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1 elax smooth muscle cells in a process termed flow-mediated dilatation.
2         Endothelial function was assessed by flow-mediated dilatation.
3 renergic blockade resulted in an increase in flow-mediated dilatation.
4 ype mice, CHF markedly impaired NO-dependent flow-mediated dilatation.
5 P=0.27), but did ameliorate the reduction in flow-mediated dilatation (0 hours, 4.0+/-1.0; 2 hours, 3
6 0 days after therapy (absolute difference in flow-mediated dilatation, 0.9%; 95% CI, 0.1 to 1.7; P=0.
7                                              Flow-mediated dilatation, a marker of endothelial functi
8 ature was investigated using brachial artery flow-mediated dilatation and carotid artery intima-media
9                  METHODS AND Brachial artery flow-mediated dilatation and cIMT were measured in 213 n
10 there was no consistent relationship between flow-mediated dilatation and free oxidized homocysteine,
11 stiffness), brachial artery reactivity (both flow-mediated dilatation and sublingual glycerol trinitr
12 diameter of the brachial artery during flow (flow-mediated dilatation), and inflammatory biomarkers a
13 ne artery pulsatility index, brachial artery flow-mediated dilatation, and serum concentrations of pl
14 fy endothelial function (via brachial artery flow-mediated dilatation) at sea level (344 m) and high
15 fy endothelial function (via brachial artery flow-mediated dilatation) at sea level (344 m) and high
16 (by 39.7+/-11.8%; P=0.02) but also inhibited flow-mediated dilatation by approximately 80% (P<0.01).
17   In both loading studies, peak reduction in flow-mediated dilatation coincided with maximal reduced
18 n improved endothelial function (measured as flow-mediated dilatation) compared with placebo (dark ch
19             In regression analysis, improved flow-mediated dilatation correlated closely with the red
20                    Peripheral conduit artery flow-mediated dilatation decreases with ageing in humans
21 ved arterial dilatation Deltabrachial artery flow-mediated dilatation/Deltadilation response to glyce
22                        Endothelium-dependent flow-mediated dilatation (ED-FMD) of the brachial artery
23                              Brachial artery flow-mediated dilatation (endothelium dependent) and nit
24  hours, 31.2+/-3.1 micromol/l, P<0.001), and flow-mediated dilatation fell (0 hours, 4.3+/-0.7; 2 hou
25                                              Flow-mediated dilatation fell, and concentrations of tot
26 on and structure, we examined conduit artery flow mediated dilatation (FMD), an index of nitric oxide
27 binders and endothelial function measured by flow mediated dilatation (FMD).
28 tion, there was a significant improvement in flow-mediated dilatation (FMD) (endothelial-dependent di
29 al endothelial function as assessed by using flow-mediated dilatation (FMD) and arterial compliance a
30 chial artery diameter, endothelial-dependent flow-mediated dilatation (FMD) and endothelial-independe
31                                              Flow-mediated dilatation (FMD) and the dilatation after
32                                              Flow-mediated dilatation (FMD) improved after chronic (1
33                                              Flow-mediated dilatation (FMD) of conduit arteries is de
34 imary and secondary outcomes were changes in flow-mediated dilatation (FMD) of the brachial artery, a
35                 ED was evaluated by means of flow-mediated dilatation (FMD) of the brachial artery.
36                 At baseline, brachial artery flow-mediated dilatation (FMD) was 55% lower in older (n
37                                              Flow-mediated dilatation (FMD) was impaired in the famil
38 thelial function assessed by brachial artery flow-mediated dilatation (FMD) was measured before, imme
39 ma-media thickness (IMT) and brachial artery flow-mediated dilatation (FMD) were measured.
40 a-3) PUFAs on vascular function measured via flow-mediated dilatation (FMD), laser Doppler iontophore
41                              Brachial artery flow-mediated dilatation (FMD), normalized for local she
42            Endothelial function, assessed by flow-mediated dilatation (FMD), was measured before, at
43 nction determined with the use of ultrasound flow-mediated dilatation (FMD).
44 ld contribute to post-exercise reductions in flow-mediated dilatation (FMD).
45 P and DBP), central pulse pressure (cPP) and flow-mediated dilatation (FMD).
46 dothelial function in the brachial artery by flow-mediated dilatation (FMD).
47 ctors--ie, lipoproteins, blood pressure, and flow-mediated dilatation (FMD).
48     Endothelium-dependent (hyperemia-induced flow-mediated dilatation [FMD]) and -independent (glycer
49        Eight weeks of heat therapy increased flow-mediated dilatation from 5.6 +/- 0.3 to 10.9 +/- 1.
50                                              Flow-mediated dilatation improved after treatment with B
51 een changes in HDL-induced NO production and flow-mediated dilatation improvement by ET was evident.
52 ascorbic acid supplementation did not affect flow-mediated dilatation in any group.
53 tors and endothelial function as assessed by flow-mediated dilatation in children and adults.
54       The present article reviews the use of flow-mediated dilatation in clinical cardiovascular rese
55         IR caused a significant reduction in flow-mediated dilatation in control subjects (-5.1%; 95%
56                                     Impaired flow-mediated dilatation in previously preeclamptic wome
57       Ascorbic acid administration increased flow-mediated dilatation in previously preeclamptic wome
58  hypertensive patients exhibited a decreased flow-mediated dilatation in response to postischemic hyp
59 C), on the human coronary circulation and on flow-mediated dilatation in the forearm.
60 rations > 15-fold in all groups and restored flow-mediated dilatation in the sedentary older men (to
61 l arteries, carotid arteries and aortae, and flow-mediated dilatations in third-order mesenteric resi
62 ontrol subjects during endothelium-dependent flow-mediated dilatation induced by hand skin heating.
63 ement therapy on inflammatory markers and on flow-mediated dilatation is largely beneficial, although
64 s largely beneficial, although the effect on flow-mediated dilatation is modulated according to endot
65                     Other outcomes (brachial flow-mediated dilatation, microvascular reactive hyperem
66                                 At baseline, flow-mediated dilatation (normalized for the hyperaemic
67                                However, only flow-mediated dilatation, not risk factors or Framingham
68                                              Flow-mediated dilatation of the brachial artery increase
69 econdary end points of endothelial function (flow-mediated dilatation of the brachial artery) and car
70 tive than longitudinal imaging for measuring flow-mediated dilatation of the brachial artery.
71 essel endothelial function was assessed with flow-mediated dilatation of the brachial artery.
72 d not significantly affect the percentage of flow-mediated dilatation or other measures of vascular f
73 nship between homocysteine concentration and flow-mediated dilatation (P<0.001).
74 6.0+/-6.4% (n=10; P=0.03) but did not affect flow-mediated dilatation (P=0.55).
75 r n-6 PUFAs did not affect the percentage of flow-mediated dilatation (primary endpoint) or other mea
76  biomarker and vascular function measured as flow-mediated dilatation (R = -0.3, P < 0.01) or endothe
77 tral water immersion, heat therapy increased flow-mediated dilatation, reduced arterial stiffness, re
78 nction was assessed from the brachial artery flow-mediated dilatation response.
79              The primary outcome measure was flow-mediated dilatation; secondary outcome measures inc
80                                              Flow-mediated dilatation showed a reciprocal relationshi
81                                              Flow-mediated dilatation testing provides an integrated
82 ere markedly attenuated, and brachial artery flow-mediated dilatation was depressed.
83                              Brachial artery flow-mediated dilatation was determined at baseline and
84                                              Flow-mediated dilatation was determined before IR and af
85                                              Flow-mediated dilatation was expressed as mean and compa
86                                     However, flow-mediated dilatation was greater and the plasma leve
87                                              Flow-mediated dilatation was higher during a pregnancy w
88                                    Mean (SD) flow-mediated dilatation was lower in women with previou
89                              Brachial artery flow-mediated dilatation was measured at baseline and at
90                                 In patients, flow-mediated dilatation was not affected by fluconazole
91           Similarly, IR-induced reduction in flow-mediated dilatation was not observed in p47-chronic
92                 In controls, heating-induced flow-mediated dilatation was reduced by fluconazole, L-N
93                                              Flow-mediated dilatation was significantly impaired 8 ho
94           Twenty-four hours after treatment, flow-mediated dilatation was significantly lower in the
95                                              Flow-mediated dilatation was used to assess endothelial
96 dentary human ageing and the preservation of flow-mediated dilatation with physically active ageing.
97  the impairment in peripheral conduit artery flow-mediated dilatation with sedentary human ageing and

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