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1 cture of human TS can confer resistance to 5-fluorodeoxyuridine.
2 the TS inhibitor and chemotherapeutic drug 5-fluorodeoxyuridine.
3         It also confers 8-fold resistance to fluorodeoxyuridine.
4 that mediates radiosensitization produced by fluorodeoxyuridine.
5 current continuous-infusion hepatic arterial fluorodeoxyuridine (0.2 mg/kg/d) during the first 4 week
6 hat in humans the dose-limiting toxicity for fluorodeoxyuridine [2-fluoro-5'-deoxyuridine (FdUrd)] wh
7  and obtain variants that are resistant to 5-fluorodeoxyuridine (5-FdUR).
8  other nucleoside analogs modified at C-5, 5-fluorodeoxyuridine and 5-fluorocytidine, did not induce
9              Trifluoromethyl thymidine and 5-fluorodeoxyuridine are good substrates for CpTK, and bot
10 tagens, including ultraviolet irradiation, 5-fluorodeoxyuridine, ethyl methanesulfonate, and methyl m
11  chromosomes that are induced by agents like fluorodeoxyuridine (FdU) that affect intracellular thymi
12 s each containing either zero, one, or two 5-fluorodeoxyuridine (FdUrd) or 5-fluorouridine (FUrd) nuc
13 ines, in particular, fluorouracil (5-FU) and fluorodeoxyuridine (FdUrd), are active alone and in comb
14 amage and cytotoxicity caused by exposure to fluorodeoxyuridine (FdUrd).
15   5-Fluorouracil (5-FU) and its metabolite 5-fluorodeoxyuridine (FdUrd, floxuridine) are chemotherapy
16                  5-Fluorouracil (5-FU) and 5-fluorodeoxyuridine (FdUrd, floxuridine) have activity in
17                               Floxuridine (5-fluorodeoxyuridine, FdUrd), a U.S. Food and Drug Adminis
18 h the anticancer agents 5-fluorouracil and 5-fluorodeoxyuridine (floxuridine), a 5-fluorouracil metab
19          Treatment regimen included HAI with fluorodeoxyuridine (FUDR) 60 mg/m2/d and L 15 mg/m2/d co
20 se the thymidylate synthetase (TS) inhibitor fluorodeoxyuridine (FUdR) can alter expression of cellul
21 y to the thymidylate synthase (TS) inhibitor fluorodeoxyuridine (FUdR) in human cancer cell lines.
22 frequent pattern of disease progression when fluorodeoxyuridine (FUDR) is given by the intraarterial
23 nsport of the pyrimidine nucleoside analog 5-fluorodeoxyuridine (FUdR) would inhibit growth, PhtC and
24 s delivered with concurrent hepatic arterial fluorodeoxyuridine (HA FUdR).
25 DNA and then selected mutants resistant to 5-fluorodeoxyuridine in a bacterial complementation system
26  chemotherapy agents that are converted to 5-fluorodeoxyuridine monophosphate (FdUMP) and 5-fluorodeo
27 onophosphates (deoxyuridine monophosphate, 5-fluorodeoxyuridine monophosphate, 1-beta-D-arabinofurano
28                                     HAI with fluorodeoxyuridine plus dexamethasone was given on days
29 ransfected cells, respectively, also lead to fluorodeoxyuridine resistance (26- and 97-fold in IC50 v
30 etween UDG and duplex DNA containing 2'-beta-fluorodeoxyuridine shows similar increases in the level
31 3-Oxo-C12-(L)-HSL enhances the activity of 5-fluorodeoxyuridine, tomudex, and taxol but not the activ
32 uorodeoxyuridine monophosphate (FdUMP) and 5-fluorodeoxyuridine triphosphate (FdUTP).
33 HT29 and SW620 human colon cancer cells with fluorodeoxyuridine under conditions that produced nearly
34  selected marker and cells grown in 0.5 mM 5-fluorodeoxyuridine, we have recovered up to 50 transform
35 S was established through the use of [6-3H]5-fluorodeoxyuridine, which forms a tight ternary complex

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