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1 d at pressure-bearing sites of the mammalian footpad.
2 0 s on the bottom surface of a tumor-bearing footpad.
3 of Freund's complete adjuvant into the hind footpad.
4 ith AdZ.F(RGD) or AdZ subcutaneously via the footpad.
5 ensory neurons in the DRG that innervate the footpad.
6 suppressed in another anatomic location, the footpad.
7 swelling when mice received challenge in the footpad.
8 s following CFA injection in the adult mouse footpad.
9 ed with low doses of anti-CD3 Ab s.c. in the footpad.
10 mor or intracutaneously in the contralateral footpad.
11 ted HSV-1 replication in the DRG but not the footpad.
12 injection of viable M. leprae into each hind footpad.
13 ed by enumerating the M. leprae bacteria per footpad.
14 eaches the sensory nerve fibers in the mouse footpad.
15 ted onto microneedles and delivered to mouse footpad.
16 n after they were sensitized with SRW in the footpad.
17 a scaling similar to that of a natural gecko footpad.
18 onstruct and mutant-specific siRNAs to mouse footpads.
19 the tail and, by day 7, a thickening of the footpads.
20 thetic innervation of sweat glands in rodent footpads.
21 exclusively localized to the stress-bearing footpads.
22 after inoculation of guinea pig genitals and footpads.
23 essively up-regulated in WT but not in Tabby footpads.
24 itment to draining lymph nodes following the footpad administration of TLR4 and TLR5 agonists, is dra
25 B, and beta 2 receptors are expressed in rat footpads; alpha 1 and beta 2 receptors are localized spe
27 density of nonpeptidergic nociceptors in the footpad and exhibit enhanced sensitivity to mechanical f
29 with virulent EEE virus in their right rear footpad and were followed in a time-course study for 4 d
31 d susceptible C3H/HeJ (C3H) mice in the hind footpads and monitored arthritis development for 21 days
32 l structures indicated by absence of ventral footpads and presence of supernumerary ventral nails.
33 tin-3 impaired neutrophil recruitment in the footpads and the draining lymph nodes 1 d following infe
35 noma cells were implanted into the subcutis, footpad, and pancreas of syngeneic IFN-gamma(+/+) and IF
36 infected neutrophils than inoculation of the footpad, and these higher frequencies were associated wi
38 tion of claws, interdigital webbing, reduced footpads, and trans-differentiation of sweat glands into
39 neous vaccination with live parasites in the footpad are even greater than previously appreciated.
40 ting bands encircle the tail and digits, the footpads are thickened and scaled, and loricrin staining
41 TH response to mycobacterial proteins in the footpad assay, indicating that the accumulation of blood
42 sferred with recipient APC into a SCID mouse footpad, CD4(+) T cells were hyporesponsive in DTH to do
43 12, or 24 h prior to an otherwise lethal VEE footpad challenge were completely protected from death,
45 te topographically segregated targets in the footpad; cholinergic sympathetic axons innervate sweat g
47 on of adjuvants (CFA or Pam(3)CSK(4)) in the footpad decreases experimental autoimmune encephalomyeli
49 ory, SDR-MEM mice responded with an enhanced footpad delayed-type hypersensitivity response, and more
54 zed to the somatotopic representation of the footpad dermatome within the dorsal root ganglia and spi
55 gnificantly in murine sweat gland-containing footpads during the time period when the gland innervati
56 with an ED40 of 16 mg/kg in the carrageenan footpad edema (CFE) assay and caused no gastrointestinal
60 mice, VSV, injected subcutaneously into the footpad, entered the proximal lymph node, where it repli
62 by confirming previous observations made in footpad epidermis by showing that the full-length involu
64 gered the formation of sebaceous glands from footpad epidermis, in regions normally devoid of hair fo
65 odeling, we demonstrate that in normal mouse footpad epidermis, transition of KCs from basal epiderma
69 njected into the subcutaneous space of mouse footpads, film-embedded IgG were retained locally, with
70 also reveal similarities in propatagial and footpad form between basal paravians and modern birds, e
71 generated following s.c. immunization in one footpad generate secondary responses to soluble Ag given
73 h the pVR1012-Ag2 construct mounted a strong footpad hypersensitivity and their spleen cells secreted
74 coccidioidomycosis and elicited delayed-type footpad hypersensitivity responses in Coccidioides-immun
75 he recombinant Ag2-GST protein did not mount footpad hypersensitivity to C-ASWS or the recombinant Ag
79 usly (s.c.) into the dorsal area or the hind footpad in three-dose schedules; the PorB VRP-immunized
80 The comparable levels of parasites in the footpads in the two diet groups and the higher lymph nod
81 activation by deacylation before it left the footpad; in animals that lacked acyloxyacyl hydrolase, t
89 r) and become activated following a low-dose footpad infection, although the mechanism of activation
90 y, and in contrast to subcutaneous M. leprae footpad infection, systemic M. leprae-specific gamma int
92 ow important differences between genital and footpad infections, including independence of spread to
94 pontaneous metastasis model originating from footpad injection in severe combined immunodeficient mic
95 o elicit delayed-type hypersensitivity after footpad injection into mice previously immunized against
97 ort de novo generation of T(R) upon a single footpad injection of Ag mixed with a classic proinflamma
101 skin painting and twice more donor DC after footpad injection, whereas migrating DC expressed less C
102 sfer, DTH to H. pylori Ags was determined by footpad injection; gastritis and bacterial colonization
105 SV latency active promoter 2 (vector SLN) by footpad inoculation 2 weeks after STZ administration pro
109 tion results in enhanced infection following footpad inoculation of cowpox virus, a natural pathogen
110 or anti-B7-2 at the time of sensitization to footpad inoculation of Schistosoma mansoni eggs or induc
111 r results were obtained following ocular and footpad inoculation with KOS/62, a LAT deletion mutant i
112 ce of delayed-type hypersensitivity (DTH) by footpad inoculation with sterile H. pylori sonicate and
114 ose of these non-neuroinvasive viruses after footpad inoculation, but in cyclophosphamide-suppressed
115 genes was detected by 1 to 3 h following VRP footpad inoculation, reaching peak expression of >100-fo
116 ug susceptibility testing method using mouse footpad inoculation, which requires at least 6 months to
120 and Leishmania amazonensis leads to a healed footpad lesion, whereas co-infection of C57BL/6 (B6) mic
121 astigotes, in which the rapid development of footpad lesions was associated with an increasing number
122 owing inoculation of B. burgdorferi into the footpad, lymph node NK cells from susceptible C3H/HeJ (C
124 inistration of SC-560 in the rat carrageenan footpad model did not affect acute inflammation or hyper
128 nfiltrate within the draining lymph node and footpad of B6.TNF(-/-) mice resembled that of B6.WT mice
130 e 1 (HSV-1) infections initiated in the hind footpad of C57BL/6 (B6, H-2b) mice is dependent upon the
131 und that B16-F10 melanoma growth in the rear footpad of immunocompetent mice induces marked B cell ac
132 on of the NRF2 activator sulforaphane to the footpad of Krt16-/- mice prevented the development of PP
134 pon subcutaneous (s.c.) inoculation into the footpad of mice, a VEE vector containing the complete in
138 opolysaccharide (LPS) was injected into hind footpads of C3H/HeN mice; and AqH, collected at 6, 12, 2
140 metacyclic promastigotes inoculated into the footpads of genetically resistant C57BL/6 mice was studi
141 At 4 months, growth of the bacilli in the footpads of GKO mice plateaued a log(10) higher than tha
142 K16 expression is absent in the thinned footpads of irhom2(-/-) mice compared with irhom2(+/+)mi
143 sisting of granuloma Mphi harvested from the footpads of M. leprae-infected athymic nu/nu mice, were
144 with control DCs, isc DCs injected into the footpads of mice demonstrated enhanced migration, which
146 jection of Schistosoma mansoni eggs into the footpads of mice results in a localized Th2 cytokine res
148 ion) or coinjected with OVA-pulsed APCs into footpads of naive DO11.10 mice whose draining lymph node
150 ers (fLUC and DsRed, respectively), into the footpads of outbred CD-1 mice to simulate transmission b
151 nuclear cells (PBMCs) were injected into the footpads of severe combined immunodeficiency mice to mea
152 cells, but not 3T3control fibroblasts, into footpads of syngeneic and SCID mice results in lesions t
158 y, we demonstrated that coinfection of mouse footpads or rabbit eyes with rAAV vectors and HSV-1 resu
159 ear, subcutaneous (s.c.) inoculation of the footpad, or inoculation of the peritoneal cavity (intrap
165 of sections of M. leprae-infected nude mouse footpads resulted in strongly positive staining in macro
169 PPK onset is preceded by oxidative stress in footpad skin of Krt16-/- mice and correlates with an ina
171 of the suprabasal layer of the rat and human footpad/sole epidermis in both immunoblotting and immuno
172 o-cultures, and extracts of gland-containing footpads stimulated tyrosine phosphorylation of LIFRbeta
173 ermore, extracts from sweat gland-containing footpads suppressed BH4 in cultured mouse sympathetic ne
177 sitivity, CP-481,715 significantly inhibited footpad swelling and decreased the amount of IFN-gamma a
178 type hypersensitivity responses by measuring footpad swelling and measuring in vitro proliferation of
180 ens after resolving this infection: enhanced footpad swelling in response to intradermal C. albicans
181 nificantly reduced magnitude and duration of footpad swelling observed in VCAM-1 mutant mice compared
185 meters measured, it did cause a reduction in footpad swelling when mice received challenge in the foo
190 e operative in delayed-type hypersensitivity footpad-swelling reaction and in debilitating meningitis
193 ce were inoculated with CHIKV SL15649 in the footpad, they displayed reduced weight gain and swelling
194 we observed that inflammation as measured by footpad thickness and neutrophil recruitment occurred in
198 ation of 51Cr-labeled, skin-derived DCs from footpads to draining LNs by 50% (n = 9, p < 0.005).
200 negative effect of plasminogen deficiency on footpad tumor growth was entirely relieved by superimpos
201 ents, single cells, and emboli shed from the footpad tumor were easily distinguished with the labeled
202 in tumors from plasminogen-deficient mice or footpad tumors from mice that also lacked fibrinogen.
203 icrovascular thrombi were commonplace within footpad tumors from plasminogen-deficient mice, whereas
206 node alterations, as macrophages infiltrated footpad tumors, whereas lymphocytes accumulated in tumor
209 inflammatory s.c. challenge in contralateral footpads, unlike lymphadenectomized mice lacking the ori
211 to mouse skin by intradermal injection into footpads using in vivo bioluminescence imaging over mult
213 LLC tumors--T241 fibrosarcoma growth in the footpad was also restricted by plasminogen deficiency in
215 the DLN after injection of Ag and CFA into a footpad was dramatically reduced after FTY720 treatment.
216 ensory and sympathetic fibers arrived in the footpad was not affected, the normal partitioning of axo
217 autonomic innervation of sweat glands in the footpads was significantly reduced in db/db mice compare
221 fter immunization with rrPorB in Ribi in the footpad while the same vaccine given in the dorsal area
222 e we report C57BL/6 WT mice infected via the footpad with 10(3) to 10(6) CFU of Brucella spp. display
223 on of 3-week-old weanling BALB/c mice in the footpad with DENV2 VRP resulted in high levels of DENV-s
227 kout (KO) BALB/c mice were sensitized in the footpad with short ragweed (SRW) allergen and challenged
231 y, athymic (nu/nu) mice were infected in the footpads with Mycobacterium leprae and fed a linoleic ac
232 Mice were infected orally or in the hind footpads with reovirus, and the repertoires of TCR beta-
233 cal allodynia developed in the corresponding footpad within two weeks and persisted throughout the ex
234 observed that following low-dose challenge, footpads without apparent lesions provided an efficient
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