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1 asticity in the primary somatosensory cortex following therapy.
2 ome measure for recovery of binocular vision following therapy.
3 m, and Corynebacterium species were observed following therapy.
4 ons were carried out at baseline and 8 weeks following therapy.
5 mplete resolution of Guillain-Barre syndrome following therapy.
6 n imaging data were acquired at baseline and following therapy.
7 t high frequencies (42 to 50%) 70 to 90 days following therapy.
8 epeat liver biopsy were assessed at 24 weeks following therapy.
9 of Aa at baseline as well as 3 and 12 months following therapy.
10 se activity during the study or in the month following therapy.
11 oups and HGV was not associated with outcome following therapy.
12 mined during the course of HIV infection and following therapy.
13 were measured at baseline and 6 and 9 months following therapy.
14 g indolent phases, and undergo rapid changes following therapy.
15 patients under 60 years of age received the following therapy: 45 mg/m(2) daunorubicin on days 1-3,
16 s perceived their disease to be in remission following therapy, 78% of these patients required contin
19 from the end of the successful weaning trial following therapy and the previously failed one, were an
20 ll clones, elimination of clonal populations following therapy, and subsequent appearance of a clone
22 t therapy, and patients with detectable DTCs following therapy are at substantially increased risk fo
24 KLF4 at disease acceleration, which resolves following therapy, but reoccurs following relapse and de
25 ng period, the defects were treated with the following therapies: collagen membrane (GTR), human demi
26 ibute to the survival of breast cancer cells following therapy could aid in the development of more e
27 immunomodulators, and predictors of relapse following therapy discontinuation have become available.
28 viral therapy era; however, data on outcomes following therapy for anal dysplasia (infrared coagulato
29 delines are commonly used to assess response following therapy for hepatocellular carcinoma (HCC).
31 ion that gonococcal DNA is detectable by LCR following therapy for uncomplicated gonococcal infection
32 cted an increase in Ki-67+ PD-1+ CD8 T cells following therapy in approximately 70% of patients, and
34 nd to analyze changes in cytokine expression following therapy in order to understand its primary mec
36 mice harbored abnormal lymphoproliferations following therapy--in these cases, comparison of the loc
37 the assessments of minimal residual disease following therapy, including for patients who have under
38 ed by diverse therapy-sensitive cancer cells following therapy-induced caspase cleavage of the tumor
44 ssessment of restoration of retinal function following therapy renders achromatopsia a very attractiv
45 (IgE) and other Th2 responses in the months following therapy, responses that have been associated w
46 ividuals experiencing serious adverse events following therapy (see the related articles beginning on
47 showing a time-dependent response up to 3 h following therapy, suggesting a diffusion of soluble Fas
48 ared with T1DM patients and can be monitored following therapies targeted at improving inflammation a
50 eventual secondary decline of DP thymocytes following therapy was associated with renewed viral expr
53 lapse) and 10 patients with no response (NR) following therapy were identified through the Virahep-C
54 ite APBI developed peak distortion 21 months following therapy, which may be sooner than distortion f
55 rinary storage symptoms (overactive bladder) following therapy with an alpha-blocker, the addition of
56 ssociated with DNA-damaging agents can occur following therapy with FND, with or without rituximab, a
58 myeloid leukemia (CML) SCs that is enriched following therapy with tyrosine kinase inhibitors (TKIs)
59 hat the reduction of DMN-insula connectivity following therapy would correlate with diminished pain.
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