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1 n by chemoattractants, such as the peptide N-formyl-methionyl-leucyl-phenylalanine.
2 ith both phorbol myristate acetate (PMA) and formyl-methionyl-leucyl-phenylalanine.
3 f neutrophil elastase after stimulation with formyl-methionyl-leucyl-phenylalanine.
4 ation in the presence of the chemoattractant formyl-methionyl-leucyl-phenylalanine.
5 les in the cremaster muscle in response to N-formyl- methionyl-leucyl-phenylalanine.
7 Ab to CD18 on the surface of unactivated and formyl methionyl leucyl phenylalanine-activated PMN as d
9 hemotactic responses upon stimulation with N-formyl methionyl leucyl phenylalanine and CC chemokine l
10 when cells are simultaneously treated with N-formyl-methionyl-leucyl-phenylalanine and IgE plus polyv
11 f PMN to two biologically relevant agents, N-formyl-methionyl-leucyl-phenylalanine and leukotriene B4
12 o the chemoattractants interleukin 8, C5a, N-formyl-methionyl-leucyl-phenylalanine, and interleukin 1
13 obulin G latex beads, Staphylococcus aureus, formyl-methionyl-leucyl-phenylalanine, and zymosan were
14 gents such as tumor necrosis factor (TNF) or formyl-methionyl-leucyl-phenylalanine, as described for
15 similar to those of the chemotactic peptide formyl-methionyl-leucyl-phenylalanine at 10(-10)-10(-8)
16 ted adhesion to ICAM-1, and stimulation with formyl-methionyl-leucyl-phenylalanine boosted capture ef
18 bility by measuring the transmucosal flux of formyl-methionyl-leucyl-phenylalanine (f-MLP), a ubiquit
19 lation following neutrophil stimulation with formyl-methionyl-leucyl-phenylalanine, FcgammaR cross-li
20 wever, P. stomatis significantly increased N-formyl-methionyl-leucyl phenylalanine (fMLF)-stimulated
21 k translocates to the plasma membrane upon N-formyl-methionyl-leucyl-phenylalanine (fMLF) stimulation
22 e of superoxide elicited by phorbol ester or formyl-methionyl-leucyl-phenylalanine (fMLF) was unaffec
25 rbol 12-beta-myristate 13-alpha-acetate or N-formyl methionyl-leucyl-phenylalanine (fMLP) for stimulu
26 nd that the chemotactic bacterial peptide, N-formyl- methionyl-leucyl-phenylalanine (fMLP), was able
27 lung isolation and ex vivo perfusion with n-formyl-methionyl-leucyl-phenylalanine (fMLP) (10(-)(7) M
28 eceptor (FPR) on neutrophils, which binds to formyl-methionyl-leucyl-phenylalanine (fMLP) and plays a
29 oxidative species and elastase) exposed to N-formyl-methionyl-leucyl-phenylalanine (fMLP) and/or mult
30 until superfusion of the chemotactic peptide formyl-methionyl-leucyl-phenylalanine (fMLP) caused a do
31 Moreover, when cells were exposed to an N-formyl-methionyl-leucyl-phenylalanine (FMLP) gradient wh
32 rally decreasing chemoattractant signal of N-formyl-methionyl-leucyl-phenylalanine (FMLP) in the abse
35 ndent decreases in secondary activation by N-formyl-methionyl-leucyl-phenylalanine (FMLP) or phorbol
37 (TNF-alpha)-primed human neutrophils with N-formyl-methionyl-leucyl-phenylalanine (fMLP) results in
39 g factor (GM-CSF) followed by treatment with formyl-methionyl-leucyl-phenylalanine (fMLP) stimulates
40 nases (MAPK)] were assessed in response to N-formyl-methionyl-leucyl-phenylalanine (fMLP) stimulation
42 Secondary stimulation of PMNs with 1 muM N-formyl-methionyl-leucyl-phenylalanine (fMLP) triggered e
43 interleukin-8 (IL-8), formylpeptides (e.g. N-formyl-methionyl-leucyl-phenylalanine (fMLP)), and plate
44 was stimulated using N-formylated peptide (N-formyl-methionyl-leucyl-phenylalanine (fMLP)), platelet
45 -8 X 10(5)) were coincubated with 0.5 microM formyl-methionyl-leucyl-phenylalanine (fMLP), 1.3 microM
46 by HT29-Cl.19A cells permits absorption of N-formyl-methionyl-leucyl-phenylalanine (fMLP), as occurs
47 ing factor (G-CSF) and then activated with N-formyl-methionyl-leucyl-phenylalanine (FMLP), C(2)-ceram
48 tions in response to the chemotactic peptide formyl-methionyl-leucyl-phenylalanine (FMLP), colony sti
51 receptor in the CNS, and also reduces the N-formyl-methionyl-leucyl-phenylalanine (fMLP)-induced neu
52 lpha, was used to examine the mechanism of N-formyl-methionyl-leucyl-phenylalanine (fMLP)-mediated fo
60 myristate acetate, opsonized zymosan, and N-formyl-methionyl-leucyl-phenylalanine) induce p22(phox)
61 ctivation with platelet-activating factor or formyl-methionyl-leucyl-phenylalanine induced beta(2)-in
62 80, was able to abolish the TNF-alpha- and N-formyl-methionyl-leucyl-phenylalanine-induced activation
64 g was examined by quantitative analysis of N-formyl-methionyl-leucyl-phenylalanine-induced increase i
65 trophils were deficient in spontaneous and N-formyl-methionyl-leucyl-phenylalanine-induced polarizati
66 th hypertonic saline before stimulation with formyl methionyl-leucyl-phenylalanine inhibited A3 recep
67 igration in response to multiple agonists (N-formyl-methionyl-leucyl-phenylalanine, interleukin-8, an
68 In neutrophils activated to secrete with formyl-methionyl-leucyl-phenylalanine, intermediate fila
69 However, in intact cells stimulated with formyl-methionyl-leucyl-phenylalanine, intermediate fila
70 eled chemotactic peptide receptor agonist (N-formyl-methionyl-leucyl-phenylalanine-lysine; N-For-MLFK
71 LA(2)-X to eosinophils under conditions of N-formyl-methionyl-leucyl-phenylalanine-mediated cPLA(2)al
72 h activities when cells were stimulated by N-formyl-methionyl-leucyl-phenylalanine or opsonized zymos
73 n perforated-patch configuration with PMA, N-formyl-methionyl-leucyl-phenylalanine, or anti-IgE great
74 13, in basophils stimulated with anti-IgE, N-formyl-methionyl-leucyl-phenylalanine, or phorbol 12-myr
75 suppressed basophil activation induced by N-formyl-methionyl-leucyl-phenylalanine, phorbol 12-myrist
76 e rate of pseudopod extension induced with N-formyl-methionyl-leucyl-phenylalanine, platelet activati
77 reased Fgr activity in cells stimulated with formyl-methionyl-leucyl phenylalanine plus dihydrocytoch
78 The products were assayed in vitro for N-formyl-methionyl-leucyl-phenylalanine receptor binding a
79 (CD11b/CD18, the mannose receptor, and the N-formyl-methionyl-leucyl-phenylalanine receptor) did not
80 /18 expression and doubling of the number of formyl-methionyl-leucyl-phenylalanine receptors on the c
81 impaired migration and O(2)(-) responses to formyl-methionyl-leucyl-phenylalanine, reflecting the sa
82 sis, cell polarization, and TNFalpha-primed, formyl-methionyl-leucyl-phenylalanine-stimulated respira
83 y, it inhibited arachidonate production in N-formyl-methionyl-leucyl-phenylalanine-stimulated U937 ce
86 n, whereas hypertonic saline-treatment after formyl methionyl-leucyl-phenylalanine-stimulation augmen
87 ulocyte macrophage-colony-stimulating factor/formyl-methionyl-leucyl-phenylalanine stimuli, which can
88 crosis factor-alpha (TNF-alpha) as well as N-formyl-methionyl-leucyl-phenylalanine treatment leads to
89 ation method that retains coupling between N-formyl-methionyl-leucyl-phenylalanine tripeptide (FMLP)
90 inophil responsiveness upon stimulation with formyl-methionyl-leucyl phenylalanine was found to ident
91 fter activation by the chemoattractant fMLF (formyl methionyl leucyl phenylalanine) was observed by R
92 ponse to the prototype N-formylpeptide fMLF (formyl-methionyl-leucyl-phenylalanine) were both absent
93 axis defects were also seen in response to N-formyl-methionyl-leucyl-phenylalanine, zymosan-activated
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