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1 ere assigned based on percentage of expected forced expiratory volume.
2 FEV1/forced vital capacity (FVC) ratio, and forced expiratory volume after exhaling 75% of vital cap
3 scores and lung parameters such as decreased forced expiratory volume and increased residual volume c
5 values were significantly increased, whereas forced expiratory volume at 0.5 seconds (FEV(0.5)) signi
6 in baseline airflow were not significant for forced expiratory volume at 0.5 seconds (mean z score fo
9 quantitative spirometric indices, including forced expiratory volume at 1 s (FEV(1)) and the ratio o
10 is was linearly associated with the ratio of forced expiratory volume at 1 s to forced vital capacity
13 ypes including moderate airflow obstruction [forced expiratory volume at one second (FEV(1)) < 60% pr
14 n quantitative spirometric indices including forced expiratory volume at one second (FEV(1)) and the
15 49); resting Pa(O(2)), 0.97 (0.90-1.05); and forced expiratory volume at one second as a percent of p
17 In those with low predicted postoperative forced expiratory volume during first second (FEV(1)) or
18 ciation with the pulmonary function measure--forced expiratory volume (FEV(1)) % of predicted value.
19 on was estimated by calculating the ratio of forced expiratory volume (FEV) after 1 second (FEV1)/for
20 ort uses symptoms, exacerbation history, and forced expiratory volume (FEV1)% to categorise patients
21 stage 2 airflow limitation (ratio of FEV1 to forced expiratory volume [FEV1/FVC] less than 70% plus F
22 ican Thoracic Society criteria, based on the forced expiratory volume/forced vital capacity (FEV1/FVC
23 ene, has been previously associated with the forced expiratory volume/forced vital capacity ratio.
24 high-density lipoprotein (HDL) cholesterol, forced expiratory volume, grip strength, HbA1c, longevit
26 lung function measures as z scores at birth (forced expiratory volume in 0.5 seconds [FEV0.5], forced
27 between groups except for the mean change in forced expiratory volume in 0.5 seconds, which was 38 mL
29 pronounced, with boys demonstrating reduced forced expiratory volume in 0.5 seconds/forced vital cap
31 h Arg117His-CFTR and percentage of predicted forced expiratory volume in 1 s (% predicted FEV1) of at
32 , height, body-mass index, percent predicted forced expiratory volume in 1 s (%FEV1), risk of Pseudom
33 - 37.4 vs. 396.2 +/- 32.1 L/min; P = 0.007), forced expiratory volume in 1 s (75.2 +/- 4.6 vs. 88.4 +
34 e change from baseline in pre-bronchodilator forced expiratory volume in 1 s (FEV(1) ); eosinophilic
35 forced vital capacity (FVC) (P </= 0.01) and forced expiratory volume in 1 s (FEV(1)) (P </= 0.05) (m
36 anges correlated with the late-phase fall in forced expiratory volume in 1 s (FEV(1)) after whole-lun
37 y endpoints were change in prebronchodilator forced expiratory volume in 1 s (FEV(1)) and the rate of
39 ial demonstrated significant improvements in forced expiratory volume in 1 s (FEV(1)) from baseline,
40 dy, significantly improves prebronchodilator forced expiratory volume in 1 s (FEV(1)) in a subset of
41 ge caused an early change (0-2 h) in minimum forced expiratory volume in 1 s (FEV(1)) of -1.091 l (95
42 e relation between dietary food patterns and forced expiratory volume in 1 s (FEV(1)) or respiratory
43 history of 10 or more pack-years, a ratio of forced expiratory volume in 1 s (FEV(1)) to forced vital
44 mass (FFM), arm muscle circumference (AMC), forced expiratory volume in 1 s (FEV(1)), and functional
46 d in 107 patients with stable COPD [x +/- SD forced expiratory volume in 1 s (FEV(1)): 34.5 +/- 16.5]
47 had substantial deficits in 8-year growth of forced expiratory volume in 1 s (FEV(1), -81 mL, p=0.01
50 point was change from baseline at week 12 in forced expiratory volume in 1 s (FEV1 in L) in patients
51 dication, were enrolled in pairs matched for forced expiratory volume in 1 s (FEV1) according to whet
53 utcomes were on-treatment rate of decline in forced expiratory volume in 1 s (FEV1) and a composite o
54 irometrically; that is, as the ratio between forced expiratory volume in 1 s (FEV1) and forced vital
55 ble and evidence for major genes influencing forced expiratory volume in 1 s (FEV1) and forced vital
56 ) Normative Aging Study whose lung function [forced expiratory volume in 1 s (FEV1) and forced vital
59 sociation study (GWAS) so far (n=48,201) for forced expiratory volume in 1 s (FEV1) and the ratio of
60 secondary endpoints were pre-bronchodilator forced expiratory volume in 1 s (FEV1) and total asthma
61 y secondary endpoints were prebronchodilator forced expiratory volume in 1 s (FEV1) and total asthma
62 ol was non-inferior to tiotropium for trough forced expiratory volume in 1 s (FEV1) at week 12 (prima
63 ere, upper lobe-predominant emphysema with a forced expiratory volume in 1 s (FEV1) between 20% and 4
64 ed 40-80 years and had a post-bronchodilator forced expiratory volume in 1 s (FEV1) between 50% and 7
65 Biobank, from the middle and extremes of the forced expiratory volume in 1 s (FEV1) distribution amon
66 Although documents have traditionally used forced expiratory volume in 1 s (FEV1) for staging, clin
67 been associated with accelerated decline in forced expiratory volume in 1 s (FEV1) in patients with
69 randomly assigned (1:1) adults with COPD, a forced expiratory volume in 1 s (FEV1) less than 50% pre
70 ystic fibrosis, age at least 18 years, and a forced expiratory volume in 1 s (FEV1) of 40% or more th
72 symptomatic asthma and a pre-bronchodilator forced expiratory volume in 1 s (FEV1) of 60-90% predict
73 fibrosis, aged 8-18 years, and with a median forced expiratory volume in 1 s (FEV1) of 61% (range 33-
74 gnosis of cystic fibrosis, percent predicted forced expiratory volume in 1 s (FEV1) of 70 or more, an
75 d with a postbronchodilator reversibility in forced expiratory volume in 1 s (FEV1) of at least 12% a
76 onic obstructive pulmonary disease who had a forced expiratory volume in 1 s (FEV1) of less than 50%
77 gible patients had COPD, post-bronchodilator forced expiratory volume in 1 s (FEV1) of less than 50%,
78 e patients with COPD had post-bronchodilator forced expiratory volume in 1 s (FEV1) of lower than 50%
79 efficacy endpoint of both studies was trough forced expiratory volume in 1 s (FEV1) on day 169, which
80 r study 1 was maximum percentage decrease in forced expiratory volume in 1 s (FEV1) over 4-10 h after
81 lternative to and more sensitive method than forced expiratory volume in 1 s (FEV1) to assess treatme
82 -reported asthma and a postbronchodilatatory forced expiratory volume in 1 s (FEV1) to forced vital c
84 lowed up by questionnaires until age 5, when forced expiratory volume in 1 s (FEV1) was measured by s
86 hen adjusted for sex, body-mass index (BMI), forced expiratory volume in 1 s (FEV1), and PA:A greater
87 condary endpoints included prebronchodilator forced expiratory volume in 1 s (FEV1), Asthma Control Q
89 duals, we studied genome-wide association of forced expiratory volume in 1 s (FEV1), forced vital cap
90 hese functional volumes were correlated with forced expiratory volume in 1 s (FEV1), forced vital cap
91 and pulmonary function levels, including the forced expiratory volume in 1 s (FEV1), in general popul
93 ve comparator groups, as was the decrease in forced expiratory volume in 1 s (net difference -0.038 L
94 onary function testing demonstrated improved forced expiratory volume in 1 s (p=0.003) in the hMSC-tr
96 IL-25(+) cells correlated inversely with the forced expiratory volume in 1 s (r = -0.639; P = 0.01).
97 ose-ranging study in 424 patients with COPD (forced expiratory volume in 1 s [FEV(1)] 46.8% of predic
98 ntial difference; sweat chloride >40 mmol/L; forced expiratory volume in 1 s [FEV1] >/= 40% and </= 9
99 ithin 1 year (defined as post-bronchodilator forced expiratory volume in 1 s [FEV1] to forced vital c
100 ung function measures sequentially (ratio of forced expiratory volume in 1 s [FEV1] to forced vital c
101 te asthma were enrolled in pairs matched for forced expiratory volume in 1 s and ethnic origin, accor
104 xidant diet had a lower percentage predicted forced expiratory volume in 1 s and percentage predicted
105 ower mean residual forced vital capacity and forced expiratory volume in 1 s in men after adjustment
106 and to decrease future risk (as predicted by forced expiratory volume in 1 s level and exacerbations
107 iciency (serum concentration <11 muM) with a forced expiratory volume in 1 s of 35-70% (predicted).
109 ase in FVC (forced vital capacity) and FEV1 (forced expiratory volume in 1 s) of 0.03 L [95% confiden
114 cipants, methacholine PC20 (20% reduction in forced expiratory volume in 1 s; a prespecified secondar
115 ed as at least a 12% and 200 mL reduction in forced expiratory volume in 1 sec (FEV(1)) or forced vit
117 ) on specific airway resistance (sR(aw)) and forced expiratory volume in 1 sec (FEV1) before and afte
118 ng mass was the only measure associated with forced expiratory volume in 1 sec (FEV1) decline, with e
119 We measured forced vital capacity (FVC), forced expiratory volume in 1 sec (FEV1), and blood DNA
120 in D (25-OHD) levels and pulmonary function (forced expiratory volume in 1 sec [FEV(1)] %predicted) w
121 , we separated patients with an obstructive (forced expiratory volume in 1 sec [FEV(1)]: vital capaci
122 ive decline in pulmonary allograft function (forced expiratory volume in 1 sec [FEV1]) in absence of
123 er lung function [average reduction in FEV1 (forced expiratory volume in 1 sec) for a 10% increase in
124 at both moderate and elevated temperatures (forced expiratory volume in 1 sec, -12.4% vs. -7.5%, p >
128 d with forced vital capacity % predicted and forced expiratory volume in 1 second % predicted (P < 0.
129 Extended donor recipients also had lower forced expiratory volume in 1 second % predicted at 1 ye
130 talized 58 times and had significantly worse forced expiratory volume in 1 second ( FEV1 forced expir
131 intake was also associated with deficits in forced expiratory volume in 1 second (-2.7%, 95% confide
132 2) and 4.0 percentage points lower predicted forced expiratory volume in 1 second (95% CI, -6.6 to -1
133 flation, r = -0.8; 95% CI: -0.94, 0.42), and forced expiratory volume in 1 second (airway obstruction
134 icant correlation with the percent predicted forced expiratory volume in 1 second (FEV(1)%) (r = 0.67
135 e of total fiber intake had a 60.2-ml higher forced expiratory volume in 1 second (FEV(1)) (p for tre
136 with findings at quantitative CT (r = 0.75), forced expiratory volume in 1 second (FEV(1)) (r = -0.68
137 n whose mothers had received vitamin A had a forced expiratory volume in 1 second (FEV(1)) and a forc
138 African ancestry was inversely related to forced expiratory volume in 1 second (FEV(1)) and forced
139 the authors developed prediction models for forced expiratory volume in 1 second (FEV(1)) and the pr
140 val, Pseudomonas aeruginosa acquisition, and forced expiratory volume in 1 second (FEV(1)) below 70%
141 nt predicted forced vital capacity (FVC) and forced expiratory volume in 1 second (FEV(1)) compared w
142 nts who quit smoking in the first year, mean forced expiratory volume in 1 second (FEV(1)) expressed
143 was the relative change in prebronchodilator forced expiratory volume in 1 second (FEV(1)) from basel
145 tients were at least 40 years of age, with a forced expiratory volume in 1 second (FEV(1)) of 70% or
146 were symptomatic, had a post-bronchodilator forced expiratory volume in 1 second (FEV(1)) of 80% or
147 Using linear mixed models, we analyzed the forced expiratory volume in 1 second (FEV(1)) of both ac
148 When compared with smokers with the largest forced expiratory volume in 1 second (FEV(1)) to forced
151 ated with clinical improvement measured with forced expiratory volume in 1 second (FEV(1)), forced vi
152 y function impairment, as evidenced by lower forced expiratory volume in 1 second (FEV(1)), was assoc
153 of 75 g of glucose was inversely related to forced expiratory volume in 1 second (FEV(1)), with a di
157 In mixed models, the rate of decline in forced expiratory volume in 1 second (FEV(1))/forced vit
159 re decline in lung function measurements for forced expiratory volume in 1 second (FEV1) (388 mL), fo
160 siblings (P = 0.010) and is associated with forced expiratory volume in 1 second (FEV1) (P = 0.030).
161 , P = .02) were significant variables, while forced expiratory volume in 1 second (FEV1) and airway d
162 ions between exposures and annual changes in forced expiratory volume in 1 second (FEV1) and FEV1 as
163 ermined by their history and their values of forced expiratory volume in 1 second (FEV1) and forced v
164 e, 6574 had COPD, defined as a ratio between forced expiratory volume in 1 second (FEV1) and forced v
165 io was used to assess abdominal obesity, and forced expiratory volume in 1 second (FEV1) and forced v
166 iation between lung function, as measured by forced expiratory volume in 1 second (FEV1) and forced v
167 5 years of age, measured as the increases in forced expiratory volume in 1 second (FEV1) and forced v
170 icacy end points were percent changes in the forced expiratory volume in 1 second (FEV1) and the 6-mi
171 by measuring forced vital capacity (FVC) and forced expiratory volume in 1 second (FEV1) at baseline,
172 from baseline in the percentage of predicted forced expiratory volume in 1 second (FEV1) at week 24.
173 ys, with a difference of 0.079 (P=0.01); the forced expiratory volume in 1 second (FEV1) before bronc
174 solute change in the percentage of predicted forced expiratory volume in 1 second (FEV1) from the bas
175 ents with lymphangioleiomyomatosis, the mean forced expiratory volume in 1 second (FEV1) increased by
177 omonas aeruginosa for 1 or more years, and a forced expiratory volume in 1 second (FEV1) of 30% or mo
178 eal-life study was to compare the changes in forced expiratory volume in 1 second (FEV1) of omalizuma
179 ght to result from an accelerated decline in forced expiratory volume in 1 second (FEV1) over time.
181 te change in the percentage of the predicted forced expiratory volume in 1 second (FEV1) through week
182 pulmonary disease (COPD) requires a ratio of forced expiratory volume in 1 second (FEV1) to forced vi
183 and moderately correlated with the ratio of forced expiratory volume in 1 second (FEV1) to forced vi
184 bstruction by spirometry, using the ratio of forced expiratory volume in 1 second (FEV1) to forced vo
185 .77; P < .0001) with percentage predicted of forced expiratory volume in 1 second (FEV1) was observed
186 men with skin lesions, the average adjusted forced expiratory volume in 1 second (FEV1) was reduced
187 h intermittent culture positivity and higher forced expiratory volume in 1 second (FEV1) were most li
188 deficits in flows that were larger in girls (forced expiratory volume in 1 second (FEV1), -3.3%, 95%
189 miquantitative food frequency questionnaire, forced expiratory volume in 1 second (FEV1), and respira
190 ardised format (CRQ-SAS), pre-bronchodilator forced expiratory volume in 1 second (FEV1), and safety.
191 effect of either benralizumab regimen on the forced expiratory volume in 1 second (FEV1), as compared
192 es were changes from baseline to 6 months in forced expiratory volume in 1 second (FEV1), forced vita
194 h and decline on the basis of graphs showing forced expiratory volume in 1 second (FEV1), representin
199 s ratio (OR) = 3.10, 95% CI: 1.65, 5.78) and forced expiratory volume in 1 second (OR = 2.35, 95% CI:
200 alized subjects had significantly worse FEV1 forced expiratory volume in 1 second (P = .02) and (3)He
201 ficant correlation (P < .01) with changes in forced expiratory volume in 1 second (r = 0.70), forced
202 forced expiratory volume in 1 second ( FEV1 forced expiratory volume in 1 second ) (P < .0001), CT R
203 measure of lung function (prebronchodilator forced expiratory volume in 1 second [FEV(1)]) in more t
204 adiographic and PFT (percentage of predicted forced expiratory volume in 1 second [FEV(1)], percentag
205 year, asthma hospitalization in prior year, forced expiratory volume in 1 second [FEV1 ; FEV1 <65% v
206 0 to 80 years of age, had COPD (defined by a forced expiratory volume in 1 second [FEV1] of less than
207 Spirometry (forced vital capacity [FVC] and forced expiratory volume in 1 second [FEV1]) was perform
209 ss index grade, respiratory function levels (forced expiratory volume in 1 second [FEV1], forced vita
210 viation of Pao2 correlated with PFT metrics (forced expiratory volume in 1 second [FEV1]/forced vital
211 oxide (Dlco%) than with airflow obstruction (forced expiratory volume in 1 second [FEV1]/vital capaci
212 s syndrome, anti-TNFalpha treatment improved forced expiratory volume in 1 second and 6-min walk dist
214 Trolox)-equivalent antioxidant capacity with forced expiratory volume in 1 second and forced vital ca
215 showed an inverse association of TBARS with forced expiratory volume in 1 second and forced vital ca
217 -1998 the authors studied the association of forced expiratory volume in 1 second and forced vital ca
220 ated 5-year survivorship model included age, forced expiratory volume in 1 second as a percentage of
221 atients (101 male; mean age, 88.5 years; and forced expiratory volume in 1 second as percent predicte
223 ion of methacholine required to decrease the forced expiratory volume in 1 second by 20% (PC20).
224 hanges in nasal examinations, or declines in forced expiratory volume in 1 second during the celecoxi
226 ion tests revealed stable vital capacity and forced expiratory volume in 1 second in all cases after
227 bacco in the home was weakly associated with forced expiratory volume in 1 second in girls, accountin
229 e model that predicted PRM gas trapping, the forced expiratory volume in 1 second normalized to the f
231 piratory volume in 1 second and the ratio of forced expiratory volume in 1 second to forced vital cap
232 e (renal), serum albumin (hepatic), ratio of forced expiratory volume in 1 second to forced vital cap
233 difference in the z scores for the ratio of forced expiratory volume in 1 second to forced vital cap
234 % CI: -0.93, -0.64; P<.001) and the ratio of forced expiratory volume in 1 second to forced vital cap
235 d mean reduction from baseline in the trough forced expiratory volume in 1 second was 38 ml greater i
236 were available for 84%; when available, mean forced expiratory volume in 1 second was 51% predicted (
237 ge from baseline in the percent of predicted forced expiratory volume in 1 second was 8.7% (range, 2.
239 change in SUV(max) and percentage predicted forced expiratory volume in 1 second was negatively corr
240 Patients with an irreversible decline in forced expiratory volume in 1 second were identified and
241 owth rates of maximal midexpiratory flow and forced expiratory volume in 1 second were reduced by app
242 y in poor pulmonary function patients [FEV1 (forced expiratory volume in 1 second) or DLCO (diffusion
243 (exacerbations, asthma-control days, and the forced expiratory volume in 1 second) to determine wheth
244 ate, renal dysfunction, atrial fibrillation, forced expiratory volume in 1 second, and C-reactive pro
245 of response included forced vital capacity, forced expiratory volume in 1 second, and diffusing capa
246 forced vital capacity, total lung capacity, forced expiratory volume in 1 second, and diffusing lung
247 ity of the lungs for carbon monoxide (DLCO), forced expiratory volume in 1 second, and forced vital c
248 measures of respiratory function, including forced expiratory volume in 1 second, forced vital capac
249 of effect modification by race/ethnicity for forced expiratory volume in 1 second, forced vital capac
250 correlated with the percentage of predicted forced expiratory volume in 1 second, or FEV(1), (r = -0
254 e in 1 second, forced vital capacity, or the forced expiratory volume in 1 second/forced vital capaci
255 also decreased in patients with HLA-Ab (mean forced expiratory volume in 1 second=49%) when compared
257 onchial hyperresponsiveness to methacholine, forced expiratory volume in 1s (FEV1 ) and atopy and ast
258 The primary outcome was the pretreatment forced expiratory volume in 1s (FEV1) after 12 months tr
260 ction (specific airways resistance [sR(aw)], forced expiratory volume in 1s [FEV1]) and airway reacti
261 rd some measure of expiration, such as FEV1 (Forced Expiratory Volume in 1s), because airway constric
262 preceding day was associated with a reduced forced expiratory volume in 8-yr-olds; -32.4 ml; 95% CI:
263 ent contributors to ACQ-6 score were R20 and forced expiratory volume in one second (% pred.), and th
264 ine the relationship of air pollution to the forced expiratory volume in one second (FEV(1)) and othe
265 asthma-related traits: (i) percent predicted forced expiratory volume in one second (FEV(1)); (ii) do
266 /= 0.35 kUA /L) (n = 418) and lung function [forced expiratory volume in one second (FEV1 ) and force
267 val included an older age (P=0.003), a lower forced expiratory volume in one second (FEV1) (P=0.004),
269 hypertonic saline with placebo improved the forced expiratory volume in one second (FEV1) between th
270 defined by the lowest or highest quartile of forced expiratory volume in one second (FEV1), respectiv
271 ation; secondary measures included change in forced expiratory volume in one second (FEV1), weight, a
273 odest reduction in forced vital capacity and forced expiratory volume in one second following broncho
274 ry of cancer, family history of lung cancer, forced expiratory volume in one second percent predicted
275 albuterol Treatment 1, the median change in forced expiratory volume in one second was 14.6% in the
276 cations, vital signs, oxygen saturation, and forced expiratory volume in one second were not differen
277 ization, serum total immunoglobulin E (IgE), forced expiratory volume in one-second (FEV1) and forced
278 the results from a standard spirometry test, forced expiratory volume in one-second percent (FEV1 %),
279 ence in change for the secondary outcomes of forced expiratory volume in the first second (0.0 L; 95%
280 clinically important lung-function measures: forced expiratory volume in the first second (FEV(1)) an
281 The primary outcome measure was change in forced expiratory volume in the first second (FEV1) at 2
282 We performed GWAS of the rate of change in forced expiratory volume in the first second (FEV1) in 1
283 ounds, and a prolonged expiratory phase; his forced expiratory volume in the first second (FEV1) is 1
284 ratory Questionnaire (MCID, 4) and change in forced expiratory volume in the first second (FEV1; MCID
285 de Park led to an increase in lung function (forced expiratory volume in the first second [FEV1] and
287 very 6 months for 5 years and measured child forced expiratory volume in the first second of expirati
288 f 5 patients, 4 (80%) had improvement of the forced expiratory volume in the first second of expirati
290 difference for change in postbronchodilator forced expiratory volume in the first second of expirati
291 ty criteria included age of 6 to 18 years, a forced expiratory volume in the first second of expirati
292 -3.05; P < .001; I(2) = 0%), lowest baseline forced expiratory volume in the first second of expirati
293 mean BMI of 21.6 [IQR, 18.2-26.1], mean [SD] forced expiratory volume in the first second of expirati
294 (95% CI, 0.03 L to infinity) (P = .002) for forced expiratory volume in the first second, +21 m (95%
295 ustment for age, sex, race, body mass index, forced expiratory volume in the first second, pack-years
297 tcome; secondary outcomes included change in forced expiratory volume, Mini Asthma Quality of Life Qu
299 s of peak VO2 were peak heart rate (r=0.33), forced expiratory volume (r=0.33), pulmonary hypertensio
300 After adjustment for sex, age, and length, forced expiratory volume was significantly reduced by an
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