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1 released by eEPCs into CM, were crucial for folliculogenesis.
2 is severely defective at multiple stages of folliculogenesis.
3 of GATA4 and FOG2 in ovarian development and folliculogenesis.
4 id they contribute to oocytes during de novo folliculogenesis.
5 re oocyte-specific and detectable throughout folliculogenesis.
6 n of Pou5f1 and Gdf9 in oocytes during early folliculogenesis.
7 e polyovular follicles, suggesting a role in folliculogenesis.
8 of gonad development-germ cell migration and folliculogenesis.
9 fringe-deficient ovary demonstrated aberrant folliculogenesis.
10 ling pathway and lunatic fringe in mammalian folliculogenesis.
11 a highly regulated expression pattern during folliculogenesis.
12 em most likely plays a pivotal role in early folliculogenesis.
13 early as one-layer follicles and throughout folliculogenesis.
14 hogenetic proteins (BMPs) in early stages of folliculogenesis.
15 ding how oocyte growth factors contribute to folliculogenesis.
16 atrix and the oolemma, that perturbed normal folliculogenesis.
17 not PP1gamma, in oocytes from all stages of folliculogenesis.
18 tor-9 (GDF-9), which is required for ovarian folliculogenesis.
19 n the ovary during oocyte cyst breakdown and folliculogenesis.
20 showed that reconstituted ovaries exhibited folliculogenesis after transplantation of PGCs-aggregate
21 promotes an ovarian environment defective in folliculogenesis and conducive to teratoma formation.
22 ound that Calr fcKO female mice had impaired folliculogenesis and decreased ovulatory rates due to de
23 ies have revealed key roles of the oocyte in folliculogenesis and established that bidirectional comm
26 Given the importance of FSH in regulating folliculogenesis and fertility, the development of FSH m
28 epithelial basement membrane is crucial for folliculogenesis and is controlled by endothelial cell i
29 llicles within the ovary, but their roles in folliculogenesis and pregnancy, as well as the necessity
30 s an increase in EGFR expression during late folliculogenesis and provide evidence that the FSH-depen
31 ary homeobox gene) deficiency disrupts early folliculogenesis and the expression of oocyte-specific g
32 pecific genes, including those that initiate folliculogenesis and those that encode the zona pellucid
35 nesis) and their enclosure by somatic cells (folliculogenesis) are processes not limited to the perin
36 pe of BMP-15-null mice, which exhibit normal folliculogenesis but have defects in the ovulation proce
37 Static images provide a thumbnail view of folliculogenesis but imperfectly capture the dynamic cel
38 abnormal zona matrix does not affect initial folliculogenesis, but there is a significant decrease in
39 of oocyte meiotic progression and primordial folliculogenesis by decreasing intra-oocyte cAMP levels.
40 ions in oocyte-GC communication during early folliculogenesis can induce GCT by activating an autocri
41 viable but infertile because of a defect in folliculogenesis correlating with restricted expression
42 thelial progenitor cells (eEPCs) rescued the folliculogenesis defects of both Smad1/5(dKO) and Vegfa(
45 yte can help us better understand oogenesis, folliculogenesis, fertilization, and embryonic developme
46 multitude of signaling pathways required for folliculogenesis have been identified, downstream transc
47 of both Egfr and EGFR increases during late folliculogenesis in Fshb(+/-) females, these increases f
51 ion of a subset of genes required for proper folliculogenesis in the ovary and establishes TAFII105 a
54 demonstrates normal thecal layer, defects in folliculogenesis including many degenerating antral foll
57 ation of two genes that are known to disrupt folliculogenesis: newborn ovary homeobox gene (Nobox) an
58 Unique for the ovary, hormonally regulated folliculogenesis, ovulation, luteal formation/regression
59 ient females are infertile due to a block in folliculogenesis prior to antral follicle formation.
60 evelopment and lactation, failure of ovarian folliculogenesis resulting in decreased fertility, loss
62 roteins (ZP1, ZP2, ZP3) that are involved in folliculogenesis, species-specific fertilization, and pa
63 otropins during the endocrine stimulation of folliculogenesis (superovulation) may contribute to the
64 own to be co-expressed in oocytes throughout folliculogenesis, supporting the idea that BMP-15 is a p
65 s between granulosa cells and oocytes during folliculogenesis that are critical to maximize developme
68 naling and endothelial cell invasion promote folliculogenesis via assembly of the basement membrane.
69 th mutant PI3K-positive oocytes during early folliculogenesis were essential for the GC transformatio
70 n completely abrogates this wounding-induced folliculogenesis, whereas overexpression of Wnt ligand i
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