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1 and five nuclear receptors in murine primary follicular cells.
2 Dally causes ectopic accumulation of Upd in follicular cells.
3 in mediating estrogen action in the thyroid follicular cells.
4 ferative and functional stimulus for thyroid follicular cells.
5 cal cells from prolactin-treated mice become follicular cells.
6 ere expressed almost entirely in surrounding follicular cells.
7 types of the hair follicle as well as upper follicular cells.
8 r significantly from that observed in normal follicular cells.
9 ress constitutively IFN-gamma in the thyroid follicular cells.
10 cells to mimic the iodide uptake of thyroid follicular cells.
11 rNIS-transduced tumor cells and rat thyroid follicular cells.
12 y relevant model, the differentiated thyroid follicular cells, a system that requires thyroid-stimula
13 function of this pathway in primary thyroid follicular cells and a papillary thyroid carcinoma cell
14 r TSH/TSHR-mediated proliferation of thyroid follicular cells and biosynthesis of thyroid hormone.
16 ng of additional subsets, including T helper follicular cells and IL-17-producing T helper cells.
17 c instability, we transfected FTC133 thyroid follicular cells and observed increased genetic instabil
21 ne biosynthesis and proliferation of thyroid follicular cells and uncovers a mechanism by which GLIS3
22 (BCL6) is required for the development of Th follicular cells, and it has been shown to suppress Th2
24 the chimeric RET/PTC oncoproteins in thyroid follicular cells, are frequently found in radiation-indu
25 aRKO) exhibited a significant expansion of T follicular cells, as well as increases in TFH to TFR rat
26 lasma membrane of ovarian follicular and non-follicular cells, binds to E and its expression is regul
28 transport at the apical membrane of thyroid follicular cells, but experimental evidence for this con
29 ine whether programmed cell death in thyroid follicular cells can be related to activation of the tum
31 b(PV), an established mouse model of thyroid follicular cell carcinogenesis, significantly increased
32 mouse strains may be due to differences in T follicular cell commitment or intrinsic B cell differenc
34 g been demonstrated in thyroid carcinomas of follicular cell derivation, but no consistent relationsh
40 (familial medullary thyroid cancer) or from follicular cells (familial nonmedullary thyroid cancer).
41 activation specifies three distinct anterior follicular cell fates, suggesting that Upd is a morphoge
42 ape of thyroid cancers that are derived from follicular cells has been substantially elucidated throu
46 thyroid carcinomas (BRAF(V600E)) in thyroid follicular cells in a doxycycline-inducible (dox-inducib
47 litates the apoptotic destruction of thyroid follicular cells in experimental autoimmune thyroiditis,
49 transported DA and cAMP produced by somatic follicular cells in response to NE-induced beta-adrenore
50 ter that is found at the luminal membrane of follicular cells in the thyroid gland as well as in the
52 e-tracing studies and showed an expansion of follicular cells into the interfollicular epidermis, as
53 onditional loss of Pten in the mouse thyroid follicular cells is sufficient to stimulate continuous a
55 lume and nuclear-to-cytoplasmic ratio of the follicular cells may provide important information about
56 on pattern; RASAL is highly expressed in the follicular cells of the thyroid and the adrenal medulla
57 Braf was appropriately activated in thyroid follicular cells of these mice, they had a lower mitotic
59 s either by directing the differentiation of follicular cells or assisting cells in interpreting a gr
60 somatic genetic changes in thyroid cancer of follicular cell origin (RET/PTC, NTRK, RAS, BRAF, PAX8-P
62 the tumor cell biology of thyroid cancer of follicular cell origin has improved and modern genomic t
65 sex determination and differentiation of the follicular cell progenitors, but is downregulated after
67 1R interactions influence the phenotype of T follicular cells, reducing the expression of CXCR4 and i
70 lts revealed that 6 had a paucity of thyroid follicular cells, suggesting insufficient sampling of th
71 hways, alone, is unable to transform thyroid follicular cells, their simultaneous activation is highl
72 with thyroiditis, and unexpectedly, thyroid follicular cells themselves could be induced to express
74 use diminished export of iodide from thyroid follicular cells to the colloid and are associated with
75 the thyroid that could interact with thyroid follicular cell TRAIL receptors, RNase protection assays
76 he use of agents to improve iodine uptake in follicular cell tumors, are in early clinical investigat
78 ell proliferation of GLIS3-deficient thyroid follicular cells was due to the inhibition of TSH-mediat
79 Furthermore, the number of apoptotic thyroid follicular cells was increased only in the thyroids from
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