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1 antially affected in the presence of cis-(Z)-flupentixol.
2 p-mediated drug transport by both isomers of flupentixol.
3 ent reward deliveries was also unaffected by flupentixol.
4    Rats were then injected systemically with flupentixol, a dopamine receptor antagonist, or vehicle
5                                       cis(Z)-Flupentixol, a modulator of Pgp function, preferentially
6 on of Pgp-mediated drug transport by cis-(Z)-flupentixol, a thioxanthene derivative, occurs through a
7 howed greater response suppression following flupentixol administration than rats experiencing no shi
8 mechanism by which the Pgp modulator cis-(Z)-flupentixol allosterically inhibits drug transport.
9 based allosteric modulators, such as cis-(Z)-flupentixol and its closely related analogs, induce rege
10 nthene-based Pgp modulators, such as cis-(Z)-flupentixol and its closely related analogues, effective
11 ibition of drug transport by both isomers of flupentixol and plays an important role in stimulation a
12 ibit P-GP and that the P-GP inhibitors trans-flupentixol and quinidine inhibit VMAT2.
13    In F983A the stimulatory effect of cis(Z)-flupentixol and the inhibitory effect of trans(E)-flupen
14 s accessible and responsive to modulation by flupentixol (and its closely related analogs), which can
15 ug transport by the cis and trans isomers of flupentixol are mediated through a common site of intera
16 such as cyclosporin A and verapamil, cis-(Z)-flupentixol does not interfere with substrate ([(125)I]i
17  dissociates susceptibility to inhibition by flupentixol from drug translocation, indicating the allo
18                      Interestingly, although flupentixol had a modest effect on the immediate respons
19                                Specifically, flupentixol had a stronger suppressive effect in group F
20             The allosteric modulator cis-(Z)-flupentixol has no effect on [alpha-(32)P]-8-azido-ATP b
21                             Finally, cis-(Z)-flupentixol has no effect on dissociation of [alpha-(32)
22                                         Oral flupentixol (HR, 0.92; 95% CI, 0.74-1.14), quetiapine (H
23 tant F983A that is impaired in modulation by flupentixols, indicating involvement of the allosteric m
24                                      cis-(Z)-Flupentixol-induced complex formation requires involveme
25  isomers of the dopamine receptor antagonist flupentixol inhibit drug transport and reverse drug resi
26 ion, indicating the allosteric nature of the flupentixol interaction.
27             Allosteric modulation by cis-(Z)-flupentixol involves a conformational change in Pgp dete
28 12 of Pgp that impairs inhibition by cis-(Z)-flupentixol of Pgp-mediated drug transport also affects
29 date-trapped Pgp shows protection by cis-(Z)-flupentixol of two Pgp fragments (approximately 60 kDa)
30 ntixol and the inhibitory effect of trans(E)-flupentixol on ATP hydrolysis and [125I]IAAP labeling we
31 the nonspecific dopamine receptor antagonist flupentixol on response invigoration and action bias in
32  MPTP and a P-GP inhibitor (quinidine, trans-flupentixol or cyclosporine A), the elimination of MPP(+
33 croinjections of the dopamine antagonist cis-flupentixol or the cholinergic antagonist atropine into
34 n of either the dopamine receptor antagonist flupentixol or the nicotinic receptor antagonist mecamyl
35 s injection of either a dopamine antagonist (flupentixol) or an opioid antagonist (naloxone) into the
36                       At test, we found that flupentixol pretreatment blocked the response invigorati
37  [125I]IAAP labeling by cis(Z)- and trans(E)-flupentixol, respectively.
38                            A second round of flupentixol tests was conducted using the rats' maintena
39 , which is impaired in modulation by cis-(Z)-flupentixol, the modulator has a minimal effect on subst
40 o shift in cost (group FR-10/FR-10), whereas flupentixol treatment had no effect on rats experiencing
41  C-site was increased 5- to 6-fold by cis(Z)-flupentixol without any effect on the N-site.

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