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1 ceptance criteria, and testing schedule) for fludeoxyglucose (18)F ((18)F-FDG) injection listed in th
2 in neurodegeneration was assessed by MRI and fludeoxyglucose-18 positron emission tomography ((18)FDG
3 e association study (GWAS) of decline in PCC fludeoxyglucose F 18 ([(18) F] FDG) positron emission to
4 relates with brain metabolism as measured by fludeoxyglucose F 18 ([(18)F]-FDG) positron emission tom
5 ent-naive HIV-infected individuals underwent fludeoxyglucose F 18 ([18F]-FDG) positron emission tomog
6 tron emission tomography (PET) combined with fludeoxyglucose F 18 (FDG) is recommended for the noninv
9 ment for apolipoprotein epsilon4, concurrent fludeoxyglucose F 18 PET scan, and baseline cognitive st
11 omography, AD hypometabolic pattern shown on fludeoxyglucose F 18 positron emission tomography, and/o
13 We used positron emission tomography and fludeoxyglucose F 18 to measure brain metabolism in Viet
14 etabolism (positron emission tomography with fludeoxyglucose F 18), extracted from Alzheimer disease-
16 ow reduced glucose metabolism, as indexed by fludeoxyglucose F 18-labeled positron emission tomograph
17 of the PET scan was estimated by multiplying fludeoxyglucose F18 radioactivity with dose coefficients
19 Dynamic Contrast-Enhanced MRI (DCE-MRI) and Fludeoxyglucose Positron Emission Tomography(FDG-PET).
20 lorbetapir positron emission tomography, 18F-fludeoxyglucose positron emission tomography, structural
21 tive study was to assess predictive value of fludeoxyglucose-positron emission tomography (FDG-PET) a
22 d no family history of dementia has abnormal fludeoxyglucose-positron emission tomography imaging and
23 This study aimed to determine whether 18F-fludeoxyglucose-positron emission tomography/computed to
24 at is the sensitivity and specificity of 18F-fludeoxyglucose-positron emission tomography/computed to
25 emission tomography with 18F-florbetapir and fludeoxyglucose was used to quantify amyloid retention a
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