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3 ing microhemorrhages and hyperintensities on fluid-attenuated inversion recovery and diffusion-weight
4 dical records and magnetic resonance imaging fluid-attenuated inversion recovery and gradient-echo sc
6 cts and cerebral microbleeds was assessed on fluid-attenuated inversion recovery and T1-weighted and
8 ceptibility-weighted perfusion, T2-weighted, fluid-attenuated inversion recovery, and high-dose contr
9 re- and postcontrast transverse T1-weighted, fluid-attenuated inversion recovery, and T2-weighted ima
10 mor MRI indices at baseline (tumor volume on fluid-attenuated inversion recovery, baseline intratumor
11 duced diffusion and high signal intensity on fluid-attenuated inversion recovery brain MRI within a v
12 new hyperintensity on diffusion-weighted and fluid-attenuated inversion recovery cerebral MRI sequenc
13 onal and fast spin echo T2-weighted imaging, fluid-attenuated inversion recovery, detection of blood-
15 cally ill cats, which included multifocal T2 fluid attenuated inversion recovery (FLAIR) signal hyper
16 s) or regions that were only hyperintense on fluid-attenuated inversion recovery (FLAIR) 1H images (e
18 tney U test), (d) relationships between fast fluid-attenuated inversion recovery (FLAIR) data and enh
19 rain ratio (VBR) was measured on T2-weighted fluid-attenuated inversion recovery (FLAIR) images at di
20 signal evolution on magnetic resonance (MR) fluid-attenuated inversion recovery (FLAIR) images betwe
21 -state acquisition (FIESTA), and T2-weighted fluid-attenuated inversion recovery (FLAIR) imaging were
23 layer appearance of the cerebral cortex with fluid-attenuated inversion recovery (FLAIR) magnetic res
24 euroradiologists independently reviewed 2572 fluid-attenuated inversion recovery (FLAIR) MRI scans fr
25 ent with a conventional two-dimensional (2D) fluid-attenuated inversion recovery (FLAIR) sequence wit
26 nds in brain imaging are reviewed, including fluid-attenuated inversion recovery (FLAIR), diffusion-w
27 ched patients without MS, sagittal 2-mm fast fluid-attenuated inversion-recovery (FLAIR) imaging was
29 t SE, gradient-echo and SE (GRASE), and fast fluid-attenuated inversion-recovery (FLAIR) imaging.
30 ted supra- and infratentorially on simulated fluid-attenuated inversion-recovery (FLAIR) magnetic res
31 ee-dimensional (3D) fast spin-echo (FSE) and fluid-attenuated inversion-recovery (FLAIR) T2-weighted
32 methods (T1- and T2-weighted MR imaging plus fluid-attenuated inversion recovery [FLAIR] at 3-mm sect
33 cm, 15 of 18), subcortical (18 of 18), T2 or fluid-attenuated inversion recovery hyperintense (18 of
34 es revealed confluent bifrontal white matter fluid-attenuated inversion recovery hyperintensities, as
35 sets with a volume resolution of 1 mm(3) and fluid-attenuated inversion-recovery image sets with a vo
37 ions and hyperintense regions at nonenhanced fluid-attenuated inversion recovery imaging) information
38 (contrast material-enhanced T1-weighted and fluid-attenuated inversion-recovery imaging sequences) a
39 weighted imaging lesions and a corresponding fluid-attenuated inversion recovery lesion 48 hours afte
40 te models including brain GM and WM volumes, fluid-attenuated inversion recovery lesion load, T1 lesi
41 esion number, brain WM volumes, brain T1 and fluid-attenuated inversion recovery lesion loads, age, s
42 story of optic neuritis, was associated with fluid-attenuated inversion recovery lesion volume (P=.00
43 eatment; P < .02), and mean number of new T2/fluid-attenuated inversion recovery lesions per year (7.
45 ities and brain infarcts were measured using fluid-attenuated inversion recovery magnetic resonance i
46 Clinic Study of Aging who had a baseline 3 T fluid-attenuated inversion recovery magnetic resonance i
47 ical white matter hyperintensities (WMHs) on fluid-attenuated inversion recovery magnetic resonance i
48 ck, and definitive ischemic brain lesions on fluid-attenuated inversion recovery magnetic resonance i
50 -weighted magnetic resonance (MR) images, or fluid-attenuated inversion-recovery MR images were obtai
51 (and metrics obtained) were: (i) optic nerve fluid-attenuated inversion-recovery (optic nerve cross-s
52 predicted outcomes with higher accuracy than fluid-attenuated inversion recovery or diffusion-weighte
53 ighted gradient echo sequences combined with fluid attenuated inversion recovery, or saccades error r
57 intense white matter abnormalities on T2 and fluid attenuated inversion recovery sequences predominan
58 , and/or susceptibility-weighted imaging and fluid-attenuated inversion recovery sequences, were cons
61 ic resonance imaging showed increased T2 and fluid-attenuated inversion recovery signals in the putam
62 l dystrophy and patchy increased T2-weighted fluid-attenuated inversion recovery (T2/FLAIR) signal in
63 with diffusion-weighted MR imaging and with fluid-attenuated inversion recovery, T2-weighted fast sp
64 tical signal intensities within T1-weighted, fluid-attenuated inversion recovery, T2-weighted, and pr
65 ronic MS lesions on conventional T2-weighted fluid-attenuated inversion recovery, T2-weighted, and T1
66 uded sagittal T1-weighted images, axial fast fluid-attenuated inversion-recovery/T2-weighted images,
67 cluding contrast-enhanced and T2-weighted or fluid-attenuated inversion recovery-weighted images) is
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