ライフサイエンスコーパス: fluorosis

1 rosis) and enamel development (dental/enamel fluorosis).

2 MMP20 plays a nominal role in dental enamel fluorosis.

3 ession is involved in the etiology of dental fluorosis.

4 ression is not solely responsible for dental fluorosis.

5 ctional Mmp20 allele are more susceptible to fluorosis.

6 understanding of the biological mechanism of fluorosis.

7 as a model organism for the study of dental fluorosis.

8 ing that ER stress may play a role in dental fluorosis.

9 sis may also play a role in the mechanism of fluorosis.

10 train is least affected, with minimal dental fluorosis.

11 sceptibility or resistance to develop dental fluorosis.

12 etic component in the pathogenesis of dental fluorosis.

13 unding areas suffer from dental and skeletal fluorosis.

14 e of ready to feed infant formula and enamel fluorosis.

15 rall, only 8% of children showed evidence of fluorosis.

16 uorosis; and 3.7%, mild, moderate, or severe fluorosis.

17 Of 5,484 examined students, 71.8% had no fluorosis; 24.4%, questionable to very mild fluorosis; a

18 is a serious health hazard as it may lead to fluorosis, a serious bone disease.

19 been an increase in the prevalence of dental fluorosis, a side-effect of exposure to greater than opt

20 No associations between fluorosis and any OHRQoL scales met statistical or MID t

21 study were to determine the impact of enamel fluorosis and dental caries on oral health-related quali

22 association between mild-to-moderate enamel fluorosis and exposure during early childhood to fluorid

23 o disturbances of bone homeostasis (skeletal fluorosis) and enamel development (dental/enamel fluoros

24 fluorosis; 24.4%, questionable to very mild fluorosis; and 3.7%, mild, moderate, or severe fluorosis

25 sms that underlie the pathogenesis of dental fluorosis are not known.

26 he molecular mechanisms that underlie dental fluorosis are poorly understood.

27 mechanism or mechanisms resulting in dental fluorosis are unknown.

28 es) were analyzed for their association with fluorosis categories and sum of d2-3fs and D2-3MFS accor

29 rapid onset and severe development of dental fluorosis compared with that in the other strains tested

30 All students were examined for fluorosis (Dean's index) and caries experience (d2-3fs o

31 en developing incisors are most sensitive to fluorosis development.

32 en in the Birth Cohort through Cohort 3, and fluorosis for children in Cohorts 1 through 5.

33 experience negatively affects OHRQoL, while fluorosis has little impact.

34 this break on the development of caries and fluorosis in children.

35 studies of intra-oral distribution of dental fluorosis in low-fluoride areas reported that teeth that

36 l study investigated risk factors for enamel fluorosis in optimally fluoridated children, born after

37 lanation for the higher occurrence of dental fluorosis in posterior teeth.

38 occurrence of very mild or greater levels of fluorosis in the upper anterior teeth was 7 to 10% in th

39 In the fluoridated area, the occurrence of fluorosis increased from anterior to posterior teeth.

40 The severity of dental fluorosis is also dependent upon fluoride dose and the t

41 Dental fluorosis is caused by chronic high-level fluoride (F(-)

42 he break had little effect on caries, dental fluorosis is sensitive to even small changes in fluoride

43 association between mild-to-moderate enamel fluorosis on early forming (FRI classification I) enamel

44 association between mild-to-moderate enamel fluorosis on early forming (FRI classification I) enamel

45 formula was not found to be associated with fluorosis on either FRI classification I or II surfaces.

46 strong association between mild-to-moderate fluorosis on later forming (FRI classification II) ename

47 strong association between mild-to-moderate fluorosis on later forming (FRI classification II) ename

48 olars according to the decayed-filled index; fluorosis on the labial surfaces of the upper permanent

49 ive, but nonsignificant, association between fluorosis on these enamel surfaces and infant formula in

50 Fluorosis prevalence for seven age cohorts whose birth y

51 sm of how excessive fluoride exposure causes fluorosis remains uncertain.

52 The mechanism of how fluoride causes fluorosis remains unknown.

53 Case-control status was determined using the Fluorosis Risk Index (FRI).

54 s of a clinical dental examination using the Fluorosis Risk Index (FRI).

55 The fluorosis status of the subjects was determined on the b

56 strains of mice showed differences in dental fluorosis susceptibility/resistance.

57 ak, and those born 1 year after it, had less fluorosis than those aged 4-5 years at the break.

58 Overall prevalence of fluorosis was 44%.

59 Fluorosis was measured by the TF index, and parents or g

60 sess caries and the presence of sealants and fluorosis were performed on 2921 third-grade students in