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1  as it relates to the occurrence of diabetic foot ulcer.
2 icient to prevent the occurrence of diabetic foot ulcer.
3 iological deficit in the nonhealing diabetic foot ulcer.
4 und healing deficiency, typified by diabetic foot ulcer.
5 serts to diabetic patients with a history of foot ulcer.
6 MRSA into the care of diabetic patients with foot ulcers.
7 phropathy, 4.5% for neuropathy, and 5.7% for foot ulcers.
8  maintained in a commensal state in diabetic foot ulcers.
9 ure in patients with neuroischaemic diabetic foot ulcers.
10 lammation predisposing to polyneuropathy and foot ulcers.
11 to develop therapies for nonhealing diabetic foot ulcers.
12 uction studies and occurrence of new plantar foot ulcers.
13  local treatment for neuroischaemic diabetic foot ulcers.
14 diabetes are attributed to deep infection of foot ulcers.
15                    Most wounds were diabetic foot ulcers (19.0%), venous leg ulcers (26.1%), and pres
16 namics of the microbiota colonizing diabetic foot ulcers, a common and costly complication of diabete
17 roving diabetic management and prevention of foot ulcers and neurogenic arthropathy.
18 viduals with a high ABI have higher odds for foot ulcers and neuropathy, as well as lower scores on s
19 crease amputations in patients with diabetic foot ulcers and possibly accelerate closure of venous ul
20                                              Foot ulcers are more likely to be of neuropathic origin,
21                                     Diabetic foot ulcers are serious and challenging wounds associate
22 tant to remember that rates of recurrence of foot ulcers are very high, being greater than 50% after
23                 People with diabetes develop foot ulcers because of neuropathy (sensory, motor, and a
24 tic neuropathy patients developed a diabetic foot ulcer by the one-year follow-up.
25                                     Diabetic foot ulcer (DFU) infections are challenging.
26                          Nonhealing diabetic foot ulcers (DFUs) are a common and costly complication
27                                     Diabetic foot ulcers (DFUs) are a debilitating complication of di
28                                     Diabetic foot ulcers (DFUs) are a severe complication of diabetes
29                                     Diabetic foot ulcers (DFUs) threaten limbs and prompt hospitaliza
30                                     Diabetic foot ulcers (DFUs), a leading cause of amputations, affe
31                                     Diabetic foot ulcers (DFUs), a life-threatening complication of d
32 effect of NETosis on the healing of diabetic foot ulcers (DFUs).
33 otential of S. aureus isolated from diabetic foot ulcers (DFUs).
34 the development and failure to heal diabetic foot ulcers (DFUs).
35 icantly predicted the occurrence of diabetic foot ulcer, even after controlling for demographic varia
36                Critical limb ischemia (CLI), foot ulcers, former amputation, and impaired regeneratio
37 s and a non-infected neuroischaemic diabetic foot ulcer greater than 1 cm(2) and of grade IC or IIC (
38 care behavior on the development of diabetic foot ulcer has received little empirical investigation.
39                                     Diabetic foot ulcers have been neglected in health-care research
40 graphic variables and the number of diabetic foot ulcer hospitalizations, however, the effect was non
41 ors (neuropathy severity, number of diabetic foot ulcer hospitalizations, insulin treatment, and peri
42 antly associated with the risk of developing foot ulcers (HR=1.04, 95% CI=1.01-1.07, p=0.004).
43 ehavior predicted the occurrence of diabetic foot ulcers in diabetic patients with neuropathy.
44  S. aureus isolates from uninfected diabetic foot ulcers in French patients harbor a prophage, ROSA-l
45                    Management of neuropathic foot ulcers in patients with diabetes (DFUs) has changed
46 e base for many aspects of the management of foot ulcers in people with diabetes is weak, and good-qu
47 in the prevention and management of diabetic foot ulcers, including studies that focus on off-loading
48 t, and while its pathogenic role in diabetic foot ulcers is difficult to establish, it may be a previ
49                                     Diabetic foot ulcer microbiota were found to exist in one of four
50  ABI group had significantly higher odds for foot ulcers (p < 0.005) and borderline associations with
51 al in a timely manner, for example, diabetic foot ulcers, pose a health, economic, and social problem
52 lling for demographic variables and diabetic foot ulcer predictors (neuropathy severity, number of di
53                 When infection complicates a foot ulcer, the combination can be limb or life-threaten
54 in and as a possible participant in diabetic foot ulcers, we used a selective medium to culture both
55 e-associated infections, as well as infected foot ulcers, which often lead to amputation.
56 ved wound closure of neuroischaemic diabetic foot ulcers without affecting safety after 20 weeks of t
57 ar has been implicated as a primary cause of foot ulcers, yet research is limited on the efficacy of

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