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1 cial function of BG in organizing cerebellar foliation.
2 e cell progenitors (GCP) regulate cerebellar foliation.
3 of lobules that arises during the process of foliation.
4 required in Bergmann glia during cerebellar foliation.
5 rved normal cerebellar anlage patterning and foliation.
6 tionally required to pattern anterior vermis foliation.
7 duced in size and exhibits defects in vermal foliation.
8 kness of the granule cell layer and abnormal foliation.
9 ebrinII/AldolaseC) independent of regulating foliation.
10 itical for patterning PC gene expression and foliation.
11 ting in smaller cerebella that are devoid of foliation.
12 signaling regulates the extent of cerebellar foliation.
13 escues GNP cell proliferation and cerebellar foliation.
14 edial posterior transverse migrations during foliation.
15 ion correlates spatially and temporally with foliation.
16 was associated with reduction in cerebellar foliation.
17 n in vivo, and tested whether they influence foliation.
18 displayed an abnormal pattern of cerebellar foliation.
19 in the cerebellum, which is small and lacks foliation.
20 ck cortical laminar structure and cerebellar foliation.
22 enes caused severe cerebellar hypoplasia and foliation abnormalities, primarily attributable to a fai
24 Furthermore, En1/En2 continue to regulate foliation after embryonic day 14, at which time Fgf8 ist
25 n anterior region in which there is abnormal foliation, agranularity, and Purkinje cell (PC) ectopia.
26 entation of olivine crystals defining a weak foliation and a well-defined lattice-preferred orientati
30 ormal neuronal migration, lack of cerebellar foliation, and chromatolytic changes of neurons in the b
32 gmann glia-basement membrane adhesion during foliation, and provide new insights into the signaling p
33 uclei; produced defects in the patterning of foliation; and also affected litter size, body weight, a
36 ll mice, which include attenuated cerebellar foliation as well as defective radial migration and incr
38 ome of which have a strong layering-parallel foliation, confirm a long-held belief that such rocks ar
39 General deficiency of CerS1 in mice caused a foliation defect, progressive shrinkage, and neuronal ap
40 c4 in mice produces both cerebellar size and foliation defects similar to human DWM, confirming a req
42 (Atoh1(S193A/lacZ)) exhibit mild cerebellar foliation defects, motor impairments, partial pontine nu
46 ocampal abnormalities, absence of cerebellar foliation, disorganization of cortical neurons, and leth
48 in vitro, it is possible that Shh influences foliation during cerebellum development by regulating th
50 ly restricted gene expression occur prior to foliation in mutants, and foliation is altered from the
51 c features such as compositional banding and foliation in rocks that are reacting or dissolving while
54 of cortical layers and absence of cerebellar foliation in these animals precisely mimic the phenotype
55 ion occur prior to foliation in mutants, and foliation is altered from the onset and is accompanied b
56 receptor smoothened, we found the extent of foliation is gradually reduced, and that this correlates
58 more, cerebellar cortical development (size, foliation, layering, cell number, and position), which p
59 arkably, in En1/2 mutants with almost normal foliation, ML molecular code patterning is severely disr
61 d in the disruption of the normal process of foliation of the cerebellar cortex and the alteration of
64 nitors leads to strong defects in growth and foliation owing to its crucial role in the differentiati
65 m and spinal cord tracts; reduced cerebellar foliation, patchy loss of Purkinje cells, multifocal thi
66 s in cerebellar growth and the rostro-caudal foliation pattern reflect a reduced granule neuron popul
67 rmal domain does not grossly alter the basic foliation pattern, but does lead to prolonged proliferat
73 reveal that the abnormal anterior cerebellar foliation results from local disruptions of the basement
74 - cerebellum revealed gross abnormalities in foliation; specifically, lobule VII in the anterior verm
75 hat are fundamental to patterning cerebellum foliation throughout the mediolateral axis and that act
77 ernal granule layer thickness and defects in foliation were seen in embryonic and new-born LGI1 knock
78 e during late embryogenesis, at the onset of foliation, when ric-8a mutant Bergmann glia fail to main
79 sible for regional differences in cerebellum foliation, which interestingly are accompanied by region
80 expansion, resulting in abnormal post-natal foliation, while deregulated transcriptional programs co
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