ライフサイエンスコーパス: focal nodular hyperplasia

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1 sion levels in H-HCA, IHCA, b-HCA, UHCA, and focal nodular hyperplasia.

2 recursor, hepatic adenoma, but not in benign focal nodular hyperplasia.

3 atients (42.2%) had adenomas, 29 (19.7%) had focal nodular hyperplasia, 25 (17.0%) had hemangiomas, 1

4 Significant differences were seen between focal nodular hyperplasia and all other lesion types (P

5 This finding completely separated focal nodular hyperplasia and malignancies.

6 necrosis, in alcoholic liver disease, and in focal nodular hyperplasia are compared with liver progen

7 s encompassed cysts (70), hemangiomata (37), focal nodular hyperplasia (FNH) (23), adenomata (47), an

8 Focal nodular hyperplasia (FNH) and hepatocellular adeno

9 Nonoperative management of focal nodular hyperplasia (FNH) is an accepted paradigm

10 Focal nodular hyperplasia (FNH), hepatocellular adenoma

11 ed with nodular regenerative hyperplasia and focal nodular hyperplasia (FNH), which finally evolved t

12 radiologically indeterminate for adenoma or focal nodular hyperplasia (FNH): (1) continue to observe

13 (CCC), hepatocellular carcinomas (HCC), and focal nodular hyperplasias (FNH).

14 Focal nodular hyperplasia had stimulated acoustic emissi

15 nal uptake of SH U 508A is characteristic of focal nodular hyperplasia, is seen in some hemangiomas,

16 cclusive thrombosis, and lack of visibility; focal nodular hyperplasia-like nodules (six [14%] of 42

17 inoma (n = 6), cavernous hemangioma (n = 4), focal nodular hyperplasia (n = 2), hamartoma (n = 1), an

18 liver disease (n = 9), hydatid cyst (n = 6), focal nodular hyperplasia (n = 3), and adenoma (n = 9).

19 ellular carcinoma, n = 4; hemangioma, n = 9; focal nodular hyperplasia, n = 7) received 2.5 g SH U 50

20 Recent evidence suggests that focal nodular hyperplasia of the liver (FNH) may represe

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