ライフサイエンスコーパス: fluid volume

1 area, microvascular volume, and interstitial fluid volume.

2 atogenesis, and the control of extracellular fluid volume.

3 d despite an increase in total extracellular fluid volume.

4 s to alterations in sodium and extracellular fluid volume.

5 ontent in the cartilage and a lower synovial fluid volume.

6 y a factor of at least 20 in the bulk of the fluid volume.

7 ements were effective in determining ascites fluid volume.

8 ip between sodium intake and changes in body fluid volume.

9 ssary to replenish circulating intravascular fluid volume.

10 ase bone scintigraphic findings and synovial fluid volume.

11 in body weight, hematocrit, or extracellular fluid volume.

12 (HA) has a major role in regulating synovial fluid volume.

13 subjects to quantify brain and cerebrospinal fluid volumes.

14 ful regulators of arterial pressure and body fluid volumes.

15 rain tissue volumes and larger cerebrospinal fluid volumes.

16 devices are used for dispensing well-defined fluid volumes.

17 ite expanded blood, plasma and extracellular fluid volumes.

18 0.9% bolus and infusion for 3 hr [equivalent fluid volume]).

19 Median fluid volume (~15,774 m(3)) and volume per perforated in

20 rresponding to an average loss of 22% plasma fluid volume (227 mul), and this was almost completely r

21 d with baseline illness severity rather than fluid volume administered.

22 e was greater expansion of the extravascular fluid volume after saline (P = 0.029).

23 rd of death even after controlling for total fluid volume, age, and severity (p = 0.0015) over 1 year

24 erval, 5 to 15 /1 to 6 mm Hg), extracellular fluid volume, albuminuria, and proteinuria in patients w

25 rocal increase in frontal lobe cerebrospinal fluid volume also occurred at a more rapid rate in patie

26 compensatory increase in total cerebrospinal fluid volume also was found (mean +/- SD: PVL, 64.5 +/-

27 hormone aldosterone increases extracellular fluid volume and blood pressure by activating epithelial

28 n important role in regulating extracellular fluid volume and blood pressure, as well as airway surfa

29 cial role in the regulation of extracellular fluid volume and blood pressure.

30 regulatory system controlling extracellular fluid volume and blood pressure.

31 key role in the regulation of extracellular fluid volume and blood pressure.

32 angiotensin system (RAS) in controlling body fluid volume and blood pressure.

33 BCG vaccination increased fluid volume and cellular accumulation, significantly en

34 day, pleural exudates were removed, and the fluid volume and characteristics of the infiltrating cel

35 cellular fluid volume homeostasis and airway fluid volume and composition, we investigated whether th

36 anti-TGF-beta1 resulted in decreased pleural fluid volume and decreased cell numbers in the pleural s

37 e was no relationship between changes in leg fluid volume and either DeltaNC or apnea-hypopnea index.

38 V, CNV volume, retinal thickness, subretinal fluid volume and height of neurosensory detachment befor

39 early and uniform expansion of extracellular fluid volume and increased cardiac output.

40 LBPP reduced leg fluid volume and increased neck circumference in both ob

41 Pressure increased as a function of fluid volume and infusion rate in wild-type animals, but

42 functions of epithelia and determine bodily fluid volume and ionic composition, among other things.

43 have a key role in regulating extracellular fluid volume and the volume of airway surface liquids.

44 were computer digitized and analyzed for hip fluid volume and visually assessed for the presence and

45 (brain volume/[brain volume + cerebrospinal fluid volume), and magnetization transfer ratio histogra

46 r volume, white matter volume, cerebrospinal fluid volume, and hippocampal volume), while questions s

47 in volume, gray matter volume, cerebrospinal fluid volume, and hippocampal volume, were independently

48 r volume, white matter volume, cerebrospinal fluid volume, and hippocampal volume.

49 hree are lateral casing diameter, fracturing fluid volume, and length of the lateral.

50 Blood volume, extracellular fluid volume, and regional blood flow were estimated fro

51 al lobes), lateral ventricular cerebrospinal fluid volume, and symptom severity from the Scale for th

52 for multiple treatments, injection of large fluid volumes, and decreased efficacy in treatment of no

53 ent antibiotic administration, lower mean IV fluids volume, and suffered higher in-hospital mortality

54 ventricle enlargement; larger cerebrospinal fluid volume; and smaller volumes of the basal ganglia,

55 linearly until old age, while cerebrospinal fluid volumes are stable in adulthood (age 20-50 years).

56 Larger residual interface fluid volume, area, and thickness at the end of surgery

57 Tumor extracellular fluid volume as measured with (99m)Tc DTPA [(3.1 +/- 0.2

58 us abortion, preterm labor, and low amniotic fluid volume at the time of the pathogen isolation.

59 ed overnight changes in NC (DeltaNC) and leg fluid volume before and after polysomnography.

60 ent antibiotics, and emergency department IV fluids volume, being afebrile remained a significant pre

61 ule, that features the largest difference in fluid volume between the scala vestibuli (SV) and scala

62 27.9+/-1 mmol/L, P < 0.05) and extracellular fluid volume bicarbonate by an estimated 39+/-10 mmol.

63 27 mmol acid, did not increase extracellular fluid volume bicarbonate over the 90-min period.

64 ) was ingested and the gain in extracellular fluid volume bicarbonate was compared with renal acid el

65 ion and thus in the control of extracellular fluid volume, blood pressure, and sodium homeostasis.

66 tributes to the maintenance of extracellular fluid volume by regulating sodium transport in the nephr

67 The regulation of extracellular fluid volume by renal sodium excretion lies at the centr

68 e intravascular portion of the extracellular fluid volume, can be measured using standard dilution te

69 technique to measure brain and cerebrospinal fluid volume changes.

70 Furthermore, although in men, changes in leg fluid volume correlated inversely with DeltaNC (r=-0.755

71 ese conditions coexist because extracellular fluid volume depletion is often treated rapidly with 0.9

72 iology of both dehydration and extracellular fluid volume depletion must be understood if these condi

73 As with extracellular fluid volume depletion, arterial underfilling secondary

74 ular volume and is best termed extracellular fluid volume depletion.

75 Although the changes in leg fluid volume did not differ significantly between men an

76 windows, combined with it having the biggest fluid-volume difference between the SV and ST, is though

77 setting of normal or expanded extracellular fluid volume, e.g., in primary aldosteronism.

78 with measurement of total body extracellular fluid volume (ECFV), and ECFV of the neck, thorax, and r

79 gray matter, as well as faster cerebrospinal fluid volume expansion in right frontal sulci, left late

80 Intravascular fluid volume expansion is an acute compensatory adaptati

81 Extracellular fluid volume expansion occurs in clinically stable HTRs

82 kely related to the effects of extracellular fluid volume expansion, also regulate kidney AQP-2 expre

83 ts show that in the setting of extracellular fluid volume expansion, excessive water retention with h

84 , including several related to extracellular fluid volume expansion, increased in the intervention gr

85 nd the natriuresis produced by extracellular fluid volume expansion.

86 used to treat hypertension and extracellular fluid volume expansion.

87 ated with an increased risk of extracellular fluid volume expansion.

88 correcting for the aftershock rate, the net fluid volume (extracted-injected) provides the best corr

89 t may involve alterations in renal function, fluid volume, fluid-regulatory hormones, the vasculature

90 tionally measured concentration, (2) minimum fluid volume for analysis is usually less than 0.005 mic

91 may autonomously regulate the local surface fluid volume for homeostasis while permitting acute resp

92 ite matter volumes; and larger cerebrospinal fluid volumes for temporal lobe sulci and the 3 ventricu

93 ent levels (RAL); and 5) gingival crevicular fluid volume (GCF).

94 showed significant associations between high fluid volume given on the day of surgery with both incre

95 For patients in the fourth quartile of fluid volume (> 7 L), the odds ratio for mortality for %

96 Flow in these devices with small fluid volumes has mainly been characterized by following

97 has important implications in extracellular fluid volume homeostasis and airway fluid volume and com

98 rticipate in the regulation of extracellular fluid volume homeostasis and blood pressure.

99 derstanding of the role of NCC in sodium and fluid volume homeostasis and in the pathogenesis of Gite

100 Natriuretic regulation of extracellular fluid volume homeostasis includes suppression of the ren

101 uses only subtle perturbations of sodium and fluid volume homeostasis, but renal handling of Mg2+ and

102 ysiological pathways involving extracellular fluid volume homeostasis, cardiac contractility and vasc

103 exchanger impairs acid-base balance and Na+-fluid volume homeostasis.

104 abnormalities of electrolyte, acid-base, and fluid-volume homeostasis occur because of defective NaCl

105 perturbations of electrolyte, acid-base, and fluid-volume homeostasis, reduced absorption of NaCl in

106 the primary cause of expanded cerebrospinal fluid volume in newborns, intracranial and retinal haemo

107 Ohm/m, p = 0.009 for men), indicating lower fluid volume in the patients with CI-AKI.

108 n to attenuate drainage, conserving synovial fluid volume in the presence of raised joint pressure.

109 This process results in a locally mixed fluid volume in the range of 0.5-1.5 nL that is convecte

110 ma volume, and no increases in extracellular fluid volume in vivo.

111 tter and increased ventricular cerebrospinal fluid volumes in patients with schizophrenia in the whol

112 3.10 to -2.63%; P < 0.001) and extracellular fluid volume increased by 0.62 L/1.73 m(2) (95% CI 0.26

113 ike response with expansion of extracellular fluid volume, increased vascular permeability, and vasod

114 Fluid volume infused during hypotensive resuscitation wa

115 f the field as the number of earthquakes per fluid volume injected decreases over time.

116 It is known that extracellular fluid volume is expanded (12% to 15%) in HTRs who develo

117 Extracellular fluid volume is expanded (12%) in clinically stable HTRs

118 filled pores, such that the combined solid + fluid volume is reduced and the inefficiencies in space

119 ow sensitivity of NMR is aggravated by small fluid volumes leading to low NMR signal and geometric co

120 tegies involving haemoglobin replacement and fluid volume loading to regain tissue perfusion and oxyg

121 used lungs using 125I-albumin as an airspace fluid volume marker.

122 l pressure, cerebral perfusion pressure, and fluid volume may be detrimental to severe brain injury o

123 Hydraulic fracturing required a median fluid volume of 11,350 m(3) per horizontal well in Oklah

124 Computer analysis yielded an average fluid volume of 2.7 mL (range, 0.7-5.6 mL) in asymptomat

125 ted from 14-mm test tubes, with an aspirated fluid volume of 201 mL +/- 64.

126 econd dimension was capable of analyzing all fluid volumes of interest from the IEF dimension, as IEF

127 requirements and to determine the effects of fluid volumes on outcome.

128 ically relevant features, such as subretinal fluid volume or pigment epithelial detachment volume.

129 When using high gavage fluid volumes or injection into ligated intestinal loops

130 e slow, lack single-cell specificity, or use fluid volumes out of scale with those of cells.

131 here was a significant reversal of the daily fluid volume output/input ratio from 0.8 +/- 0.1 to 1.2

132 nd hypertonic saline dextran reduced the net fluid volume over 8 hrs by 48% and 74%, respectively, co

133 ing the operating room identified that total fluid volume (P = .002), largest fluid volume pocket (P

134 volume (P < .001, SE = .0021), cerebrospinal fluid volume (P = .01, SE = .0024), and hippocampal volu

135 volume (P = .008, SE = .0687), cerebrospinal fluid volume (P = .012, SE = -.0667), and hippocampal vo

136 scan scores (P = 0.015), as well as synovial fluid volumes (P < 0.0001).

137 that total fluid volume (P = .002), largest fluid volume pocket (P = .002), max fluid area (P = .006

138 duce the costs of highly parallelized, small fluid volume, point-of-care and home-based diagnostics.

139 Median fluid volume provided in the first 6 hrs of hospitalizat

140 ociated with increase in total cerebrospinal fluid volume (r = 0.83, p < 0.001), and change in total

141 Average fluid volume received during the first 24 hours after in

142 Increased fluid volume received increased risk of development of p

143 f micro-electrodes for the analysis of small fluid volumes recovered from the oral environment.

144 -0.12 to 1.87%; P = 0.095) and extracellular fluid volume reduction of -0.75 L/1.73 m(2) (95% CI -1.5

145 How is fluid volume regulated in joints?

146 ify aquaporin 1 (AQP1), a potent effector of fluid volume regulation and angiogenic activity, as a no

147 Body fluid volume regulation by the kidney relies upon the co

148 be involved in inner ear ion homeostasis and fluid volume regulation for the maintenance of hearing a

149 exclusion, is of importance for interstitial fluid volume regulation.

150 Therefore, establishing peri-procedural fluid volume related to increased risk of CI-AKI develop

151 A comprehensive 2D analysis of a fluid volume spanning 15% of the total IEF channel lengt

152 y outcome measures comprised time to anuria, fluid volume status, peritonitis-free survival, techniqu

153 should be accompanied by close monitoring of fluid volume status.

154 er right hippocampi and larger cerebrospinal fluid volumes than healthy subjects of the same sex.

155 other material and to contain a substantial fluid volume that can rival the volume of the dry stratu

156 trimentally to the gradual expansion of cyst fluid volume that is a hallmark of ADPKD.

157 and make use of rapid temperature changes in fluid volumes that are commensurate with the size of sin

158 icted age with grey matter and cerebrospinal fluid volumes (themselves strong predictors) not did imp

159 en with bed rest, without alteration in limb fluid volumes thus validating the technique and raising

160 al signaling but by regulating extracellular fluid volume to modulate ligand-receptor interactions.

161 tion due to contraction of the extracellular fluid volume (vAKI) or due to intrinsic kidney injury (i

162 Associations among total cerebrospinal fluid volume, ventricular volume, serum sodium, and Glas

163 Synovial fluid volume was also decreased in the sedentary group c

164 < 0.001), and change in total cerebrospinal fluid volume was associated with ventricular volume chan

165 grams microliters-1, the endogenous synovial fluid volume was calculated to be 50 microliters (mass/c

166 Therefore, cerebral spinal fluid volume was decreased (Delta-40ml, 95% CI = -67 to

167 Tumor extracellular fluid volume was determined in a separate group of rats.

168 The intraoperative fluid volume was higher for all patients with pancreatic

169 The ascitic fluid volume was modeled as a segment of a sphere.

170 nd enlargement in frontal lobe cerebrospinal fluid volume were associated with greater negative sympt

171 body water, blood volume, and extravascular fluid volume were calculated.

172 All measures of crystalloid fluid volume were reduced while patients were maintained

173 ermeability index, and the epithelial-lining fluid volume were similar with the two techniques and de

174 result from processes that alter the pore or fluid volume, which in turn implies crustal changes happ

175 The fluid volume within the eye was discretized using 6 x 10

176 le bridges as a novel method of manipulating fluid volumes within paper-based devices.