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1 to pressure elevation (e.g. effusions, joint flexion).
2 ws slightly flexed (20 degrees-30 degrees of flexion).
3 ed in the posterior direction (ankle plantar flexion).
4 arger when the ipsilateral hip was moving in flexion.
5 eral leg was moving either into extension or flexion.
6 ectromyography (EMG) before and during ankle flexion.
7 ring hip extension and reinforced during hip flexion.
8 oronal plane with the elbow in 20 degrees of flexion.
9 stsynaptic neuron firing and a contralateral flexion.
10 les for generating uropod flaring and telson flexion.
11 complete axial rotation and reduced cervical flexion.
12  stretch injury from cervical hyperextension/flexion.
13 aracterised by abnormal thoracolumbar spinal flexion.
14  was successful with all methods with forced flexion.
15 nd studied at flexion, extension, and forced flexion.
16 on was substantially larger than that during flexion.
17 xing the knee and ankle joints at 90 degrees flexion.
18  was significantly greater in extension than flexion.
19 on and was mildest when it was around 20 deg flexion.
20 ts and, to a lesser extent, extension versus flexion.
21 e activated during neck rotation and lateral flexion.
22  PADs evoked by both sources were maximal in flexion.
23 ation and may not be associated with lateral flexion.
24 isting of repeated dorsal-ventral whole-body flexions.
25 onsisting of alternating left and right body flexions.
26 .93 degrees (95% CI 0.19-1.66 degrees ); for flexion, 1.11 degrees (95% CI 0.38-1.85 degrees ).
27                                     Upon hip flexion, 23 CIA and 116 EIA stenoses showed kinking (mea
28  subjects (age 25-45 years) performed finger flexion (7 % maximal voluntary contraction at 0.67 Hz) u
29 ocomotion [5], the nerve chord for abdominal flexion [9], antennal muscles [2, 10], and the flight mu
30 3.2 degrees [13.6 degrees ], P =.03; passive flexion: 90.5 degrees [6.8 degrees ] vs 81.8 degrees [13
31 pearance as either taut or lax in extension, flexion, abduction, adduction, and internal and external
32 hograde posture and (2) lateral side-to-side flexion about the C6-T3 axis while in a pronograde postu
33  infants toward airway collapse include neck flexion, airway secretions, gastroesophageal reflux, and
34  and extension, and concentric ankle plantar flexion and dorsiflexion, and 3) body mass index and a k
35 The mean maximal range of motion of shoulder flexion and elbow extension increased significantly.
36 and elbow flexion) and 'reach out' (shoulder flexion and elbow extension).
37 vements were a sagittal 'reach up' (shoulder flexion and elbow flexion) and 'reach out' (shoulder fle
38                            In contrast, head flexion and extension exerted nonspecific bilateral effe
39 ol subjects' was over 40% higher during both flexion and extension movements when compared with the p
40 iceps head over the medial epicondyle during flexion and extension movements.
41 ssing neurons in legs were stretched by both flexion and extension of corresponding joints.
42 ension, and in four patients during repeated flexion and extension of the wrist.
43 ension of the wrist and during repeated slow flexion and extension of this joint.
44 hase-related amplitudes, which resembled the flexion and extension sequence of the fingers.
45                                      Truncal flexion and extension strength, hand grip strength, leg
46 ed tone of the limbs at rest and with active flexion and extension were observed in the survivors of
47  running, loss of righting reflex, and tonic flexion and extension, and are followed by a postictal p
48 th measures of concentric and eccentric knee flexion and extension, and concentric ankle plantar flex
49 and visible adjustment of the knee with each flexion and extension.
50 d from the normal phasing pattern underlying flexion and extension.
51 ne of the two dominant phases of locomotion: flexion and extension.
52  of phase locking at the transitions between flexion and extension.
53 ions commenced from a position of full elbow flexion and full wrist extension.
54   The composite is sensitive to pressure and flexion and recovers its mechanical and electrical prope
55 ite resistance varies inversely with applied flexion and tactile forces.
56 rooming of the ear, hindleg tibial extension/flexion and tibial extensor/flexor muscle bursts can occ
57 rgery resulted in a decrease in both truncal flexion and truncal extension.
58                 Indeed both voluntary finger flexion and voluntary leg extension produced significant
59 ffect was strongest during voluntary plantar flexion and weaker during dorsiflexion or at rest.
60 iew radiographs of the knee in 30 degrees of flexion and with weight bearing were obtained at baselin
61 ittal 'reach up' (shoulder flexion and elbow flexion) and 'reach out' (shoulder flexion and elbow ext
62 sed the initial phase of the movement (wrist flexion) and assisted the reverse phase, so that recruit
63 s of paw placement, weight-bearing, and limb flexion, and (3) the lowest level of body weight support
64 eptive acuity was assessed in varus, valgus, flexion, and extension using threshold to detection of p
65  power and isometric knee extension, plantar flexion, and hand grip strength measures.
66 orientation behavior either before or during flexion, and only larvae that were within a given maximu
67 wild running, loss of righting reflex, tonic flexion, and tonic extension in response to high-intensi
68 ror when attempting to reproduce a test knee flexion angle (a measure of joint position sense).
69 r valgus load while maintaining a fixed knee flexion angle and thigh and ankle immobilization.
70 , 40, 60, 80, and 100%), during which lumbar flexion angle and trunk moment were recorded.
71            There were significant effects of flexion angle on initial moment, moment drop, changes in
72 ed neutral zone increased exponentially with flexion angle.
73  reproduce the in vivo knee position at each flexion angle.
74 ing BO bisect offset measurements at various flexion angles with 4D four-dimensional CT.
75 ion exposures and the influence of different flexion angles.
76 lunge motion from 0 degrees to 90 degrees of flexion as images were recorded with a dual fluoroscopic
77 ed position, exhibiting progressively longer flexions as a function of training (Experiment 1).
78 rs were much stiffer for stretching than for flexion, as expected from their diameter and length.
79 s, ten healthy men performed isometric elbow flexion at 20% to 70% of their maximal force.
80 ignificant only for abduction at 1 month and flexion at 3 months.
81 ealthy subjects sustained an isometric elbow flexion at 30% maximal level until exhaustion while thei
82    As the K/L score was increasing, the knee flexion at the heel strike and 50% of the stance phase i
83  the TF joint and weight-bearing, 30 degrees flexion, axial views of the PF joint.
84 or tasks, contributing to an upper extremity flexion bias following stroke.
85          Sixteen subjects performed 36 elbow flexions ("biceps curls") at one of two submaximal workl
86 ere measured with the elbow at 30 degrees of flexion, both at rest and with valgus stress.
87 interneurons mediates flexion reflex and the flexion components of locomotion and scratching.
88 se fasciitis, with severe loss of motion and flexion contractures in multiple joints.
89 isorder of the palmar fascia, which leads to flexion contractures of the digits, and is associated wi
90 hrodesis is generally recommended for severe flexion contractures of the interphalangeal joints, othe
91 neous nodular tumors, gingival fibromatosis, flexion contractures of the joints, and an accumulation
92  skin thickness score (MRSS), and knee joint flexion contractures were measured with a goniometer.
93  Over 50% of patients with skin involvement, flexion contractures, or oral manifestations achieved co
94  progressive fibrosis of the palm leading to flexion deformities of the digits that impair hand funct
95  descending stairs, palpable effusion, fixed-flexion deformity, restricted-flexion range of motion, a
96 on in the limbs to stand still, but how much flexion depends directly on where its CM is assumed to l
97 sonance scanner using a custom-built plantar flexion device.
98 en the wrist was spontaneously moving in the flexion direction, extension direction or at random.
99 tingent shock) do not exhibit an increase in flexion duration and fail to learn when tested with cont
100 s extended exhibit a progressive increase in flexion duration that minimizes net shock exposure, a si
101 ed (contingent shock) exhibit an increase in flexion duration, a simple form of instrumental learning
102 f leg position do not exhibit an increase in flexion duration.
103  set of spinal cord neurons that produce hip flexion during flexion reflex, locomotion, and scratchin
104 exions (LR) and rhythmic dorsal-ventral body flexions (DV).
105  locking at the transition from extension to flexion (E to F) is stronger than at the transition from
106 rog (0.33-0.58)], as well as tonic and ankle flexion-evoked EMG activity.
107        Comparison between knee extension and flexion exercise at the same RPE with and without PTV fo
108                         An energetic plantar flexion exercise fatigability test and magnetic resonanc
109           Symptomatic fatigue during plantar flexion exercise occurs at a common energetic limit in a
110 matched control (CON) subjects after plantar flexion exercise that lowered muscle glycogen to approxi
111 eatine recovery kinetics following a plantar flexion exercise using an efficient sampling scheme with
112 lower operating pressure (P < 0.05) and knee flexion exercise with PTV (increased CC activation) rese
113 cts also performed static knee extension and flexion exercise without PTV at a force development that
114 rmed duplicate MR experiments during plantar flexion exercise, three weeks apart.
115 and WBRT groups performed knee extension and flexion exercises, and the WBRT group also performed che
116 elaxation of the lumbar spine in response to flexion exposures and the influence of different flexion
117 s identified in the 16 patients who obtained flexion extension radiographs at 6 months.
118   Pulley lesions were created and studied at flexion, extension, and forced flexion.
119 activation in the motor cortex during simple flexion-extension finger movements.
120  getting each subject to perform an isolated flexion-extension movement at each of the shoulder, elbo
121                          The metronome-timed flexion-extension movement speed varied 36-fold from "sl
122 inger and both one-finger passive and active flexion-extension movement tasks for the three groups.
123 atients were scanned while performing a hand flexion-extension movement twice before and twice after
124 attached semi-isolated preparations, passive flexion-extension movements applied to a single hindlimb
125 st, slow or moderately paced voluntary wrist flexion-extension movements dramatically increase the ha
126 onkeys performed visually cued, individuated flexion-extension movements of the fingers and wrist.
127 lthy subjects performed a series of imagined flexion-extension movements of the fingers.
128 of six healthy subjects while they performed flexion-extension movements of the right index finger (f
129 contributes to their differential actions on flexion-extension movements.
130        Two animals performed an index finger flexion-extension task to track a target presented on a
131 xion phase of contralateral rhythmical wrist flexion-extension.
132 g (fMRI) while tracking complex targets with flexion/extension at right finger, elbow and ankle separ
133 ted a virtual table through continuous wrist flexion/extension movements with the goal to position a
134 uring 10, 30, and 70% of maximal right wrist flexion force.
135 erosseous during the generation of fingertip flexion forces.
136  Patients with PD and Pisa syndrome (lateral flexion &gt;10 degrees in the standing position) were exami
137 ccuracy of reproduction of the angle of knee flexion had modest effects on the trajectory of pain and
138 d other discrete motor responses (e.g., limb flexion, head turn, etc.) learned with an aversive uncon
139 ensor tone, increased resistance to hindlimb flexion, hypermetria during positive support responses,
140        Quadriceps strength deficits and knee flexion impairments persisted.
141 ion and at 30 degrees and 60 degrees of knee flexion in six cadaveric knees.
142                             It required some flexion in the limbs to stand still, but how much flexio
143 echanics between 0 degrees and 60 degrees of flexion in the medial compartment of the knee.
144 rmation from 0 degrees to 30 degrees of knee flexion in the presence of ACL deficiency.
145 tolith organs and involved both rotation and flexion in the spine.
146 low-intensity (0.5-2.0 kg), rhythmic plantar flexion in the supine posture.
147 e stance phase increased while the peak knee flexion in the swing phase decreased.
148                22 subjects performed plantar flexions in a 7T MR-scanner, leading to PCr changes rang
149                                              Flexion-induced changes in viscous properties and neutra
150  Further, the DeltaMAP during 0.5 kg plantar flexion inversely correlated with the ankle-brachial ind
151 sual; obligatory external rotation of hip in flexion is classic.
152 ammable stimulator, and the resultant finger flexion joint angles were recorded using a motion captur
153  goniometer, 2) a posteroanterior (PA) fixed-flexion knee radiograph (anatomic(PA) axis), and 3) an a
154 with knee OA underwent nonfluoroscopic fixed-flexion knee radiography.
155 valent of which are rhythmic left-right body flexions (LR) and rhythmic dorsal-ventral body flexions
156  with the placebo group at discharge (active flexion: mean [SD], 84.2 degrees [11.1 degrees ] vs 73.2
157 lain knee radiography-ie, the degree of knee flexion, misalignment of the x-ray beam, magnification o
158 aretic subjects tended to produce concurrent flexion motions of shoulder and elbow joints when attemp
159 nked online neural decoding of extension and flexion motor states with stimulation protocols promotin
160 ments associated with rapid, voluntary elbow flexion movements (focal movements) originate as a selec
161  an ataxic gait characterized by exaggerated flexion movements and marked alterations to the step cyc
162 nce of unconstrained syncopated index finger flexion movements in patients with PD, older adult subje
163     The subjects executed single rapid wrist flexion movements in response to a flash of light.
164  by an exercise protocol (repetitive plantar-flexion movements in supine position; n=28).
165 onkey was trained to perform wrist extension/flexion movements in the horizontal plane to align a poi
166 ral nervous system control for extension and flexion movements is unknown.
167 d on both sides by impaired fingers, and the flexion movements of a given finger could be unaffected
168 rtical activation during thumb extension and flexion movements of eight human volunteers was measured
169 rget on a computer screen with extension and flexion movements of the paretic index finger.
170  (FCO) of the hindleg monitors extension and flexion movements of the tibia and provides the main sou
171    Intermittent Head Drops are episodic head flexion movements that can occur in a number of conditio
172                       During ballistic wrist flexion movements, the latency of the second agonist EMG
173  the timely initiation and execution of limb flexion movements.
174 sic EMG pattern accompanying ballistic wrist flexion movements; and reciprocal inhibition between for
175  structure lies near one end of the range of flexion observed.
176                                 Mean maximum flexion of affected knees was 114.08 degrees .
177 scillatory activity in the STN and voluntary flexion of either the index or little finger at differen
178 t individuation abnormalities were excessive flexion of joints that should have been held fixed durin
179 sults demonstrated a significant increase in flexion of the digits (P < 0.001) and elbow (P < 0.005),
180 ions: dorsiflexion of the dominant ankle; or flexion of the dominant wrist.
181 in the impaired leg was active during finger flexion of the impaired hand in the stroke survivors and
182  to be triggered by self-initiated voluntary flexion of the index finger.
183 ns, as did MEPs evoked during unopposed weak flexion of the index finger.
184 on achievable with fluoroscopically-assisted flexion of the knee and rotation of the foot with comput
185                           Passive and active flexion of the proximal and distal interphalangeal joint
186 f biceps tendons in normal subjects elicited flexion of the stimulated arm at the elbow and a matchin
187 e an arbitrary number of at least 45 degrees flexion of the thoracolumbar spine when the individual i
188 n normal individuals, this stimulus produces flexion of the vibrated arm around the elbow joint.
189 antly extended three-domain molecule exhibit flexions of <40 degrees .
190 e, instantly peaking pain) and "limited neck flexion on examination" resulted in the Ottawa SAH Rule,
191 ion of the limbs on the side moving down and flexion on the opposite side.
192 which stroking the skin on one side leads to flexion on the other side, is disynaptic.
193 vements from neutral to targets at 20 deg of flexion or extension in response to an auditory cue.
194  at the elbow and simultaneously feeling for flexion or extension of the contralateral (paretic) arm.
195 nificantly lower than IOP measured with neck flexion or extension or in the recumbent positions.
196  and PADs showed maximal amplitude in either flexion or extension phases.
197   Both of these effects depend on the phase (flexion or extension) of the rhythm in which the stimuli
198                    No significant changes in flexion or grip strength, no systemic allergic reactions
199 g of the muscular annular perimeter, annular flexion, or angular excursion of the anterior or posteri
200  measurements obtained at 30 degrees of knee flexion (P = .047) had an association with the presence
201 ignificantly greater DeltaMAP during plantar flexion, particularly at 0.5 kg with the most affected l
202 plit-belt walking, whereas that of the swing/flexion phase decreased.
203 n phase decreased, whereas that of the swing/flexion phase increased.
204 e the limbs were at rest, and during the mid-flexion phase of contralateral rhythmical wrist flexion-
205 iculospinal inputs were increased during the flexion phase, whereas sensory-evoked DRPs and PADs show
206 rn, but with increased firing centred on the flexion phase.
207 the data show that the Q loop is the central flexion point where the aspect of the drug-binding cavit
208        The same stimulation delivered during flexion produces a temporary resetting to extension with
209 ne and 30 months using posteroanterior fixed-flexion radiographs and Kellgren/Lawrence (K/L) grading,
210 ondral tibial plateau was performed on fixed flexion radiographs of 248 nonreplaced knees, using a co
211 lity of the nonfluoroscopically guided fixed-flexion radiography protocol to detect knee joint space
212                                 Hip and knee flexion range contribute significantly to walking veloci
213 aminer variation, revealed that hip and knee flexion range explained 6% of the variance in walking ve
214 ffusion, fixed-flexion deformity, restricted-flexion range of motion, and crepitus.
215 correlation (r) between walking velocity and flexion range of the hip and knee were 0.40 and 0.35, re
216 ed the variance attributable to hip and knee flexion range only slightly, to 5%.
217 ocity over 50 feet explained by hip and knee flexion range, adjusting for the combined influence of d
218 imental studies suggest that prolonged trunk flexion reduces passive support of the spine.
219 t are strongly activated during both fictive flexion reflex and fictive scratching.
220                            We show here that flexion reflex and swimming also share key spinal cord c
221    However, similar interactions between leg flexion reflex and swimming have not been reported.
222 a common set of spinal interneurons mediates flexion reflex and the flexion components of locomotion
223 s that are strongly activated during fictive flexion reflex but inhibited during fictive scratching a
224             Leg cutaneous stimuli that evoke flexion reflex can alter the timing of (i.e., reset) cat
225                                              Flexion reflex can interrupt and reset the rhythm of scr
226                               Therefore, leg flexion reflex circuits likely share key spinal interneu
227 escending control of both spinal nociceptive flexion reflex EMG activity and individual dorsal horn n
228 icate that dorsal horn neurons that underlie flexion reflex generation (SMR) and the rostral transmis
229 a long-term potentiation (LTP) in the spinal flexion reflex in mammals, presumably to provide enhance
230         These two phase-dependent effects of flexion reflex on the swim rhythm and vice versa togethe
231 and vice versa together demonstrate that the flexion reflex spinal circuit shares key components with
232 ecordings of the H-reflex and nonnociceptive flexion reflex were obtained from pentobarbital-anesthet
233 or nerve activity underlying leg withdrawal (flexion reflex) and the rhythmic, alternating hip flexor
234 The spinal cord can generate leg withdrawal (flexion reflex), locomotion, and scratching in limbed ve
235 f limb movements, including limb withdrawal (flexion reflex), scratching, and locomotion, and thus is
236 cord neurons that produce hip flexion during flexion reflex, locomotion, and scratching based on evid
237 hat spinal cord neuronal networks underlying flexion reflex, multiple forms of locomotion, and scratc
238 the ipsilateral hip flexor bursts of fictive flexion reflex.
239 bar flexion specified relative to individual flexion-relaxation angles (i.e., 30, 40, 60, 80, and 100
240 subjects' ability to reacquire the prolonged flexion response during testing.
241 drug baclofen dose-dependently decreased the flexion response of Chronic Spinal rats (A(50)=4.3 mg/kg
242 ally transected rats can acquire a prolonged flexion response to prevent the delivery of shock.
243  patients with dystonia, there was a similar flexion response to the vibratory stimulus in the stimul
244  spinal transections can learn to maintain a flexion response when shock delivery is paired with leg
245  (contingent shock) will learn to maintain a flexion response.
246  acquisition and retention of this prolonged flexion response.
247 guidance of trajectory or by "nongiant" tail flexion responses that allow for sensory guidance but oc
248  rotor and the C ring also exhibited angular flexion, resulting in a slight narrowing of both structu
249                                        These flexion-selective cells are physiologically and morpholo
250                                        These flexion-selective interneurons are typically rhythmicall
251 also show that our material is pressure- and flexion-sensitive, and therefore suitable for electronic
252               Here we develop a new model of flexion spasms based on prenatal exposure to betamethaso
253 n therapy on the development of age-specific flexion spasms were determined and electroencephalograph
254 in structures involved in the development of flexion spasms.
255 rain structures involved in the induction of flexion spasms.
256  16 min to each of five magnitudes of lumbar flexion specified relative to individual flexion-relaxat
257 raining states and accurately generate wrist flexion states that are intermediate to training levels.
258 AWR demonstrated an increase of both truncal flexion strength (from mean 505.6 N to 572.3 N, P < 0.00
259 ed arm strength (P < 0.05), and improved hip flexion strength (P < 0.05) with treatment.
260                               Poorer plantar flexion strength (p trend = 0.004), lower baseline leg p
261                             The reduction in flexion strength did not reach statistical significance.
262 lower calf muscle density and weaker plantar flexion strength, knee extension power, and hand grip we
263                  Observed responses to trunk flexion suggest nonlinearity in viscoelastic properties,
264             The Gaussian spread of angles of flexion suggests that flexibility is driven thermally, f
265                                    The wrist flexion task yielded no differences in onset latencies b
266 ce of a finger tapping task, but not a wrist flexion task, improved significantly with anesthesia of
267 ved in all five tested subjects in imaginary flexion tasks at very short latencies (26.4 +/- 3.7 msec
268 ormed a series of submaximal isometric elbow flexion tasks.
269 tarod, inclined-plane, and forelimb/hindlimb flexion tests.
270 ent walking pattern marked by excessive knee flexion that worsens with age.
271 y either giant neuron-mediated "reflex" tail flexions that occur with very little delay but do not al
272  no consensus on the degree of thoracolumbar flexion to define camptocormia.
273 o F) is stronger than at the transition from flexion to extension (F to E).
274  at low obstacle height), and increased head flexion to look down at more immediate areas of the grou
275 ts also generated 0.2 Newton meter voluntary flexion torque in preloading tasks before stretch.
276 ions reveals a pronounced asymmetry favoring flexions toward the central protrusion.
277        fMRI data obtained during rest, thumb flexion (trained movement) and thumb extension (untraine
278 undertaken before unilateral ballistic wrist flexion training.
279 iscrete behavioral responses (eyeblink, limb flexion) under all conditions; however, in the "trace" p
280         Radiography of the knee in the fixed-flexion view provides a sensitive and valid measure of j
281                                         Knee flexion was increased in the rofecoxib group compared wi
282 observed with complete rings, normal annular flexion was maintained with the Tailor ring before and d
283 ion and at 30 degrees and 60 degrees of knee flexion was observed.
284                         As well, IOP in neck flexion was significantly higher than IOP in neck extens
285 ed by isokinetic knee extension and shoulder flexion, was significantly higher in controls than all i
286 the knee in extension and lateral 30 degrees flexion were obtained and assessed for the Kellgren/Lawr
287 g PECO following electrically evoked plantar flexion, where only muscle chemosensitive afferents were
288 d a pattern of finger extension reversing to flexion, whereas the second principal component became i
289 d and torso in the anterior direction (torso flexion) while the hips shifted in the posterior directi
290  contraction (MVC) static knee extension and flexion with manipulation of central command (CC) by pat
291 tral neck position, neck extension, and neck flexion, with the order of measurements randomized.
292  pain modulation other than the well-studied flexion withdrawal pathways.
293 e pulley system was studied at extension and flexion without and with MR tenography.
294 ion of the behavioral response (extension or flexion wrist movement).

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