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1 d microtubule cytoskeletons were examined by fluorescence staining.
2 etermined by dihydroethidium (hydroethidine) fluorescence staining.
3 uorescence in situ hybridization and G-actin fluorescence staining.
4                                              Fluorescence staining, along with gene expression measur
5 of Abeta as determined by thioflavin T (ThT) fluorescence staining and atomic force microscopy (AFM)
6                                 Furthermore, fluorescence staining and confocal microscopy revealed a
7 inguishable from native cell surface WI-1 by fluorescence staining and restored adhesivity to the kno
8 verified by both electron spin resonance and fluorescence staining assay.
9 y H(2)O(2) and O( minus sign, dot below )(2) fluorescence staining assays in JB6 Cl 41 cells.
10  was measured further by H(2)O(2) and O(-.2) fluorescence staining assays.
11 ite the extensive efforts along the lines of fluorescence staining, autoradiography, and mass spectro
12     Western blotting and immunohistochemical fluorescence staining demonstrates that EphB1 receptor p
13 reased: the proportion of laboratories using fluorescence staining for acid-fast microscopy has incre
14 ned by transmission electron microscopy, and fluorescence staining for adhesion site protein expressi
15 n by quantitative immunogold staining and by fluorescence staining for lysosomotropic agents.
16 k and corneal endothelium--revealed positive fluorescence staining for peroxidized carbonyl products.
17                                          The fluorescence staining identified the same particles as t
18      Subcellular colocalization studies with fluorescence staining indicate SV can colocalize with AR
19                                      PIKfyve fluorescence staining largely coincided with trans-Golgi
20                                            A fluorescence staining method was used to show that ChrS
21 ator acridine orange (AO) identified intense fluorescence staining of acidic cytoplasmic vesicles wit
22 dual NSCs and glial cells were compared with fluorescence staining of cell nuclei and cytoplasm to sh
23 ific cellular uptake of LPS was suggested by fluorescence staining of cells along the abrasion site.
24 ed for vasoreactivity study, dihydroethidium fluorescence staining of superoxide, immunohistochemical
25                                              Fluorescence staining of the cells with rhodamine-conjug
26           These data and the results of dual-fluorescence staining of the endogenous RBCs and syncyti
27 translocation of NF-kappaB was determined by fluorescence staining of the NF-kappaB Rel A (p65) subun
28 midino-2-phenylindole (DAPI) staining and by fluorescence staining of the nuclei of membrane-compromi
29 ntified at various periods after exposure by fluorescence staining of the nuclei of membrane-compromi
30                                   Dual-label fluorescence staining of these sediments showed a fivefo
31           The lectin MAA produced an intense fluorescence-staining pattern only at the J1/J2 interfac
32 alized in bright field by phase imaging, and fluorescence-staining patterns indicate they contain mem
33 es by the use of time-lapse and quantitative fluorescence staining procedures.
34 hilic cationic protein (ECP) was detected by fluorescence staining techniques and confocal microscopy
35 morphogenesis of the MGC by means of various fluorescence staining techniques and laser-scanning conf
36 ated by confocal microscopy following immune fluorescence staining techniques.
37                          As suggested by the fluorescence staining, the deformation lines appeared to
38  and in cholesterol accumulation (by filipin fluorescence staining) throughout the cerebral neocortex
39          The use of paper point sampling and fluorescence staining was shown to be a rapid method, ab

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