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1 tionally had schizencephaly, heterotopia, or focal cortical dysplasia.
2 euronal organization consistent with type Ia focal cortical dysplasia.
3 ing, pathological aging, and the presence of focal cortical dysplasia.
4 ay play a causal role in the pathogenesis of focal cortical dysplasias.
6 encephaly and intractable epilepsy, one with focal cortical dysplasia and intractable epilepsy, and o
7 s name is linked to the first description of focal cortical dysplasia and limbic encephalitis, the pa
9 e beneficial, particularly for patients with focal cortical dysplasia and unilateral hemispheric malf
10 ons about the prevalence of HPV infection in focal cortical dysplasias and about its potential import
11 l elements, including hemimegalencephaly and focal cortical dysplasia, are common causes of intractab
12 lish megalencephaly, hemimegalencephaly, and focal cortical dysplasia as part of a single pathogenic
15 ormations of cortical development, including focal cortical dysplasia (FCD) and hemimegalencephaly (H
17 ical and ultrastructural patterns in type II focal cortical dysplasia (FCD) have been studied to expl
21 ptom of tuberous sclerosis complex (TSC) and focal cortical dysplasia (FCD) is early life refractory
25 complex (TSC)-associated cortical tubers and focal cortical dysplasia (FCD), we performed qualitative
29 neuroepithelial tumours (DNTs; eight cases), focal cortical dysplasia (FCD; 14 cases) and hippocampal
30 immunohistochemistry in the core of type II focal cortical dysplasias (FCD-II), at the FCD boundary
34 s of the complex structural abnormalities of focal cortical dysplasia have been proposed - from Taylo
36 hemimegalencephaly, two polymicrogyria, two focal cortical dysplasia, one diffuse cortical dysplasia
37 ession suggests that new cells may arrive in focal cortical dysplasia, perhaps deriving in part from
38 some of the most common or enlightening MCD: focal cortical dysplasia, periventricular heterotopia, p
39 tal retardation and seizures associated with focal cortical dysplasias, subependymal nodules, and sub
40 dictor for surgical outcome in patients with focal cortical dysplasia than the presence of a lesion o
41 astic megalencephaly, hemimegalencephaly and focal cortical dysplasia, the most common cause of intra
42 and astrocytes of all hemimegalencephaly and focal cortical dysplasia type II specimens, regardless o
48 of the association between HPV infection and focal cortical dysplasia type IIb, and its reproducibili
49 high proportion of pathological specimens of focal cortical dysplasia type IIb, but not in control sp
50 istopathological diagnosis among adults, and focal cortical dysplasia was the most common diagnosis a
51 formations of cortical development in 19.8% (focal cortical dysplasia was the most common type, 52.7%
52 of cortical dysplasia are recognized.Type I focal cortical dysplasia with mild symptomatic expressio
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