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1 flock house virus, human rhinovirus-14, and foot and mouth disease virus.
2 n, livestock can be made less susceptible to foot and mouth disease virus.
3 ere experimentally infected via aerosol with foot-and-mouth disease virus.
4 xsackievirus, rhinovirus, enterovirus 71 and foot-and-mouth disease virus.
5 lved in genome packaging of the picornavirus foot-and-mouth disease virus.
6 ctive peptide (A20FMDV2) derived from VP1 of foot-and-mouth disease virus.
7 2A self-processing peptide derived from the foot-and-mouth disease virus.
8 polymerases from poliovirus, rhinovirus, and foot-and-mouth disease viruses.
9 sh-ble antibiotic resistance gene, with the foot and mouth disease virus 2A self-cleaving sequence p
10 peptide binding and explains the ability of foot-and-mouth disease virus 3C(pro) to cleave sequences
13 on mediated a salient genome segmentation of foot-and-mouth disease virus, an important animal pathog
15 nked to the gene encoding the 2A protease of foot-and-mouth disease virus and then inserted in frame
16 henotype has been documented for poliovirus, foot-and-mouth disease virus, and coxsackievirus B3 and
17 cally important members, such as poliovirus, foot-and-mouth disease virus, and endomyocarditis virus.
18 icornavirus family, including poliovirus and foot-and-mouth disease virus, are widespread pathogens o
19 AVPNLRGDLQVLAQKVART (A20FMDV2), derived from foot-and-mouth disease virus, as a potent inhibitor of a
23 n from genetic and epidemiological data in a Foot and Mouth Disease Virus (FMDV) veterinary outbreak
27 ck in secretion are induced by expression of foot-and-mouth disease virus (FMDV) 3C(pro) and that thi
30 ished data from transmission experiments for foot-and-mouth disease virus (FMDV) and African swine fe
31 s were tested in this study: one recognizing foot-and-mouth disease virus (FMDV) and another recogniz
32 IRESs) of encephalomyocarditis virus (EMCV), foot-and-mouth disease virus (FMDV) and other picornavir
34 the extent to which the genetic diversity of foot-and-mouth disease virus (FMDV) arising over the cou
36 shown that the leader proteinase (L(pro)) of foot-and-mouth disease virus (FMDV) blocks cap-dependent
39 use in stabilizing SAT2 vaccines.IMPORTANCE Foot-and-mouth disease virus (FMDV) causes a highly cont
46 for the differential laboratory detection of foot-and-mouth disease virus (FMDV) from viruses that ca
48 hin the RNA genome of all seven serotypes of foot-and-mouth disease virus (FMDV) has been developed.
52 ocked the replication of poliovirus (PV) and foot-and-mouth disease virus (FMDV) in a variety of cell
53 ociated with clearance versus persistence of foot-and-mouth disease virus (FMDV) in micro-dissected c
58 ole of T-lymphocyte subsets in recovery from foot-and-mouth disease virus (FMDV) infection in calves
61 ecades of investigation, the manner in which foot-and-mouth disease virus (FMDV) interacts with the i
62 shown that the leader proteinase (L(pro)) of foot-and-mouth disease virus (FMDV) interferes with the
63 iral vectors were constructed containing the foot-and-mouth disease virus (FMDV) internal ribosome en
68 One of the final steps in the maturation of foot-and-mouth disease virus (FMDV) is cleavage of the V
69 We have previously shown that replication of foot-and-mouth disease virus (FMDV) is highly sensitive
72 n the initiation of immune responses against foot-and-mouth disease virus (FMDV) is poorly understood
73 domestic animals with chemically inactivated foot-and-mouth disease virus (FMDV) is widely practiced
77 eta interferon [IFN-alpha/beta]) can inhibit foot-and-mouth disease virus (FMDV) replication in cell
78 and II IFNs have proven effective to inhibit foot-and-mouth disease virus (FMDV) replication in swine
79 g a comparative analysis, of 103 isolates of foot-and-mouth disease virus (FMDV) representing all sev
83 ents from human rhinovirus type 2 (HRV2) and foot-and-mouth disease virus (FMDV) to control the trans
84 previously demonstrated that the ability of foot-and-mouth disease virus (FMDV) to form plaques in c
88 The development of a serological test for foot-and-mouth disease virus (FMDV) which is quick and e
91 the integrin receptors on cultured cells for foot-and-mouth disease virus (FMDV), and high-efficiency
93 ily, and for several diverse species such as foot-and-mouth disease virus (FMDV), hemagglutinin (HA)
98 " We show that the key replication enzyme of foot-and-mouth disease virus (FMDV), the RNA-dependent R
99 s of representatives of several serotypes of foot-and-mouth disease virus (FMDV), we discovered a put
101 mutants were used to map antigenic sites on foot-and-mouth disease virus (FMDV), which resulted in t
111 , we apply the method to two UK epidemics of Foot-and-Mouth Disease Virus (FMDV): the 2007 outbreak,
115 n a 4H junction derived from domain 3 of the foot-and-mouth disease virus internal ribosome entry sit
116 (6) and 2 x 10(7) c.f.u./ml, indicating that foot-and-mouth disease virus IRES provides high-titer bi
118 a vector with a multiple cloning site 3' to foot-and-mouth disease virus IRES, was used to construct
119 of the O(1) British field strain serotype of foot-and-mouth disease virus is a high-affinity ligand f
122 from high affinity ligands of alpha v beta6 (foot-and-mouth-disease virus, latency associated peptide
123 ar to those of core catalytic domains of the foot-and-mouth disease virus leader protease and coronav
124 we use these methods to analyze data from a foot-and-mouth disease virus outbreak in the United King
125 the prominent G-H loop of the VP1 protein of foot-and-mouth disease virus, raised substantial levels
126 The larger picornavirus IRESs (those of foot-and-mouth disease virus, rhinovirus, encephalomyoca
127 Venus and a puromycin-resistant gene via the foot-and-mouth disease virus self-cleaving peptide T2A.
128 odels to predict the antigenic similarity in foot-and-mouth disease virus strains and in influenza st
129 ar viruses, including all seven serotypes of foot-and-mouth disease virus, two serotypes of vesicular
130 opy of the genome-linked protein, VPg wheras foot-and-mouth disease virus uniquely encodes three copi
131 imal pathogens: classical swine fever virus; foot-and-mouth disease virus; vesicular stomatitis virus
132 s in innate responses against infection with foot-and-mouth disease virus was analyzed on consecutive
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