ライフサイエンスコーパス: food allergy

1 patients with asthma, atopic dermatitis, and food allergy.

2 posure may be a risk factor for IgE-mediated food allergy.

3 n and suggest HRF as a therapeutic target in food allergy.

4 ications for the prevention and treatment of food allergy.

5 rly phenotypes were strongly associated with food allergy.

6 ound SPINK5 are associated with IgE-mediated food allergy.

7 lusions on the cost-effectiveness of AIT for food allergy.

8 cases of food sensitization and 14 cases of food allergy.

9 5) were underrepresented among subjects with food allergy.

10 rs20541) is associated with challenge-proven food allergy.

11 to development of more targeted treatment of food allergy.

12 une responses in the setting of pre-existing food allergy.

13 to the patient, to confirm the diagnosis of food allergy.

14 reducing anxiety in mothers of children with food allergy.

15 estigating the cost-effectiveness of AIT for food allergy.

16 lergen immunotherapy (AIT) on pollen-related food allergy.

17 tary allergens, promoting the development of food allergy.

18 old school students in Melbourne has current food allergy.

19 he other four loci increase the risk for any food allergy.

20 development of challenge-proven IgE-mediated food allergy.

21 stress response in mothers of children with food allergy.

22 , emphasizing the role of both mechanisms in food allergy.

23 problems were higher among adolescents with food allergy.

24 e sought to investigate the role of ILC2s in food allergy.

25 nts would protect against the development of food allergy.

26 to innovative therapies in the treatment of food allergy.

27 h birch pollen improves birch pollen-related food allergy.

28 adulthood among individuals with and without food allergy.

29 rtium of Food Allergy observational study of food allergy.

30 history of eczema, asthma, hay fever, and/or food allergy.

31 respectively, and resistance to experimental food allergy.

32 of birch pollen AIT on birch pollen-related food allergy.

33 genesis of EoE is distinct from IgE-mediated food allergy.

34 -threatening immune responses in the case of food allergy.

35 n of allergen-specific Treg cells and favors food allergy.

36 xpansion and the development of IgE-mediated food allergy.

37 nquired about wheeze, hay fever, eczema, and food allergy.

38 control allergy in an experimental model of food allergy.

39 idelines for the diagnosis and management of food allergy.

40 of a conventional disease model and/or human food allergy.

41 may have a causal role in the development of food allergy.

42 ression from atopic dermatitis to asthma and food allergy.

43 early infancy to subsequent challenge-proven food allergy.

44 rs9325071 and challenge-proven IgE-mediated food allergy.

45 ing 81 mothers of children with IgE-mediated food allergy.

46 IgE are not definitive for the diagnosis of food allergy.

47 hes C11orf30 as a risk locus for both PA and food allergy.

48 rom the food in our diet are predisposing to food allergy.

49 E titers as observed in humans with clinical food allergy.

50 amined changes in patient QOL during OIT for food allergy.

51 icacy of immunotherapy for environmental and food allergies.

52 lergens is associated with the appearance of food allergies.

53 ence-based school policies for children with food allergies.

54 ltaneous treatment of Bet v 1 and associated food allergies.

55 he loss of oral tolerance and development of food allergies.

56 ms) for understanding details of pupils with food allergies.

57 There is growing evidence for an increase in food allergies.

58 ses including asthma, atopic dermatitis, and food allergies.

59 for patients with concurrent PN/TN and other food allergies.

60 the prevention and treatment of IgE-mediated food allergies.

61 idence interval) (1.78; 1.28-2.48), non-milk food allergy (1.65; 1.27-2.14), and other allergies (3.0

62 mia and Hodgkins'disease); - 3 patients with food allergy; - 1 patient with acute gastroenteritis; -

63 Of 547 students with possible IgE-mediated food allergy, 243 (44.4%; 95% CI, 40.3% to 48.7%) report

64 es, and 426 (4.5%) and 447 (4.4%) pupils had food allergies, 61 (0.6%) and 61 (0.6%) had anaphylaxis,

65 Bet v 1-mediated birch pollen and associated food allergies, a single wild-type allergen does not pro

66 was not associated with primary outcomes of food allergy (adjusted odds ratio (aOR) 0.77; 95% CI: 0.

67 Atopic dermatitis (AD), food allergy, allergic rhinitis, and asthma are common a

68 Measures: Wheeze, eczema, allergic rhinitis, food allergy, allergic sensitization, type 1 diabetes me

69 We sought to determine the prevalence of food allergy among a population-based sample of 10- to 1

70 GOS/inulin prebiotics were protected against food allergies and displayed lower clinical scores, spec

71 This subclass distribution is atypical for food allergies and distinct from natural alpha-gal IgG r

72 iggered by exposure to food allergens (e.g., food allergies and eosinophilic gastrointestinal disorde

73 Although eczema resolved in all, food allergies and high IgE levels persisted in some pat

74 of basic mechanisms underlying IgE-mediated food allergies and novel therapeutic approaches under in

75 latory DC immunotherapy can be effective for food allergies and suggest that induction of Foxp3(-) re

76 The increasing prevalence of food allergies and the intake of packing foods in the pa

77 arise in the setting of naturally resolving food allergy and accompany the acquisition of food aller

78 Self-reported food allergy and adverse reaction details, including ana

79 primer for those embarking on understanding food allergy and also details advances and updates in ep

80 Community practitioners who manage childhood food allergy and anaphylaxis need to be educated about t

81 hildhood allergic sensitization, inhalant or food allergy and eczema, and whether any association was

82 ltiple novel loci as risk factors for PA and food allergy and establishes C11orf30 as a risk locus fo

83 ing to explain the immunologic correlates of food allergy and food desensitization.

84 atients is a window into the pathogenesis of food allergy and food tolerance development.

85 tients with suspected histamine intolerance, food allergy and healthy controls.

86 huNSG mice provide a novel tool for studying food allergy and IgE-mediated anaphylaxis.

87 y of life (QOL) is impaired in patients with food allergy and improves following oral immunotherapy (

88 mine intolerance, in 21 patients with proven food allergy and in 10 healthy control patients.

89 comitant diagnoses of asthma, hay fever, and food allergy and increased disease severity.

90 h positive and negative associations between food allergy and infection with the gastric bacterium He

91 00 and 2013, we determined the prevalence of food allergy and intolerance by sex, racial/ethnic group

92 We sought to determine the prevalence of food allergy and intolerance documented in the EHR aller

93 The prevalence of food allergy and intolerance was higher in females (4.2%

94 ociations between VDI, UVR exposure dose and food allergy and investigate potential confounding.

95 ensitivity disorders frequently triggered by food allergy and manifested by mucosal eosinophilic infi

96 rived prevalence data on challenge-confirmed food allergy and other allergic diseases in preschool-ag

97 antibiotic use with subsequent occurrence of food allergy and other allergies in childhood using elec

98 We also saw new studies on the diagnosis of food allergy and potential approaches to the treatment o

99 AT can also be used to monitor resolution of food allergy and the clinical response to immunomodulato

100 ematic reviews on allergic rhinitis, asthma, food allergy and venom allergy, respectively.

101 rgic rhinitis, allergic asthma, IgE-mediated food allergy and venom allergy.

102 kine receptors that are important in asthma, food allergies, and atopic dermatitis and their respecti

103 allergic diseases, such as asthma, rhinitis, food allergies, and atopic dermatitis, are generally cla

104 en antibiotic use and milk allergy, non-milk food allergies, and other allergies.

105 for hay fever, 4.2% (95% CI, 4.1%-4.3%) for food allergy, and 1.1% (95% CI, 1.1%-1.2%) for anemia.

106 were assessed for asthma, allergic rhinitis, food allergy, and atopic dermatitis.

107 tates, and history of asthma, hay fever, and food allergy, anemia was associated with eczema in 14 of

108 95% CI, 1.1-2.6]) and those with more than 2 food allergies (aOR, 1.9 [95% CI, 1.1-3.1]) were at grea

109 Food allergies are a growing health problem, and the dev

110 ultiple food allergens is thus imperative as food allergies are becoming increasingly recognized as a

111 Food allergies are believed to be on the rise, and curre

112 ure of fish allergy, associations with other food allergies are not well understood.

113 Food allergies are recognized as an increasing health co

114 Adolescents with food allergy are frequently exposed to food allergens.

115 Clinical manifestations of food allergy are largely mediated by IgE.

116 Genetic factors and mechanisms underlying food allergy are largely unknown.

117 f allergen-specific B cells in patients with food allergy are not fully understood but are of major p

118 of apnea attack associated with IgE-mediated food allergy are rare.

119 Many consider food allergy as the "second wave" of the allergy epidemi

120 ts with CID, early-onset asthma, eczema, and food allergies, as well as autoimmunity.

121 and potential approaches to the treatment of food allergy, as well as novel mechanistic studies helpi

122 ell beyond the march to allergic conditions (food allergy, asthma, allergic rhinitis, allergic conjun

123 to initiate the atopic march, which includes food allergy, asthma, and allergic rhinitis.

124 hole cohort, and to report the prevalence of food allergy, asthma, eczema, and allergic rhinitis at a

125 rst 6 months of infancy is a risk factor for food allergy at 1 year of age.

126 or 6 months (aRR 0.93, 95% CI 0.41-2.14) and food allergy at 1 year.

127 ween VDI during infancy and challenge-proven food allergy at 1 year.

128 beneficial effect on psychosocial impact of food allergy at 3 and 12 months after end-of-treatment.

129 The prevalence of challenge-confirmed food allergy at age 1 and 4 years was 11.0% and 3.8%, re

130 dy aimed to report the updated prevalence of food allergy at age 1 year from the whole cohort, and to

131 The prevalence of clinic-defined current food allergy based on history, sensitization data, and O

132 The diagnosis of IgE-mediated food allergy based solely on the clinical history and th

133 at vitamin D insufficiency may contribute to food allergy, but findings vary between populations.

134 The prevalence of asthma and food allergy by 6 years of age was strongly increased am

135 tests, and physician-diagnosed inhalant and food allergy by a postal questionnaire.

136 ed the question of whether the prevention of food allergy by means of early introduction of multiple

137 re were 484 milk allergy cases, 598 non-milk food allergy cases and 3652 other allergy cases.

138 id not differ between food sensitization and food allergy cases and controls.

139 IgE-mediated food allergy caused by jellyfish is rare worldwide.

140 successful desensitization in patients with food allergy completing clinical trials and, in some stu

141 Although the prevalence of food allergy decreased between age 1 year and age 4 year

142 non-food-allergic), mean age 2.5 years, with food allergy defined by either clinical history of react

143 sample of adolescents and young adults with food allergy; determine whether food allergy is associat

144 others during pregnancy and breastfeeding on food allergy development in offspring mice.

145 arding the role of microbiome alterations in food allergy development.

146 we report a genome-wide association study on food allergy diagnosed by oral food challenge in 497 cas

147 There have been no reports on the use of "foods allergy disease lifestyle guidance and management

148 Current hypotheses and models of food allergy do not adequately explain the dramatic incr

149 e on the potential for precision medicine in food allergy, drug allergy, and anaphylaxis under the au

150 ates, ever history of asthma, hay fever, and food allergy, eczema was found to be associated with hea

151 A number of food allergies (eg, fish, shellfish, and nuts) are lifel

152 is after cow's milk allergy the most common food allergy; eggs are used in many food products and th

153 molecules, affects the onset and severity of food allergy, either positively or negatively.

154 risk of asthma, eczema, and aeroallergen and food allergy, especially pronounced after a duration of

155 changes observed during the establishment of food allergy (FA) desensitization in FA patients is a wi

156 Food allergy (FA) is an increasing problem that has no a

157 sociation between atopic dermatitis (AD) and food allergy (FA) is not fully understood, although a ca

158 ges as the preferred diagnostic standard for food allergy (FA) was harmonized over the last years.

159 hat explain the recent increase in childhood food allergy (FA), the dual-allergen exposure hypothesis

160 n increase in the prevalence of IgE-mediated food allergy (FA).

161 n if students were suspected to have current food allergy from parent response.

162 By using an adjuvant-free model of food allergy generated by epicutaneous sensitization and

163 Few food allergy genetic risk variants have yet been identif

164 The prevalence of food allergies has been increasing at an alarming rate o

165 ng increase in the incidence and severity of food allergies has coincided with lifestyle changes in W

166 to induce oral tolerance as a treatment for food allergy has been hampered by a lack of sustained cl

167 The incidence of food allergy has increased dramatically in the last few

168 Food allergy has increased rapidly in recent years affec

169 e lacking, there is a strong impression that food allergy has increased, and rates as high as approxi

170 However, the role of IDO in food allergy has not yet been elucidated.

171 Mothers of children with food allergy have increased anxiety, which may be influe

172 Adolescents with food allergy have poorer psychosocial outcomes compared

173 The spectrum of severity observed with food allergy highlights the critical need for more aller

174 overall association between delayed DTaP and food allergy; however, children with delayed DTaP had le

175 QOL of patients with food allergy improves in some but deteriorates in others

176 ation of diagnoses of asthma, hay fever, and food allergy improves PPV and specificity.

177 ed apple allergy is among the most prevalent food allergies in adolescent/adult subjects and mainly r

178 milk allergy (CMA) is one of the most common food allergies in children.

179 guidance form will make it easier to manage food allergies in children.

180 n how breastfeeding can prevent the onset of food allergies in offspring by instructing T reg formati

181 ionally and industrially, but can also cause food allergies in some individuals.

182 omotes a long-term protective effect against food allergies in the offspring.

183 gy is one of the most common and most severe food allergies in Western countries and its accurate dia

184 studies, asthma in 11, hay fever in 12, and food allergy in 12.

185 asing factor (HRF) interactions with IgE and food allergy in a murine model.

186 ased studies have examined the prevalence of food allergy in adolescence using objective measures suc

187 the allergen molecules causative for type I food allergy in animals, which, like in human patients,

188 s governing the persistence or resolution of food allergy in childhood are not understood.

189 d therapeutic target for this common form of food allergy in childhood.

190 nological driver and predictor of persistent food allergy in childhood.

191 Hen's egg is the most common cause of food allergy in early childhood.

192 While the prevalence of food allergy in humans has been well studied for some al

193 Asian infants, and rates of challenge-proven food allergy in infants have been found to be unexpected

194 he highest prevalence of challenge-confirmed food allergy in infants internationally.

195 in an accurate diagnosis of gastrointestinal food allergy in neonates, OFC should be performed proact

196 tization to dietary antigens and may lead to food allergy in some children.

197 published, suggesting differential rates of food allergy in specific racial and ethnic groups.

198 71 (AG) was associated with challenge-proven food allergy in the discovery sample (P=.001, OR=2.95, C

199 1295686 was associated with challenge-proven food allergy in the discovery sample (P=.003; OR=1.75; C

200 rotection against clinical manifestations of food allergy in two mouse models with OVA and peanut.

201 Spontaneous food allergy in Was(-/-) mice was compared to an adjuvan

202 adequately explain the dramatic increase in food allergy in Western countries.

203 Food allergies, in particular, have increased dramatical

204 symptomatic characteristics of experimental food allergy, including incidence of diarrhea, incidence

205 Quality of Life Questionnaire (FAQLQ-PF) and Food Allergy Independent Measure (FAIM) at pre-treatment

206 ergen, Dau c 1, has been suggested to induce food allergy independently from Bet v 1.

207 bjected them to four different protocols for food allergy induction, using either ovalbumin or peanut

208 Although specific immunotherapy for food allergies is becoming more effective, it is still l

209 A goal of immunotherapy for food allergies is to induce sustained desensitization or

210 Food allergy is a growing health problem with very limit

211 Food allergy is a major health issue, but its pathogenes

212 Food allergy is an important public health problem becau

213 Food allergy is an increasingly common health problem in

214 adults with food allergy; determine whether food allergy is associated with adolescent and maternal

215 Although the reporting on food allergy is inadequate to perform systematic reviews

216 The diagnosis of food allergy is largely reliant on medical history, test

217 However their role in food allergy is largely unknown.

218 Food allergy is major public health concern affecting ne

219 In the cases that the BAT is positive, food allergy is sufficiently confirmed without OFC; in t

220 ta-analyses, the available data suggest that food allergy is underdiagnosed.

221 (PN) and tree nuts (TN), the most dangerous food allergies, is common.

222 allergic symptoms of hay fever, eczema, and food allergy later in life.

223 nella spiralis infection or the induction of food allergy-like disease.

224 Families were assessed using the Food Allergy Management and Adaptation Scale.

225 litis syndrome (FPIES) is a non-IgE-mediated food allergy manifesting within 1 to 4 hours of food ing

226 Ninety-two patients with IgE-mediated food allergy (mean age 18.6 years) completed Coping Orie

227 Food allergy naturally resolves in a proportion of food-

228 e most highly dose-sensitive population with food allergy not otherwise identifiable by using routine

229 ere enrolled at infancy in the Consortium of Food Allergy observational study of food allergy.

230 These findings can explain lifelong food allergies observed in human subjects as the consequ

231 litis syndrome (FPIES) is a non-IgE-mediated food allergy of infancy whose pathophysiology is poorly

232 roteins (LTPs) are important causes of plant-food allergies often associated with severe allergic rea

233 tified 97,482 patients (3.6%) with 1 or more food allergies or intolerances (mean, 1.4 +/- 0.1).

234 e a questionnaire regarding the adolescent's food allergy or food-related reactions.

235 potentially IgE-mediated (affecting 50.8% of food allergy or intolerance patients) and 15.9% were ana

236 AC microarray was performed in patients with food allergy or WAS.

237 ared allergic sensitization in patients with food allergy or Wiskott-Aldrich syndrome (WAS) and defin

238 window during which gut microbiota may shape food allergy outcomes in childhood.

239 =50 nM/L) at age 1 years was associated with food allergy, particularly among infants with the GG gen

240 n D supplementation was associated with less food allergy, particularly in infants with the GT/TT gen

241 Food allergies pose a considerable world-wide public hea

242 lergies/atopies including hay fever, eczema, food allergy, positive skin prick testing (SPT), or elev

243 Food allergy prevalence at 1 year was 7.7% (61/786), and

244 Food allergy prevalence is reported to be increasing, bu

245 important implications for the evaluation of food allergy prevention strategies.

246 ators appears to be a promising strategy for food allergy prevention.

247 Patients with food allergy produce high-titer IgE antibodies that bind

248 Administration of specific IgG to food allergy-prone IL4raF709 mice during initial food ex

249 ; placebo 27) from the PPOIT trial completed Food Allergy Quality of Life Questionnaire (FAQLQ-PF) an

250 roup may reflect an increasing prevalence of food allergy rather than simply increased reporting of a

251 Adolescents have the highest risk for food allergy-related fatalities.

252 Food allergy-related quality of life improved from basel

253 The definite diagnosis of food allergy relies-as in humans-on elimination diet and

254 genic foods on the subsequent development of food allergy remains uncertain.

255 severe allergic reactions with IgE-mediated food allergy, reports of apnea attack associated with Ig

256 The world of food allergy research continues to rapidly accelerate, w

257 hma, conjunctivitis, drug allergies, eczema, food allergy, rhinitis, urticaria, venom allergy and oth

258 d allergic sensitization in their effects on food allergy risk (relative excess risk due to interacti

259 trong interactive effects on both asthma and food allergy risk at age 3 years.

260 or adverse effects of low serum vitamin D on food allergy risk.

261 Children with food allergies spend a large proportion of time in schoo

262 IgE-mediated food allergy status at 1 year was determined by formal c

263 4800 adolescents who had only self-reported food allergy status available, the prevalence of self-re

264 th previously validated methods for studying food allergy, suggesting that the EHR's allergy module h

265 Since wheat flour, a cause of food allergy, tends to disperse rapidly in air, it can u

266 ocolitis (FPIES) is a non-IgE cell- mediated food allergy that can be severe and lead to shock.

267 This review highlights research advances in food allergy that were published in the Journal in 2015.

268 t present, there is no cure or treatment for food allergy that would result in an induction of tolera

269 Among students with current IgE-mediated food allergy, those with resolved or current asthma (adj

270 The patient had food allergy to egg until 5 years old.

271 Patients with food allergy to fish and chicken meat (n = 29) or chicke

272 The primary outcome was food allergy to one or more of the six foods between 1 y

273 In the intention-to-treat analysis, food allergy to one or more of the six intervention food

274 notherapy (OIT) is an effective experimental food allergy treatment that is limited by treatment with

275 ontributes to human diseases such as asthma, food allergies, type 1 and type 2 diabetes, hepatic stea

276 SNPs were tested for association with food allergy using the Cochran-Mantel-Haenszel test adju

277 min D3 (25(OH)D3 ) levels among infants with food allergy vs a random subcohort (n = 274).

278 rize the current knowledge on immediate-type food allergy vs other food adverse reactions in companio

279 ble, the prevalence of self-reported current food allergy was 5.5% (95% CI, 4.9-6.2), with peanut, 2.

280 Food allergy was also associated with depressive symptom

281 ses, mast cell homeostasis, and anaphylactic food allergy was assessed in these animals.

282 Food allergy was associated with increased odds of eleva

283 eonates suspected of having gastrointestinal food allergy was conducted in 126 neonatal intensive car

284 Food allergy was defined as caretaker report of healthca

285 e foods between 2013 and 2014.Immediate-type food allergy was significantly more frequent in 2014 tha

286 When a phenotype of any food allergy was used for meta-analysis, the C11orf30 lo

287 A meta-analysis of 2 phenotypes (PA and food allergy) was conducted by using 7 studies from the

288 tization affects offspring susceptibility to food allergy, we epicutaneously sensitized female mice w

289 Those with asthma and more than 2 food allergies were at the greatest risk for adverse foo

290 at age 1 year or reported symptoms of a new food allergy were invited for an assessment that include

291 15, important studies on the epidemiology of food allergy were published, suggesting differential rat

292 Two hundred mothers of children with food allergy were recruited from allergy clinics.

293 icity and comparable sensitivity to diagnose food allergy, when compared with skin prick test and spe

294 us exposure to food allergens predisposes to food allergy, which is commonly associated with atopic d

295 knowledge gaps were identified in veterinary food allergy, which need to be filled by systematic comp

296 ment option to be considered for adults with food allergy who were not able to acquire immune toleran

297 Three forms of food allergy with distinct clinical features are now wel

298 multinational European birth cohort study on food allergy with gold-standard diagnostic methods showe

299 Was(-/-) mice provide a model of food allergy with the advantage of mimicking polysensiti

300 ld infants (discovery sample) phenotyped for food allergy with the gold standard oral food challenge.