea

17 are the most potent of all toxins that cause flaccid muscle paralysis leading to death.

18 is characterised by episodes of often severe flaccid muscle paralysis, in which the muscle fibre memb

19 nsmitter acetylcholine, thereby resulting in flaccid muscle paralysis.

20 atients also suffer from hypotonia (weak and flaccid muscles) and balance problems.

21 Enterovirus D68 (EV-D68)-associated acute flaccid myelitis (AFM) is a devastating neurological dis

22 e summer/fall 2014, pediatric cases of acute flaccid myelitis (AFM) occurred in the United States, co

23 One child with acute flaccid myelitis and a sibling with only upper respirato

24 ssociation between enterovirus D68 and acute flaccid myelitis and the contention that acute flaccid m

25 ve for enterovirus D68 with those with acute flaccid myelitis but negative for enterovirus D68 using

26 rovirus D68 and the recent increase in acute flaccid myelitis cases in the United States.

27 o temporally and geographically linked acute flaccid myelitis clusters at the height of the 2014 outb

28 ebrospinal fluid from 14 patients with acute flaccid myelitis did not reveal evidence of an alternati

29 between enterovirus D68 infection and acute flaccid myelitis during the 2014 enterovirus D68 respira

30 erovirus D68 sequences associated with acute flaccid myelitis grouped into a clade B1 strain that eme

31 accid myelitis and the contention that acute flaccid myelitis is a rare yet severe clinical manifesta

32 and from 12 (48%) of 25 patients with acute flaccid myelitis overall.

33 Acute flaccid myelitis refers to acute flaccid limb weakness w

34 Patients with acute flaccid myelitis who presented to two hospitals in Color

35 We compared patients with acute flaccid myelitis who were positive for enterovirus D68 w

36 re respiratory illness in children and acute flaccid myelitis, raising concerns about its potential i

37 48 patients were included: 25 with acute flaccid myelitis, two with enterovirus-associated enceph

38 in a polio-like neurological disorder, acute flaccid myelitis.

39 reported sporadically in patients with acute flaccid myelitis.

40 a spectrum of motility, from paralysis with flaccid or twitching flagella as other spoke mutants to

41 h a higher rate of reporting non-polio acute flaccid paralysis (AFP) (OR = 1.13, 95% CI 1.02-1.26 for

42 t noted an abnormal number of cases of acute flaccid paralysis (AFP) among adults, which were later c

43 In 1986, surveillance of acute flaccid paralysis (AFP) cases among children <15 years o

44 Polio cases were defined as acute flaccid paralysis (AFP) cases reported from 7 October to

45 Surveillance for acute flaccid paralysis (AFP) is a fundamental cornerstone of

46 n <36 mo old identified with non-polio acute flaccid paralysis (AFP) reported through polio surveilla

47 We used acute flaccid paralysis (AFP) surveillance data from Nigeria c

48 ES complements clinical acute flaccid paralysis (AFP) surveillance for possible polio

49 Gaps in acute flaccid paralysis (AFP) surveillance have been consisten

50 io enteroviruses (NPEVs) isolated from acute flaccid paralysis (AFP) surveillance in Shandong Provinc

51 tionnaires collected information about acute flaccid paralysis (AFP) surveillance resources, training

52 VAPP cases were identified using LAC's acute flaccid paralysis (AFP) surveillance system.

53 kistan during July 2009 to support the acute flaccid paralysis (AFP) surveillance system.

54 cle reviews the epidemiology of polio, acute flaccid paralysis (AFP) surveillance, and the implementa

55 ect process indicators associated with acute flaccid paralysis (AFP) surveillance, routine immunizati

56 lan as an activity that can complement acute flaccid paralysis (AFP) surveillance.

57 A case of acute flaccid paralysis (AFP) was defined as a child <15 years

58 averaged only 57% and surveillance for acute flaccid paralysis (AFP) was suboptimal (AFP rate<1 per 1

59 1 isolates obtained from patients with acute flaccid paralysis (AFP) were compared by nucleotide sequ

60 Twenty-one cases of acute flaccid paralysis (AFP) were reported on the island of H

61 A case was defined as any case of acute flaccid paralysis (AFP) with virological confirmation of

62 ani and Afghani children with nonpolio acute flaccid paralysis (AFP).

63 35 South Asian children with nonpolio acute flaccid paralysis (AFP).

64 of South Asian children with nonpolio acute flaccid paralysis (AFP).

65 ted HFMD (n = 47), meningitis (n = 8), acute flaccid paralysis (n = 1), encephalitis (n = 21), and en

66 the release of neurotransmitters that cause flaccid paralysis and are considered potential bioweapon

67 d to assess a cluster of children with acute flaccid paralysis and cranial nerve dysfunction geograph

68 ally and temporally defined cluster of acute flaccid paralysis and cranial nerve dysfunction in child

69 ne depolarization, action potential failure, flaccid paralysis and cytopathology that are characteris

70 eins required for neurotransmission, causing flaccid paralysis and death by asphyxiation.

71 caviruses in human diseases, including acute flaccid paralysis and diarrhea, will require further epi

72 er investigators that highlighted persistent flaccid paralysis and electrophysiological evidence of a

73 self-limiting acute febrile illness to acute flaccid paralysis and lethal encephalitis.

74 of a surveillance system for cases of acute flaccid paralysis and poliovirus infection.

75 Using acute flaccid paralysis and routine, monthly countrywide envir

76 al features of the increasing cases of acute flaccid paralysis associated with anterior myelitis note

77 e of human motor neurons are responsible for flaccid paralysis associated with botulism.

78 children aged 0-14 years with onset of acute flaccid paralysis between Jan 1, 2001, and Dec 31, 2011.

79 ) is a highly potent neurotoxin that elicits flaccid paralysis by enzymatic cleavage of the exocytic

80 oteases that cleave SNARE proteins to elicit flaccid paralysis by inhibiting neurotransmitter-carryin

81 oteases that cleave SNARE proteins to elicit flaccid paralysis by inhibiting the fusion of neurotrans

82 oteases that cleave SNARE proteins to elicit flaccid paralysis by inhibiting the fusion of neurotrans

83 An analysis was conducted of 10,486 acute flaccid paralysis cases diagnosed as Guillain-Barre synd

84 ean numbers of infected and uninfected acute flaccid paralysis cases investigated in a season are der

85 like poliovirus isolates from Nigerian acute flaccid paralysis cases obtained from routine surveillan

86 lic Health received several reports of acute flaccid paralysis cases of unknown etiology.

87 nergic nerve terminals, causing a descending flaccid paralysis characteristic of the disease botulism

88 tates between 2009 and 2014, using the acute flaccid paralysis database at the World Health Organizat

89 rable outcomes, although patients with acute flaccid paralysis did not recover limb strength.

90 as compared with that of children with acute flaccid paralysis due to other causes to estimate the cl

91 West Nile encephalitis; a poliomyelitis-like flaccid paralysis due to West Nile virus was recognised,

92 23 August 1991, a 2-year-old boy with acute flaccid paralysis due to wild poliovirus was detected in

93 meeting diagnostic criteria for other acute flaccid paralysis etiologies were excluded.

94 Since then, >9000 cases of acute flaccid paralysis have been reported and thoroughly inve

95 documented in 27 percent of the patients and flaccid paralysis in 10 percent; in all of the latter, n

96 in 20 (35%), encephalitis in 6 (11%), acute flaccid paralysis in 4 (7%), and autonomic dysregulation

97 cribed as a bilateral, symmetric, descending flaccid paralysis in an afebrile and alert patient witho

98 ism manifests with cranial nerve palsies and flaccid paralysis in children and adults.

99 dium botulinum (Clb), the causative agent of flaccid paralysis in humans that can be fatal in 5 to 10

100 emics of meningitis, encephalitis, and acute flaccid paralysis in humans.

101 wild poliovirus and cases of non-polio acute flaccid paralysis in India.

102 MP2 and VAMP4 in cultured neurons and causes flaccid paralysis in mice.

103 here has been limited surveillance for acute flaccid paralysis in North America since the regional er

104 Case series of acute flaccid paralysis in patients with radiological or neuro

105 erosurvey was conducted among cases of acute flaccid paralysis in the 25 high-polio-incidence distric

106 us system, is the most common cause of acute flaccid paralysis in the post-polio era.

107 d in 12 of 192 patients with non-polio acute flaccid paralysis in Tunisia and Nigeria and 0 of 96 hea

108 ingitis, and concomitant muscle weakness and flaccid paralysis may provide a clinical clue to the pre

109 pisodes), oculogyric crisis (four episodes), flaccid paralysis of all extremities (four episodes), tr

110 m is characterized by symmetric, descending, flaccid paralysis of motor and autonomic nerves, usually

111 lysis (hypoKPP) is characterized by episodic flaccid paralysis of muscle and acute hypokalemia during

112 sure for botulism, a fatal illness caused by flaccid paralysis of muscles due to botulinum neurotoxin

113 BoNT/A application causes long-lasting flaccid paralysis of muscles through inhibiting the rele

114 Clusters of acute flaccid paralysis or cranial nerve dysfunction in childr

115 Patients initially seen with acute flaccid paralysis or pulmonary edema had significantly g

116 Surveillance for cases of acute flaccid paralysis provides a means for detecting circula

117 dose of OPV among 47 574 children with acute flaccid paralysis reported during 2005-2009.

118 s of stool samples taken from cases of acute flaccid paralysis revealed the presence of mixtures of r

119 posed national surveillance system for acute flaccid paralysis should capture at a minimum the 796 GB

120 , the importance of maintaining strong acute flaccid paralysis surveillance even in adults, and the n

121 We conducted a retrospective review of acute flaccid paralysis surveillance in the security-compromis

122 lected from children identified by the acute flaccid paralysis surveillance program in India during 2

123 from among cases reported through the acute flaccid paralysis surveillance system between November 2

124 r understanding the sensitivity of the acute flaccid paralysis surveillance system is presented by fi

125 Health Organization requested that the acute flaccid paralysis surveillance system of Latin American

126 The acute flaccid paralysis surveillance system represents a usefu

127 A robust acute flaccid paralysis surveillance system, including a multi

128 supplementary immunization activities, acute flaccid paralysis surveillance, and routine immunization

129 ages of human resources and strengthen acute flaccid paralysis surveillance.

130 f field teams, (3) field work, and (4) acute flaccid paralysis surveillance.

131 cted in Bangladesh in 2007 to 2008 for acute flaccid paralysis surveillance.

132 Botulinum neurotoxins (BoNTs) elicit flaccid paralysis through cleavage of SNARE proteins wit

133 e zinc proteases (serotypes A-G) which cause flaccid paralysis through the cleavage of SNARE proteins

134 Botulinum neurotoxin causes rapid flaccid paralysis through the inhibition of acetylcholin

135 nce database, in which 27,379 cases of acute flaccid paralysis were recorded between 2001 and 2007.

136 icated a rare but distinct syndrome of acute flaccid paralysis with evidence of spinal motor neuron i

137 AE in rodents typically results in ascending flaccid paralysis with inflammation primarily targeting

138 rodents is manifested typically as ascending flaccid paralysis with inflammation targeting the spinal

139 ibe a group of patients with acute segmental flaccid paralysis with minimal or no encephalitic or sen

140 apon would cause acute symmetric, descending flaccid paralysis with prominent bulbar palsies such as

141 spital Colorado (Aurora, CO, USA) with acute flaccid paralysis with spinal-cord lesions involving mai

142 rome (GBS) is the most common cause of acute flaccid paralysis worldwide, and is thought to be immune

143 ive disease (meningitis, encephalitis, acute flaccid paralysis) develops in less than 1% but carries

144 ncluding neonatal sepsis-like disease, acute flaccid paralysis, and acute hemorrhagic conjunctivitis.

145 hagic disease, encephalitis, biphasic fever, flaccid paralysis, and jaundice are typical manifestatio

146 polio vaccine (OPV), surveillance for acute flaccid paralysis, and OPV "mop-up" campaigns.

147 aseptic meningitis, poliomyelitis-like acute flaccid paralysis, brainstem encephalitis, and other sev

148 d population groups, and surveillance (acute flaccid paralysis, enterovirus, and environmental) needs

149 After an initial contralateral flaccid paralysis, motor function rapidly recovered, aft

150 e release at neuromuscular junctions causing flaccid paralysis, specifically synaptosomal-associated

151 While BoNTs cause flaccid paralysis, TeNT causes spastic paralysis.

152 Botulism is characterized by flaccid paralysis, which can be caused by intoxication w

153 s act locally within motor neurons to elicit flaccid paralysis, while retrograde TeNT traffics to inh

154 Timely investigation of children with acute flaccid paralysis, with collection of stool specimens fo

155 utants move poorly, exhibiting an incomplete flaccid paralysis, yet have normal muscle ultrastructure

156 ation coverage from data for non-polio acute flaccid paralysis.

157 phalitis, and 3 had poliomyelitis-like acute flaccid paralysis.

158 s conducting surveillance for cases of acute flaccid paralysis.

159 s investigated during surveillance for acute flaccid paralysis.

160 3 and early 2014 in the absence of any acute flaccid paralysis.

161 re neurological complications, such as acute flaccid paralysis.

162 sting stool samples from patients with acute flaccid paralysis.

163 naptic vesicle (SV) exocytosis, resulting in flaccid paralysis.

164 romes of meningitis, encephalitis, and acute flaccid paralysis/poliomyelitis.

165 sm (WB) is a potentially lethal, descending, flaccid, paralysis that results when spores of Clostridi

166 ute encephalomyeloradiculitis had a moderate flaccid paraparesis, and the patient with subacute menin

167 and, after 6 months, culminated in areflexic flaccid quadriplegia.

168 Sharper lateral inflection in the flaccid rather than the tense LR seems inconsistent with

169 For a flaccid red cell, we show that the total adhesion energy

170 h RhoA-mediated Ca(2+) sensitization, to the flaccid state of CC that can be reversed by a water-solu

171 inct signs of CNS disease that ranged from a flaccid tail to complete paralysis of hind limbs.

172 yed, the clinical signs will ascend, causing flaccid tetraparesis and other lower motor neuron signs.

173 ation of the latter drugs, most had diffuse, flaccid weakness with failure to wean from mechanical ve