ライフサイエンスコーパス: foot

1 e ways (e.g., kicking is making contact with foot).

2 nd flattening of the medial arch of the left foot.

3 domains has been characterized from Atrina's foot.

4 l contacts at the vitreal surface of the end-foot.

5 ands controlled by their two hands and right foot.

6 lements, including one nearly-complete adult foot.

7 athetic fibres innervating the dorsum of the foot.

8 ning the forces acting underneath the stance foot.

9 that places the technique on a quantitative footing.

10 lavins/flavoproteins on a firm photophysical footing.

11 eating place and non-place cells on the same footing.

12 ctronic interactions are treated on an equal footing.

13 ls, zeta-tubulin is a component of the basal foot, a centriolar appendage that connects centrioles to

14 heral bypass surgery or angioplasty, limb or foot amputation, intermittent claudication with objectiv

15 Foot anaesthesia reduced ankle adaptation to external fo

16 istribution among various subsections of the foot and ankle can be difficult, in large part due to a

17 mediated oxidative damage in the ipsilateral foot and ankle joint compared to wild-type mice which wa

18 We found that various subsections within the foot and ankle showed disparate work distribution, parti

19 us studies of human locomotion indicate that foot and ankle structures can interact in complex ways.

20 The human foot and ankle system is equipped with structures that c

21 evolution included anatomical changes of the foot and ankle, altering the moment arms and control of

22 lcium influx was initiated at the Muller end-foot and apical process, triggering centrifugal propagat

23 -dependent relaxation of apical muscle, tube foot and cardiac stomach preparations from A. rubens.

24 It also exhibits a humanlike foot and lower limb.

25 Hand Foot and Mouth Disease (HFMD) constitutes a considerable

26 To monitor search trends on Hand, Foot and Mouth Disease (HFMD) in Guangdong Province, Chi

27 s recently emerged as a major cause of hand, foot and mouth disease in children worldwide but no vacc

28 ccines.Coxsackievirus A6 (CVA6) causes hand, foot and mouth disease in children.

29 n from genetic and epidemiological data in a Foot and Mouth Disease Virus (FMDV) veterinary outbreak

30 byssal secretion is enabled by a specialized foot and multiple proteins including expanded tyrosinase

31 utine weight-bearing radiography of her left foot and weight-bearing computed tomography (CT) of both

32 nephropathy, peripheral neuropathy, diabetic foot, and ischemic heart disease were 21.9%, 17.6%, 28.0

33 (EV71) is an emerging pathogen causing hand, foot, and mouth disease (HFMD) and fatal neurological di

34 viridae family and are major causes of hand, foot, and mouth disease (HFMD) and pediatric respiratory

35 f enterovirus 71 (EV71) and associated hand, foot, and mouth disease (HFMD) are recognized as emergin

36 Epidemiology and etiology of hand, foot, and mouth disease (HFMD) based on large sample siz

37 87% (9.8 million/11.3 million) of all hand, foot, and mouth disease (HFMD) cases reported to WHO dur

38 Hand, foot, and mouth disease (HFMD) has spread throughout the

39 Hand, foot, and mouth disease (HFMD) is a common childhood ill

40 Hand, foot, and mouth disease (HFMD) is a reemerging illness c

41 are emerging pathogens associated with hand, foot, and mouth disease and pediatric respiratory diseas

42 (EV-A71) is the major cause of severe hand, foot, and mouth disease and viral encephalitis in childr

43 Foot-and-mouth disease (FMD) can cause large disruptive

44 Foot-and-mouth disease (FMD) in Turkey is controlled usi

45 Foot-and-mouth disease (FMD) is one of the most feared v

46 ly more stable vaccine candidates.IMPORTANCE Foot-and-mouth disease (FMD) is the most devastating dis

47 Foot-and-mouth disease (FMD) remains one of the most dev

48 Production of foot-and-mouth disease (FMD) vaccines requires cytosolic

49 Foot-and-mouth disease (FMD) virus (FMDV) circulates as

50 ent for control and potential eradication of foot-and-mouth disease (FMD).

51 Enterovirus 71 (EV71) can cause hand-foot-and-mouth disease (HFMD) in young children.

52 Hand-foot-and-mouth disease is a serious public health threat

53 Foot-and-mouth disease outbreaks in non-endemic countrie

54 The Global Foot-and-Mouth Disease Research Alliance (GFRA), an inte

55 The foot-and-mouth disease virus (FMDV) "carrier" state was

56 The foot-and-mouth disease virus (FMDV) afflicts livestock i

57 ished data from transmission experiments for foot-and-mouth disease virus (FMDV) and African swine fe

58 Foot-and-mouth disease virus (FMDV) causes a fast-spread

59 use in stabilizing SAT2 vaccines.IMPORTANCE Foot-and-mouth disease virus (FMDV) causes a highly cont

60 Foot-and-mouth disease virus (FMDV) causes a highly cont

61 ocked the replication of poliovirus (PV) and foot-and-mouth disease virus (FMDV) in a variety of cell

62 ociated with clearance versus persistence of foot-and-mouth disease virus (FMDV) in micro-dissected c

63 The pathogenesis of persistent foot-and-mouth disease virus (FMDV) infection was invest

64 Foot-and-mouth disease virus (FMDV) is a highly contagio

65 Foot-and-mouth disease virus (FMDV) is an important anim

66 Foot-and-mouth disease virus (FMDV) mediates cell entry

67 Foot-and-mouth disease virus (FMDV) RNA-dependent RNA po

68 Foot-and-mouth disease virus (FMDV), particularly strain

69 merous human and animal pathogens, including foot-and-mouth disease virus (FMDV).

70 lved in genome packaging in the picornavirus foot-and-mouth disease virus (FMDV).

71 agents that produce vesicular lesions, e.g., foot-and-mouth disease virus and others.

72 ar to those of core catalytic domains of the foot-and-mouth disease virus leader protease and coronav

73 we use these methods to analyze data from a foot-and-mouth disease virus outbreak in the United King

74 lved in genome packaging of the picornavirus foot-and-mouth disease virus.

75 cellosis) and animal (bovine brucellosis and foot-and-mouth disease) infections clearly differentiati

76 an important animal pathogen responsible for foot-and-mouth disease.

77 We investigated this foot-ankle interplay during walking by adding stiffness

78 The ankle and foot are commonly injured during sporting activities.

79 ntrolling the placement of the participants' foot as it contacted a ground-mounted force platform.

80 ty of putting sequence alignment on the same footing as statistical phylogenetics, theorists have str

81 ty was 20/70 (20/20 to counting fingers at 1 foot) at time of recurrence and declined to counting fin

82 in ischemic tissues, therefore retarded the foot blood perfusion recovery.

83 vironmental samples, consisting of collected foot borne debris, were taken at SRS over an eleven year

84 The lesion is usually on the foot but all parts of the body can be affected.

85 e male and full-sibling female African black-footed cat developed vision deficits and mydriasis as ea

86 Analysis of the black-footed cat studbook suggests additional captive cats are

87 African black-footed cats (Felis nigripes) are endangered wild felids.

88 T2D); however, their contribution to Charcot foot (CF) disease is not known.

89 r) in 43 hospitals with specialised diabetic foot clinics in France, Spain, Italy, Germany, and the U

90 EM analysis of the end-foot compartment showed high-density ER cisternae that s

91 unrelated families characterized by a split-foot defect, nail abnormalities of the hands, and hearin

92 The structure of the foot defines the input and output lever arms that influe

93 gressive distal muscle weakness and atrophy, foot deformities, distal sensory loss, as well as dimini

94 However, the H. naledi foot differs from modern humans in having more curved pr

95 rotubules, adherens-junctions and apical end-foot dimensions.

96 The end-foot disproportionately expresses the depletion sensor s

97 t a single-stranded DNA with one leg and two foot domains for walking, and one arm and one hand domai

98 eedback from the superficial peroneal nerve (foot dorsum) and medial plantar nerve (foot sole) during

99 Foot drop and toe walking are frequent concerns in child

100 albuminuria, nephrinuria, FSGS, and podocyte foot effacement in Ang II-induced hypertension; and earl

101 ey disease (OR = 1.31; CI, 1.08-1.59), prior foot examination (OR = 1.49; CI, 1.28-1.74), prior hemog

102 Domestic pigeons have striking variation in foot feathering within a single species, providing a tra

103 ctopic expression of Tbx5 is associated with foot feathers in chickens, suggesting similar molecular

104 administration of testosterone (T) increases foot flagging behavior under seminatural conditions.

105 limb gestural signals, called "foot flags." Foot flagging is a derived display that emerged in the r

106 The results show that foot flagging is an androgen-dependent gestural signal,

107 o other anuran species, which do not produce foot flags (Rana pipiens and Xenopus laevis).

108 ions and hind limb gestural signals, called "foot flags." Foot flagging is a derived display that eme

109 The existence of a prespike foot for many events suggests the formation of an initia

110 ce and recovery, sustained a catastrophic 50-foot free-fall from the top of the rainforest canopy to

111 obe to bone," "osteomyelitis," and "diabetic foot" from 1946 to May 2015.

112 facilities were commonly reached rapidly by foot (>70%), transportation to secondary facilities diff

113 ce of an energy-saving longitudinally arched foot in H. erectus.

114 ed in quantum contexts and then, on an equal footing, in classic wave systems.

115 control feet or other areas of the diseased foot (including the coronary band or interdigital space)

116 absent from invasive isolates from diabetic foot infections, including osteomyelitis.

117 pired structural materials, petals and gecko foot-inspired adhesive films, lotus and mosquito eye ins

118 We show here that a tap to the foot interrupted and reset the rhythm of forward swimmin

119 tens the plantar soft tissues to convert the foot into a stiff propulsive lever.

120 sponses, or reflexes, if they occur when the foot is contacting the ground or in the air.

121 ched and relaxed by ankle movements when the foot is on the ground, helping to fulfil one function of

122 ailing this, any straight-line flow into the foot is pursued.

123 eattaches to the track in front of the bound foot it forms an (S)-stereocenter, which is resistant to

124 higher eukaryotes, including split hand and foot malformation 1 in humans.

125 A Q186H mutation linked to split hand and foot malformation 1 likely affects affinity of DNA bindi

126 egulation of Trp63, a known split-hand/split-foot malformation disease gene.

127 At the same time, deformation of the foot may dissipate some of the mechanical energy generat

128 itative microperfusion data from gated human foot microvasculature.

129 test our solution in a reanalysis of a white-footed mouse data set.

130 (EV71) is a human pathogen that causes hand, foot, mouth disease and neurological complications.

131 for motor pools controlling hip, ankle, and foot muscles, revealing a variable circuit architecture

132 With MASC, a theoretical footing now exists to generate and test computationally

133 Here we describe the foot of Homo naledi from Dinaledi Chamber, South Africa,

134 talyzes the selective hydrolysis of the rear foot of macrocyclized walkers (an information ratchet me

135 genitals are represented displaced below the foot of the cortical body map [10-12] or whether they ar

136 rom 243 BP measurements, the landmark at the foot of the oscillometric pulse was found to be associat

137 olonized French area nestled at the northern foot of the Pyrenees.

138 rs, eventually resulting in the reduced hind foot of these sauropods.

139 nput compensation and entrainability: on the footing of equal phase-response curves, it exhibits the

140 of variable scan rate cyclic voltammetry and foot-of-the-wave analysis (FOWA) can be used to detect t

141 ical methods, current-potential analysis and foot-of-the-wave analysis (FOWA), were performed on 1 to

142 Foot-of-the-wave analysis gives a catalytic rate constan

143 the PTB test can accurately rule in diabetic foot OM in the high-risk patients and rule out OM in low

144 ccuracy of the PTB test to diagnose diabetic foot OM.

145 al palsy who had been prescribed fixed ankle-foot orthoses as an example.

146 The main findings indicate that ankle-foot orthoses exert significant effects on coronal and s

147 swing phase using an electrohydraulic ankle-foot orthosis.

148 tiffness decreased energy dissipation at the foot (p < 0.001) and increased the gear ratio (i.e., rat

149 .251, beta = 0.309, P = .002), self-reported foot pain (r = 0.162, beta = .114, P = .009), and self-r

150 f progressive long-standing left dorsomedial foot pain, which was made worse with weight bearing.

151 thus step to step adjustments in posture and foot placement across a range of walking speeds in respo

152 e second half of the preceding step, and (2) foot placement is guided by information about the positi

153 nilateral hemisection to walk with a precise foot placement on the rungs fixed to an ordinary flat tr

154 tep adjustments in postural sway and lateral foot placement positively correlated with those of postu

155 ymmetry, participants gradually adjust their foot placement to adopt steps of equal length.

156 ient measurement of ballistocardiography and foot PPG waveforms - and thus PTT through larger, more e

157 parameters of 20 healthy subjects with right-foot preference during treadmill walking at speeds of 1.

158 Torque moments around the center of foot pressure on the force platform were measured, and t

159 A high resolution (3 km foot print) SM/ST dataset prepared from a land data assi

160 We found that the foot-print-area per ligand was unaffected by the nanocry

161 ioR binding site was confirmed using DNase I foot-printing.

162 ical for glomerular permselectivity; loss of foot process architecture results in proteinuria and FSG

163 anization of the slit diaphragm, followed by foot process disappearance, flattening and fusion of maj

164 pression, which is a key factor for podocyte foot process effacement and proteinuria.

165 hese mice were protected from acute podocyte foot process effacement following protamine sulfate perf

166 amination of podocytes confirmed more robust foot process effacement in the knockout animals.

167 zebrafish knockdown model and mild podocyte foot process effacement in the mouse model, whereas all

168 We observed pronounced podocyte foot process effacement on long stretches of the filtrat

169 ocytes of adult mice results in proteinuria, foot process effacement, and glomerulosclerosis.

170 iculum, resulted in progressive albuminuria, foot process effacement, and histology consistent with E

171 in increased proteinuria, increased podocyte foot process effacement, and to decreased podocyte numbe

172 n the filtration barrier, including podocyte foot process effacement, irregular thickening of the glo

173 SGS, including mesangial sclerosis, podocyte foot process effacement, tubular atrophy, interstitial f

174 ion barrier development, leading to podocyte foot process effacement.

175 in vivo, shedding new light on mechanisms in foot process effacement.

176 dramatic change in cell morphology known as foot process effacement.

177 th loss of nephrin-Nck1/2 association during foot process effacement.

178 cterized by proteinuria and partial podocyte foot process effacement.

179 aotic spatial patterns that could impair the foot process morphology.

180 found a corresponding reduction in podocyte foot process number.

181 est that Ubr4 is a key regulator of podocyte foot process proteostasis.

182 l renal diseases characterized by pathologic foot process remodeling, prompting the hypothesis that p

183 onal (albuminuria and azotemia), structural (foot-process effacement and glomerulosclerosis) and mole

184 a mechanism associated to a reduction in the foot-process fusion and desmin, and a recovery of synapt

185 ses characterized by loss of interdigitating foot processes and decreased expression of components of

186 complex expansions resembling interdigitated foot processes at the basal surface.

187 gate how regulation of actin dynamics within foot processes controls local morphology.

188 esses that connect the primary processes and foot processes in Alport mice.

189 ndrial degeneration, mitophagy, and deformed foot processes in podocytes.

190 ue 3D structure of major and interdigitating foot processes which is the prerequisite for renal blood

191 Podocyte cell protrusions (foot processes) are critical for glomerular permselectiv

192 function depends on fingerlike projections (foot processes) that interdigitate with those from neigh

193 filtration apparatus consisting of podocyte foot processes, glomerular basement membrane and endothe

194 ecifically bind to murine THSD7A on podocyte foot processes, induce proteinuria, and initiate a histo

195 y via a series of interdigitating actin-rich foot processes.

196 appearance of TJ-like structures between the foot processes.

197 rol of dynamics of actin cytoskeleton in the foot processes.

198 (an information ratchet mechanism), the rear foot producing an (R)-stereocenter at its point of attac

199 ain simulations of an amputee using an ankle-foot prosthesis by simultaneously optimizing human movem

200 terfacial load-bearing protein (A. pectinata foot protein-1, apfp-1) with L-3,4-dihydroxyphenylalanin

201 mussels use catechol-rich interfacial mussel foot proteins (mfps) as primers that attach to mineral s

202 Mussel foot proteins (Mfps) exhibit remarkably adaptive adhesio

203 g sticky biological molecules-such as mussel foot proteins (MFPs)-into synthetic, cost-effective unde

204 If the hydrolyzed foot reattaches to the track in front of the bound foot

205 Quantitative sensory testing of the foot revealed striking impairments in thermal detection

206 ring the swing phase or the sensation of the foot rolling on the floor while walking.

207 tion; and both sagittal and horizontal plane foot rotation.

208 The energy-sparing spring theory of the foot's arch has become central to interpretations of the

209 technique that restricted compression of the foot's longitudinal arch, this study provides the first

210 has become central to interpretations of the foot's mechanical function and evolution.

211 d by isolating the forces acting within each foot segment through controlling the placement of the pa

212 t-Yangming (SMFY) or Gallbladder Meridian of Foot-Shaoyang (GMFS) in healthy male Sprague Dawley (SD)

213 The foot-shaped yeast U1 snRNP contains a core in the "ball-

214 ditioning variants motivated aversively with foot shock and appetitively with food.

215 lesions drastically impaired the ability of foot shock to suppress operant responding for food.

216 ntext after conditioning and responsivity to foot shock were unaffected by optogenetic silencing.

217 g consisting of an auditory CS paired with a foot shock, and the auditory CS was re-presented during

218 Importantly, foot-shock alone did not increase spinogenesis.

219 the effect is seen only when high-intensity foot-shock is used in training.

220 atement procedure in mice, we show that both foot-shock stress and the pharmacological stressor yohim

221 reinstatement of cocaine seeking induced by foot-shock stress, but in the absence of continued globa

222 which is dependent upon the intensity of the foot-shock used for training; that is, the effect is see

223 unpredictably either in punishment (0.45 mA foot-shock) or the opportunity to make a taking response

224 The foot-shock-driven excitation within the LHb requires glu

225 in KOR conditional knock-out mice prevented foot-shock-induced CPP reinstatement.

226 Pregnant dams were exposed to mild stress (foot shocks at 1 week intervals) throughout pregnancy.

227 lever presses were punished by mild electric foot shocks.

228 nile and adult rats were presented with mild foot-shocks and their USV frequency, duration, and relat

229 We observe that aversive stimuli, including foot-shocks, excite LHb neurons and promote escape behav

230 plantar flexors, as walking on a more rigid foot/shoe surface compromises the plantar flexors' mecha

231 Foot sites exhibited the most variability; individuals d

232 ed MCs is increased in unwounded forearm and foot skin of patients with diabetes and in unwounded dor

233 al disorders (18 [11%] vs 12 [8%]), and hand-foot skin reaction (12 [8%] and none).

234 Adverse events included hand-foot skin reaction (22 [54%]), generalized pigment dilut

235 ne patients [5%] in the placebo group), hand-foot skin reaction (47 patients [13%] vs one [1%]), fati

236 d with a cutaneous adverse event called hand-foot skin reaction (HFSR), in which sites of pressure or

237 related serious adverse events included hand-foot skin reaction (ten [2%]), abnormal hepatic function

238 of common adverse effects, most notably hand-foot skin reaction, diarrhea, and hypertension, compared

239 erve (foot dorsum) and medial plantar nerve (foot sole) during walking.

240 lic cost during walking increased with added foot stiffness (p < 0.001).

241 Added foot stiffness also altered soleus muscle behaviour, lea

242 the swimming rhythm and the response to each foot stimulation can itself be altered by the swim rhyth

243 Foot stimulation can reset the timing of the swimming rh

244 e hip flexor nerve response to an electrical foot stimulus was reduced or eliminated during the swim

245 y + 6.0% (p < 0.001, d = 0.67; unaffected by foot strike technique).

246 Delivery of fast, forceful and accurate foot strikes that are sufficient to stun and kill prey r

247 stal to the shank (i.e., ankle joint and all foot structures), these structures resembled an energy-n

248 ans, with the exception of the autopod (hand/foot) structures, which have no clear correspondence wit

249 IRF-1 expression limited CHIKV-induced foot swelling in joint-associated tissues and prevented

250 ) in the patients treated with CMF, and hand-foot syndrome (129 [12%]) and diarrhoea (67 [6%]) in the

251 st common grade 3-4 adverse events were hand-foot syndrome (201 [21%] of 963 in the capecitabine alon

252 b and 102 [16%] patients on sorafenib), hand-foot syndrome (94 [15%] patients on sunitinib and 208 [3

253 ]), stomatitis (none vs five [9%]), and hand-foot syndrome (four [8%] vs none).

254 Hand-foot syndrome (HFS) is a common adverse effect of capeci

255 in both arms, although higher rates of hand-foot syndrome and diarrhea occurred in patients randomly

256 DSS1, a candidate gene for split hand/split foot syndrome, provides the ability to recognize RPA-coa

257 back in paraplegics by remapping missing leg/foot tactile sensations onto the skin of patients' forea

258 Thus, the 555-foot-tall Washington Monument often looms large against

259 were semantically unrelated (e.g., milk and foot) than when they were related (e.g., milk and juice)

260 across traces-may now be treated on an equal footing, thereby eliminating user-dependent trace select

261 ay during walking by adding stiffness to the foot through shoes and insoles, and characterized the re

262 CODD lesions (n = 58) and healthy sheep foot tissues (n = 56) were analyzed by PCR for the three

263 Healthy foot tissues did not amplify BDD-associated Treponema ph

264 ons and 22/56 (39%) and 5/56 (9%) of healthy foot tissues, respectively.

265 eling places the technique on a quantitative footing to allow the response of the carbon electrode an

266 d with Wiwaxia's mollusc-like mouthparts and foot; together these point to a deep phylogenetic positi

267 ere to a variety of substrates, and tolerate foot traffic (>1000 steps) after moderate wear and heali

268 o change features of locomotion, such as the foot trajectory as well as diminished paw drag often obs

269 s and a non-infected neuroischaemic diabetic foot ulcer greater than 1 cm(2) and of grade IC or IIC (

270 Diabetic foot ulcer microbiota were found to exist in one of four

271 Diabetic foot ulceration is a severe complication of diabetes tha

272 Diabetic foot ulcers (DFUs) are a debilitating complication of di

273 Diabetic foot ulcers (DFUs) are a severe complication of diabetes

274 Diabetic foot ulcers (DFUs) threaten limbs and prompt hospitaliza

275 Diabetic foot ulcers (DFUs), a life-threatening complication of d

276 effect of NETosis on the healing of diabetic foot ulcers (DFUs).

277 Diabetic foot ulcers are serious and challenging wounds associate

278 S. aureus isolates from uninfected diabetic foot ulcers in French patients harbor a prophage, ROSA-l

279 Management of neuropathic foot ulcers in patients with diabetes (DFUs) has changed

280 e base for many aspects of the management of foot ulcers in people with diabetes is weak, and good-qu

281 ved wound closure of neuroischaemic diabetic foot ulcers without affecting safety after 20 weeks of t

282 namics of the microbiota colonizing diabetic foot ulcers, a common and costly complication of diabete

283 Critical limb ischemia (CLI), foot ulcers, former amputation, and impaired regeneratio

284 in the prevention and management of diabetic foot ulcers, including studies that focus on off-loading

285 e-associated infections, as well as infected foot ulcers, which often lead to amputation.

286 local treatment for neuroischaemic diabetic foot ulcers.

287 diabetes are attributed to deep infection of foot ulcers.

288 phropathy, 4.5% for neuropathy, and 5.7% for foot ulcers.

289 maintained in a commensal state in diabetic foot ulcers.

290 MRSA into the care of diabetic patients with foot ulcers.

291 ure in patients with neuroischaemic diabetic foot ulcers.

292 cerebellar Functional System score, Timed 25-Foot Walk Test, 9-Hole Peg Test (9-HPT), Symbol Digit Mo

293 By week 120, performance on the timed 25-foot walk worsened by 38.9% with ocrelizumab versus 55.1

294 however the sympathetic skin response of the foot was impaired and sweat gland innervation was reduce

295 peroneal nerve innervating the dorsum of the foot were recorded by microneurography in seven healthy

296 ermatome (laser Doppler flowmetry; dorsum of foot) were measured during whole-body cooling (mean skin

297 ell flux (laser Doppler flowmetry; dorsum of foot) were measured during whole-body cooling (water-per

298 es calcium influx via TRPV4 in the glial end foot, which regulates expression of Aqp4 and Kir4.1 gene

299 Although the coordination of the foot with the natural hands increased trial after trial,

300 d at acupoints of either Stomach Meridian of Foot-Yangming (SMFY) or Gallbladder Meridian of Foot-Sha