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1 tation: "current FEV1/ baseline FEV1" (FEV1: forced expiratory volume in 1 second).
2 alth care use and lung function, measured by forced expiratory volume in 1 second.
3 x, estimated glomerular filtration rate, and forced expiratory volume in 1 second.
4 he severity of asthma and inversely with the forced expiratory volume in 1 second.
5 intake was also associated with deficits in forced expiratory volume in 1 second (-2.7%, 95% confide
6 also decreased in patients with HLA-Ab (mean forced expiratory volume in 1 second=49%) when compared
7 as measured by percentage of predicted FEV1 (forced expiratory volume in 1 second) (51% in patients r
9 2) and 4.0 percentage points lower predicted forced expiratory volume in 1 second (95% CI, -6.6 to -1
10 flation, r = -0.8; 95% CI: -0.94, 0.42), and forced expiratory volume in 1 second (airway obstruction
11 s syndrome, anti-TNFalpha treatment improved forced expiratory volume in 1 second and 6-min walk dist
13 showed an inverse association of TBARS with forced expiratory volume in 1 second and forced vital ca
15 -1998 the authors studied the association of forced expiratory volume in 1 second and forced vital ca
17 Trolox)-equivalent antioxidant capacity with forced expiratory volume in 1 second and forced vital ca
20 ate, renal dysfunction, atrial fibrillation, forced expiratory volume in 1 second, and C-reactive pro
21 of response included forced vital capacity, forced expiratory volume in 1 second, and diffusing capa
22 forced vital capacity, total lung capacity, forced expiratory volume in 1 second, and diffusing lung
23 ity of the lungs for carbon monoxide (DLCO), forced expiratory volume in 1 second, and forced vital c
24 ated 5-year survivorship model included age, forced expiratory volume in 1 second as a percentage of
25 atients (101 male; mean age, 88.5 years; and forced expiratory volume in 1 second as percent predicte
28 hanges in nasal examinations, or declines in forced expiratory volume in 1 second during the celecoxi
30 icant correlation with the percent predicted forced expiratory volume in 1 second (FEV(1)%) (r = 0.67
31 e of total fiber intake had a 60.2-ml higher forced expiratory volume in 1 second (FEV(1)) (p for tre
32 with findings at quantitative CT (r = 0.75), forced expiratory volume in 1 second (FEV(1)) (r = -0.68
33 n whose mothers had received vitamin A had a forced expiratory volume in 1 second (FEV(1)) and a forc
34 African ancestry was inversely related to forced expiratory volume in 1 second (FEV(1)) and forced
35 the authors developed prediction models for forced expiratory volume in 1 second (FEV(1)) and the pr
36 val, Pseudomonas aeruginosa acquisition, and forced expiratory volume in 1 second (FEV(1)) below 70%
37 nt predicted forced vital capacity (FVC) and forced expiratory volume in 1 second (FEV(1)) compared w
38 nts who quit smoking in the first year, mean forced expiratory volume in 1 second (FEV(1)) expressed
39 was the relative change in prebronchodilator forced expiratory volume in 1 second (FEV(1)) from basel
41 tients were at least 40 years of age, with a forced expiratory volume in 1 second (FEV(1)) of 70% or
42 were symptomatic, had a post-bronchodilator forced expiratory volume in 1 second (FEV(1)) of 80% or
43 Using linear mixed models, we analyzed the forced expiratory volume in 1 second (FEV(1)) of both ac
44 When compared with smokers with the largest forced expiratory volume in 1 second (FEV(1)) to forced
47 ated with clinical improvement measured with forced expiratory volume in 1 second (FEV(1)), forced vi
48 y function impairment, as evidenced by lower forced expiratory volume in 1 second (FEV(1)), was assoc
49 of 75 g of glucose was inversely related to forced expiratory volume in 1 second (FEV(1)), with a di
55 measure of lung function (prebronchodilator forced expiratory volume in 1 second [FEV(1)]) in more t
56 adiographic and PFT (percentage of predicted forced expiratory volume in 1 second [FEV(1)], percentag
57 talized 58 times and had significantly worse forced expiratory volume in 1 second ( FEV1 forced expir
58 re decline in lung function measurements for forced expiratory volume in 1 second (FEV1) (388 mL), fo
59 siblings (P = 0.010) and is associated with forced expiratory volume in 1 second (FEV1) (P = 0.030).
60 end point was the extent of the decrease in forced expiratory volume in 1 second (FEV1) 10 minutes a
61 , P = .02) were significant variables, while forced expiratory volume in 1 second (FEV1) and airway d
62 ions between exposures and annual changes in forced expiratory volume in 1 second (FEV1) and FEV1 as
63 io was used to assess abdominal obesity, and forced expiratory volume in 1 second (FEV1) and forced v
64 iation between lung function, as measured by forced expiratory volume in 1 second (FEV1) and forced v
66 5 years of age, measured as the increases in forced expiratory volume in 1 second (FEV1) and forced v
67 ermined by their history and their values of forced expiratory volume in 1 second (FEV1) and forced v
68 e, 6574 had COPD, defined as a ratio between forced expiratory volume in 1 second (FEV1) and forced v
71 icacy end points were percent changes in the forced expiratory volume in 1 second (FEV1) and the 6-mi
73 by measuring forced vital capacity (FVC) and forced expiratory volume in 1 second (FEV1) at baseline,
74 from baseline in the percentage of predicted forced expiratory volume in 1 second (FEV1) at week 24.
75 ys, with a difference of 0.079 (P=0.01); the forced expiratory volume in 1 second (FEV1) before bronc
76 solute change in the percentage of predicted forced expiratory volume in 1 second (FEV1) from the bas
77 ents with lymphangioleiomyomatosis, the mean forced expiratory volume in 1 second (FEV1) increased by
78 of 607.5 mg/d for 2 weeks, prebronchodilator forced expiratory volume in 1 second (FEV1) increased fr
80 toxic death by univariate analysis included forced expiratory volume in 1 second (FEV1) less than 78
81 omonas aeruginosa for 1 or more years, and a forced expiratory volume in 1 second (FEV1) of 30% or mo
82 eal-life study was to compare the changes in forced expiratory volume in 1 second (FEV1) of omalizuma
83 ght to result from an accelerated decline in forced expiratory volume in 1 second (FEV1) over time.
85 te change in the percentage of the predicted forced expiratory volume in 1 second (FEV1) through week
86 pulmonary disease (COPD) requires a ratio of forced expiratory volume in 1 second (FEV1) to forced vi
87 and moderately correlated with the ratio of forced expiratory volume in 1 second (FEV1) to forced vi
88 bstruction by spirometry, using the ratio of forced expiratory volume in 1 second (FEV1) to forced vo
89 .77; P < .0001) with percentage predicted of forced expiratory volume in 1 second (FEV1) was observed
90 men with skin lesions, the average adjusted forced expiratory volume in 1 second (FEV1) was reduced
91 h intermittent culture positivity and higher forced expiratory volume in 1 second (FEV1) were most li
92 ssociation between lung function measured as forced expiratory volume in 1 second (FEV1) with 1) leve
93 efore age 40 had an age- and height-adjusted forced expiratory volume in 1 second (FEV1)(in liters) t
94 deficits in flows that were larger in girls (forced expiratory volume in 1 second (FEV1), -3.3%, 95%
95 miquantitative food frequency questionnaire, forced expiratory volume in 1 second (FEV1), and respira
96 ardised format (CRQ-SAS), pre-bronchodilator forced expiratory volume in 1 second (FEV1), and safety.
97 ma and compared with clinical improvement in forced expiratory volume in 1 second (FEV1), arterial pa
98 effect of either benralizumab regimen on the forced expiratory volume in 1 second (FEV1), as compared
99 es were changes from baseline to 6 months in forced expiratory volume in 1 second (FEV1), forced vita
101 ere was an average improvement of 47% in the forced expiratory volume in 1 second (FEV1), of 20% in a
102 h and decline on the basis of graphs showing forced expiratory volume in 1 second (FEV1), representin
103 ramic fibers on forced vital capacity (FVC), forced expiratory volume in 1 second (FEV1), the ratio o
110 year, asthma hospitalization in prior year, forced expiratory volume in 1 second [FEV1 ; FEV1 <65% v
111 g function (the mean percentage of predicted forced expiratory volume in 1 second [FEV1] improved fro
112 0 to 80 years of age, had COPD (defined by a forced expiratory volume in 1 second [FEV1] of less than
113 Spirometry (forced vital capacity [FVC] and forced expiratory volume in 1 second [FEV1]) was perform
115 ss index grade, respiratory function levels (forced expiratory volume in 1 second [FEV1], forced vita
116 viation of Pao2 correlated with PFT metrics (forced expiratory volume in 1 second [FEV1]/forced vital
117 oxide (Dlco%) than with airflow obstruction (forced expiratory volume in 1 second [FEV1]/vital capaci
118 e emphysema indexes and percentage predicted forced expiratory volume in 1 second, forced expiratory
119 measures of respiratory function, including forced expiratory volume in 1 second, forced vital capac
120 of effect modification by race/ethnicity for forced expiratory volume in 1 second, forced vital capac
121 dicted forced expiratory volume in 1 second, forced expiratory volume in 1 second/forced vital capaci
122 e in 1 second, forced vital capacity, or the forced expiratory volume in 1 second/forced vital capaci
123 ion tests revealed stable vital capacity and forced expiratory volume in 1 second in all cases after
124 bacco in the home was weakly associated with forced expiratory volume in 1 second in girls, accountin
126 e model that predicted PRM gas trapping, the forced expiratory volume in 1 second normalized to the f
128 s ratio (OR) = 3.10, 95% CI: 1.65, 5.78) and forced expiratory volume in 1 second (OR = 2.35, 95% CI:
129 y in poor pulmonary function patients [FEV1 (forced expiratory volume in 1 second) or DLCO (diffusion
130 correlated with the percentage of predicted forced expiratory volume in 1 second, or FEV(1), (r = -0
132 alized subjects had significantly worse FEV1 forced expiratory volume in 1 second (P = .02) and (3)He
133 forced expiratory volume in 1 second ( FEV1 forced expiratory volume in 1 second ) (P < .0001), CT R
134 d with forced vital capacity % predicted and forced expiratory volume in 1 second % predicted (P < 0.
135 Extended donor recipients also had lower forced expiratory volume in 1 second % predicted at 1 ye
136 ficant correlation (P < .01) with changes in forced expiratory volume in 1 second (r = 0.70), forced
137 piratory volume in 1 second and the ratio of forced expiratory volume in 1 second to forced vital cap
138 e (renal), serum albumin (hepatic), ratio of forced expiratory volume in 1 second to forced vital cap
139 difference in the z scores for the ratio of forced expiratory volume in 1 second to forced vital cap
140 % CI: -0.93, -0.64; P<.001) and the ratio of forced expiratory volume in 1 second to forced vital cap
141 angles (< 80 degrees) [corrected], decreased forced expiratory volume in 1 second to vital capacity r
142 (exacerbations, asthma-control days, and the forced expiratory volume in 1 second) to determine wheth
143 d mean reduction from baseline in the trough forced expiratory volume in 1 second was 38 ml greater i
144 were available for 84%; when available, mean forced expiratory volume in 1 second was 51% predicted (
145 ge from baseline in the percent of predicted forced expiratory volume in 1 second was 8.7% (range, 2.
147 change in SUV(max) and percentage predicted forced expiratory volume in 1 second was negatively corr
148 high levels of alcohol use, cholesterol, and forced expiratory volume in 1 second were associated wit
149 Patients with an irreversible decline in forced expiratory volume in 1 second were identified and
150 owth rates of maximal midexpiratory flow and forced expiratory volume in 1 second were reduced by app
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