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1 ials undergo fusion to form surface-damaging foreign body giant cells.
2 es including macrophages in the formation of foreign body giant cells.
3 adherent cells that included macrophages and foreign body giant cells.
4 Xid mice also failed to form multinucleated foreign body giant cells.
6 ytic macrophages, wound-healing macrophages, foreign body giant cells, and bone-resorbing osteoclasts
7 sia, encapsulation, mononuclear infiltrates, foreign body giant cells, and eosinophilic infiltrates.
8 ocyte-macrophage adhesion and fusion to form foreign body giant cells are provided by substrates with
9 eripheral blood derived macrophage adhesion, foreign body giant cell (FBGC) formation and inflammator
11 acterized by the presence of macrophages and foreign body giant cells (FBGC), can result in structura
14 s endocytic/phagocytic receptor in mediating foreign body giant cell formation at sites of chronic in
15 (MCP-1) was demonstrated to be required for foreign body giant cell formation in the foreign body re
17 is characterized by macrophage infiltration, foreign body giant cell formation, and fibrotic encapsul
18 IL-13 acts independently of IL-4 to promote foreign body giant cell formation, it may trigger a comm
20 anti-human IL-13 Abs inhibited IL-13-induced foreign body giant cell formation; the fusion-inducing e
21 ent studies involving mainly osteoclasts and foreign body giant cells have revealed a number of commo
22 ty on the formation of interleukin-4-induced foreign body giant cells in vitro Giant cell formation w
24 vascular occlusion and moderate intraluminal foreign body giant cell reaction; the acutely embolized
25 nstrated variable inflammatory reactions and foreign-body giant cell reaction and no angionecrosis or
26 tly as a result of degradation by an ongoing foreign-body giant cell reaction that peaked at 8-12 d p
27 monocyte adhesion and macrophage fusion into foreign-body giant cells while inducing adherent-macroph
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