ライフサイエンスコーパス: food deprivation

1 onal shift to a 'high-drive' condition (18-h food deprivation).

2 crose pellets under 24-hr but not under 0-hr food deprivation).

3 nable weanling animals to survive periods of food deprivation.

4 drial beta-oxidation genes were repressed by food deprivation.

5 ct any alterations in ARC NPY mRNA following food deprivation.

6 ed by the refeeding hyperphagia that follows food deprivation.

7 5, 10, 20 and 40 microg) following overnight food deprivation.

8 ther of these responses when the stimulus is food deprivation.

9 sweeter nonnutritive sugar after periods of food deprivation.

10 in response to systemic/metabolic aspects of food deprivation.

11 ht to mediate the neuroendocrine response to food deprivation.

12 both physiological/psychological stress and food deprivation.

13 no inhibitory effects on feeding after 24 h food deprivation.

14 tion during the lengthening period of summer food deprivation.

15 or SHU9119 to rats before a 24 hr period of food deprivation.

16 elimination of odors as cues, and the use of food deprivation.

17 ody weight, and become hyperphagic following food deprivation.

18 o give rise to interoceptive cues like 24-hr food deprivation.

19 ity in other systems may be downregulated by food deprivation.

20 isual interneurons, an effect not altered by food deprivation.

21 failed to recover normal pumping rates after food deprivation.

22 did not show an altered feeding response to food deprivation.

23 ahepatic tissues during periods of prolonged food deprivation.

24 uced fibre atrophy caused by denervation and food deprivation.

25 to exhibit exaggerated locomotor response to food deprivation.

26 ll Period mutant mice was also robust during food deprivation.

27 ed by control floxed mice only after 24 h of food deprivation.

28 n than controls under conditions of complete food deprivation.

29 ing neurons increases in response to chronic food deprivation.

30 kness with ad libitum food and after 48 h of food deprivation.

31 y and suppressed 6-h refeeding after 24 h of food deprivation.

32 limited food is presented after a period of food deprivation.

33 bohydrate content of the diet consumed after food deprivation.

34 te for the reduced contribution from chronic food deprivation.

35 ally contiguous phases: 1-h chow access, 2-h food deprivation, 10-min chow access, and 10-min access

36 In Experiment 1, the impact of food deprivation (24 and 48 h) was examined.

37 ntal area upon feeding responses elicited by food deprivation (24 h), 2-deoxy-D-glucose-induced gluco

38 DAMGO (90% maximal decrease), and 24 h acute food deprivation (60% maximal decrease).

39 eus (Arc) and their synthesis increases with food deprivation, a naturally-occurring stimulus that ma

40 Both restraint and food deprivation affected select limbic areas associated

41 etailed profile of cognitive consequences of food deprivation, affected by time of day, task demands,

42 e, and bacterial translocation compared with food deprivation (all p < 0.05).

43 on the GH-IGF-I axis differed from those of food deprivation alone; and b) whether administration of

44 culating FFA for glucose-stimulated IS after food deprivation also applies in the case of nonglucose

45 Food deprivation also challenges the gut microbiota, whi

46 Food deprivation alters the processing of sensory inform

47 Included among these are (1) food deprivation and (2) acute microinjection of the neu

48 Food deprivation and concentration interacted to increas

49 The effects of food deprivation and concentration on burst size (BS), b

50 he mammalian body provide resilience against food deprivation and dietary stress.

51 oth the depletion of energy induced by acute food deprivation and excessive storage of energy by high

52 bling induction of beta-oxidation genes upon food deprivation and facilitating activation of SCD in f

53 t small intestine OEA levels decrease during food deprivation and increase upon refeeding, suggesting

54 y a biological role for AGS-3 in response to food deprivation and indicate the mechanism for its acti

55 havioral shift occurs more rapidly following food deprivation and is modulated by cAMP and insulin si

56 Both food deprivation and MA (600 micromol/kg), but not 2-DG,

57 f the CRR, and thus the ability to withstand food deprivation and maintain euglycemia, is not known.

58 However, the role of food deprivation and metabolic needs in the selection of

59 om environmental energetic stressors such as food deprivation and physical exertion.

60 Acb shell GABA(A) receptor stimulation or by food deprivation and produced a syndrome of forepaw trea

61 (dNPF), an ortholog of mammalian NPY, mimics food deprivation and promotes memory performance in sati

62 g despite normalizing feeding in response to food deprivation and PYY.

63 on the nutritional state-they decreased with food deprivation and recovered after subsequent feeding.

64 ationally related manipulations of the task (food deprivation and reward magnitude) produced some sub

65 The ZI cells were excited by food deprivation and the gut hunger signal ghrelin.

66 multaneous variations in motivational state (food deprivation) and reinforcer magnitude (food present

67 iven to rats in a 'low-drive' condition (2-h food deprivation), and also after a motivational shift t

68 For example, ghrelin levels rise following food deprivation, and ghrelin administration stimulates

69 obility is regulated by caloric restriction, food deprivation, and heat shock.

70 muli, including exposure to dauer pheromone, food deprivation, and high temperature, can induce C. el

71 as markedly increased by lipin 1 deficiency, food deprivation, and obesity, often independent of chan

72 Twenty-eight peptides were increased >50% by food deprivation, and some of these were increased by 2-

73 tis elegans to alter several behaviors after food deprivation, apparently so that the animals can mor

74 food deprivation, suggesting that effects of food deprivation are mediated by modifying this protein.

75 severe childhood undernutrition (SCU) during food deprivation are not clear.

76 e increases in food intake following 24 h of food deprivation are reduced by systemic and central adm

77 s water maze without requiring foot shock or food deprivation as motivating factors.

78 Animals were studied after overnight food deprivation, awake, unrestrained, and unstressed; a

79 ate different protein breakdown responses to food deprivation between children with edematous and non

80 tabolic state, we examined whether overnight food deprivation blunts stress-induced recruitment of th

81 The results demonstrated that NPY and food deprivation both produced dose- or deprivation-depe

82 Peptides found to be elevated by food deprivation but not exercise included a number of f

83 Conversely, food deprivation but not NMDA injection produced marked

84 Finally, food deprivation, but not other stressors, stimulated cA

85 ly hatched Caenorhabditis elegans respond to food deprivation by halting development and promoting lo

86 of liquid diet or 10% sucrose solution after food deprivation by rats with APX also were considerably

87 Here we report that this effect of food deprivation can be blocked by leptin, a hormone inv

88 These data indicate that food deprivation can provoke relapse to heroin seeking v

89 the delay was extended beyond 1 h, and that food deprivation could attenuate the degree of impairmen

90 gely on elevated responding to nonreinforced food deprivation cues.

91 Food deprivation decreased drinking latencies, did not c

92 by a state of negative-energy balance (24-hr food deprivation), demonstrating a specific influence of

93 duced rise in hypothalamic malonyl-CoA after food deprivation, demonstrating that leptin was not requ

94 Food deprivation did not affect BS but rather increased

95 esponse of chromatin-associated proteins, as food deprivation did not affect the mobility of heteroch

96 Following recovery from surgery, food deprivation discrimination performance was compared

97 lt mice, leptin was not acutely regulated by food deprivation during the early postnatal period.

98 We find that food deprivation elevates excitatory synaptic input, whi

99 lows survival during the frequent periods of food deprivation encountered during evolution.

100 s were tested for reinstatement after 1 d of food deprivation (experiment 1) or exposure to intermitt

101 ic gene expression resulting from short-term food deprivation (fasting) in Caenorhabditis elegans.

102 We found that short-term food deprivation (fasting) potently induced p21 expressi

103 adapted to prolonged periods (1-2 months) of food deprivation (fasting) which result in metabolic cha

104 erian hamster feeding behaviors; (2) whether food deprivation (FD) co-increases agouti-related protei

105 completely blocked the effects of leptin on food deprivation (FD)-induced feeding.

106 EXPERIMENTS 2 AND 3: Following overnight food deprivation, five groups of rats were injected with

107 We found short-term food deprivation for 24 h, already induces SIRT2 express

108 Food deprivation for 48 hr also increased CRE binding, w

109 In contrast, food deprivation had little effect.

110 Food deprivation has been shown to deleteriously affect

111 bing neurons controls behavioral response to food deprivation in C. elegans.

112 d tanycytes, were significantly decreased by food deprivation in male mice in a manner that was parti

113 nment in vivo, resulting in overeating after food deprivation in mice.

114 ke and body weight changes following 24 h of food deprivation in rats.

115 vation induced by 2-deoxy-D-glucose (2DG) or food deprivation in rodents.

116 lects calorie-rich foods following prolonged food deprivation in the absence of taste-receptor signal

117 mediating the short-term feeding response to food deprivation in the nucleus accumbens shell as well

118 of food and over an extended time course of food deprivation in wild-type and mutant worms to determ

119 immunoreactivity in this area is enhanced by food deprivation; in contrast, it is reduced by injectio

120 mice at this chronological age, because 24-h food deprivation increased arcuate NPY mRNA in wild-type

121 Food deprivation increased c-fos expression and spike fr

122 context of diet-induced and genetic obesity; food deprivation increased Deptor mRNA in both the ARC a

123 Food deprivation increases hypothalamic syndecan-3 level

124 easure of eating is modulated by satiety and food deprivation increases the reward value of food, the

125 We also establish that food deprivation increases the worm's willingness to cro

126 MTII administration for 24 h prevents food deprivation-induced alterations in hypothalamic neu

127 nfused into the basolateral amygdala blocked food deprivation-induced changes in sucrose-related acti

128 In addition, IV-SUnSET detected food deprivation-induced decreases in PS in the heart, k

129 These similarities between NPY-induced and food deprivation-induced feeding are consistent with a s

130 oraging and food hoarding were not affected, food deprivation-induced increased food hoarding was sur

131 suggests an intact Arc is not necessary for food deprivation-induced increases in food foraging and

132 Like obesity, prolonged food deprivation induces severe hepatic steatosis; howev

133 Rats were trained to solve a food deprivation intensity discrimination problem in whi

134 lating concentration of FFA that accompanies food deprivation is a sine qua non for efficient GSIS wh

135 ce of energy and nutrient homeostasis during food deprivation is accomplished through an increase in

136 circulating FFA levels after 24 and 48 h of food deprivation is critically important for pancreatic

137 Results suggest that food deprivation may act as a novel stress, thereby incr

138 er infusion of milk to assess the effects of food deprivation on sleep.

139 Lipid stores are mobilized in the case of food deprivation or high energy demands--for example, du

140 ntaining neurons or fibers are unaffected by food deprivation or MA, and the antimetabolite 2-DG has

141 ine [TMT]), a paradigm that does not involve food deprivation or operant performance.

142 Here, we show in mice that food deprivation or optogenetic activation of AgRP neuro

143 hich can be altered by manipulations such as food deprivation or prior exposure to MOP receptor agoni

144 stimuli (footshocks, shock-predictive cues, food deprivation, or reward omission) and inhibitions af

145 learned schedules of food intake rather than food deprivation per se.

146 at another environmental stressor, acute 1 d food deprivation, potently reinstates heroin seeking in

147 ess NMDA-elicited feeding by PTK inhibitors, food deprivation readily drives PTK activity in vivo.

148 Acute food deprivation reinstated heroin seeking, an effect th

149 corticosterone levels were reduced, whereas food deprivation resulted in significantly enhanced plas

150 In Caenorhabditis elegans, pairing odor with food deprivation results in aversive olfactory learning,

151 in which stimuli produced by 0-hr and 24-hr food deprivation served as discriminative cues for the d

152 Here, we show that after food deprivation, SIRT1 levels fall dramatically in type

153 ternation in a T-maze and on a task in which food-deprivation state was used as a contextual cue.

154 effects on food intake both under normal and food deprivation states.

155 ine or sucrose, it effectively blocked acute food deprivation stress-induced reinstatement of cocaine

156 results independent from changes related to food deprivation stress.

157 change in its biochemical fractionation upon food deprivation, suggesting that effects of food depriv

158 , whereas nutrient supplementation following food deprivation suppressed it; the former and latter co

159 Food deprivation suppresses animal growth and developmen

160 Food deprivation suppresses sleep, presumably to increas

161 Following food deprivation, the expression and acetylation of the

162 In response to food deprivation, the pumping rate rapidly declines by a

163 responses that are otherwise associated with food deprivation; thus, unlike forced dieting, cessation

164 at an increase in locomotor speed induced by food deprivation was accompanied by an activity- and oct

165 In addition, food deprivation was tested as a possible modulating fac

166 portance of considering prolonged periods of food deprivation when assessing chemical risks posed to

167 ach 4-min session that took place under 0-hr food deprivation, whereas no pellets were delivered duri

168 Food deprivation which enhances aPVN GAL produces a mark

169 other, naturally occurring stressors such as food deprivation, which is being exacerbated by global w

170 Food deprivation, which transiently favors feeding over

171 /DA and HVA/DA ratios were normalized by age/food-deprivation while that of 3MT/DA was not.

172 ice, otherwise apparently normal, respond to food deprivation with markedly reduced reflex hyperphagi

173 crose palatability induced by an increase in food deprivation without affecting the performance of su