ライフサイエンスコーパス: foreskin

1 penile shaft to 95.0% (kappa = 0.89) for the foreskin.

2 ible increased HIV-1 susceptibility of human foreskin.

3 important SIV-specific cellular immunity in foreskin.

4 ce potentially important immune responses in foreskin.

5 une responses to candidate HIV-1 vaccines in foreskin.

6 specific immune responses or inflammation in foreskin.

7 POT) assay in cell suspensions made from the foreskin.

8 dily found in cell suspensions made from the foreskin.

9 1.05) compared with sites more distal to the foreskin.

10 nile shaft, 16% from the glans, 28% from the foreskin, 17% from the scrotum, and 6% in urine.

11 sulcus (31.9% and 33.1%, P = 0.84), and the foreskin (33.3% and 28.6%, P = 0.74).

12 ong uncircumcised men, HPV prevalence in the foreskin (44%) was comparable to that in the glans/coron

13 we show that TLR3 expression was detected on foreskin, adult skin, and lung fibroblasts, and TLR3 lev

14 in primary human keratinocytes from neonatal foreskin and adult female skin.

15 HIV-1 DNA copies in foreskin and cervical mucosal tissue were compared and t

16 a complete population of immune cells in the foreskin and glans of normal RMs, although B cells were

17 In the foreskin and glans of SIV-infected RMs, although B cells

18 Both human foreskin and HSE successfully engrafted onto naive SCID

19 und in the epidermal basal layer of neonatal foreskin and in the follicular bulge region.

20 or (TGF)B1-dependent transdifferentiation of foreskin and tongue, but not gingival fibroblasts into m

21 16 and HPV18 in organotypic raft cultures of foreskin and vaginal keratinocytes as determined by miRN

22 f cultured human keratinocytes from neonatal foreskins and cultured murine dermal papilla cells from

23 Foreskins and melanoma cell cultures were irradiated wit

24 nvelopes prepared from human breast skin and foreskin, and from spontaneous and induced envelopes pre

25 nocyte cultures isolated from human neonatal foreskin, and human organotypic keratinocyte cultures.

26 abs were used to obtain penile shaft, glans, foreskin, and scrotum samples from 318 male university s

27 Foreskin biopsies were infected ex vivo in organotypic c

28 cific RNA in human fetal lung (HFL) or human foreskin (BJ) fibroblasts increased substantially after

29 vels of CRH-R1 and HDC as compared to normal foreskin, breast skin and cultured human keratinocytes.

30 of SIV-specific cellular immune responses in foreskin by adenovirus serotype 26 and 35 vaccine vector

31 While frequencies of foreskin C-C chemokine receptor type 5(+) (CCR5(+)) T ce

32 Foreskin CD4 T-cell density and CCR5 expression were not

33 n (108.8 vs 23.1 cells/mm(2); P < .001); but foreskin CD4 T-cell density was unchanged (43.0 vs 33.7/

34 although vaccination did not further enhance foreskin CD4(+) T cell activation.

35 Foreskin CD8 T-cell density was increased, but decreased

36 eptides accounted for about 20% of the total foreskin CE ceramides.

37 owth of primary human keratinocytes from the foreskin, cervix, and tonsils.

38 We have treated human foreskin CEs with methanol/KOH (saponification) to hydro

39 s more readily detectable in facial skin and foreskin compared with skin from abdomen and breast.

40 We found that penile (glans, foreskin, coronal sulcus) T cells and, to a lesser exten

41 tibody brightly stained LC on human neonatal foreskin cryosections.

42 e predominantly expressed spontaneously when foreskin-derived early-passage KCs undergo confluency-in

43 Under current culture conditions, neonatal foreskin-derived human keratinocytes possess a relativel

44 By partial saponification of isolated foreskin epidermal CEs followed by limited proteolysis,

45 c dynein was identified in extracts of human foreskin epidermis and in isolated human keratinocytes.

46 median CD1a(+) dendritic cell density in the foreskin epidermis was significantly higher among males

47 elopes prepared from cultured cells and from foreskin epidermis.

48 solated from the inner living cell layers of foreskin epidermis.

49 ell clones derived from primary tonsillar or foreskin epithelia immortalized with HPV-16 genomes.

50 nd in HPV16- and 18-transformed cervical and foreskin epithelial cell lines showed that expression of

51 periments, human foreskin fibroblasts, human foreskin epithelial cells, and, to a lesser extent, HEp-

52 All newborn foreskins examined (n = 5) expressed hTER in epidermal b

53 iency virus (SIV) strain SIVmac251 by penile foreskin exposure.

54 SG) expression, and CMV replication in human foreskin fibroblast (HFF) and peripheral blood mononucle

55 ta15kb), was tested for growth in both human foreskin fibroblast (HFF) cells and SCID-hu mice.

56 scular endothelial (HMVEC-d) cells and human foreskin fibroblast (HFF) cells in vitro by Kaposi's sar

57 scular endothelial (HMVEC-d) cells and human foreskin fibroblast (HFF) cells is characterized by the

58 rmal microvascular endothelial (HMVEC-d) and foreskin fibroblast (HFF) cells were examined by real-ti

59 hese spliced species occur in infected human foreskin fibroblast (HFF) cells, with accumulation kinet

60 at the G1/S transition in p53-positive human foreskin fibroblast (HFF) cells.

61 ocytosis for its infectious entry into human foreskin fibroblast (HFF) cells.

62 Vero, human foreskin fibroblast (HFF), and Jurkat cells could be pro

63 low multiplicity of infection (MOI) in human foreskin fibroblast (HFF)- or NTera2-derived neuronal ce

64 lls (HMVECd)] and foreskin fibroblast [human foreskin fibroblast (HFF)] cells and human B cell line (

65 icrovascular endothelial cells (HMVECd)] and foreskin fibroblast [human foreskin fibroblast (HFF)] ce

66 f fibronectin are potent inhibitors of human foreskin fibroblast cell adhesion to fibronectin-coated

67 nanoUPLC-MS/MS to examine 500 mug of a human foreskin fibroblast cell lysate a total of 1,944 unique

68 class I molecules in B virus-infected human foreskin fibroblast cells and macaque kidney epithelial

69 -PMEG demonstrated increased uptake in human foreskin fibroblast cells and was readily converted in v

70 cytomegalovirus inhibits the growth of human foreskin fibroblast cells by 12 h after infection.

71 These were evaluated in human foreskin fibroblast cells challenged with the vaccinia v

72 n numerous transfections of permissive human foreskin fibroblast cells with these three BAC DNA clone

73 n effect not observed in normal cells (human foreskin fibroblast cells).

74 r for the transcription of IE genes in human foreskin fibroblast cells, at lower MOIs, the murine CMV

75 n pAD/Cre was transfected into primary human foreskin fibroblast cells, Cre was expressed and mediate

76 n microvascular dermal endothelial and human foreskin fibroblast cells, they exhibited much lower vir

77 I also induced proliferation of normal human foreskin fibroblast cells.

78 repared from mock-infected or infected human foreskin fibroblast cells.

79 hree human cell types: HeLa, 293T, and human foreskin fibroblast cells.

80 r compaction with these cells, whereas human foreskin fibroblast migration was positively correlated

81 We used a serum-starved human foreskin fibroblast model to determine changes in COX-2

82 transcription from the US3 promoter in human foreskin fibroblast or HeLa cells.

83 o-2) but not three other lines (HepG2, human foreskin fibroblast, and KD).

84 ral growth by using human fibroblasts (human foreskin fibroblast-1) infected with the hCMV Towne stra

85 hat early during in vitro infection of human foreskin fibroblasts (2 to 4 h), HSV-1 induced the activ

86 s observed in different cell contexts: human foreskin fibroblasts (BJ), human colon and lung carcinom

87 tors by lovastatin treatment of normal human foreskin fibroblasts (FSF cells) resulted in an increase

88 5'-modified derivatives was greater in human foreskin fibroblasts (HFF cells) than in L1210 or KB tum

89 To address this discrepancy, human foreskin fibroblasts (HFF) and three ATM(-) lines (GM025

90 factor-3 (IRF-3) signaling in primary human foreskin fibroblasts (HFF) infected with herpes simplex

91 infection of beta-estradiol stimulated human foreskin fibroblasts (HFF) or HFF/DeltaB-Raf([FF]):ER (e

92 uman cell lines (KB, CEM-SS cells, and human foreskin fibroblasts (HFF)).

93 ritis synovial tissue (RASF), human neonatal foreskin fibroblasts (HFF), and the histiocytic cell lin

94 scular endothelial cells (HMVEC-d) and human foreskin fibroblasts (HFF), and these interactions are f

95 sed to examine the entry of HHV-8 into human foreskin fibroblasts (HFF).

96 n decreased adherence of H. ducreyi to human foreskin fibroblasts (HFF); Flp1 and Flp2, the determina

97 oxidative stress in hTERT-immortalized human foreskin fibroblasts (HFF-hTERT).

98 irus was attenuated in primary human newborn foreskin fibroblasts (HFFs) and embryonic lung fibroblas

99 Comparison between normal human foreskin fibroblasts (HFFs) and HFFs in which p53 was el

100 ilitates viral DNA synthesis in normal human foreskin fibroblasts (HFFs) but not in primary epithelia

101 sing rat dermal fibroblasts (RDFs) and human foreskin fibroblasts (HFFs) demonstrated that these cell

102 is showed induction of amyloid-beta in human foreskin fibroblasts (HFFs) infected with each of 3 clin

103 e investigated their relative roles in human foreskin fibroblasts (HFFs) infected with HSV or transfe

104 of resistance to Toxoplasma in primary human foreskin fibroblasts (HFFs) that does not depend on the

105 PS) cells and the fully differentiated human foreskin fibroblasts (HFFs) they were derived from.

106 The standard was prepared by infecting human foreskin fibroblasts (HFFs) with cytomegalovirus (CMV) s

107 -phase cell attachment assays in HSFs, human foreskin fibroblasts (HFFs), and human corneal stroma fi

108 d deltaTMgpK8.1A proteins bound to the human foreskin fibroblasts (HFFs), human dermal microvascular

109 ease of IFI16 protein levels in normal human foreskin fibroblasts (HFFs), normal oral keratinocytes (

110 were induced or inhibited in infected human foreskin fibroblasts (HFFs), T cells, and skin in respon

111 for their cytopathic activity against human foreskin fibroblasts (HFFs).

112 = 0.40-2.0 muM) and less toxic against human foreskin fibroblasts (IC(50) = 1.1-2.9 muM) and Vero cel

113 mphocytes in vivo as well as in normal human foreskin fibroblasts (NHFs) in vitro in normal (10%) ser

114 ng experiments on synchronized primary human foreskin fibroblasts across the cell cycle.

115 ved vimentin architecture in migrating human foreskin fibroblasts and found that network organization

116 l replication, telomerase-immortalized human foreskin fibroblasts and HEp-2 cells were transduced wit

117 sed an in vitro human skin model composed of foreskin fibroblasts and keratinocytes to examine host s

118 combinant TR1 induced proliferation of human foreskin fibroblasts and potentiated TNF-induced prolife

119 l mechanical properties in nonadherent human foreskin fibroblasts and THP-1 human monocytes before an

120 The three mutant strains adhered to human foreskin fibroblasts and to a human keratinocyte cell li

121 characterized molecular differences in human foreskin fibroblasts and WI-38 TRAIL-resistant cells and

122 cytoplasm of wild-type virus-infected human foreskin fibroblasts as early as 2 h and in HEp-2 cells

123 lls, foreskin fibroblasts, or LDLr-deficient foreskin fibroblasts at 4 degrees C by flow cytometry an

124 ary rat embryo fibroblasts (REF52) and human foreskin fibroblasts become senescent in tetraploid G1 a

125 Human foreskin fibroblasts bind to mouse S4ED, and both mouse

126 Transient transfection experiments in human foreskin fibroblasts demonstrate that proteins binding t

127 pled receptors for bioactive lipids, whereas foreskin fibroblasts expressed Edg-2, Edg-3, and Edg-4.

128 size and length of focal adhesions of human foreskin fibroblasts gradually decreased from short to l

129 Cellular RNA extracted from quiescent human foreskin fibroblasts harvested at 1, 3, 7, or 12 h after

130 endogenous MMP-1 mRNA expression in 34 human foreskin fibroblasts homozygous or heterozygous for the

131 e or binding to HaCaT keratinocytes or human foreskin fibroblasts in vitro.

132 ability to attach to both plastic and human foreskin fibroblasts in vitro.

133 1P to human osteosarcoma MG63 cells or human foreskin fibroblasts increased cell-mediated binding and

134 HCMV infection of human foreskin fibroblasts induced NOD2, the downstream recept

135 levels from the early UL4 promoter in human foreskin fibroblasts infected by recombinant viruses wit

136 r changes, gene expression profiles of human foreskin fibroblasts infected with Toxoplasma were studi

137 inal epithelial cell lines, but not in human foreskin fibroblasts or in HT-1080 fibrosarcoma cells.

138 t wild-type levels with Shield-1 or in human foreskin fibroblasts overexpressing hemagglutinin (HA)-t

139 Normal human foreskin fibroblasts overexpressing icIL-1ra showed high

140 r purified plasma FBG with cultures of human foreskin fibroblasts resulted in FBG deposition in the E

141 COX-2 in human foreskin fibroblasts stimulated by PMA (100 nm) or inter

142 abolic screen of VACV-infected primary human foreskin fibroblasts suggested that glutamine metabolism

143 reconstitution assay in HeLa cells and human foreskin fibroblasts to explore the processes by which E

144 Human foreskin fibroblasts transduced with a retrovirus encodi

145 6 increases the telomerase activity of human foreskin fibroblasts transduced with the hTERT gene, and

146 c AMP (-59/-53) regulatory elements in human foreskin fibroblasts treated with phorbol 12-myristate 1

147 onectin assembly by myrAkt1-expressing human foreskin fibroblasts was abolished by treatment with ant

148 Migration of human foreskin fibroblasts was also inhibited by S. marcescens

149 ced collagen synthesis in both NRK and human foreskin fibroblasts was effectively blocked with specif

150 Here, primary human foreskin fibroblasts were embedded within collagen gels

151 n human umbilical vein endothelial cells and foreskin fibroblasts were evaluated.

152 Human embryonic kidney cells and foreskin fibroblasts were first immortalized by combinat

153 derived from healthy and diseased tissue and foreskin fibroblasts were grown to confluency, photograp

154 ects of VZV on this pathway, confluent human foreskin fibroblasts were infected with cell-associated

155 In contrast, human foreskin fibroblasts were susceptible to the prototype a

156 We found that infection of primary foreskin fibroblasts with human cytomegalovirus (HCMV) c

157 An infection of human foreskin fibroblasts with human cytomegalovirus induced

158 iled in MCF7 (breast cancer) and HFF1 (human foreskin fibroblasts) cell cultures, is less potent than

159 In permissive human foreskin fibroblasts, a fluorescence-based double-strand

160 nously administered into HCMV-infected human foreskin fibroblasts, a reduction of about 80-90% in the

161 d LPs to CD4+ lymphoblastoid (MOLT-4) cells, foreskin fibroblasts, and hepatocytes.

162 imary cell lines: aorta smooth muscle cells, foreskin fibroblasts, and umbilical vein endothelial cel

163 tingly, infectivity of tachyzoites for human foreskin fibroblasts, cells that are commonly used to gr

164 r, induced both E4-orf6/7 and TAp73 in human foreskin fibroblasts, emphasizing the importance of cell

165 In these experiments, human foreskin fibroblasts, human foreskin epithelial cells, a

166 In primary human foreskin fibroblasts, ICP0 was localized predominantly i

167 We used human foreskin fibroblasts, infected by replication-incompeten

168 ines with minimum growth inhibition in human foreskin fibroblasts, mouse fibroblasts, and immortalize

169 oteins, we assessed binding to MOLT-4 cells, foreskin fibroblasts, or LDLr-deficient foreskin fibrobl

170 8-fold higher in retinal endothelium than in foreskin fibroblasts, the cell subtype often used to inv

171 microvascular endothelium, as well as human foreskin fibroblasts, were grown to confluence in 24-wel

172 types, retinal pigment epithelial cells and foreskin fibroblasts, were transfected with vectors enco

173 essible genes, we identified a cDNA in human foreskin fibroblasts, which by GenBankTM DNA data base s

174 eriodically expressed genes in primary human foreskin fibroblasts.

175 ary TBCs compared with HeLa cells or primary foreskin fibroblasts.

176 SCs isolated from adipose tissue, but not on foreskin fibroblasts.

177 marked inhibition of its growth within human foreskin fibroblasts.

178 tivity in hTERT-negative, nonimmortalized BJ foreskin fibroblasts.

179 onies when cultured in the presence of human foreskin fibroblasts.

180 form microcolonies in the presence of human foreskin fibroblasts.

181 crosis factor-alpha (TNFalpha)-treated human foreskin fibroblasts.

182 were compared to those in VZV-infected human foreskin fibroblasts.

183 natal murine epidermal cells or human infant foreskin fibroblasts.

184 est inhibitory effect on the growth of human foreskin fibroblasts.

185 te of a dinucleotide repeat in diploid human foreskin fibroblasts.

186 ient transfection assays using primary human foreskin fibroblasts.

187 nd induced the proliferation of normal human foreskin fibroblasts.

188 n in Vero and SK-N-SH cell lines or in human foreskin fibroblasts.

189 sis in both neuroblastoma cells and in human foreskin fibroblasts.

190 lmarks of Ras-induced senescence in human BJ foreskin fibroblasts.

191 tants had reduced ability to attach to human foreskin fibroblasts; the defect correlated with reduced

192 e main agent of cervical cancer, using human foreskin fragments implanted in severe combined immunode

193 or extraction of infectious virus, and human foreskin fragments were incubated with the virus suspens

194 veloped from simultaneous infection of human foreskin fragments with HPV-11, -40, and -LVX82/MM7 viri

195 Foreskins from eight pediatric and six adult patients wi

196 l cancer and is likely due to elimination of foreskin harboring human papilloma virus.

197 addition of EGF to COS-7 cells and to human foreskin Hs27 fibroblasts results in a rapid tyrosine ph

198 Foreskin immune cells expressed predominantly the CCR5 H

199 Abs capable of causing acantholysis of human foreskin in culture and blistering in neonatal mice.

200 Laboratory findings suggest that the foreskin is enriched with HIV-1 target cells.

201 Foreskin is the principal site of heterosexual HIV-1 inf

202 licited immune responses and inflammation in foreskin is warranted.

203 nces of LMP2A expression in the normal human foreskin keratinocyte (HFK) cell line were investigated

204 Three human foreskin keratinocyte (HFK) cell lines were established

205 A was transfected into an immortalized human foreskin keratinocyte cell line shown previously to supp

206 Undifferentiated normal human foreskin keratinocyte cells responded to increased extra

207 Northern analysis of the normal human foreskin keratinocyte cells treated with NPS R-467 demon

208 fic probe for TGX on Northern blots of human foreskin keratinocyte mRNA, indicating the presence of a

209 LMP2A expression induced migration in human foreskin keratinocytes (HFK) and HaCaT keratinocytes mea

210 amined markers of apoptosis in primary human foreskin keratinocytes (HFK) transduced with either a re

211 study a new cellular target in primary human foreskin keratinocytes (HFK), the serine/threonine kinas

212 inhibits markers of differentiation in human foreskin keratinocytes (HFK).

213 ng pathways in telomerase-immortalized human foreskin keratinocytes (HFK).

214 an NFkappaB inhibitor in both primary human foreskin keratinocytes (HFKs) and HCT116 cells.

215 nsufficient for the immortalization of human foreskin keratinocytes (HFKs) and human mammary epitheli

216 t genomes were transfected into normal human foreskin keratinocytes (HFKs) and selected for drug resi

217 Primary human foreskin keratinocytes (HFKs) cultured in keratinocyte s

218 We show that human foreskin keratinocytes (HFKs) expressing several betapap

219 retention of p27 and the migration of human foreskin keratinocytes (HFKs) in a phosphoinositide-3 ki

220 a integrin alpha(2)beta(1) by cultured human foreskin keratinocytes (HFKs) requires RhoGTP, a regulat

221 rotection assays in transduced primary human foreskin keratinocytes (HFKs) shows that the E6 gene (bu

222 ell lines with or without HPV16 and in human foreskin keratinocytes (HFKs) with or without HPV16 E6,

223 l for the efficient immortalization of human foreskin keratinocytes (HFKs).

224 ion with E7 were used to infect normal human foreskin keratinocytes (HFKs).

225 the entire genome, in primary human neonatal foreskin keratinocytes (HFKs).

226 Suspension of cultured human foreskin keratinocytes (HKs) with trypsin phosphorylates

227 on single targeted individual primary human foreskin keratinocytes (PHFK) cells cultured and maintai

228 retroviruses, we acutely transduced neonatal foreskin keratinocytes (PHKs) with a lacZ reporter gene

229 were used to infect low-passage-number human foreskin keratinocytes and a variety of epithelial cell

230 We detected H4R mRNA on foreskin keratinocytes and on outer root sheath keratino

231 E5 and E6 also induce koilocytosis in human foreskin keratinocytes but not in primate COS cells.

232 In cultured foreskin keratinocytes containing replicating HPV11 DNA,

233 in experimental observations, pools of human foreskin keratinocytes from multiple sources were infect

234 Quercetin arrested primary human foreskin keratinocytes in G1.

235 mainly as a nuclear protein in primary human foreskin keratinocytes in monolayer cultures and their d

236 f cornified envelopes isolated from cultured foreskin keratinocytes releases several discrete involuc

237 g the high risk HPV-16 oncoprotein E7, human foreskin keratinocytes stably expressing E7 were treated

238 ubstratum, as in wound repair, enables human foreskin keratinocytes to interact via alpha(6)beta(4) a

239 religated, and transfected into normal human foreskin keratinocytes together with a neomycin-selectab

240 tion by pathogenic bacteria in both oral and foreskin keratinocytes was blocked by inhibitors of NF-k

241 Human foreskin keratinocytes were transfected with wild-type H

242 Cultured human oral and foreskin keratinocytes were treated separately with inhi

243 n the other hand, infection of primary human foreskin keratinocytes with AAV2 resulted in upregulatio

244 Transfection of primary human foreskin keratinocytes with LT did not immortalize cells

245 emonstrated that lipofection of normal human foreskin keratinocytes with recircularized cloned HPV-31

246 Infection of primary human foreskin keratinocytes with recombinant retroviruses exp

247 Supplementing the medium of primary foreskin keratinocytes with TLCK or TPCK during their im

248 Adherence to keratin types 1 and 10, human foreskin keratinocytes, and nasal epithelial cells was e

249 d its stability in HPV16 E6-expressing human foreskin keratinocytes, and NFX1-123 increased the stabi

250 duced by expression of viral oncoproteins in foreskin keratinocytes, and not seen in HPV-related prec

251 Stimulation of foreskin keratinocytes, atopic dermatitis outer root she

252 HPV-6 E7 (6E7G22D) and showed that, in human foreskin keratinocytes, HPV-6 E7G22D decreased the level

253 the elevated levels of p53 protein in human foreskin keratinocytes, relative to levels in dermal fib

254 ere seen following infection of normal human foreskin keratinocytes, the natural host cell.

255 rker into monolayer cultures of normal human foreskin keratinocytes, the natural host cell.

256 CaT and N/TERT-1 cells and low-passage human foreskin keratinocytes.

257 nd stimulate differentiation in normal human foreskin keratinocytes.

258 issues derived from primary human vaginal or foreskin keratinocytes.

259 In organ cultures of neonatal human foreskin, mast cell degranulation induced by either subs

260 Foreskin mucosa contained higher mean proportions of CD4

261 Adult foreskin mucosa had greater susceptibility to infection

262 +) cellular subpopulations in colorectal and foreskin mucosa were similar in both groups.

263 characterized antiviral immune responses in foreskin of male rhesus macaques (RMs) inoculated with s

264 ) and CD8(+) T cells were also detectable in foreskin of SIV- and SHIV-infected animals and were at l

265 sured using immunohistochemistry analysis in foreskins of 79 males randomly selected from participant

266 s substantial influx of CD8 T-cells into the foreskins of HIV+ men (108.8 vs 23.1 cells/mm(2); P < .0

267 HIV-specific CD8 T cells were present in the foreskins of HIV+ men, although their frequency and func

268 mmunodeficiency virus (HIV) infection in the foreskin or glans of the human penis, although this is a

269 Envelopes prepared from human breast skin or foreskin, or spontaneous or induced envelopes prepared f

270 Foreskin organ culture experiments confirmed our in vitr

271 Hyaluronidase activity in human foreskin primary keratinocyte cultures was also quantita

272 ision device causes ischemic necrosis of the foreskin, raising concerns of anaerobic overgrowth.

273 ing engraftment and vascularization of human foreskin resulted in marked CD3+ T cell infiltrates and

274 d CD8(+) T cell densities were quantified in foreskin samples obtained from medical circumcision in R

275 ls, SIV-specific IgG antibody was present in foreskin secretions.

276 In newborn foreskins, squamous cell carcinomas, and basal cell carc

277 in the basal and suprabasal layer of newborn foreskins, suggesting that hTER expression is present bo

278 e of antiviral effector B and T cells in the foreskin suggests that vaccines may be able to elicit im

279 ke peripheral blood T cells, the majority of foreskin T cells exhibited transitional memory or effect

280 Foreskin T cells were more activated than peripheral blo

281 activated than peripheral blood T cells, but foreskin T cells were not further activated by vaccinati

282 d that HIV induces immune alterations in the foreskin that may impact the subsequent acquisition/clea

283 ecific CD4(+) and CD8(+) T cell responses in foreskin that were detectable for more than 1 year follo

284 Foreskin tissue and blood were obtained from 70 HIV-unin

285 Immunostaining of paraffin-embedded foreskin tissue for the EGFR confirmed that there is an

286 In situ hybridization of human foreskin tissue sections demonstrated that sciellin is e

287 ny that were isolated directly from neonatal foreskin tissue.

288 n cervical mucosa or the external surface of foreskin tissue.

289 estimated immunologic cellular densities in foreskin tissue.

290 r endothelial cells in placenta and neonatal foreskin, two tissues with ongoing growth of microvessel

291 membrane fraction was prepared from newborn foreskins using sucrose gradient centrifugation, followe

292 In contrast, human foreskin was rejected by 21 days posttransplant in hu-PB

293 epidermal melanocytes isolated from neonatal foreskin were evaluated by similar techniques and served

294 derived from the same dark-skinned neonatal foreskins were seeded onto both acellular human dermis a

295 To study this question, neonatal foreskins were transplanted onto immunodeficient mice.

296 f 158 RAG-1 mice, grafted with human newborn foreskin, were separated into four groups and observed f

297 na, possibly because of its proximity to the foreskin, which may be particularly vulnerable to infect

298 ity is localized to the epidermis of newborn foreskin, which suggests that telomerase is expressed co

299 have genetically marked xenografts of human foreskin with a lentivirus encoding a fluorescent marker

300 resent in both the patient condyloma and the foreskin xenografts, and passage of both types was achie