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1 as it relates to the occurrence of diabetic foot ulcer.
2 icient to prevent the occurrence of diabetic foot ulcer.
3 iological deficit in the nonhealing diabetic foot ulcer.
4 und healing deficiency, typified by diabetic foot ulcer.
5 serts to diabetic patients with a history of foot ulcer.
6 MRSA into the care of diabetic patients with foot ulcers.
7 phropathy, 4.5% for neuropathy, and 5.7% for foot ulcers.
8 maintained in a commensal state in diabetic foot ulcers.
9 ure in patients with neuroischaemic diabetic foot ulcers.
10 lammation predisposing to polyneuropathy and foot ulcers.
11 to develop therapies for nonhealing diabetic foot ulcers.
12 uction studies and occurrence of new plantar foot ulcers.
13 local treatment for neuroischaemic diabetic foot ulcers.
14 diabetes are attributed to deep infection of foot ulcers.
16 namics of the microbiota colonizing diabetic foot ulcers, a common and costly complication of diabete
18 viduals with a high ABI have higher odds for foot ulcers and neuropathy, as well as lower scores on s
19 crease amputations in patients with diabetic foot ulcers and possibly accelerate closure of venous ul
22 tant to remember that rates of recurrence of foot ulcers are very high, being greater than 50% after
35 icantly predicted the occurrence of diabetic foot ulcer, even after controlling for demographic varia
37 s and a non-infected neuroischaemic diabetic foot ulcer greater than 1 cm(2) and of grade IC or IIC (
38 care behavior on the development of diabetic foot ulcer has received little empirical investigation.
40 graphic variables and the number of diabetic foot ulcer hospitalizations, however, the effect was non
41 ors (neuropathy severity, number of diabetic foot ulcer hospitalizations, insulin treatment, and peri
44 S. aureus isolates from uninfected diabetic foot ulcers in French patients harbor a prophage, ROSA-l
46 e base for many aspects of the management of foot ulcers in people with diabetes is weak, and good-qu
47 in the prevention and management of diabetic foot ulcers, including studies that focus on off-loading
48 t, and while its pathogenic role in diabetic foot ulcers is difficult to establish, it may be a previ
50 ABI group had significantly higher odds for foot ulcers (p < 0.005) and borderline associations with
51 al in a timely manner, for example, diabetic foot ulcers, pose a health, economic, and social problem
52 lling for demographic variables and diabetic foot ulcer predictors (neuropathy severity, number of di
54 in and as a possible participant in diabetic foot ulcers, we used a selective medium to culture both
56 ved wound closure of neuroischaemic diabetic foot ulcers without affecting safety after 20 weeks of t
57 ar has been implicated as a primary cause of foot ulcers, yet research is limited on the efficacy of
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