ライフサイエンスコーパス: formazan

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1 te via the formation of an intensely colored formazan.

2 -yl)-2,5-diphenyltetrazolium bromide to blue formazan.

3 ce nitrotetrazolium blue chloride to produce formazan.

4 t cell proliferation (relative absorbance of formazan: 23.4% +/- 7, 44.6% +/- 7.5, 95.8% +/- 2, 100%,

5 t cell proliferation (relative absorbance of formazan: 23.4% +/- 7, 44.6% +/- 7.5, 95.8% +/- 2, 100%,

6 the reduction of nitroblue tetrazolium into formazan, and hydroxyl radicals (OH( *)) were detected b

7 zol-2-yl)-2,5-diphenyltetrazolium bromide to formazan] and apoptotic index (cytoplasmatic release of

8 hiazol-2-yl)-2,5-diphenyltetrazolium bromide formazan assay (MTT assay) as a reporter of Abeta-mediat

9 r serum-free culture conditions, with both a formazan assay and [3H]thymidine uptake.

10 Effects were assayed by a formazan-based colorimetric assay, [(3)H]thymidine incor

11 ation assays (tetrazolium salt conversion to formazan colorimetric assay) were performed beginning 2-

12 ium-5-carboxanilide) to water-soluble orange formazan compounds, the intensity of which can then be d

13 els also modulate the trafficking of the MTT formazan-containing vesicles, suggesting that the traffi

14 (MTT formazan), with the formation of unique formazan crystals on the cell surface.

15 munohistochemical staining co-localized with formazan deposits resulting from nitro blue tetrazolium

16 limit (10 muM in a coupled assay producing a formazan dye) enables accurate kinetic readouts sensitiv

17 By means of a formazan dye-based spectrophotometric assay of cell viab

18 reduction to a yellow colored water-soluble formazan dye.

19 -dependent reduction of a tetrazolium to the formazan enables the measurement of nanomole quantities

20 he ability of amyloid peptides to induce MTT formazan exocytosis is closely associated with both thei

21 Cell growth was assayed by formazan formation and 5-bromo-2'-deoxyuridine (BrdU) in

22 ligomers to rapidly induce the exocytosis of formazan formed by 3-(4,5-dimethylthiazol-2-yl)-2,5-diph

23 The dark-blue reaction product (formazan) formed a pattern that was consistent with mito

24 in a greater than 50% decline in the rate of formazan generation in the CA1 pyramidal neuronal layer

25 1) and the conversion of tetrazolium salt to formazan in four SCC cell lines (P = 0.01-0.004).

26 injury by spectrophotometric measurement of formazan produced from 2,3,5-triphenyltetrazolium chlori

27 -yl)-2,5-diphenyltetrazolium bromide, to its formazan product (MA); DNA synthesis reflected as bromod

28 with a tetrazolium salt (NBT(2+)), forming a formazan product that is visible at 530 nm.

29 rsible and irreversible inhibitors decreased formazan production, whereas conventional enzyme assays

30 ogenase, trypan blue uptake, morphology, and formazan production.

31 es (NAD(H)) was the rate limiting factor for formazan production.

32 Time course analysis using a formazan salt reduction assay to monitor metabolic activ

33 itro blue tetrazolium (NBT) resulted in blue formazan staining of the adventitia, and lucigenin chemi

34 -2-yl)-2, 5-diphenyltetrazolium bromide (MTT formazan), the formation of which is a widely used cell

35 te, NADPH, reduces tetrazolium violet to its formazan, the color of which reflects the number of livi

36 These results suggest that the MTT formazan-transporting vesicles may be involved in cellul

37 by reduction of the tetrazolium compound to formazan, trypan blue dye exclusion, and clonogenic assa

38 tonation of the NH group, a C6F5-substituted formazan undergoes facile cyclization as a result of int

39 sodium salt] to water-soluble yellow-colored formazan with 1-methoxy-5-methylphenazium methylsulfate

40 l-2-yl)-2,5-diphenyltetrazolium bromide (MTT formazan), with the formation of unique formazan crystal

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