ライフサイエンスコーパス: fluke

1 tremely small head and a long tail without a fluke.

2 l nematode, and Schistosoma mansoni, a blood fluke.

3 the 363 megabase nuclear genome of the blood fluke.

4 ase caused by infection with parasitic blood flukes.

5 Stable positioning using the fluke and flipper was confirmed by video data for both t

6 63 family tegument tetraspanins in parasitic flukes and support efforts to target these proteins for

7 l fibrosis, which is a precursor of CCA, the flukes and their microbiota may together drive this dist

8 ng hair and hind limbs, and having flippers, flukes, and a streamlined body.

9 The fluke-associated microorganisms included potential patho

10 ential vaccine candidates could reduce total fluke burden and egg output by up to 43% and 99%, respec

11 ttle, but it is unclear at what levels liver fluke burdens cause production losses.

12 y is maintained with distantly related blood flukes but we find extreme losses of genes and pathways

13 immature and adult Fasciola hepatica (liver fluke) but widespread resistance to the drug greatly com

14 ely related to CsRn1 from the Oriental liver fluke Clonorchis sinensis and to kabuki from Bombyx mori

15 d resistance to the drug greatly compromises fluke control in livestock and humans.

16 EVs were identified in fluke culture medium and bile specimens from infected ho

17 mated that cattle classified as having liver fluke damage had on average 10 days greater slaughter ag

18 nzyme-linked immunosorbent assay (ELISA) and fluke egg counts.

19 terizing the metabolome of the hepatobiliary fluke Fasciola hepatica , using ultra performance liquid

20 Only marginal activity against the liver fluke Fasciola hepatica and Trichostrongylus colubriform

21 The liver fluke Fasciola hepatica is a major pathogen of livestock

22 For instance, the giant liver fluke, Fascioloides magna, is a major parasite of severa

23 dedicated to the dissemination of data from flukes, flatworm parasites of the class Trematoda, phylu

24 roundworm, whipworm, pinworm, Chinese liver fluke, fleas and lice.

25 for associations among infection with liver fluke, Helicobacter and hepatobiliary fibrosis.

26 O. viverrini were examined in this model of fluke-induced CCA.

27 f infection and a tissue microarray of liver fluke-induced human cholangiocarcinomas (CCAs).

28 Relationships between fluke infection and immune responses were analyzed by us

29 ent progress in tracking the spread of rumen fluke infection in Europe, and propose some research que

30 are poorly understood, though chronic liver fluke infection is a risk factor for disease.

31 ertainties by estimating the impact of liver fluke infection on UK beef cattle productivity and inves

32 Microbial community analyses revealed that fluke infection perturbed the gastrointestinal tract mic

33 iated risks for food security posed by rumen fluke infection, it is imperative that we develop a bett

34 t cagA-positive H. pylori than those free of fluke infection.

35 Liver fluke infections occur in people worldwide.

36 targeting hookworm and liver and intestinal fluke infections through current MDA approaches.

37 nd the prospects for future control of liver fluke infections using an integrated parasite management

38 is - in addition to the intestinal and liver fluke infections, as some of the most important gastroin

39 Fasciola hepatica, common liver fluke, infects cattle and sheep causing disease and prod

40 oma mansoni, a multicelluar eukaryotic blood fluke, is a major cause of morbidity worldwide in humans

41 Following ingestion, the flukes locate to the biliary tre where chronic infection

42 ments are encouraging a renaissance in liver fluke neurobiology that can now support flukicide discov

43 293 and 260 positive and negative for liver fluke O. viverrini eggs, of residents in Northeastern Th

44 The coevolution of blood flukes of the genus Schistosoma and their human hosts is

45 is a major parasitic disease caused by blood flukes of the genus Schistosoma.

46 --is a parasitic disease caused by trematode flukes of the genus Schistosoma.

47 between parasitism by the carcinogenic liver fluke Opisthorchis viverrini and this coregulator using

48 The liver fluke Opisthorchis viverrini infects 10 million people i

49 The liver fluke Opisthorchis viverrini, highly prevalent in Southe

50 e of granulin from the human parasitic liver fluke Opisthorchis viverrini, known as Ov-GRN-1, induces

51 langiocarcinoma and infection with the liver fluke Opisthorchis viverrini.

52 sease caused by the carcinogenic Asian liver fluke, Opisthorchis viverrini.

53 ses worldwide, is caused by flatworms (blood flukes or schistosomes) that live in the bloodstream of

54 Adult Schistosoma mansoni blood flukes reside in the mesenteric veins of their vertebrat

55 observation of tsp-2 and tsp-3 dsRNA-treated flukes resulted in phenotypes with increased tegument th

56 The blood fluke Schistosoma haematobium causes urogenital schistos

57 acterised from the parasitic trematode blood fluke Schistosoma haematobium.

58 resent on the surface of the trematode blood fluke Schistosoma has been implicated in the regulation

59 (AChE) on the surface of the parasitic blood fluke Schistosoma is the likely target for schistosomici

60 The taurocyamine kinase from the blood fluke Schistosoma mansoni (SmTK) belongs to the phosphag

61 glabrata, a natural host for the human blood fluke Schistosoma mansoni Granulins are growth factors t

62 The blood fluke Schistosoma mansoni is the causative agent of the

63 In the case of the human blood fluke Schistosoma mansoni, responsible for intestinal bi

64 trumental in transmission of the human blood fluke Schistosoma mansoni.

65 abrata, intermediate host of the human blood fluke Schistosoma mansoni.

66 , snail intermediate host of the human blood fluke Schistosoma mansoni.

67 The blood flukes Schistosoma japonicum and Schistosoma mansoni are

68 The blood flukes Schistosoma mansoni and Schistosoma japonicum inf

69 iquine resistance evolved in the human blood fluke (Schistosoma mansoni) in Brazil in the 1970s.

70 The metazoan parasitic blood flukes, Schistosoma spp., infect over 200 million people

71 itional evidence that O. viverrini and other flukes secrete proteins that directly modulate host cell

72 Fluke secreted and tegumental proteins contribute to the

73 with substantial morbidity and several liver fluke species are recognised as biological carcinogens.

74 ge by weight and different measures of liver fluke status, while accounting for sex, breed, season, y

75 of aquatic locomotion is that cetaceans use fluke strokes to power their swimming while relying on l

76 riptomes of O. viverrini, elucidate how this fluke survives in the hostile environment within the bil

77 lifestyle evolved in each group, such as the fluked tails, dorsal fins and wing-shaped limbs of ichth

78 alization of Ov-TSP-2 and TSP-3 to the adult fluke tegument.

79 complement of Schistosoma mansoni, the blood fluke that causes schistosomiasis, ranked among the most

80 Schistosoma mansoni is a parasitic fluke that infects millions of people in the developing

81 um, S. mansoni and S. haematobium, are blood flukes that have complex life cycles involving a snail i

82 he intricacies of the biology of these blood flukes, their host relationships, and the diseases that

83 l model to assess the effectiveness of liver fluke vaccines under simulated field conditions.

84 The model describes development of fluke within a group of animals and includes heterogenei