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1 antially affected in the presence of cis-(Z)-flupentixol.
2 p-mediated drug transport by both isomers of flupentixol.
3 ent reward deliveries was also unaffected by flupentixol.
6 on of Pgp-mediated drug transport by cis-(Z)-flupentixol, a thioxanthene derivative, occurs through a
7 howed greater response suppression following flupentixol administration than rats experiencing no shi
9 based allosteric modulators, such as cis-(Z)-flupentixol and its closely related analogs, induce rege
10 nthene-based Pgp modulators, such as cis-(Z)-flupentixol and its closely related analogues, effective
11 ibition of drug transport by both isomers of flupentixol and plays an important role in stimulation a
13 In F983A the stimulatory effect of cis(Z)-flupentixol and the inhibitory effect of trans(E)-flupen
14 s accessible and responsive to modulation by flupentixol (and its closely related analogs), which can
15 ug transport by the cis and trans isomers of flupentixol are mediated through a common site of intera
16 such as cyclosporin A and verapamil, cis-(Z)-flupentixol does not interfere with substrate ([(125)I]i
17 dissociates susceptibility to inhibition by flupentixol from drug translocation, indicating the allo
23 tant F983A that is impaired in modulation by flupentixols, indicating involvement of the allosteric m
25 isomers of the dopamine receptor antagonist flupentixol inhibit drug transport and reverse drug resi
28 12 of Pgp that impairs inhibition by cis-(Z)-flupentixol of Pgp-mediated drug transport also affects
29 date-trapped Pgp shows protection by cis-(Z)-flupentixol of two Pgp fragments (approximately 60 kDa)
30 ntixol and the inhibitory effect of trans(E)-flupentixol on ATP hydrolysis and [125I]IAAP labeling we
31 the nonspecific dopamine receptor antagonist flupentixol on response invigoration and action bias in
32 MPTP and a P-GP inhibitor (quinidine, trans-flupentixol or cyclosporine A), the elimination of MPP(+
33 croinjections of the dopamine antagonist cis-flupentixol or the cholinergic antagonist atropine into
34 n of either the dopamine receptor antagonist flupentixol or the nicotinic receptor antagonist mecamyl
35 s injection of either a dopamine antagonist (flupentixol) or an opioid antagonist (naloxone) into the
39 , which is impaired in modulation by cis-(Z)-flupentixol, the modulator has a minimal effect on subst
40 o shift in cost (group FR-10/FR-10), whereas flupentixol treatment had no effect on rats experiencing
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