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1  found that PL projects to caudal cerebellar folia.
2 tical areas and underneath the summit of its folia.
3  of Purkinje cells distributed over multiple folia.
4 ked to variation in the number of cerebellar folia.
5 orming distinct parasagittal bands in caudal folia.
6 ized lamination and an absence of cerebellar folia.
7                        In the pars anterior (folia 1-3) of the paramedian lobule, the projection to t
8                       In the pars posterior (folia 4-6), cells targeting the two zones arose from lar
9 ng fibers that project to the uvula-nodulus (folia 8-10).
10 pontaneous discharge of SSs in contralateral folia 8-10, but blocked their modulation during vestibul
11 rded from Purkinje cells and interneurons in folia 8-10, identified by juxtacellular labeling with Ne
12 limbing fiber signaling to the contralateral folia 8-10, while leaving intact vestibular primary and
13 e cell layers with the densest AMGBi were in folia adjacent to the fourth ventricle.
14 ellum with disrupted layers in poorly formed folia and strikingly reduced granule cell production.
15 usion of cerebral hemispheres and cerebellar folia, and aberrant migration of granule cells.
16 eometry and size of clones in long and short folia are distinct.
17  at P3 in caudal lobules and extended to all folia by P9.
18                         Cerebellar deficient folia (cdf) is a recessive mouse mutation causing ataxia
19 Mice homozygous for the cerebellar deficient folia (cdf) mutation are ataxic and have cerebellar hypo
20 eover, in engrailed 1/2 mutants with shorter folia, clone cell number and geometry are most similar t
21 at both En genes are required to ensure that folia exclusive to the vermis or hemispheres form in the
22 rkinje cell development and the formation of folia in the cerebellum.
23  have defects in the formation of lamina and folia in the cerebral and cerebellar cortices that are c
24 ed for appropriate development of cerebellar folia layering and structure.
25 stretches of Purkinje cell bodies in various folia of cerebellum.
26 erograde tracers were microinjected into the folia of crus I of the cat cerebellum to investigate spa
27 r an area of elevated density in the ventral folia of lobules V and VI.
28 d tactile cerebellar map within the crus IIa folia of the cerebellar hemispheres reorganizes after de
29 ellar cortical c1 and c2 zones within apical folia of the forelimb-receiving area of the rostral para
30 obule V of the anterior lobe and the rostral folia of the paramedian lobule (PML) in the posterior lo
31                                 In the lower folia of the uvula and nodulus, Purkinje cell expression
32 geometry are most similar to clones in short folia of wild-type mice.
33 atrophic cerebellar hemispheres with stunted folia, profound granule cell depletion, Bergmann gliosis
34 in structure composed of an elaborate set of folia separated by fissures of different lengths, remain
35 rcm(tg) homozygotes is smaller and has fewer folia than in the wild-type, ectopic cerebellar cells ar
36 bellum consists of a highly organized set of folia that are largely generated postnatally during expa
37 length of each zone located within different folia were also shown to relate to different groups of o
38                                          The folia were shown to be mainly occupied in rostrocaudal s

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