ライフサイエンスコーパス: follicular

1 Additionally, follicular adenoma, a benign subtype of thyroid neoplasm

2 typic and functional characteristics of both follicular and blood Tfh cells and of the IL-21/IL-21R s

3 nificantly blocks the effect of epilation on follicular and epidermal melanocyte regeneration as well

4 at after epilation of mice, McSCs regenerate follicular and epidermal melanocytes, resulting in skin

5 e, a significantly reduced frequency of both follicular and extrafollicular FOXP3(hi) CD4 T cells was

6 on with rituximab and idelalisib in relapsed follicular and mantle cell lymphoma.

7 re, which was comparable to that seen in old follicular and marginal zone B cell subsets.

8 ng B cells with up-regulated Nod1, including follicular and marginal zone B cells with natural autore

9 However, in contrast to the follicular and marginal zone cells, ABCs displayed signi

10 By calculating the surface area of follicular and the interfollicular epithelial surface it

11 NA, 5 oncocytic/Hurthle cell, 2 medullary, 1 follicular, and 4 metastases from underlying malignancy)

12 r regions, Ag production and presentation by follicular APCs represent a unique mechanism to secure e

13 balance between ovarian folliculogenesis and follicular atresia is critical for female fertility and

14 Moreover, increased apoptosis of GCs and follicular atresia, reduced ovulation rate, and a dramat

15 onal B cells become marginal zone B (MZB) or follicular B (FoB) cells in the spleen, but it is unknow

16 lts in increased marginal zone and decreased follicular B cell numbers.

17 LF2 and IRF8, which are known to enforce the follicular B cell phenotype.

18 efold and a proportional decrease in splenic follicular B cells (CD21/35(int)CD23(+)) at 1, 2, and 12

19 on-induced proliferation, differentiation of follicular B cells during germinal center formation, and

20 ne-expression pattern associated with mature follicular B cells) and also attained increased cytosine

21 resulting in the accumulation of peripheral follicular B cells.

22 brane-bound Ab, yet harbor marginal zone and follicular B cells.

23 We found that Id3 expression is high in follicular B lineage cells but declines in GC cells.

24 As a major mediator of follicular B-cell migration, the G protein-coupled recep

25 follicle appeared to occur independently of follicular bulb bioenergetics by a tractor mechanism inv

26 cated by reduced germinal centers reactions, follicular CD4 T cells, and production of class-switched

27 To gain insights into follicular CD8(+) T cell function, we characterized foll

28 sex determination and differentiation of the follicular cell progenitors, but is downregulated after

29 argets are key integrators of antibody and T follicular cell responses.

30 r TSH/TSHR-mediated proliferation of thyroid follicular cells and biosynthesis of thyroid hormone.

31 ne biosynthesis and proliferation of thyroid follicular cells and uncovers a mechanism by which GLIS3

32 vulated oocytes degenerated, the surrounding follicular cells did not begin atresia.

33 ape of thyroid cancers that are derived from follicular cells has been substantially elucidated throu

34 ell proliferation of GLIS3-deficient thyroid follicular cells was due to the inhibition of TSH-mediat

35 aRKO) exhibited a significant expansion of T follicular cells, as well as increases in TFH to TFR rat

36 1R interactions influence the phenotype of T follicular cells, reducing the expression of CXCR4 and i

37 suppressed plasma FT4 and TT4, and depleted follicular colloid and increased epithelial cell height

38 th-dependent corneocyte diffusivity; and the follicular contribution has significance for low transbi

39 Although similar follicular defects are found in Vegfa knockout (Vegfa(KO

40 rs tend to acquire the most antigen from the follicular dendritic cell (FDC) network and present the

41 tain prion strains upon stromal cell-derived follicular dendritic cells (FDC) in the Peyer's patches

42 In the host response to foreign antigens, follicular dendritic cells (FDCs) maintain GCs through t

43 We show that follicular dendritic cells are stiff cells that promote

44 eatments which prevent prions from infecting follicular dendritic cells can block their spread to the

45 Antigen is provided on follicular dendritic cells in the form of immune complex

46 xpression on the metastatic cells and within follicular dendritic cells of the metastasis-infiltrated

47 to facilitate their initial delivery toward follicular dendritic cells to establish host infection.

48 How the prions are first delivered to follicular dendritic cells to establish infection was un

49 ese mice the early accumulation of prions on follicular dendritic cells was impaired and oral prion d

50 gut to the brain, they first replicate upon follicular dendritic cells within intestinal Peyer's pat

51 ystem, intranodal prion replication by B and follicular dendritic cells, and potential prion strain s

52 Here, BTV infects and disrupts follicular dendritic cells, hindering B-cell division in

53 , including fibroblastic reticular cells and follicular dendritic cells.

54 endritic cells were unable to migrate toward follicular dendritic cells.

55 rily presented in its membrane-bound form by follicular dendritic cells.

56 plays a critical role in the balance between follicular development and atresia.

57 GS21680, following immunization suppressed T follicular differentiation, GC B cell frequency, and cla

58 Then, asymmetry with depth and follicular diffusion are studied using caffeine as an il

59 cts of heterogeneity, anisotropy, asymmetry, follicular diffusion, and location of the main barrier o

60 suppurativa (HS) is a recurrent inflammatory follicular disease that commonly affects the apocrine-be

61 Trials were in early-follicular (EF) and mid-luteal (ML) phases in dry (DRY)

62 s in the matrix that are differentiated from follicular epithelial cells expressing transcription fac

63 We established primary cultures from follicular epithelium in human homeostatic conditions (h

64 Studying the Drosophila follicular epithelium, we show that the cadherin Fat2 an

65 IL7 concentration in the follicular fluid correlates with the oocyte ability to r

66 ched when IL7 secretion is measured in human follicular fluid during in vitro fertilization cycles.

67 Here, using follicular fluid from patients with the disease endometr

68 n the homeostasis and function of peripheral follicular (Fo) B cells.

69 associated with CD4(+) T cells (including Th follicular functionality in lymphoid tissues and Th2 res

70 izing selection as the best model describing follicular gland diversification, and revealed high rate

71 genetic patterns of diversification of their follicular glands for chemical communication.

72 and that the crown squamate ancestor lacked follicular glands, which therefore originated and divers

73 Follicle-stimulating hormone (FSH) regulates follicular growth and stimulates estrogen synthesis in t

74 is dependent upon numerous factors mediating follicular growth, vascularization, and ultimately oocyt

75 t of GC B cells and germinal center CD4(+) T follicular helper (GC Tfh) cells problematic.

76 f CD4 T cell subsets, including peripheral T follicular helper (pTfh) cells, which provide help to B

77 s linked to the development of both CD4(+) T follicular helper (TFH) and central memory T (TCM) cells

78 -LSQ) was especially effective at inducing T follicular helper (Tfh) and LLPC responses to Pfs25 when

79 T follicular helper (Tfh) and Th17 cells are known to prom

80 tory T (Treg) cell responses, but inhibits T follicular helper (TFH) cell development.

81 te autoimmunity.Excessive expansion of the T follicular helper (TFH) cell pool is associated with aut

82 hat is required for the differentiation of T follicular helper (TFH) cell populations.

83 Cognate interactions between T follicular helper (Tfh) cells and B cells are essential

84 rLBNSE-IL-7 induced higher rates of T follicular helper (Tfh) cells and germinal center (GC) B

85 on in HIV bnAbs led to the hypothesis that T follicular helper (Tfh) cells and germinal centers (GC)

86 T follicular helper (TFH) cells and infiltrating stromal c

87 subset of regulatory T cells that suppress T follicular helper (Tfh) cells and the generation of high

88 Recent evidence suggests that T follicular helper (Tfh) cells are the primary producer o

89 t, activated ICOS(hi) PD-1(hi) circulating T follicular helper (Tfh) cells decreased after rituximab

90 T follicular helper (TFH) cells have been shown to be crit

91 es in the regulation of humoral responses: T follicular helper (Tfh) cells support germinal center fo

92 s engage in long-lasting interactions with T follicular helper (Tfh) cells, a process that depends on

93 forms were required for differentiation of T follicular helper (TFH) cells, but not TH1 effectors, el

94 gnature that was related to that of CD4(+) T follicular helper (TFH) cells, CD8(+) T cell memory prec

95 Similar to CD4(+) T follicular helper (Tfh) cells, IL-21-producing CD8(+) T

96 as increased on CD4(+) T cells, especially T follicular helper (Tfh) cells, in HIV-infected lymph nod

97 ion, an essential transcription factor for T follicular helper (TFH) cells, is critical as aberrant T

98 th the differentiation and accumulation of T follicular helper (TFH) cells.

99 f viral reservoirs in germinal center (GC) T follicular helper (Tfh) cells.

100 ma activation and increased recruitment of T follicular helper (TFH) cells.

101 n, where balanced CD4 T helper 1 (Th1) and T follicular helper (Tfh) responses were induced, Ad5 immu

102 characterization of human Th1, Th2, Th17, T follicular helper (Tfh), Treg, and Tr1 cells has helped

103 IL-17 and limited IL-2 production, whereas T follicular helper and double negative (DN) T cells signi

104 n particular Langerhans cells at governing T follicular helper and germinal center formation after in

105 ted with a significantly higher frequency of follicular helper CD4 T cells compared with the unvaccin

106 Infected follicular helper CD4 T cells, TFH, present inside B-cel

107 Follicular helper CD4 T cells, TFH, residing in B-cell f

108 ampened splenic germinal center B-cell and T-follicular helper cell frequencies that collaborate to p

109 ns enhanced lymph node germinal center and T follicular helper cell responses.

110 Germinal center T follicular helper cells (GCTfh) in lymphatic tissue are

111 T follicular helper cells (odds ratio [OR] = 1.34, 95% CI

112 Limited data suggest that a subset of T follicular helper cells (TFH) within germinal centers (G

113 severity may be related to development of T follicular helper cells and antiviral inflammatory seque

114 ated with KD025 had decreased frequency of T follicular helper cells and increased frequency of T fol

115 tion of IL-6 is critical for initiation of T follicular helper cells and production of high-affinity

116 s found between the frequency of tonsillar T follicular helper cells and tonsillar Ag-specific Ab-sec

117 necessitates further understanding of how T follicular helper cells are regulated in health and dise

118 T follicular helper cells contribute to the development of

119 As such, T follicular helper cells impact immunodeficiencies, autoi

120 referential accumulation of CXCR3(+)CD4(+) T follicular helper cells in the spleen and enhanced Ab re

121 cally associated with germinal centers and T follicular helper cells were examined using immunohistoc

122 Signals delivered by T follicular helper cells were not required to initiate di

123 However, CGS did abrogate germinal center T follicular helper cells, and blunted PE-specific germina

124 The IL-23R(-/-)MRL.lpr had fewer T follicular helper cells, B cells, and plasma cells, lead

125 ighlight differences between TPH cells and T follicular helper cells, including altered expression of

126 Like PD-1(hi)CXCR5(+) T follicular helper cells, TPH cells induce plasma cell di

127 a critical interaction between B cells and T follicular helper cells.

128 t-PBio treatment, which correlated with LN T follicular helper cells.

129 but it impairs the development of Th17 and T follicular helper cells.

130 This results in severe defects in T follicular helper development and TH2 polarization, as s

131 expression and rescues, at least in part, T follicular helper numbers and the abnormal Ab production

132 nflammatory T-helper 17/T-cytotoxic 17 and T-follicular helper paradigms with consequent thymic damag

133 tically increased frequencies of circulating follicular helper T (cTFH) cells.

134 It was found that LBNSE-CXCL13 recruited follicular helper T (Tfh) and germinal center (GC) B cel

135 Functional analyses included in vitro follicular helper T (TFH) cell differentiation and cTFH/

136 errantly upregulated genes characteristic of follicular helper T (TFH) cell lineage, including Bcl6,

137 TFR cells were previously included in the follicular helper T (TFH) cell subset, which consists of

138 Recent work identified PD-1(+) follicular helper T (Tfh) cells as an important cellular

139 Abnormal development of follicular helper T (TFH) cells can induce the GC respon

140 Within secondary lymphoid follicles, follicular helper T (TFH) cells have previously been sho

141 se is based upon the repertoire of different follicular helper T (Tfh) cells in germinal centers.

142 ying somatically mutated B-cell receptors by follicular helper T (TFH) cells in germinal centres.

143 f intricate interactions between B cells and follicular helper T (Tfh) cells occurring in lymphoid ge

144 Follicular helper T (Tfh) cells promote germinal center

145 Follicular helper T (TFH) cells support terminal B-cell

146 A large number of follicular helper T (Tfh) cells were also detected in dr

147 A new T-cell subset, follicular helper T (TFH) cells, is specialized in suppo

148 ugh this process is known to be regulated by follicular helper T (TfH) cells, the mechanism by which

149 ells and substantially impaired formation of follicular helper T (Tfh) cells, which are essential for

150 t the highest density of cognate peptides to follicular helper T (Tfh) cells, which provide survival

151 iated inhibitory circuit that stabilizes the follicular helper T cell program at least in part throug

152 ulatory T cell (Treg) subset that suppresses follicular helper T cell-mediated B cell responses in th

153 Follicular helper T cells (Tfh ) are located within germ

154 Aberrant population expansion of follicular helper T cells (TFH cells) occurs in patients

155 unodeficiency virus (HIV), which persists in follicular helper T cells (TFH cells), and Epstein-Barr

156 Follicular helper T cells (Tfh) are essential for B cell

157 Follicular helper T cells (TFHs) are a key component of

158 IFN-regulated genes, increased the number of follicular helper T cells and plasmablasts in the spleen

159 al CD4 memory T cells and lymph node-derived follicular helper T cells of patients with CVID compared

160 on DCs in the generation of antigen-specific follicular helper T cells, antigen-specific GC B cells,

161 ID with secondary complications and a skewed follicular helper T-cell differentiation in defined mono

162 ssing cells in DLNs acquired an alternative, follicular helper-like fate.

163 ility to expand and become germinal center T follicular helpers and enhances B cell IgG Ab production

164 revealed that the histopathologic feature of follicular hyperkeratosis distinguished these 6 patients

165 e, we observed PrP(CWD) seeding activity and follicular immunoreactivity in oropharyngeal lymphoid ti

166 iphoton microscopy showed that it opened the follicular infundibula (p<0.001).

167 res, and (2) round, large pagetoid cells and follicular localization of atypical cells, respectively.

168 cular openings by dermoscopy correlated with follicular localization of pagetoid cells by RCM (kappa

169 ovaries develop through all stages; however, follicular loss accumulated with age and unfertilized me

170 95% CI, 18 to 71) and 10 of 14 patients with follicular lymphoma (71%; 95% CI, 42 to 92).

171 mantle cell lymphoma (A051201) and relapsed follicular lymphoma (A051202).

172 oma (EMZL) (68.4% [180 of 263]), followed by follicular lymphoma (FL) (16.3% [43 of 263]), mantle cel

173 arginal-zone lymphoma (EMZL) (37% [n = 32]), follicular lymphoma (FL) (23% [n = 20]), diffuse large B

174 BBP is targeted by inactivating mutations in follicular lymphoma (FL) and diffuse large B-cell lympho

175 follicular lymphoma (PTNFL) is a variant of follicular lymphoma (FL) characterized by limited-stage

176 Most patients with follicular lymphoma (FL) experience multiple relapses ne

177 Follicular lymphoma (FL) is a clinically and molecularly

178 Follicular lymphoma (FL) is a clinically and molecularly

179 Purpose Follicular lymphoma (FL) is an indolent cancer, with eff

180 Follicular lymphoma (FL) is an indolent malignancy of ge

181 Follicular lymphoma (FL) is the most common form of indo

182 Follicular lymphoma (FL) is the most common form of indo

183 Follicular lymphoma (FL) is the most frequent indolent l

184 s a critical event in the clinical course of follicular lymphoma (FL) patients.

185 e follicular lymphoma (PTFL) is a variant of follicular lymphoma (FL) with distinctive clinicopatholo

186 ial therapy for patients with advanced-stage follicular lymphoma (FL).

187 c lymphocytic leukemia (CLL; B-cell CLL) and follicular lymphoma (FL).

188 ently identified many new genetic lesions in follicular lymphoma (FL).

189 included mantle cell lymphoma (MCL; n = 28), follicular lymphoma (FL; n = 29), diffuse large B-cell l

190 Pediatric-type follicular lymphoma (PTFL) is a B-cell lymphoma with dis

191 Pediatric-type follicular lymphoma (PTFL) is a variant of follicular ly

192 Pediatric-type nodal follicular lymphoma (PTNFL) is a variant of follicular l

193 ents with histologically documented relapsed follicular lymphoma and time to progression 6 months or

194 aneous rituximab to intravenous rituximab in follicular lymphoma and to provide efficacy and safety d

195 ls of most measured signaling nodes, whereas follicular lymphoma cells represented the opposite patte

196 Randomisation was stratified by Follicular Lymphoma International Prognostic Index risk

197 s and was stratified by country, gender, and Follicular Lymphoma International Prognostic Index score

198 ion was stratified by selected chemotherapy, Follicular Lymphoma International Prognostic Index, and

199 erapy-treated patients was additional to the Follicular Lymphoma International Prognostic Index.

200 Chemoimmunotherapy in follicular lymphoma is associated with significant toxic

201 Minor changes (2.3%) mostly consisted of follicular lymphoma misgrading and diffuse large B-cell

202 , advanced stage (Ann Arbor stage III or IV) follicular lymphoma of WHO histological grades 1, 2, or

203 slocations present in lymph node biopsies of follicular lymphoma patents.

204 Previously untreated patients with follicular lymphoma received rituximab 375 mg/m(2) on da

205 ted to be a reliable predictor of outcome in follicular lymphoma requiring treatment, and prospective

206 tients with diffuse large B-cell lymphoma or follicular lymphoma that had relapsed or was refractory

207 tients with diffuse large B-cell lymphoma or follicular lymphoma that is refractory to or that relaps

208 od for identifying bona fide contributors to follicular lymphoma transformation and may therefore gui

209 cific functional and genetic determinants of follicular lymphoma transformation remain elusive, and g

210 8 years or older with Ann Arbor stage III-IV follicular lymphoma were assigned 1:1 to CVP plus intrav

211 (95% CI, 33 to 98) and 89% of patients with follicular lymphoma who had a response (95% CI, 43 to 98

212 mediastinal B-cell lymphoma, or transformed follicular lymphoma who had refractory disease despite u

213 ree with mantle cell lymphoma and eight with follicular lymphoma) were enrolled.

214 A051201 (mantle cell lymphoma) and A051202 (follicular lymphoma) were phase 1 trials.

215 hter's transformation, mantle cell lymphoma, follicular lymphoma, and chronic lymphocytic leukemia, w

216 Risks of chronic lymphocytic leukemia, follicular lymphoma, and mantle cell lymphoma were 5%-10

217 s involving MYC in Burkitt lymphoma, BCL2 in follicular lymphoma, and MYC/BCL2/BCL6 in high-grade B-c

218 fuse large B-cell lymphoma, two patients had follicular lymphoma, and one patient had mantle cell lym

219 with rituximab-CVP (R-CVP) in patients with follicular lymphoma.

220 clinical trials in rheumatoid arthritis and follicular lymphoma.

221 rash (four [50%] of eight) for patients with follicular lymphoma.

222 patients with newly diagnosed advanced-stage follicular lymphoma.

223 ypes, in particular mantle cell lymphoma and follicular lymphoma.

224 subtype, and 1 family displayed early-onset follicular lymphoma.

225 refractory diffuse large B-cell lymphoma and follicular lymphoma.

226 patients with previously untreated advanced follicular lymphoma.

227 a new therapeutic option for advanced-stage follicular lymphoma.

228 y untreated, CD20-positive grade 1, 2, or 3a follicular lymphoma; Eastern Co-operative Oncology Group

229 e progression-free survival in patients with follicular lymphoma; however, the optimal duration of ma

230 e B-cell lymphoma vs one of 15 patients with follicular lymphoma; P = .047) and with advanced stage d

231 were diffuse large B-cell lymphomas (32.4%), follicular lymphomas (15.3%), classic Hodgkin lymphomas

232 mas (MZLs), 2 lymphoplasmacytic lymphomas, 2 follicular lymphomas, 4 CLL/small lymphocytic lymphomas

233 ing pathways to regulate genes necessary for follicular maturation.

234 ment of self-reactive specificities into the follicular mature compartment and thereby likely increas

235 ied a bias for selection of B cells into the follicular mature versus marginal zone B cell compartmen

236 However, the niche that regulates follicular melanocytes is not well characterized.

237 artly by creating an SCF-dependent niche for follicular melanocytes.

238 men, aged 18 to 35 y, were seen during their follicular menstrual phase.

239 Follicular nodular hyperplasia (FNH) is a common benign

240 fects the apocrine-bearing skin and involves follicular occlusion and hyperkeratosis.

241 Asymmetric pigmented follicular openings by dermoscopy correlated with follic

242 Using a 3D B cell follicular organoid system that mimics the GC reaction,

243 The follicular pathway gives rise to germinal centers (GCs)

244 menorrheic; HC and OW/OB were studied in the follicular phase.

245 Early-follicular-phase serum level of antimullerian hormone (A

246 es associated with LM or LMM: (1) asymmetric follicular pigmentation and targetlike structures, and (

247 ajor obstacles to successful recovery of the follicular pool within grafted ovarian tissue.

248 e spleen whereas TEFF cannot traffic through follicular regions, Ag production and presentation by fo

249 T follicular regulatory (Tfr) cells are a subset of regula

250 T follicular regulatory (Tfr) cells control the magnitude

251 e regulatory CD4(+) (Treg) cells including T follicular regulatory (Tfr) cells inhibit the germinal c

252 However, it is not clear how IL-2 affects T follicular regulatory (TFR) cells, a cell type with prop

253 ction, and also promotes immunosuppressive T follicular regulatory cells (TFR).

254 ar helper cells and increased frequency of T follicular regulatory cells, accompanied by a reduction

255 generation of both RORgammat(+) Tregs and T follicular regulatory cells, but not for adipose-residen

256 Follicular regulatory T (TFR) cells are a newly defined

257 Follicular regulatory T (TFR) cells are a subset of CD4(

258 secondary lymphoid follicles was described, follicular regulatory T (TFR) cells.

259 eg cells control TFH cell maturation, expand follicular regulatory T cells, and inhibit the TFH cell-

260 etions, and expanded Foxp3(+)CXCR5(+)PD-1(+) follicular regulatory T cells.

261 To investigate the follicular skin microbiome in patients with HS and in he

262 rovoke meiotic defects leading to a depleted follicular stock, as in Fancm(-/-) mice.

263 The chemokine CXCL13 is expressed by FDC and follicular stromal cells and modulates the homing of CXC

264 ar structures, and suggest that it was after follicular structures appeared that this cytokine evolve

265 Many of these microbes inhabit follicular structures of the skin.

266 stimulating IgM responses in the absence of follicular structures, and suggest that it was after fol

267 thyroid cancers compared with papillary and follicular subtypes (P < 0.0001).

268 n health through skin have ignored the inner follicular surface, and therefore vastly underestimated

269 We demonstrate a benefit to follicular survival and function in human ovarian tissue

270 Whole ovary vitrification provides better follicular survival compared to slow freezing and may be

271 how that depends on the stage of the ongoing follicular T cell and GC B cell response.

272 The realization that follicular T cells are heterogeneous, comprising helper

273 3 interacts with dendritic cells, Th2 cells, follicular T cells, and regulatory T cells, where IL-33

274 ith conventional T helper cell responses and follicular T helper (TFH)-B cell interactions in germina

275 affected patient displayed increased TH1 and follicular T helper cell and suppressed TH17 cell respon

276 ype I and II interferons, normalized TH1 and follicular T helper cell responses, improved TH17 differ

277 dependent on the function of CD4(+)CXCR5(+) follicular T helper cells (Tfh).

278 the umbrella of nodal T-cell lymphomas with follicular T helper phenotype.

279 lls (P < .01), and CD4+CXCR5+interleukin 21+ follicular T-helper (Tfh) cells (P < .01).

280 homa (AITL) and other lymphomas derived from follicular T-helper cells (TFH) represent a large propor

281 d microneutralisation assays and circulating follicular T-helper cells and plasmablast cells were mea

282 Follicular Th (Tfh) cells orchestrate physiological germ

283 ell activation and promotes the induction of follicular Th (TFH) cells, CD4(+) T cells that support B

284 inths, Batf(-/-) mice are unable to generate follicular Th and Th2 cells.

285 eckpoint protein ICOS on T cells and induced follicular Th cell differentiation.

286 rentiating within IL-17-secreting T cell and follicular Th cell paradigms to generate IL-21 and IL-17

287 Ag-specific follicular Th cell responses to adenovirus vectored vacc

288 serve as an immunophenotype capable of Th1, follicular Th, and CTL functionalities, yet they are una

289 nosis, no specific recommendations exist for follicular thyroid cancer and Hurthle cell carcinoma in

290 odide uptake in RAI-refractory papillary and follicular thyroid cancer cell lines.

291 Follicular thyroid cancer is the second most common diff

292 Despite certain unique aspects of follicular thyroid cancer presentation and prognosis, no

293 we comprehensively review the literature on follicular thyroid cancer to provide an evidence-based g

294 esmoplasia and inflammation in papillary and follicular thyroid cancers and the presence of multipote

295 on in gilts, the mRNA expression profiles of follicular tissue from Large White gilts in diestrus (LD

296 s a population-based surveillance survey for follicular trachoma (TF) and trachomatous trichiasis (TT

297 sociated with an increase in total Tregs and follicular Tregs (Tfr) that control the GC reaction, alo

298 illary carcinomas, with 2 of them exhibiting follicular variant.

299 lar CD8(+) T cell function, we characterized follicular virus-specific CD8(+) T cells in situ by usin

300 t interface and increased both viability and follicular volume in ExEC-assisted grafts with resumptio