ライフサイエンスコーパス: flaccid paralysis

1 phalitis, and 3 had poliomyelitis-like acute flaccid paralysis.

2 s conducting surveillance for cases of acute flaccid paralysis.

3 s investigated during surveillance for acute flaccid paralysis.

4 3 and early 2014 in the absence of any acute flaccid paralysis.

5 re neurological complications, such as acute flaccid paralysis.

6 sting stool samples from patients with acute flaccid paralysis.

7 naptic vesicle (SV) exocytosis, resulting in flaccid paralysis.

8 ation coverage from data for non-polio acute flaccid paralysis.

9 h a higher rate of reporting non-polio acute flaccid paralysis (AFP) (OR = 1.13, 95% CI 1.02-1.26 for

10 t noted an abnormal number of cases of acute flaccid paralysis (AFP) among adults, which were later c

11 In 1986, surveillance of acute flaccid paralysis (AFP) cases among children <15 years o

12 Polio cases were defined as acute flaccid paralysis (AFP) cases reported from 7 October to

13 Surveillance for acute flaccid paralysis (AFP) is a fundamental cornerstone of

14 n <36 mo old identified with non-polio acute flaccid paralysis (AFP) reported through polio surveilla

15 We used acute flaccid paralysis (AFP) surveillance data from Nigeria c

16 ES complements clinical acute flaccid paralysis (AFP) surveillance for possible polio

17 Gaps in acute flaccid paralysis (AFP) surveillance have been consisten

18 io enteroviruses (NPEVs) isolated from acute flaccid paralysis (AFP) surveillance in Shandong Provinc

19 tionnaires collected information about acute flaccid paralysis (AFP) surveillance resources, training

20 VAPP cases were identified using LAC's acute flaccid paralysis (AFP) surveillance system.

21 kistan during July 2009 to support the acute flaccid paralysis (AFP) surveillance system.

22 cle reviews the epidemiology of polio, acute flaccid paralysis (AFP) surveillance, and the implementa

23 ect process indicators associated with acute flaccid paralysis (AFP) surveillance, routine immunizati

24 lan as an activity that can complement acute flaccid paralysis (AFP) surveillance.

25 A case of acute flaccid paralysis (AFP) was defined as a child <15 years

26 averaged only 57% and surveillance for acute flaccid paralysis (AFP) was suboptimal (AFP rate<1 per 1

27 1 isolates obtained from patients with acute flaccid paralysis (AFP) were compared by nucleotide sequ

28 Twenty-one cases of acute flaccid paralysis (AFP) were reported on the island of H

29 A case was defined as any case of acute flaccid paralysis (AFP) with virological confirmation of

30 ani and Afghani children with nonpolio acute flaccid paralysis (AFP).

31 35 South Asian children with nonpolio acute flaccid paralysis (AFP).

32 of South Asian children with nonpolio acute flaccid paralysis (AFP).

33 the release of neurotransmitters that cause flaccid paralysis and are considered potential bioweapon

34 d to assess a cluster of children with acute flaccid paralysis and cranial nerve dysfunction geograph

35 ally and temporally defined cluster of acute flaccid paralysis and cranial nerve dysfunction in child

36 ne depolarization, action potential failure, flaccid paralysis and cytopathology that are characteris

37 eins required for neurotransmission, causing flaccid paralysis and death by asphyxiation.

38 caviruses in human diseases, including acute flaccid paralysis and diarrhea, will require further epi

39 er investigators that highlighted persistent flaccid paralysis and electrophysiological evidence of a

40 self-limiting acute febrile illness to acute flaccid paralysis and lethal encephalitis.

41 of a surveillance system for cases of acute flaccid paralysis and poliovirus infection.

42 Using acute flaccid paralysis and routine, monthly countrywide envir

43 ncluding neonatal sepsis-like disease, acute flaccid paralysis, and acute hemorrhagic conjunctivitis.

44 hagic disease, encephalitis, biphasic fever, flaccid paralysis, and jaundice are typical manifestatio

45 polio vaccine (OPV), surveillance for acute flaccid paralysis, and OPV "mop-up" campaigns.

46 al features of the increasing cases of acute flaccid paralysis associated with anterior myelitis note

47 e of human motor neurons are responsible for flaccid paralysis associated with botulism.

48 children aged 0-14 years with onset of acute flaccid paralysis between Jan 1, 2001, and Dec 31, 2011.

49 aseptic meningitis, poliomyelitis-like acute flaccid paralysis, brainstem encephalitis, and other sev

50 ) is a highly potent neurotoxin that elicits flaccid paralysis by enzymatic cleavage of the exocytic

51 oteases that cleave SNARE proteins to elicit flaccid paralysis by inhibiting neurotransmitter-carryin

52 oteases that cleave SNARE proteins to elicit flaccid paralysis by inhibiting the fusion of neurotrans

53 oteases that cleave SNARE proteins to elicit flaccid paralysis by inhibiting the fusion of neurotrans

54 An analysis was conducted of 10,486 acute flaccid paralysis cases diagnosed as Guillain-Barre synd

55 ean numbers of infected and uninfected acute flaccid paralysis cases investigated in a season are der

56 like poliovirus isolates from Nigerian acute flaccid paralysis cases obtained from routine surveillan

57 lic Health received several reports of acute flaccid paralysis cases of unknown etiology.

58 nergic nerve terminals, causing a descending flaccid paralysis characteristic of the disease botulism

59 tates between 2009 and 2014, using the acute flaccid paralysis database at the World Health Organizat

60 ive disease (meningitis, encephalitis, acute flaccid paralysis) develops in less than 1% but carries

61 rable outcomes, although patients with acute flaccid paralysis did not recover limb strength.

62 as compared with that of children with acute flaccid paralysis due to other causes to estimate the cl

63 West Nile encephalitis; a poliomyelitis-like flaccid paralysis due to West Nile virus was recognised,

64 23 August 1991, a 2-year-old boy with acute flaccid paralysis due to wild poliovirus was detected in

65 d population groups, and surveillance (acute flaccid paralysis, enterovirus, and environmental) needs

66 meeting diagnostic criteria for other acute flaccid paralysis etiologies were excluded.

67 Since then, >9000 cases of acute flaccid paralysis have been reported and thoroughly inve

68 documented in 27 percent of the patients and flaccid paralysis in 10 percent; in all of the latter, n

69 in 20 (35%), encephalitis in 6 (11%), acute flaccid paralysis in 4 (7%), and autonomic dysregulation

70 cribed as a bilateral, symmetric, descending flaccid paralysis in an afebrile and alert patient witho

71 ism manifests with cranial nerve palsies and flaccid paralysis in children and adults.

72 dium botulinum (Clb), the causative agent of flaccid paralysis in humans that can be fatal in 5 to 10

73 emics of meningitis, encephalitis, and acute flaccid paralysis in humans.

74 wild poliovirus and cases of non-polio acute flaccid paralysis in India.

75 MP2 and VAMP4 in cultured neurons and causes flaccid paralysis in mice.

76 here has been limited surveillance for acute flaccid paralysis in North America since the regional er

77 Case series of acute flaccid paralysis in patients with radiological or neuro

78 erosurvey was conducted among cases of acute flaccid paralysis in the 25 high-polio-incidence distric

79 us system, is the most common cause of acute flaccid paralysis in the post-polio era.

80 d in 12 of 192 patients with non-polio acute flaccid paralysis in Tunisia and Nigeria and 0 of 96 hea

81 ingitis, and concomitant muscle weakness and flaccid paralysis may provide a clinical clue to the pre

82 After an initial contralateral flaccid paralysis, motor function rapidly recovered, aft

83 ted HFMD (n = 47), meningitis (n = 8), acute flaccid paralysis (n = 1), encephalitis (n = 21), and en

84 pisodes), oculogyric crisis (four episodes), flaccid paralysis of all extremities (four episodes), tr

85 m is characterized by symmetric, descending, flaccid paralysis of motor and autonomic nerves, usually

86 lysis (hypoKPP) is characterized by episodic flaccid paralysis of muscle and acute hypokalemia during

87 sure for botulism, a fatal illness caused by flaccid paralysis of muscles due to botulinum neurotoxin

88 BoNT/A application causes long-lasting flaccid paralysis of muscles through inhibiting the rele

89 Clusters of acute flaccid paralysis or cranial nerve dysfunction in childr

90 Patients initially seen with acute flaccid paralysis or pulmonary edema had significantly g

91 romes of meningitis, encephalitis, and acute flaccid paralysis/poliomyelitis.

92 Surveillance for cases of acute flaccid paralysis provides a means for detecting circula

93 dose of OPV among 47 574 children with acute flaccid paralysis reported during 2005-2009.

94 s of stool samples taken from cases of acute flaccid paralysis revealed the presence of mixtures of r

95 posed national surveillance system for acute flaccid paralysis should capture at a minimum the 796 GB

96 e release at neuromuscular junctions causing flaccid paralysis, specifically synaptosomal-associated

97 , the importance of maintaining strong acute flaccid paralysis surveillance even in adults, and the n

98 We conducted a retrospective review of acute flaccid paralysis surveillance in the security-compromis

99 lected from children identified by the acute flaccid paralysis surveillance program in India during 2

100 from among cases reported through the acute flaccid paralysis surveillance system between November 2

101 r understanding the sensitivity of the acute flaccid paralysis surveillance system is presented by fi

102 Health Organization requested that the acute flaccid paralysis surveillance system of Latin American

103 The acute flaccid paralysis surveillance system represents a usefu

104 A robust acute flaccid paralysis surveillance system, including a multi

105 supplementary immunization activities, acute flaccid paralysis surveillance, and routine immunization

106 ages of human resources and strengthen acute flaccid paralysis surveillance.

107 f field teams, (3) field work, and (4) acute flaccid paralysis surveillance.

108 cted in Bangladesh in 2007 to 2008 for acute flaccid paralysis surveillance.

109 While BoNTs cause flaccid paralysis, TeNT causes spastic paralysis.

110 sm (WB) is a potentially lethal, descending, flaccid, paralysis that results when spores of Clostridi

111 Botulinum neurotoxins (BoNTs) elicit flaccid paralysis through cleavage of SNARE proteins wit

112 e zinc proteases (serotypes A-G) which cause flaccid paralysis through the cleavage of SNARE proteins

113 Botulinum neurotoxin causes rapid flaccid paralysis through the inhibition of acetylcholin

114 nce database, in which 27,379 cases of acute flaccid paralysis were recorded between 2001 and 2007.

115 Botulism is characterized by flaccid paralysis, which can be caused by intoxication w

116 s act locally within motor neurons to elicit flaccid paralysis, while retrograde TeNT traffics to inh

117 icated a rare but distinct syndrome of acute flaccid paralysis with evidence of spinal motor neuron i

118 AE in rodents typically results in ascending flaccid paralysis with inflammation primarily targeting

119 rodents is manifested typically as ascending flaccid paralysis with inflammation targeting the spinal

120 ibe a group of patients with acute segmental flaccid paralysis with minimal or no encephalitic or sen

121 apon would cause acute symmetric, descending flaccid paralysis with prominent bulbar palsies such as

122 spital Colorado (Aurora, CO, USA) with acute flaccid paralysis with spinal-cord lesions involving mai

123 Timely investigation of children with acute flaccid paralysis, with collection of stool specimens fo

124 rome (GBS) is the most common cause of acute flaccid paralysis worldwide, and is thought to be immune

125 utants move poorly, exhibiting an incomplete flaccid paralysis, yet have normal muscle ultrastructure