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1 menorrheic; HC and OW/OB were studied in the follicular phase.
2 phase of the menstrual cycle compared to the follicular phase.
3 EA plasma concentrations obtained during the follicular phase.
4 eas only a twentieth expressed GAL-R1 in the follicular phase.
5 lation and increased activation during early follicular phase.
6 complement system) were overexpressed in the follicular phase.
7 ffects of estradiol, we have now divided the follicular phase.
8 P = 0.0002) in the luteal phase than in the follicular phase.
9 with the rate of vaginal transmission in the follicular phase.
10 aked (increased by 9%, P = 0.01) in the late follicular phase.
11 were measured in serum collected during the follicular phase.
12 eased length and variability of segments and follicular phases.
13 cular phase, 0.86 +/- 0.09 kJ/min during the follicular phase, 0.70 +/- 0.10 kJ/min during the luteal
14 cycle: 0.94 +/- 0.05 kJ/min during the early follicular phase, 0.86 +/- 0.09 kJ/min during the follic
15 At the time of reward delivery, women in the follicular phase activated the midbrain, striatum, and l
16 per quartile) with anovulation and increased follicular phase and decreased luteal phase lengths; His
17 E2beta-mediated elevations of UBF during the follicular phase and late pregnancy are partially mediat
18 infusion inhibited the elevated UBF seen in follicular phase and late pregnant ewes in a time-depend
19 t analysis was used to examine the effect of follicular phase and prandial state on brain activation
21 from the early follicular phase to the late follicular phase and then decreased again in the luteal
22 ol/L (from values of 138.8 mmol/L during the follicular phase) and increases in urinary sodium excret
25 ductive and psychiatric interviews and early follicular-phase blood specimens were obtained at study
26 me measures were acquired at baseline in the follicular phase (cycle day 6.6 +/- 2.2) and at follow-u
28 f pain intensity, men and women during their follicular phase differ in the magnitude and direction o
30 ter odds of having baseline endogenous early follicular phase estradiol concentrations in the lowest
33 s: short luteal phase (< or = 10 days), long follicular phase (> or = 24 days), long menses (> or = 8
34 on, short luteal phase (< or =10 days), long follicular phase (> or =24 days), long cycle (> or =36 d
35 olated during exacerbations occurring in the follicular phase (high estradiol), with a variable P. ae
36 reduction in cortical GABA levels during the follicular phase in those with PMDD compared with health
37 plasma estradiol level was 30% lower in the follicular phase in women with depression than in their
38 change in estradiol concentration across the follicular phase (late minus early) was inversely correl
39 gth was 28.8 (standard deviation, 4.4) days; follicular phase length, 16.0 (standard deviation, 4.4)
41 t associated with detrimental alterations in follicular-phase length, time to pregnancy, or early pre
42 f > or = 35 years with decreased segment and follicular phase lengths; heavier weight (upper quartile
44 ased the excretion of 16alphaOHE1 during the follicular phase of the first menstrual cycle and during
46 icant increase in MVF (about 10%) during the follicular phase of the menstrual cycle when oestrogen l
50 sustained exposure to estradiol in the late follicular phase of the reproductive cycle, however, the
52 pounds from the armpits of women in the late follicular phase of their menstrual cycles accelerated t
54 n 4 times after injection: in the luteal and follicular phases of 2 cycles in the placebo group and 2
55 ion were lower in the early than in the late follicular phase (P = 0.03) on the basis of regression a
57 ical target cells compared with those in the follicular phase (p=0.0488), suggesting that a naturally
58 mol/d (from values of 65.5 mmol/d during the follicular phase) preceded periods when menstrual sympto
63 atios increased significantly from the early follicular phase to the late follicular phase and then d
64 an increase in cortical GABA levels from the follicular phase to the mid luteal and late luteal phase
65 ell by a median of 0.16 log10 from the early follicular phase to the midluteal phase (P=.03, Wilcoxon
66 evels decreased from the nonsymptomatic, mid-follicular phase to the symptomatic, late luteal phase.
67 tact, cycling sheep were synchronized to the follicular phase using progesterone, prostaglandin F2alp
70 y, sensitivity tended to be lower during the follicular phase (week 1, 72.1%; week 2, 80.4%; week 3,
71 reovulation (average of early follicular and follicular phases), when it was 0.90 +/- 0.06 kJ/min.
72 ate than during fasting but only in the late follicular phase, when estradiol concentration was high.
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